Science topic

Depression - Science topic

Depressive states usually of moderate intensity in contrast with major depression present in neurotic and psychotic disorders.
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Dear colleagues, Current Neuropharmacology (IF 4.8; Q1 WoS and Scopus) announces a Special issue on
Emotion (Dys)regulation: An integration of Pharmacological, Neurobiological, and Psychological Frameworks
Sub-topics are as follows:
Pharmacological aspects of emotion regulation
Neurotransmitters involved in emotion regulation
Pharmacological interventions in emotion dysregulation
Neurophysiology of emotion regulation
Peripheral and metabolic mechanisms of emotion regulation
Emotion dysregulation in clinical samples
Psychotherapeutic and educational practices to improve emotion regulation
Submission closes on 31st December, 2025
To submit, go to:
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Mr. Vladimir Kosonogov, I'm interested.
Please share full information on WhatsApp: +66 82 078 7423
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Hi all,
I'm currently doing my masterthesis and I'm a bit stuck. My thesis researches the link between self-esteem and social relationships and how anxiety and depression influence this relation (a moderation). However, I can't seem te find any research to back-up my moderation. My hypothesis is that there's a positive relation between self-esteem and social relationships, but with higher scores of anxiety/depression, the relation weakens.
I do find enough research that indicate that there's a positive relation between self-esteem and social relationships - and that this relation can variate with other factors, such as age, gender or ethnicity. Just not anything about anxiety or depression.
What I also find are direct links between anxiety and self-esteem, anxiety and social relationships, depression and self-esteem, and depression and social relationships. Which all indicates that there's a negative relation between the variables.
So, my question is: how can I best justify my moderation?
Thanks in advance!
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Can't it be a finding that there is less literature around the phenomenon you want to explore? I think you can put it as a limitation that finding that there is less literature around you talking about the phenomenon you want to explore. Then you can try looking into literature related to what you want to explore, in that sense you will be strengthening the significance and uniqueness of your study.
Blessed regards
Mohammed X
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I´ve been reading some articles on bipolar disorder and it seems to be defined by the extreme shift between manic an depressive moods. However I don´t understand how that shift happens. Does it need a trigger to happen?
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This is related article to the issue of bipolar disorder.
Bipolar blood test sparks research row
"A blood test that uses biomarkers to distinguish bipolar disorder from depression could slash the time it takes to get an accurate diagnosis, claims its maker Alcediag. The test uses biomarkers related to RNA editing to diagnose the condition, and has already been approved in France and Italy. However, the small size of trials that underpin the test and the lack of independent replication of their results has left some researchers unconvinced..."
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La Ansiedad y depresión son un problema de salud mental y los universitar
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A possible hypothesis for a research work on anxiety, depression, academic performance, and social behavior in university students could be:
"High levels of anxiety and depression negatively impact academic performance and social behavior in the classroom among university students."
You could further refine this by examining specific relationships, such as:
  1. "Increased anxiety is associated with lower academic performance and reduced social interaction in classroom settings."
  2. "Depression mediates the relationship between anxiety and academic performance in university students."
These hypotheses can guide your research design and data collection.
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Liu Zhaosheng. 2003. Triassic and Jurassic sporopollen assemblage from the Kuqa Depression,Tarim Basin of Xinjiang,NW China. Palaeontologia Sinica, New Series A,no. 14(Whole no. 190). Beijing:Science Press. 1‒244(in Chinese with English abstract)[刘兆生. 2003. 塔里木盆地库车凹陷三叠 纪和侏罗纪孢粉组合. 中国古生物志,总号第 190 册,新甲种第 14 号. 北京:科学出版社. 1‒244]
Zhang Shiben,Huang Zhibin,Zhu Huaicheng et al.. 2004. Phanerozoic subsurface stratigraphy of the Tarim Basin. Beijing:Petroleum Industry Press. xviii + 300 pp (in Chinese)[张师本,黄智斌,朱怀诚等. 2004. 塔里木盆地覆盖区显生宙地层. 北京:石油工业出版社. 1‒300]
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I've just received the papers!
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Condition: "particular mode of being of a person or thing" / "a requisite or prerequisite, a stipulation," / "state; behavior; social status"
Symptom: "a departure from normal function or form as an expression or evidence of a disease," / "a happening, accident, disease," / "to befall, happen; coincide, fall together,"
It's becoming very apparent that mental health labels and loose terms of diagnosis are starting to cost countries a fortune.
I wanted to ask about depression because in the UK, some people get told they have depression as a condition. That diagnosis on its own can gain extra financial benefits and medical support.
When it is labelled alongside other things, it seems to convert into a symptom. Which I would imagine meant it is WORSE that depression. It's depression, plus some.
