Questions related to Mercury Poisoning
Climate change is a hotly debated topic, and there is a lot of political fervor that finds its way into discussions about the topic. A discussion of climate change science, with someone who rejects climate change, will generally lead nowhere. But while climate change is so prominent in the media, health problems caused by burning of fossil fuels, mercury poisoning, and other areas of ecological and public health issues are often ignored.
I think that even such deniers would admit that burning of fossil fuels has led to a great deal of other hazards. Smog has long been a recognized problem of pollution. Recent studies suggest that mercury is almost universally found in freshwater fish.
So would we better serve the population by expanding our discussion about pollution, and focusing less on climate change specifically?
On-the-ground experience would suggest that sound/noise pollution and mercury contamination may be coincidental in artisanal and small-scale gold-mining stakeholders and their bodies. If so, does any literature make explicit this coincidence or explore it further?
i guess that malondialdehyde is a biomarker but what about glutathione , is it important to know if it is totally consumed or it is enough to measure malondialdehyde??
Does anyone know current (2018/2017) US reference values (cdc? FDA?) for mercury in urine (exposure to inorganic mercury)?
I can only find current reverence values for total mercury in blood
There are a number of bioassay tools and techniques available. They study LC50, LD 50 (for 24, 48, 96, ....hours). We also do EC studies. I think, mother Nature can provide some hidden physical clues showing toxic effects of toxicants. Different organisms show some defensive or biochemical struggling signals in the form of secretions that can be detected through chemosensing. If we detect the early warning signals by reading the physical or catching the chemical clues mitigation or removal of toxicants could be done in due time.
My partner and I will be truly grateful for your insights to the following:
(1) The mechanism behind Bacopa's hyperaccumulation of heavy metals
(2) Identification of the chemical compounds in the plant that may possess high affinity towards heavy metals
(3) Possibility that such compounds may be extracted and used in chelation therapies
Please do refer me to pertinent studies, both completed and ongoing (especially those containing detailed methodologies that aim to (1) extract and isolate natural chelating agents and/or (2) measure the efficacy of chelating agents).
The said model(s) will serve a twofold purpose: (1) to allow a comparison between bioeconomic models from developed countries and developing ones, and (2) to map out the possible strategies that may be employed in optimizing the tradeoff between maximum economic profit and the maintenance of public health and physical wellbeing.
While bioeconomic models exist out there, my partner and I failed to find models that seem to fit our research context and setting -- Albay, Philippines. The whole country is very much dependent on aquatic resources, fish in particular, for food. Existing research on MeHg contamination of commercial fish shows that the issue is already very alarming.
It has recently been shown that even low doses of mercury may cause toxicity. Increased release of mercury from dental amalgam restorations after exposure to electromagnetic fields has been confirmed by different researchers as well as my team. It has also been reported that the increased mercury release after exposure to electromagnetic fields may pose risks for the hypersensitive proportion of the population, children and pregnant women. Although it was previously believed that the amount of mercury released from dental amalgam cannot be hazardous, new findings indicate that mercury, even at low doses, may cause toxicity. By the way, now we are looking for the data showing mercury may cause synergistic effects with other heavy metals. Thank you very much in advance.
I knew that inorganic mercury compound such as mercury chloride or mercury iodide is toxic. Organic mercury compound such as methyl mercury or dimethyl mercury are more dangerous. From what I have read before, mostly it is because mercury accumulation occurs easily in organic mercury compound. What differs inorganic mercury compound and organic mercury compound which lead to different toxicity of mercury?
Even in inorganic mercury compound e.g mercury chloride, mercury iodide, mercury cyanide, etc, they have different level of toxicity.
If you could provide some reference about it, it'll be very helpful.
The cases of mercury poisoning of populations in Miamata, Japan and distribution of tainted seeds consumed in Northern Iraq reflect on widespread patterning in symptoms associated with severe exposure. Symptoms appear to exist on a spectrum. Known locations of long term low levels of mercury exposure must have widespread symptoms associated with them- what does this look like?
I have speculated on a bridge between mercury exposure and patterns of an unusual medical condition documented in the Languedoc region of France from approximately 1550- 1776 (as mentioned in an 1935 article by Kellett on Sir Thomas Browne and The Morgellons). The use of mercury in dyes may account for the widespread documentation of these symptoms.
How can we as the scientific, academic, and healthcare community help consumers with the new and growing threat of broken MCL bulbs? The dangers are extraordinary and economically impossible, yet those who should have addressed the issue before release of product have fallen asleep at the wheel of consumer safety. Please, see http://www.wnd.com/2007/04/41122/ and share your ideas on this very real danger to the public health.