Science topic

Pediatric Intensive Care Units - Science topic

Hospital units providing continuous surveillance and care to acutely ill infants and children. Neonates are excluded since INTENSIVE CARE UNITS, NEONATAL is available.
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How do we calculate the pediatric SOFA score if a child is on only milrinone? As per BP cut-offs or equivalent to dobutamine? Has there been any update in the score to account for milrinone/vasopressin?
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Try with this:
Pediatric SOFA score. BTW, did you try calculate the Vasoactive Inotropic Score (VIS)?
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The Health Technology Assessment (HTA) unit of the CHU de Québec – Université Laval is currently working to get data on integrated early rehabilitation interventions in pediatric intensive care unit.
We define «integrated early rehabilitation interventions» as physical, functional, nutritional, psychological, communicational, social or spiritual rehabilitation activities initiated during the first days of admission of a patient in the pediatric intensive care unit and delivered by each professional according to an intervention plan that has been developed beforehand as a team by these same professionals.
Are early interdisciplinary rehabilitation interventions an established practice in the pediatric intensive care unit of your hospital?
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Currently I am living in Bolivia and most of the Hospital don´t count with those services in ICU.
In Brazil there is a law that determine the presence of some of those professional (PT for example) 24/7. But that doesn´t translate into practice.
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I have unique insight on the parental dynamics of child cardiac hospitalization, on-site physical parental needs, parental emotional management, and issues of parental advocacy for ill or dying children.
In 1995, my youngest daughter was born with Tetraology of Fallot, a rare congenital heart defect. At day three of life, she underwent her first open-heart surgery. Her father and I were called into the Chaplain's office with devastating news - every time the chest cavity was closed following surgical repair/replacement of her heart valve, she would die. The Chaplain was resolved to simply deliver the grim news and manage the emotional aftermath.
However, I began to ask questions. "You say that every time the chest cavity is closed, she dies, right? You told us earlier that the heart was enlarged due to increased work load, right? What if it was possible to keep the chest cavity opened long enough to allow the swelling to go down and then, afterward, go back in and then close the chest? Could the chest be covered with something to protect it until the swelling decreases?"
The Chaplain immediately rose from the chair and announced to us that she would be right back. "Don't go anywhere. I'll be back. I'm going to speak with the surgeon."
My daughter's chest cavity remained open for close to a week, with a yellow plastic, adhesive covering to allow room for the swelling to decrease. She was kept in the PICU (Pediatric Intensive Care Unit) there at Cook's Children's Hospital in Fort Worth, TX before being moved to the surgical unit for closure of the chest when most advantageous.
She lived, and to this day requires no cardiac medications on a daily basis. My daughter will be 24 this August 26, and this aggressive act of child advocacy may have set a medical precedent. She has undergone two more surgeries as her heart grew naturally; one at 5 years of age, and the last at 17 years old.
Different emotional dynamics occurred at each surgical experience for her, most especially, but also for me as parent; the third was the most heart-wrenching. Any fellow researcher in need of particular input for devising measures to assist such parents are more than welcome to reach out to me. I will be more than happy to help.
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Heart-centered greetings
First, thanks a lot for sharing this valuable human experience with us.
Second, I would like to appreciate the iconic client's advocacy role that the Chaplain had played! Such a great role model for all of us in health care industry.
Third, I see a great opportunity for qualitative research focusing on such courageous acts of parents.
Once again thank you very much!
V/r,
Sadeq AL-Fayyadh, PhD.
University of Baghdad,School of Nursing
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Occasionally we used to have transcutaneous CO2 monitors for selected ICU patients, especially those who are ventilated with HFOV.
However, with its poor correlation with the blood gas analysis and the complications of the electrode site burns, I think it is out of fashion?
Is anyone using similar products with better clinical experience?
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I'm a little confused by the comments about "burn sites" because modern transcutaneous oxygen and carbon dioxide monitors don't burn the skin. When I was an anesthesia resident at UCLA, I had the opportunity to try the earliest version of a transcutaneous oxygen monitor, and with these primitive machines there was some concern about leaving the sensor in the same location for prolonged periods of time due to the possibility of causing tissue damage. They warmed the skin to "arterialize" capillary flow, but the heating element was not regulated by a thermostat. It was impossible to calibrate these original monitors. They provided a numerical "readout" for oxygen concentration that rose with increased tissue oxygenation, but they could not actually measure the partial pressure. There has been considerable improvment in the technology during the 50 years since I was a resident. I have purchased one of the new machines. They now measure transcutaneous CO2 as well as oxygen, and can be calibrated to measure the partial pressures of both. However, they remain far from ideal. They are very "tricky" to use. Their main advantage is that they are non-invasive, and can provide continuous measurement of the partial pressures of oxygen and carbon dioxide. The sensors are not likely to cause tissue damage, because they are now governed by a thermostat that maintains the skin temperature around 45 degrees celsius, so patient injury is no longer a concern.