However, if it is "just" a symptom, it doesn't have any financial weight, or it is extremely hard to get the right support, because other symptoms start leading professionals into focusing on groups of symptoms which are faster than ever turning into conditions/ing.
Depression used to be called meloncholia. After a guy won a nobel prize in conditioning, the world had two massive wars within years. Then it was labelled "the Great Depression"
Depression is a transitional position. It feels like it is actually impossible to be depressed as a condition. If you are long term depressed, you must surely have other symptoms and conditions causing the depression to last? Or you must have not acknowledged something about yourself that needs processing properly?
I can think of no person who is just "depressed" without a story of why they feel that way, or where its cause is from." It feels like a bad therapist to just leave people with the label "depression". Is it not medically a term for "unfinished business"?
And strange that it can provide extra financial benefits, especially when people with more serious diagnosis struggle to get that same help and support.
What is so special about depression that it can be used as condition and symptom?
It feels like a loop hole created on purpose to trap poor people and or to make a bad therapist look good.
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From the viewpoint of quantifying mental health or mood science, depression is a mood disorder that causes a persistent feeling of sadness and loss of interest, i.e. the affected person is no more able to regulate her moods and the mental state of chronification can set in. Consequently, depression is a symptom as chronifying mental and physical response to a condition, e.g. learned helplessness.
Psychologists first described learned helplessness in 1967 after a series of experiments on animals, suggesting that their findings could apply to humans. Learned helplessness leads to increased feelings of stress and depression. For some people, it is linked with post-traumatic stress disorder (PTSD).
The most common treatment is therapy, especially cognitive behavioral therapy (CBT). CBT helps people overcome these types of challenges by changing how they think and act.
Ref/
_________
You Keri Mobbs wrote:
Depression used to be called meloncholia. After a guy won a nobel prize in conditioning, the world had two massive wars within years. Then it was labelled "the Great Depression".
Skinner, John B. Watson and Ivan Pavlov, are considered to be the pioneers of modern behaviorism. You seem to mean Ivan Petrovich Pavlov, Nobel Prize in Physiology or Medicine (1904)?
The inventor of the phrase, the Great Depression, is Lionel Robbins, a British economist who lived during the Depression. In 1934, after Hoover’s tenure in office, Robbins wrote the book, The Great Depression, which contains what some historians, notably David F. Burg, consider to be the fist usage of the phrase we now use to to describe the economic meltdown on the 1930s. Although Hoover did not quite invent the term the Great Depression, he did play a role in its formation.
Ref/
An expansion of bank credit creates a boom that cannot be sustained, and the inevitable collapse of the boom is the depression. Robbins traces this boom/bust cycle, detailing the central bank policies of the 1920s. If the policy gurus at the Fed had read and understood this book, they could have spared us the economic havoc of recent years.
__________
Keri Mobbs Economics is still very blind to the workings of human psychology as it uses linear models for accounting purposes, which are unable to understand the cyclical movements of human economic activity.
The mentioned Great Depression was, imo, a full central banking failure (systems error), which is also state failure (planning agency put over the market forces).
_____
PS/
There have been three great inventions since the beginning of time: fire, the wheel, and central banking. '' The reason Will Rogers made this statement is central banking is so important because the money supply is so important.
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I need the Tunisian version of the Children’s Depression Inventory (CDI) for my master dissertation can anyone help please?
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Trierer Inventar zum Chronischen Stress (TICS) [Trier Inventory for Chronic Stress (TICS)]
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  • January 2004
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  • Peter Schulz📷
  • Wolff Schlotz📷
  • Peter Becker📷
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This appeared in my recommended, based on your interest box once I looked up das Trierer Inventar. I expect you might access the text in the same manner.
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What happens to all the overworked people in academia?
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It would not be correct to say so.
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Wellbeing and School Curricula
Why isn't psychological well-being (PWB) a core component of secondary school curricula?
I'm very curious to hear input from educators and policy makers on the above issue. Or from anyone reading this.
Is it because:
1) Secondary school curricula are traditionally focused more on intellectual growth than emotional growth. Parents are regarded as the source of education on emotional growth.
2) Educators and policy makers are not aware of the increase in rates of depression among teenagers globally.
3) Educators and policy makers are not focused on student depression / anxiety as they don't think it affects academic performance.
4) A lack of data on the impact of student wellbeing on student performance, resulting in skepticism that changes in curricula may have an impact on student wellbeing.
5) A different reason?
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Interesting question and replies.
Curricula are supposed to include goals about wellbeing and not only knowledge.
The crucial question is whether these goals are feasible and given the right attention or priority.
