Environmental Risk Assessment - Science topic

Please have look on our(Eminent Biosciences (EMBS)) collaborations.. and let me know if interested to associate with us

Here is our lab page: https://www.eminentbio.com/home/members

Here is our publication: https://www.eminentbio.com/home/Publication

Our recent publications In collaborations with industries and academia in India and world wide.

Our Lab EMBS's Publication In collaboration with Universidad Tecnológica Metropolitana, Santiago, Chile. Publication Link: https://pubmed.ncbi.nlm.nih.gov/33397265/

Our Lab EMBS's Publication In collaboration with Moscow State University , Russia. Publication Link: https://pubmed.ncbi.nlm.nih.gov/32967475/

Our Lab EMBS's Publication In collaboration with Icahn Institute of Genomics and Multiscale Biology,, Mount Sinai Health System, Manhattan, NY, USA. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/29199918

Our Lab EMBS's Publication In collaboration with University of Missouri, St. Louis, MO, USA. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30457050

Our Lab EMBS's Publication In collaboration with Virginia Commonwealth University, Richmond, Virginia, USA. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27852211

Our Lab EMBS's Publication In collaboration with ICMR- NIN(National Institute of Nutrition), Hyderabad Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/23030611

Our Lab EMBS's Publication In collaboration with University of Minnesota Duluth, Duluth MN 55811 USA. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27852211

Our Lab EMBS's Publication In collaboration with University of Yaounde I, PO Box 812, Yaoundé, Cameroon. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30950335

Our Lab EMBS's Publication In collaboration with Federal University of Paraíba, João Pessoa, PB, Brazil. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30693065

Our Lab EMBS's Publication In collaboration with collaboration with University of Yaoundé I, Yaoundé, Cameroon. Publication Link: https://pubmed.ncbi.nlm.nih.gov/31210847/

Our Lab EMBS's Publication In collaboration with University of the Basque Country UPV/EHU, 48080, Leioa, Spain. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27852204

Our Lab EMBS's Publication In collaboration with King Saud University, Riyadh, Saudi Arabia. Publication Link: http://www.eurekaselect.com/135585

Our Lab EMBS's Publication In collaboration with NIPER , Hyderabad, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/29053759

Our Lab EMBS's Publication In collaboration with Alagappa University, Tamil Nadu, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30950335

Our Lab EMBS's Publication In collaboration with Jawaharlal Nehru Technological University, Hyderabad , India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/28472910

Our Lab EMBS's Publication In collaboration with C.S.I.R – CRISAT, Karaikudi, Tamil Nadu, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30237676

Our Lab EMBS's Publication In collaboration with Karpagam academy of higher education, Eachinary, Coimbatore , Tamil Nadu, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30237672

Our Lab EMBS's Publication In collaboration with Ballets Olaeta Kalea, 4, 48014 Bilbao, Bizkaia, Spain. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/29199918

Our Lab EMBS's Publication In collaboration with Hospital for Genetic Diseases, Osmania University, Hyderabad - 500 016, Telangana, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/28472910

Our Lab EMBS's Publication In collaboration with School of Ocean Science and Technology, Kerala University of Fisheries and Ocean Studies, Panangad-682 506, Cochin, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27964704

Our Lab EMBS's Publication In collaboration with CODEWEL Nireekshana-ACET, Hyderabad, Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/26770024

Our Lab EMBS's Publication In collaboration with Bharathiyar University, Coimbatore-641046, Tamilnadu, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27919211

Our Lab EMBS's Publication In collaboration with LPU University, Phagwara, Punjab, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/31030499

Our Lab EMBS's Publication In collaboration with Department of Bioinformatics, Kerala University, Kerala. Publication Link: http://www.eurekaselect.com/135585

Our Lab EMBS's Publication In collaboration with Gandhi Medical College and Osmania Medical College, Hyderabad 500 038, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27450915

Our Lab EMBS's Publication In collaboration with National College (Affiliated to Bharathidasan University), Tiruchirapalli, 620 001 Tamil Nadu, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/27266485

Our Lab EMBS's Publication In collaboration with University of Calicut - 673635, Kerala, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/23030611

