Questions related to Periodontology
Dr. Manfredini: The American Academy of Periodontology lists bruxism and clenching as risk factors for periodontal disease. It is time to broaden your definition of Bruxism. The Sciences of Molecular Biomechanics and Microfluidics have defined the cellular response to force. I would like to continue this conversation. Best Regards, Dr. Max Perlitsh (email@example.com)
I will be very grateful for anyone who wants to donate personal used equipment to our periodontology practice. I am interested in equipment like the halimeter machine, diode laser machine, electrocautery machine, gas chromatography machine, periodontal endoscopy machine etc.
I plan to develop a periodontitis model and work in relation with behavior aspect as well as its effect on teeth and its cure. i tried to make a model after reading so many papers but fails to place a ligature between the rats molar teeth. If anyone of you can share his experience of making periodontitis model in form of video or in picture it will be quiet helpful for me.
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Dear ResearchGate Community,
I'm currently working on the implementation of the Ligature-Induced model of Periodontitis in Rats (Winstar - Sprague Dawley). Yesterday I had my first try... however I discovered rat molars are very distal and difficult to access. I tried to use a 4-0 silk suture (3/8 circle) but found it impossible to access!
Does someone has experience on this model? I need advice or guidance on how to reach the molars.
With best of Regards,
Ps. Sorry for my english, sadly I am not a native speaker.
There are several articles equating Periodontal Disease to an infection. There are several articles showing the relationship of infections on chronic diseases. Is it possible to cure the infection and eradicate the disease? Can it be that simple? I am building my research project around this question. The relationship between preventable infections and chronic diseases. My question to you: What are your thoughts, experiences, and observances with infections and their impact on health?
Sandy Evans, MHA
Cases of aggressive periodontitis are not plenty in number.
How to carry out a reliability study for an index on aggressive periodontitis?
Does anyone know of any literature regarding the limitations related to pooling of gingival crevicular fluid TNF-alpha samples collected from different sites before ELISA analysis?
I would like to know what are the factors- prosthodontics related or periodontology related, which are responsible for Implant failure and which can be avoided during formulation of treatment planing for that particular Implant. Any inputs will be highly appreciated.
Good afternoon colleagues, I am testing the antimicrobial effect of a new Peptide (Lye Tx 1) against Periodontal Pathogenic Bacteria in Biofilms. To create biofilms, I am using the Calgary Device. Reading the paper of Harrison et al. 2010, the protocol suggest that pegs may need to be coated with some agent in order to promote bacterial adhesion.
Do you know whether is necessary coating the pegs when growing periodontal pathogenic biofilms in the Calgary Device?
Thank you very much.
Some clinicians prefer closed tray over the open tray but some discrepancies can appear in this technique. On the other hand the open tray technique is somewhat difficult and time consuming.
I look for any kind of academic institution worldwide to collaborate in dental/periodontal research or lecturing/teaching.
Topics: periodontology / implantology / halitosis / lasers in dentistry.
Please contact me
Aggressive periodontitis attacks people at early age of life and causes extensive bone loss which might lead to early loss of dentition, early detection of patients at high risk to be attacked by this type of periodontitis will help them a lot to be involved in a strict professional and personal preventive program thus minimize the periodontal damage to a large extent .I wonder if any one has ideas about any chairside methods for early detection of risk factors and those who are at a high risk for developing this kind of periodontitis?
My question goes toward standardization of instruction, independently of technique or brush, thinking of how eliminating bias when two or more instructors teach the same technique of brushing.
Traditionally a lot of materials have been used for periodontal regeneration starting from GTR to bone grafts and root modification agents, but its difficult to evaluate true regeneration as the tooth needs to be examined histopathologically. So can we say for sure that it does work?
Since we know that PRF or PRP has a lot of growth factors for regeneration of tissues, if we put it inside an envelope flap, can it cover the exposed root surface?
Bioactive glasses have an anti-bacterial action due to incorporation of certain elements such as silver and zinc or to the presence of silica in their composition. These glasses , therefore; have been used as a coating to inhibit bacterial colonization. What is the optimized slurry dipping technique to coat surgical threads with bioactive glasses? Is there an easier, alternative method to slurry dipping technique?
Periodontal plastic surgery is the most challenging task a periodontal surgeon faces in his or her practice. Sometimes we are in a dilemma as to which technique to employ. It would be interesting to know the view points of others.
Does anyone have some experience in staining macrophage in periodontal tissue, especially in periodontally-infected (i.e., periodontitis) tissue in mice? Growing attention is paying on macrophage in periodontitis. However, it seems too few macrophages exist in periodontitis, let alone in the tiny gingiva in mice. If I hope to immunostain macrophages in paraffin-embedding jaw from mice, what are the notes during decalcification, tissue processing and immunohistochemistry?
I am intrigued by the association of periodontitis with various systemic disease. a constant explanation is the role of P.Gingivalis and other pathogens acting as source of immune hyperesponsiveness. several studies have proved this. I personally feel, if the association is true we should be able to develop a single model to prove this .
While selective media in the past have helped in the growth of Fusobacterium, their consistency is questionable.
What is the most effective and repeatable media for isolation of the oral Fusobacterium?
It is documented that Ciprofloxacin is one drug to which all strains of AAC react. Trends have focused mainly on amox , meronidazole, and their combinations mainly and more recently on azithromycin and ornidazole. Is it because today the consensus support the fact that CGP and GAP have similar clinical and microbiological expressions?
I want to do research about periodontal-endodontic lesion, and know the root surface areas of residual alvealar bone by using CBCT or another method. Any suggestions?