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Depressed? Addiction? Mindfulness?
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Am in full agreement with your observation s. Rama Gokula Krishnan that a more holistic and integrated approach of mental health (also with respect to the interplay with physical health) is in the making, i.e. the human organism is an information field, in terms of the physiology of behavior.
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AI plays a crucial role in researching neurobiological factors associated with depression by analyzing vast amounts of data, identifying patterns, and assisting in the interpretation of complex biological information. Machine learning algorithms can analyze neuroimaging data, genetic information, and other biomarkers to identify potential indicators of depression. Additionally, AI models contribute to the development of personalized treatment approaches by considering individual variations in neurobiology, ultimately advancing our understanding and treatment of depression.
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Artificial intelligence (AI) has become an integral part of various fields, including neuroscience and mental health research. In recent years, AI has significantly contributed to the ongoing research and understanding of the neurobiological factors associated with depression. By analyzing vast amounts of data, AI algorithms can identify patterns and correlations that humans may overlook. I will discuss how AI is revolutionizing depression research by enhancing diagnosis accuracy, predicting treatment outcomes, and uncovering novel insights.
Firstly, AI algorithms have shown remarkable accuracy in diagnosing depression based on neurobiological markers. Traditional diagnostic methods heavily rely on subjective assessments by clinicians, which can be prone to biases and errors. However, AI systems can analyze brain imaging scans and genetic data to identify specific biomarkers associated with depression. By comparing these patterns with a large database of known cases, AI algorithms can provide more objective diagnoses with higher precision.
Moreover, AI has the potential to predict treatment outcomes for individuals suffering from depression. By analyzing a patient's neurobiological data alongside their response to different treatments, AI algorithms can identify which interventions are most likely to be effective for specific individuals. This personalized approach not only saves time but also increases the chances of successful treatment outcomes.
Furthermore, AI is capable of uncovering novel insights into the complex neurobiology underlying depression. With its ability to process massive amounts of data from various sources simultaneously, AI algorithms can detect subtle patterns that may go unnoticed by human researchers. These discoveries could lead to breakthroughs in understanding the underlying mechanisms of depression and developing more targeted treatments.
In conclusion, artificial intelligence plays a crucial role in advancing our understanding of the neurobiological factors associated with depression. Through its ability to analyze vast amounts of data quickly and accurately, it enhances diagnosis accuracy while predicting treatment outcomes for individuals suffering from this debilitating condition. Additionally, it uncovers new insights into the complex nature of depression that could lead to improved treatments in the future. As technology continues to advance, AI will undoubtedly continue to contribute significantly to depression research and ultimately improve the lives of those affected by this mental health disorder.
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Who conducted the research of Women's Mental Health: Depression and Anxiety by Robynn Zender and Ellen Olshansky?
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Nursing Clinics of North America
Volume 44, Issue 3, September 2009, Pages 355-364
📷
Women's Mental Health: Depression and Anxiety
The authors are:
Robynn Zender MS, Ellen Olshansky DNSc, WHNP-BC, FAAN
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SO WHAT ARE THE JOBS WITH HIGHEST SUICIDE RATES?
1. Medical Doctors 2. Dentists 3. Police Officers 4. Veterinarians 5. Financial Services 6. Real Estate Agents 7. Electricians 8. Lawyers 9. Farmers 10. Pharmacists
Each year in the U.S., roughly 300 - 400 physicians die by suicide;
.
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Prof Stephen David Edwards is correct in my opinion, normally women are more empathic an compassionate. Therefore mind has gender as I wrote the article below: 18TH CENTURY ENGLISH PHILOSOPHER MARY WOLLSTONECRAFT'S FALSE CLAIM THAT "MIND HAS NO GENDER"
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Dementia affects several millions of people across the world. There are various types of dementia and Alzheimer’s disease is the most common type. This disease that is yet to have a cure affects several millions of families in several different ways.
Dementia is a global crisis, perhaps we can call it the dementia pandemic! Dementia is everyone's problem and as a result, we must take the responsibility to look after ourselves. One of the easiest ways to support people living with dementia and their caregivers is to be aware of this disease and by so doing we probably can become a little bit more compassionate and dementia-friendly.
Most people living with dementia receive care from their immediate family members or friends and neighbours. This makes informal caregivers become isolated. They become what we call the invisible patients. Most people who provide informal dementia care are female caregivers mainly spouses, female adult children, family members, friends, and neighbours.
Dementia caregivers suffer equally as they are mostly available to provide the required care for their family members who are living with dementia. Informal caregivers suffer from several conditions such as physical and psychological challenges,
loss of job/livelihoods = financial problems, and loss of social life such as their hobbies. Among other challenges, informal caregivers suffer from stress, anxiety, and depression.
