Pulmonary Medicine - Science topic
A subspecialty of internal medicine concerned with the study of the RESPIRATORY SYSTEM. It is especially concerned with diagnosis and treatment of diseases and defects of the lungs and bronchial tree.
Questions related to Pulmonary Medicine
I am looking for data related to breathing exercises that can be safely prescribed in case of spontaneous pneumothorax with / without bronchopleural fistula ; managed by inserting an intercostal drainage tube.
Can incentive spirometer be given to these patients ?
I’ll describe what I postulate provides the explanation of the CAP microarousal episodes during sleep.The information has been
based entirely on the conclusions of documented studies. Admittedly, and inasmuch as each conclusion individually is not new information per se. nevertheless, by combining and preferentially sequencing them,
it becomes possible to arrange a set of contingent propositions which facilitates the creation of new awareness.
The concept can be tested non-invasively during Cycle 1 of descending NREM sleep in a perfectly healthy individual. Additionally, it would provide support for the statement that “ ...several lines of evidence
suggest that the pulmonary circulation both has minimal neural regulation and is unresponsive to
changes in sleep state. and is both significantly important and key to understanding that peripheral
mechanisms cannot be excluded”. @ http://jap.physiology.org/content/88/3/1084
In my view the cause of lung damage during modern mechanical ventilation is due to the positive pressure. Positive pressure ventilation causes atelectasis, and subsequently all the other problems if mechanical ventilation is maintained for a long time.
Negative pressure (expanding the thorax and getting air to flow in by under-pressure e.g. by a quirass sytem, or natural breathing) takes away the atelectatis very rapidly. That is why in every manual of the Anesthesiogists it says: directly after surgery, when the patient is awake again, ask him/her to take e few deep sighs.
My question: is it possible by your thechnique to prove the development of atelectasis by positive pressure and removal of the atelectasis by negative pressure vetilation?
This is very important, because patients with severe lung problems should not be ventilated by positive pressure systems. The life of a category of patients will be saved by negative pressure ventilation, because of the above reasons. So, please show the world what is going on, for most of the ventilators still believe the "law" (spoken out as a proposition around 1900) that there can be no difference between negative and positive pressure; they claim that everything is determined only by the pressure difference. They claim this is just physics. But this is only true in a static situation. During dynamic pressure variation this is absolutely not true.
Jan van Egmond.
In respiratory research a breathing apparatus consisting of mouthpiece, filter, Pneumotachometer, and non-rebreathing valves plus some connectors are usually used. Although a non-rebreathing valve is used to reduce dead space, each of these devices has its own dead space. Though small, adding together they build a relatively large dead space sometimes. What is the max acceptable dead space in a breathing apparatus, for a study including healthy adults?
If treated, which agent is preferred agent? NOAC or vitamin K antagonist?
A case of 58 years old male PINK Puffer with very limited performance status. Pulmonary function Tests can not be done. They are going to do CABG 3 Vessels for him. They are wondering if he can benefit from lung volume reduction surgery.
Patient had only 3 months treatment with anti TB. Pulmonologists stratified him as a high risk for CABG alone. Is he going to benefit from Lung Volume reduction surgery simultaneous with CABG?
I observed a 58 yo female patient in deep pain and was diagnosed as having a left-sided atypical chest pain. And she was sent for a chest X-Ray. It was found that she has a 1.0 cm x 0.8 cm lower left lung nodule. A repeat X-Ray is warranted but the result is still unknown.
My question is -
1. Could the atypical chest pain be the result of the lung nodule?
2. Her previous mammogram showed no abnormality, and she conducts her BSE monthly NOS. Could it be that there is some abnormality in the breast instead?
3. What could best explain her condition(s)?
Best regards - Mariam
Is there any study that shows the effect of exercise on pulmonary pressure after acute pulmonary thrombo embolic (PTE) treatment?? thank you
A persistent ventilation tube could lead to bioflim formation over it . Is there a time limit beyond which it would be advisable to remove it? .
Apart from the subcutaneous tumour, is the lung tumour also primary or metastatic? If metastatic, does it still represent actual lung tumour?
Lungs are fixed by PFA and embeded in OCT and stored at -80oC. I am using the general protocol of abcam (http://www.abcam.com/protocols/immunostaining-paraffin-frozen-free-floating-protocol) to stain frozen sections of mice lungs and I get auto-fluorescence.
This was a symptomatic event where the patient experienced severe central chest pain radiating "like a vice" around the body causing severe pain between the shoulders. Diagnosed by means of positive D-dimer and typical ECG changes such as SInus Tachycardia during the acute event, then ECG observations noted SR, S1Q3-Type with new RBBB.
The patient self administered GTN spray during the acute event, was given S/C heparin thereafter and commenced on oral aspirin 100mg. Seven days later, the patient was prescribed and given two 30 mg doses of oral prednisolone for an unrelated event.
Would evidence of PE still be availalabe given the time frame and susequent medications?
65 years old lady with gradually pregressive dyspnea....HRCT s/o ILD, rest of the blood parameters are normal. She was started with IV methyl prednisolone followed by oral tablets along with N Acetyl cysteine...but, she is not responding with increasing dyspnea.....today we have added tab azathioprine. Any suggestion please...
Right lung edema has been observed in minimally invasive right thoracotomy mitral valve procedures, requiring extracorporeal membrane oxygenation in some patients. Proposed mechanisms have included right lung manipulation, ischemia/reperfusion, and reexpansion edema.
Considering the increasing epidemiological burden of TB, especially drug resistant TB, should the old TB sanatoria/TB hospitals (many of which are defunct) be upgraded?