Our Lab EMBS's Publication In collaboration with NIPER, Hyderabad, India. ) Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/29053759

Our Lab EMBS's Publication In collaboration with King George's Medical University, (Erstwhile C.S.M. Medical University), Lucknow-226 003, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/25579575

Our Lab EMBS's Publication In collaboration with School of Chemical & Biotechnology, SASTRA University, Thanjavur, India Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/25579569

Our Lab EMBS's Publication In collaboration with Safi center for scientific research, Malappuram, Kerala, India. Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/30237672

Our Lab EMBS's Publication In collaboration with Dept of Genetics, Osmania University, Hyderabad Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/25248957

Our Lab EMBS's Publication In collaboration with Institute of Genetics and Hospital for Genetic Diseases, Osmania University, Hyderabad Publication Link: https://www.ncbi.nlm.nih.gov/pubmed/26229292

Sincerely,

Dr. Anuraj Nayarisseri

Principal Scientist & Director,

Eminent Biosciences.

Mob :+91 97522 95342

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ممكن انه محجوب عن دول افريقيا

Dear dr Rehak,

It will be a pleasure to contribute to the special issue of the Sustainability journal. Please let me know the deadline for submission and will send you asp Abstract focused on Environmental Risk Assessment in Transport. All the best,

Darko

Drinking-water containg Arsenic increases the risks of cancer in the skin, lungs, bladder and kidney, as well as other skin changes such as hyperkeratosis and pigmentation changes.The effects on environments include death, inhibition of growth, photosynthesis and reproduction, and behavioral effects. Environments contaminated with arsenic contain only a few species and fewer numbers within species. If levels of arsenate are high enough, only resistant organisms, such as certain microbes, may be present. If arsenic poisoning occurs over a brief period of time, symptoms may include vomiting, abdominal pain, encephalopathy, and watery diarrhea that contains blood. Long-term exposure can result in thickening of the skin, darker skin, abdominal pain, diarrhea, heart disease, numbness, and cancer.

I am not sure how helpful my answer would be. But if you used an LCA software like GaBi or simapro, you can then select the LCIA methodology you want and it does not require that you change your inventory at all. Using GaBi, you can copy your plan and evaluate the impacts based on ReCiPe, or you can just change the LCIA methodology in the same plan. However, to do the comparison itself, you may need to transfer your results to a spreadsheet.

I suspect that you will find significant difference between the results as most LCIA methodologies are developed using similar concepts. What you may find, however, is that the two methods don't cover exactly the same impact categories.

I hope it helps.

Dear Maryell,

The acute toxicity tests, most tests are conducted to determine the nature of any toxicity that can be produced by repeatedly dosing animals over an extended period. NOAEL and LC50 and LD50 are quite popular method. The highest exposure of a chemical, determined in toxicity tests etc., having no adverse effect (e.g., onset of sickness) even when the chemical is taken (exposed) daily for the rest of one’s life. In practice, mice, rats or other animals are forced to take a chemical for a certain period of time. This test is repeated several times at varying dose levels. The highest dose level causing no adverse effect in these tests is adopted as NOAEL (No Observed Adverse Effect Level).other related terms you could use in your study like; LOAEL (Lowest Observed Adverse Effect Level) NOEC (No Observed Effect Concentration) and LOEC (Lowest Observed Effect Concentration)

Use this formula for your concern. (NOAEL (No Observed Adverse Effect Level) value calculation.

Toxicity studies etc., / UFs (Product of Uncertainty Factors) [ to convert it to human NOAEL (Unit converts in e.g., mg/kg/day)].

TDI (Tolerable Daily Intake)=

NOAEL (No Observed Adverse Effect Level)

UFs (product of Uncertainty Factors)

ADI (Acceptable Daily Intake) and RfD (Reference Dose) are also used as terms having the same meaning as TDI.Graph you ca take between frequency of hazards vs chemical exposure.

The LD50 is defined as the lethal dose at which 50% of the population if killed in a given period of time; an LC50 is the lethal concentration required to kill 50% of the population.