I am now working with the Plexus software company to enable automatic incorporation of TcO2 and TcCO2 data into my anesthetic records, and capture readings every five minutes.
Carbon dioxide chemistry and pathophysiology is very tricky stuff. I once had an engineer who helped design modern capnographs tell me that it was necessary to install a fan in the measurement chamber of the machines to constant stir the gas mixture, because carbon dioxide has a vexing tendency to "settle" and thereby frustrate accurate measurement. Transcutaneous CO2 measurement is similarly vexed by variables. The sensor is mounted in a small plastic cup that is pasted to the skin. About five drops of saline solution must be installed in the cup before the sensor is attached. The skin must be carefully "prepped" to prevent the water from leaking out of the plastic cup "chamber" where the measurement takes place. The temperature must rise to 45 degrees before the machine begins to operate. Too much or too little water in the chamber will disrupt measurement. The sensor must be calibated each time it is applied, and if you are using it in an ICU setting it's probably a good idea to re-calibrate the sensor every hour or so. I use the machine only during short dental surgeries, and must re-calibrate between each case. It's essential to have a clear understanding of the role of carbon dioxide in the pathophysiology of oxygen transport and delivery, and CO2 data alone is meaningless in the absence of O2 data.
Exactly what are you trying to accomplish in the ICU setting? Are you performing some sort of study, or are you using the machine to manage critically ill patients?
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As compared to the "traditional" intracoastal chest tube that are inserted at the bed side for ICU patients to drain pneumothorax / pleural effusion. In the last years some ICUs have been using the newer "Seldenger" over the guide wire technique.
Which style do you prefer? Why?
Is your setup Adult / Pedia or Neonatal ICU?
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In our PICU setting , the Seldenger technique is usually preferred : faster and somewhat "less invasive"
However, the team needs to keep their "traditional" technique handy , as on one instance the PICU supply was out, so going back to the original style was literally "life-saving"!
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My work is to care for babies of premature or neonatal critical care, and I always think they are too small to fix an endo tube or machine support. How to care premature infants if they have an upper respiratory problem (like endo tube)? And if the mortality rate is high, how do you talk about the death to the family?
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Every neonate admitted to a Neonatal Intensive Care Unit has a high risk of complications and death. Parents should know that their child may die due to complications of the disease, and even idiosyncratic complications, which are sometimes not predictable (eg, anaphylaxis). Parents should Know that the probability of death is real and that medical staff do their best effort to reduce this risk;  we should not give false hope, but we must have in mind that despite the great risk of death, there is also a probability of survival, which, small or big, must be fought.
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You have 2 problems, firstly the size of connections and secondly the lack of a reliable seal.
I remember being used (repeatedly) as a subject for the respiratory physiologists/pharmacologists when I was a resident. The standard set-up (breathing spontaneously) measured pressure and flow and the circuit included a device that occluded the airway with a diaphragm for 0.1 sec. The transient pressure gradient across  the diaphragm indicated inspiratory effort and was used as an index of respiratory drive. The lack of a cuff sealing the airway might interfere with this, but as the occlusion is only for 100 msec, you might get a useful reading.
I remember the equipment as being rather bulky, both from the tubing and the recording apparatus, but this was 50 years ago. 
So speak to your friendly local respiratory physiologists and ask if they can measure P0.1 in your circumstances.
Good luck! 
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What are different methods (clinical / pathological / radiological etc.) to diagnose and confirm Umbilical venous catheter (UVC) extravasation injuries?
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Sonography is the most useful tool per my own experience. Clinical sign is deceiving and we experienced large subcapsular liver hematoma due to UVL without remarkable clinical sign. We probably will obtain pathological evidence after the patient is dead (too late).
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If we dont have rapid EEG in the PICU can we use it ?
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Can BIS monitoring be used to assess the depth of propofol
anesthesia in the treatment of refractory status epilepticus?
*Tadeusz Musialowicz, yEsa Mervaala, zReetta Ka¨lvia¨inen, *Ari Uusaro,
Epilepsia, 51(8):1580–1586, 2010
doi: 10.1111/j.1528-1167.2009.02514.x