How dementia-friendly are you and how dementia-friendly are your localities?
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I must express my heartfelt thanks to you all who have been contributing to this discussion so far. Thank you so much, Ljubomir Jacić Mary C R Wilson Sundus F Hantoosh Mauro Colombo Anamitra Roy
Best regards
Muhammad
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A question that will debate and controversy...
Anxiety and Depression are only subjects of Psychiatry and Psychology?
What do you think Dear colleagues?
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Anxiety typically involves excessive worry, fear, and unease about future events or circumstances. It often manifests as physical symptoms like rapid heartbeat, restlessness, and difficulty concentrating. Whereas: Depression, on the other hand, is characterized by persistent feelings of sadness, hopelessness, and a loss of interest in activities. It can lead to changes in appetite, sleep disturbances, low energy, and thoughts of self-harm.
Moreover, anxiety focuses on apprehension and excessive worry, depression centers around a pervasive sense of sadness and disinterest in life.
However, it's important to note that both conditions can have overlapping symptoms and may require professional help for diagnosis and treatment.
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Suicide and Death Penalty, fatal and tragic acts, leave no one indifferent. These touch on the sacredness of life and therefore on the deepest convictions and beliefs. Philosophical reflection has been prolific on the subject dealing with the rationality and morality of and Death Penalty. The question also covers a societal component in relation to the debate on the "right to die within dignity"
All contributions on the topic are welcome.
Picture: Staged seppuku with ritual attire and kaishaku, 1897 https://en.wikipedia.org/wiki/Seppuku
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„Where there is love there is life.“ — Mahatma Gandhi
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There are several methods to consider the imperfection of the structure like the eigenvalue buckling mode, nonlinear buckling mode and weld depressions. But how to define imperfection as an equation in Abaqus software?
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In Abaqus software, imperfections can be defined using the "perturbation" feature. The perturbation feature allows you to introduce small geometric imperfections into your model and to analyze their effect on the structural response. The perturbation can be defined as a displacement or a rotation in one or more directions at a specific location on the model.
To define imperfections as an equation in Abaqus, you can use the expression language in the perturbation feature. The expression language allows you to specify the magnitude and direction of the perturbation using mathematical expressions. For example, to introduce a sinusoidal imperfection in the z-direction, you can use the following equation in the perturbation feature:
AMPLITUDE*sin(2*pi*x/WAVELENGTH)
Where AMPLITUDE is the magnitude of the imperfection, WAVELENGTH is the wavelength of the sinusoidal imperfection, and x is the coordinate along the direction of the imperfection.
You can also use other mathematical functions and operators in the expression language to define imperfections, such as exponential functions, trigonometric functions, and logical operators.
It is important to note that the definition of imperfections in Abaqus should be based on the specific imperfection that is relevant to your analysis and should be validated against experimental or analytical data if possible. Additionally, it is recommended to perform sensitivity analyses to investigate the effect of different imperfection magnitudes and locations on the structural response.
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corona pandemic is worldwide issue and during the lockdown period people are getting depressed. so let all discuss their way of handling stress
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Worries & anxiety remain the problem for well many human beings to come in the mode of progressive development for the individual . Worries all the time start with anxiety which remain the disturbing phase for certain individual .
Worries remain in mental attitude for our thinking phase & for every human beings one can certainly get rid worries for their action of life . For Corona or any serious problem even of our health which we have to keep our mind in silent mode to find the solution of the same peacefully .With this you have to understand that any disturbing phase of our life may also come out as a part of our destiny . With this also not in our hand .
This is my personal opinion
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Where could you find Numbers and Reports focus on Depression?
Dear research community,
In order to make the best with a focus on Depression, I tried to be aware about the different resources around Mental Health disorders.
I would like to know about updated reports in this issue not only in Europe but also if there is some other oficial reports or web observatories of this topic.
Thanks in advance.
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Thanks Jakub Opelka , we also explore these webpages before. Curiosly, the update of all have as reference the year 2019. Until 4 years these databases were not update. Why?? Perhaps the data is really scare. Anyway, we also use another sources as:
All of them have the same problem with the updating of data. There is not data after 2019. I guess this situation is worried in all senses.
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What is the appropriate way to report the relationships among three interrelated variables in an observational study using GLM or mediation models?
Variables: Depression, Neuroticism, Mindfulness
Regression models - All of the relationships are statistically significant and passed assumption checks.