Reference: https://www.nite.go.jp/en/chem/shiryo/ra/about_ra4.html

Ashish

Following.

Here are some results that yielded after a simple search query in Google Scholar:

To convert the different average time values, we use Peak to Mean Factor concepts that developed by Smith, 1973 and improved by Pringer et al., 2007. They are originally for odour, however it can be used for the other kinds of pollutants.

True, but I think the problem comes from the fact that you're using acute data which typically will entail a factor of 1000. It's not clear from your question if you know what the heavy metals in your mixture are. if you don't then I think that 1000 is not unreasonable to be conservative. If you know what they are then you might consider adjusting your AF according to the (known) relative toxicity of each metal (e.g. Zn should carry a lower AF than Ni) but you'll still need to justify why you reject the more conservative AF.

Yes, i can possible in highly polluted soils.......

You are welcome 

And I will try to find it 

Kind regards

please try this one.  You need to do web search with EPA website.  there are so many.

Interesting discussion...

Vegetation helps stabilize forested slopes by providing root strength and by modifying the saturated soil water regime. Plant roots can anchor through the soil mass into fractures in bedrock, can cross zones of weaknessto more stable soil, and can provide interlocking long fibrous binders within a weak soil mass. In Mediterranean-type climates, having warm, dry summers, forest evapotranspiration can develop a substantial soil moisture deficit which can reduce both piezometric head and slope mass. Pore water pressures change seasonally in response to precipitation and are often the driving mechanism which ultimately leads to slope failure. When trees are cut, the root system begins to decay, and the soil-root fabric progressively weakens. The loss of root strength or increased soil moisture content or both after-tree removal can lower the slope safety factor sufficiently that a moderate storm and associated rise in pore water pressure can result in slope failure. After trees are removed, the frequency of landslides can increase. Source :Robert R. Ziemer,

Pacific Southwest Forest and Range Experiment Station, Forest Service, U.S. Dept. of Agric., 1700 Bayview Dr., Arcata, CA, US A

Hi Ishaq,

The following papers may help you with this:

Williams, E. S., Berninger, J. P., and Brooks, B. W. (2011). Application of chemical toxicity distributions to ecotoxicology data requirements under REACH. Environmental Toxicology and Chemistry 30, 1943-1954 http://dx.doi.org/10.1002/etc.583

Brain, R. A., Sanderson, H., Sibley, P. K., and Solomon, K. R. (2006). Probabilistic ecological hazard assessment: Evaluating pharmaceutical effects on aquatic higher plants as an example. Ecotoxicology and Environmental Safety 64, 128-135.

Centile is just a shortened version of percentile, and means the same thing.

The screening point value is some measure of toxicity that you choose to decide whether there is a toxic hazard. In the Williams et al. paper, they have chosen the first and fifth centiles as their screening point values.

May be using particulate emission factor (PEF)?

Reference:

US EPA ,United States Environmental Protection Agency, 2002. Supplemental guidance for devoloping soil screening levels for superfund sites. OSWER 9355.4-24, Washington, DC: Office of Emergency and Remedial Response.

Dear Carmela

for some heavy metals such as lead or cadmium, the RfD is different according to the kind of food, but the references which our friend mentioned seems helpful: 

http://walidou84.free.fr/fichiers_utiles_m2pes/cours/Master%20II%202008%202009%20Intervention%202/Liste%20docs/VALEURS%20REGLEMENTAIRES/docs%20complementaires/INERIS-rap_vtr_86177-08805B%5B1%5D.pdf (in french)

http://www.atsdr.cdc.gov/mrls/pdfs/atsdr_mrls.pdf.

Best Regards

The environmentally relevant concentration is that which affects the ecosystem, independently of human exposure levels.

Here is the definition from the DHHS Environmental Health & Toxicology website:

Ecotoxicologically (environmentally) relevant concentration (ERC)

Concentration of a pesticide (active ingredient, formulations, and relevant metabolites) that is likely to affect a determinable ecological characteristic of an exposed system.

As you are looking for any clues:

You might want to consider if risk taking propensities apply to all environmental risks, or, to sub-sets, etc.

