Nutritional and Metabolic Diseases - Science topic
A collective term for nutritional disorders resulting from poor absorption or nutritional imbalance, and metabolic disorders resulting from defects in biosynthesis (ANABOLISM) or breakdown (CATABOLISM) of endogenous substances.
Questions related to Nutritional and Metabolic Diseases
I am interested in the relationship between diet and FTO gene expression. Most studies have been performed on FTO expression changes in hypothalamus. But as we know, FTO is expressed ubiquitous. We are going to do a research on this subject in adolescents in Iran and I look forward to hearing from everybody that can help us.
Hypophosphatemio is a verz dangerous metabolic situation by many patients, specillz in intenzive care unit, by status epilpticus, ect. It is necessery to proof the nvou of Phosphor, not only the standard electrolits. The correction must be very fast, because the start symptoms are unspecific, and the end can be paralysis of respiratorz muscels, coma and death.
L-carnitine is a quaternary ammonium compound biosynthesized from the amino acids lysine and methionine.In eukaryotic cells, it is required for the transport of fatty acids from the intermembraneous space in the mitochondria, into the mitochondrial matrix during the breakdown of lipids (fats) for the generation of metabolic energy.It is widely available as anutritional supplement.
I am currently looking for research on any clinical study of the direct correlation between oil extraction and specific diseases in Africa, such as cancer, asthma or other respiratory illnesses in Africa.
Overexpression of uncoupling proteins in small rodents is an effective way to alter thermogenesis. This strategy prevents diet induced obesity and insulin resistance (link 1). There is now substantial interest in alternative methods of increasing thermogenesis as a means to treat obesity (links 2 and 3). However, due to the volume : surface ratio, small rodents have a high tolerance for elevated thermogenesis (link 4) as a result of UCP overexpression. Humans have a much lower surface area : volume ratio and therefore have a reduced capacity to dissipate heat. Therefore, will overweight humans have a sufficient tolerance for enhanced thermogenesis for this to be a safe and viable treatment option?
I have induced type 2 diabetes mailitus using high fat diet (45% fat) plus multiple low dose of streptozotocin (30mg). So can any please tell me Which type insulin (Short, log, Intermidiate acting or Biphasic insuline) should be used to carry out Insulin Tolerance Test in type 2 diabetic rats?