Mindfulness predicts Depression: R2 = .247, unstandardized coefficient = -5.32
Mindfulness predicts Neuroticism: R2 = .503, unstandardized coefficient = -3.88
Neuroticism predicts Depression: R2 = .732, unstandardized coefficient = 1.02
Should I simply report all findings, starting with the "strongest" relationship, Neuroticism predicts Depression?
Multiple Regression models - All of the relationships are statistically significant and passed assumption checks. Is there a benefit to reporting these model results beyond what is gained from the above regression models?
Mediation models - All of the relationships are statistically significant. All Sobel test results are statistically significant.
See attached file with results of mediation models and Sobel tests. Would I report all models or only that Neuroticism mediates the role of Mindfulness on reducing Depression, and Depression mediates the role of Mindfulness on reducing Neuroticism?
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What is your hypothesis? It might be, for example, that mindfulness moderates the relationship between neuroticism and depression.
I'd strongly recommend drawing a causal diagram (wonderful online tool here http://www.dagitty.net which is really helpful in defining the relationships implied by your model).
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I'd like to know if there are other measures of hopelessness. As far as I know, there is only the Beck's Hopelessness Scale.
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Positive and Negative Suicide Ideation (PANSI)The Positive and Negative Suicide Ideation (PANSI) (Osmanet al., 1998)
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High school physics does not have the best affective appeal - not reputation among general student population-possibly due to content emphasis. Affective aspects however are important in early to mid stages of education.
Torques, circular motion and coulomb fields are depressing topics to the scientifically inadept; even more modern physics topics and high esteem terms like "constructive superposition" of waves and related phenomena do not faire better.
One however has to keep in mind that the mission of k 12 is to build some conceptual& skillsets/deep understandings that the next generations of scientists should have. Appeal is quarabteed what ever the topic choice for the later.
So the compromise is hard-leading to current win-lose solutions.
Suggestions such as for incorporation of whole year curricula on exciting, high contemporary culture status topics like radioactivity, laser apps, Vacuum conductivity have been raised to be balanced with half year courses on scientific method-a facelifted version of mentioned depressing content with deenpasis on content to teach skills/conceptual areas but keep things attractive to general population are still to gain persuasive power to be adopted.
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Another key point is the current philosophy of easy to hard progression: Laws of science as "connecting changing terms and being immanent in what it govers, are relations" and then a theory (higher grades).
The choice of theory has been critisized (constructively and with best interest in mind), what about choice of laws?
Newton is choses, adding to the depression. Not because there is something wrong with it - but because of its conceptual inconcistency (not generally admited but see my articles of vaqueness of concepts in physical science), its abstraction and its nearness to a Theory of Everything (a unifying principle of nature) that makes it hard and above the threshold challenging. Easiest laws-based year-long theme-based units should be considered and Newton as an appendix and certainly not a repeated year by year, topic.
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Depression duration varies by individuals and recurrent episodes can be observed. What are the determinants of duration?
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Inability to control moods, resulting in pathological mood chronification.
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What do you think are the factors that have depressed this generation?
Why do you think it's hard for them to open up to others when it comes to telling how they feel?
The deterioration of mental health is practically notorious in Generation Z.
There are many young people today who do not share their problems and decide to deal with them alone.
The truth is that there is more and more use of antidepressants, more assistance to psychological therapy and more self-reported expression of anxiety and sadness in these generations.
List of some of the things that some people tend to do when they feel bad : listen to music, take refuge in drugs, buy clothes, dye your hair, don't eat, insomnia, etc.
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I believe the depression age was the age of the industrial revolution and the end of the extended family, today is rather the age of eating disorders.
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How should you properly handle the relationship between yourself and your teachers in a situation where your advisor does not give you any academic guidance while you are pursuing your master's degree and you have to worry about graduating all the time,
while your teacher also attacks you psychologically? I thought I was going to get depressed.
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Speak to your graduate department head.
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Do I need to request permission to use Beck Depression Inventory and Beck Anxiety Inventory or I can simply use them since they are available in the public domain. How I can decide that any given survey is in the public domain or not ?!
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If it has already been psychometrically tested in your country, you can use it, otherwise, you must send a message to the medical author and designer and get permission to psychometrically test it.
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I have been searching about the preventive intervention for depression and found several interventions but they are mostly for adolescents, I am searching for preventive intervention that are specific for adults.
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Moderate aerobic exercise for at least 30 minutes per day, 6-7 days per week, has been shown to improve depression and anxiety in several age groups.
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The scales scoring guide (https://www.childfirst.ucla.edu/wp-content/uploads/sites/163/2018/03/RCADSUsersGuide20150701.pdf) only applicable for children till 12th grade. If I wanted to use it for adults (for which it has been validated but scoring guide unavailable. ), how should I score it?