For example, some decisions might not be processed in terms of risk per se, and some might not be processed in terms of risk to the environment.

Examples:

A person might change their car's oil, and decide to dump it into the nearby storm sewer at his curb rather than store it for later recycling.  Was doing that potentially based on their propensity for taking risks that could harm the environment, or, on their propensity to take risks regarding getting caught polluting?  Etc.  (Short term/own actions/risk/benefit issues/variables regarding political leanings, etc)

A person might vote to preserve green areas and not to allow development in an area.  Is that potentially because they have a propensity to not take risks with the environment?  Is a vote not to preserve the land, and to allow development potentially due to a propensity to take risks towards the environment?  Are the votes more aligned with their political leanings than an actual risk taking propensity? (Long term/other's actions, etc.)

Are the propensities above truly tied to risk taking disposition, or, to simple pro/con environmental biases?  You may need to establish that their actual conceptualization is actually processed in terms of environmental risks.

IE: IS THERE a risk based PROCESS that applies to environmental risks, that can BE modeled...and which would not simply be an overlay of political/socioeconomic factors, etc.

THAT might be the primary issue to resolve, teasing out a disposition that is not in line with those other factors, and which can be unique to the environment...or, at least potentially, to a sub-set of environmental issues.  Finding sub-sets with usable data may be job #1.

Does that help?

Personally, I think they are the same concept.  There are caculated as ratio of the exposure estimate over an acceptable effect level, which used for lower tiered risk assessment. 

In addition, you can learn more methods of risk assessment methods in the follow reference.

Generally speaking, environmental engineer should do environmental impact assessment for any project including the transportation systems, participating in planning phase, risk assessment studies, nature protection, and sustainability. as any new project has direct impact on environment and Eco system. As we considering autonomous car many questions will rise such as is there any special services for such cars? Any extra infra structure? How do they interact with environment and its elements? Lastly, Environmental engineering is related to civil engineering in educational levels so both can have many overlaps.

On the other hand, chemical engineers can participate in control systems for example controlling the combustion rate of fuel; develop more efficient fuel types for autonomous cars.

This may be of interest: The Impact on Health of Emissions to Air from Municipal Waste Incinerators RCE 13

Movement is definitely a possible endpoint with ecological relevance. It is often used in ecological assessment and mode of action studies using fish, fish embryo or invertebrates (e.g. nematodes). For practical reasons it is mostly done in laboratory settings using tracking camera's and software, either 2D or 3D. Using it in the field, seems to me, a difficult task. Maybe it is possible to transplant invertebrates from a pollution gradient to a lab situation and register differences, but that remains to be tried.

dear Ivan i send you one of my paper about risk management. i think that you can find something of interesting

Dear Marc,

Under an ecotoxicological point of view, there are some indications that acrylamide may pose significant threats, likely following prolonged exposure to "low" concentrations, to aquatic vertebrates and invertebrates. By low concentrations I mean a few mg/L (ca. 1-10), which is found only at wastewater outlets. We have a publication coming out soon with a study on mussels. I'll render it available as soon as possible. The main effects we found so far relate to oxidative stress and gonadotoxicity, in females. However, the effects of chronic exposure in aquatic wildlife are not yet well understood. In general, acrylamide acute toxicity is low (e.g. LC50 = 400 mg/L in some freshwater fish).

Cheers

Pedro

You could check out The Environment as Hazard by Burton, Kates and White. Also, Patricia Martel, a PhD student at Wilfred Laurier University, Ontario, Canada is doing her thesis on disaster taxonomies, and might have some useful information for you.

Certainly you can, provided you have a proper time series for each. These variables are measures of a part of the climate system, hence they have stochastic and stationary components in their time behaviour which make them suitable for statistical analysis involving uncertainty.

Yes, thanks.

Nevertheless, personally I do not support the use of TEQ in sediments. Dominant compounds in this compartment may not be taken up evenly by organisms. Moreover, CCME guidelines are based on toxicity to sediment dwelling organisms, while dioxins toxicity is much more of concern for vertebrates, in particular mammals.