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Thank you for the suggestion Dr. S. Béatrice Marianne Ewalds-Kvist
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Alcohol is a central nervous system depressant
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Alcohol is the most commonly used drug among athletes. And as you probably know, excessive drinking has its downsides. But the timing, dose, and frequency of alcohol use are critical factors in whether or not it disadvantages you.
For example, a few beers before a game can cause more than enough impairment to hurt your performance. But a glass of red wine a few days prior to a competition might relax you, which may actually be helpful.
However, there are also cumulative, long-term effects of drinking alcohol that might undermine you no matter what. A lot of it comes down to how much, and how often you drink.
Does Alcohol Affect Your Athletic Performance? - Ria Health
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Anxiety is something which happened before any situation and Depression is that which happened after any situation. Up to what extent it is true?
Waiting your response. Do you agree with it?
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When you are worrying about something for a period and it makes everyday life hard, then feeling of panic or fear is called Anxiety.
Worrisome when felt intensely for a long period of time, it effects mental health and is referred as Depression. Muhammad Rashid
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I want to find the correlation coefficients between two signals and to plot the deference in values using matlab. I have two eeg.edf datasets with 20 electrodes each, of healthy and depressed subjects. I have to find the correlation coefficient between electrode Fp1 healthy and Fp1 depressed, F3 healthy and F3 depressed and so on..
Can anyone help me with the code? so far I used edfread to transform my datasets in timetable variables.
Thank you!
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you can use corrcoef function in matlab
A = [1 2 5 7 5 1 7 3 4 7 1 2 ]
B = [7 3 3 2 8 4 7 9 4 8 2 5 ]
C = corrcoef (A,B)
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I am doing a research project on prolonged social media use and the mental well-being of college students. I want to compare time spent as well as gender to the results of the Beck Anxiety Inventory and the Beck Depression Inventory. How would I go about comparing these and what statistical test would I perform?
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It sounds like you want to do a pair of regression analyses with social media use and gender as your predictors in each. You might also want to include interaction effects to see if the effects of social media use differ by gender.
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I have to run a pearson correlation analysis between HPA axis markers- cortisol , ACTH and inflammatory marker IL-6(analysed through ELISA) and Depression anxiety questionnaires which were recorded at baseline and post interventions. I also need check the data for normality before running the analysis. If someone can provide an appropriate suggestion.
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Thank you so much Anna. Would like to ask when running a correlation analysis between hormones and questionnaire data for post-intervention do you correlate it directly with the post scores or first calculate the change score and then correlate it with hormones concentration ?
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Apologises I'm really confused and don't know how to do, appreciate any guidance you can give
Aim
1. To explore if anxiety is predicted by stress and treatment delay and whether this is moderated by Brief Cope strategies (Brief COPE) .
Design
1 continuous outcome variable – anxiety (let’s call this H)
2 continuous predictor variables (let’s call these D, S)
3 Continous Moderator - (lets call these BC - ef, bc pf and bc avoidant). These are inputted into SPSS as 3 separate variables as the questionnaire b-cope does NOT allow you to create a total score (by adding ef + pf + avoidant).
- D – delay
- S – Stress (measured by pss-10)
- BC pf - Brief Cope - 1 (consists of 4 questions with each questions represent a different factor)
- BC ef - Brief Cope – 2 (consists of 9 questions with each questions represent a different factor)
- BC avoidant - Brief Cope – 3 (consists of 4 questions which each questions represent a different factor)
To answer the aim I know i need to complete a hierarchial multiple regression but I don't know what to enter on what model or whether I need to do separate regressions and again what should be entered with what.
Q1. Can you please advise how my regression models would look as I can't work this out given my predictors, moderators and outcome variable listed below.
E.g. Model 1 ...
Model 2 ...
Q2. Do I need to look at interactions? If so which ones, how would this be put into SPSS ie in which models.
Possible Interaction examples ?
Stress x bc ef
Stress x bc pf
Stress x avoidant
Delay x bc ef
Delay x bc pf
Delay x avoidant
Q3. Do I need to run separate hierachial multiple regressions? If so can you please write how the model would look ie. Model 1 ..
Model 2...
To confirm I have completed only parametric tests. I have 1 group completing all predictor /moderators variables.
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It sounds like you would end up with a regression model in which you have a very large number of predictor (independent) variables as well as many interaction terms (since you have so many different COPE variables). My advice would be to first think about meaningful ways to reduce the number of COPE variables to be included in the model, for example, by aggregation (calculation of a summary COPE score), factor analysis, or simply selection of the theoretically most meaningful COPE variables. Otherwise you might run into various problems when entering all individual COPE items into the regression (e.g., potential collinearity, too many individual significance tests, large model with many predictors, loss of power to detect interaction and other effects).
Other than that, you could run a hierarchical regression model with only the main effects (predictors, no interaction terms) in the first model, then add the interaction terms in Model 2 to see if they add anything to the prediction of the outcome. But, once again, I would try to reduce the overall number of predictors first to avoid problems in the analysis.
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Depression is a mood disorder that involves a persistent feeling of sadness and loss of interest. What are the causes, effects and probable solutions to depression? Sharing is caring. Thanks!!!
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Depression is also a leading cause of disability globally and contributes greatly to the global burden of disease. Even though psychological and pharmacological treatments exist for depression, however, in low- and middle-income countries (LMIC), treatment and support services for depression are often absent. According to report from the World Health Organization, over 75% of individuals suffering from mental disorders in LMIC countries do not receive treatment. Depression is a serious public health challenge.
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Any studies provide this?
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Reading can help reduce the rate of depression
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Hello, I have a question related to the metabolism of Serotonin. I am studying its metabolism in case of stress and depression and I could not find out the relationship between serotonin and 5HIAA in this particular case. MAO-A (the enzyme which metabolizes the serotonin hormone) inhibition is usually the mechanism of a lot of anti deprdepressantgs, but I have found several articles where the concentration of 5HIAA was lowered in case of depression and also suggestive of a lower concentration of Serotonin itself. But it doesn't justify the mechanism of action of antidepressant drugs. so I am confused now and want an answer for the exact relation of Serotonin and 5HIAA.
If serotonin is metabolized in 5HIAA, then 5HIAA concentration increases and serotonin descreases, so as serotoni conc. decreases, depression will increase. Is it right or no?
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Hi,
The referenced article given below can clarify your doubt:
Jayamohananan H, Manoj Kumar MK, T P A. 5-HIAA as a Potential Biological Marker for Neurological and Psychiatric Disorders. Adv Pharm Bull. 2019 Aug;9(3):374-381. doi: 10.15171/apb.2019.044. Epub 2019 Aug 1. PMID: 31592064; PMCID: PMC6773935.
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Dear colleagues in the filed of psychology and similar disciplines,
Do you know some open call for cross-cultural project regarding post-COVID effects (or what is happening after the 2 years of pandemic) on various aspects of mental health (life satisfaction, stress, anxiety, optimism...), emotional status, interpersonal relationships, risky behavior, conspiracy theories and other beliefs, etc.?
Thank you.
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Dear Prof. Dinić!
I found the following websites hoping you consider these of value:
1) Coronavirus and COVID-19 Related Funding Opportunities from Research Professional and Pivot, Research Professional News is part of Ex Libris Group. Available at:
In this list I noticed the following: CDC-RFA-IP22-2203 "Tracking the burden, distribution, and impact of Post COVID-19 conditions in diverse populations for children, adolescents, and adults (Track PCC)" Department of Health and Human Services, Centers for Disease Control - NCIRD
Current Closing Date for Applications: Mar 07, 2022  Electronically submitted applications must be submitted no later than 11:59 pm ET on the listed application due date. Estimated Total Program Funding:$45,000,000
Further details are available at:
I really think these funds are also available for EU institutions as well.
Yours sincerely, Bulcsu Szekely
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I have been able to access the 10-item clinician-administered version but am interested in using the self-report form. I'm able to find articles that cite its use and those testing its viability but none that include the actual measure.
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Looking for this as well. Have you found it?
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Would it harm if an MDD patient receives TMS sessions 6 days a week instead of 5?
On a different note, is it required for the patient to receive 5 sessions in a row, or a patient could have TMS sessions simply 5 days a week in any order. Thanking you in advance.
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Some references:
Lefaucheur JP, Aleman A, Baeken C, Benninger DH, Brunelin J, Di Lazzaro V, Filipović SR, Grefkes C, Hasan A, Hummel FC, Jääskeläinen SK, Langguth B, Leocani L, Londero A, Nardone R, Nguyen JP, Nyffeler T, Oliveira-Maia AJ, Oliviero A, Padberg F, Palm U, Paulus W, Poulet E, Quartarone A, Rachid F, Rektorová I, Rossi S, Sahlsten H, Schecklmann M, Szekely D, Ziemann U. Evidence-based guidelines on the therapeutic use of repetitive transcranial magnetic stimulation (rTMS): An update (2014-2018). Clin Neurophysiol. 2020 Feb;131(2):474-528. doi: 10.1016/j.clinph.2019.11.002
Yang LL, Zhao D, Kong LL, Sun YQ, Wang ZY, Gao YY, Li N, Lu L, Shi L, Wang XY, Wang YM. High-frequency repetitive transcranial magnetic stimulation (rTMS) improves neurocognitive function in bipolar disorder. J Affect Disord. 2019 Mar 1;246:851-856. doi: 10.1016/j.jad.2018.12.102. Epub 2018 Dec 25. PMID: 30795490.
Buchholtz PE, Ashkanian M, Hjerrild S, Hauptmann LK, Devantier TA, Jensen P, Wissing S, Thorgaard MV, Bjerager L, Lund J, Alrø AJ, Speed MS, Brund RBK, Videbech P. Low-frequency rTMS inhibits the anti-depressive effect of ECT. A pilot study. Acta Neuropsychiatr. 2020 Dec;32(6):328-338. doi: 10.1017/neu.2020.
Chou YH, Ton That V, Sundman M. A systematic review and meta-analysis of rTMS effects on cognitive enhancement in mild cognitive impairment and Alzheimer's disease. Neurobiol Aging. 2020 Feb;86:1-10. doi: 10.1016/j.neurobiolaging.2019.08.020
Kaur M, Michael JA, Fitzgibbon BM, Hoy KE, Fitzgerald PB. Low-frequency rTMS is better tolerated than high-frequency rTMS in healthy people: Empirical evidence from a single session study. J Psychiatr Res. 2019 Jun;113:79-82. doi: 10.1016/j.jpsychires.2019.03.015
Ribeiro JA, Marinho FVC, Rocha K, Magalhães F, Baptista AF, Velasques B, Ribeiro P, Cagy M, Bastos VH, Gupta D, Teixeira S. Low-frequency rTMS in the superior parietal cortex affects the working memory in horizontal axis during the spatial task performance. Neurol Sci. 2018 Mar;39(3):527-532. doi: 10.1007/s10072-017-3243-8.
Lefaucheur JP. Transcranial magnetic stimulation. Handb Clin Neurol. 2019;160:559-580. doi: 10.1016/B978-0-444-64032-1.00037-0
Zhang H, Sollmann N, Castrillón G, Kurcyus K, Meyer B, Zimmer C, Krieg SM. Intranetwork and Internetwork Effects of Navigated Transcranial Magnetic Stimulation Using Low- and High-Frequency Pulse Application to the Dorsolateral Prefrontal Cortex: A Combined rTMS-fMRI Approach. J Clin Neurophysiol. 2020 Mar;37(2):131-139. doi: 10.1097/WNP.0000000000000528
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I just came across a really interesting point of view in a clinical manual by a leading author in clinical treatment in Spain, Miguel Ángel Vallejo. He claims, based on a study by Rude and Rehm (1991)*, that psychotherapy is most effective when it boosts already-existing capacities and skills, rather than when it focuses on ameliorating deficits. That seems to run counter to much of what I have seen before in psychotherapy; does this idea match your clinical experience, or do you have any additional bibliography that might support this claim Thanks a lot!
*The citation to this article is given as "Rude, S. S., & Rehm, L. P. (1990). Cognitive and behavioral predictors of response to treatments for depression. Clinical Psychology Review, 11, 493–514"; however, all I can find online is this other article, with a different name, although (apparently) similar content: https://psycnet.apa.org/record/1992-06180-001
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Respected Researcher
We, as experts in the field of clinical psychology, before we judge the individual as being sick with a certain disease, we prepare an initial interview for him/her to identify his personality through his/her words, appearance, facial features, and behavior, and then conduct him/her after that interview after two or three diagnostic tests in Intelligence, personality and thinking (mental and cognitive tests), and of course we rely on collecting information from some people close to him/her (family, children, brothers, friends, peers, etc.), because we cannot judge an individual who suffers from blindness for example, that he/she is a disabled person as soon as he/she lost his/her eyes, and we cannot judge a healthy person, who does not suffer from any disability, that he/she has several skills.
That is why we have to take proper and correct measures in the diagnosis process first, and this is explained by the published research that you read, and indeed this is what is meant, because if we focus on the individual’s disability, this may affect the loss of his/her important skills remaining, and this increases his feeling that he/she is a helpless person who is useless, and then he/she will make dangerous decisions that lead him/her to a tendency towards suicide or any other serious illness, so it is the responsibility of the psychiatrist or psychologist first and undoubtedly, to pay attention to the development of the skills available to the helpless person, and not to care about the extent feeling the level of disability, because the development of skills leads to the treatment of disability.
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I guess there must be some data collected regarding Covid and related to the field of psychology/psychiatry, considering its psychological impact. It might be gathered from the patients, family members or the society at large, either a public or private collection. Does anybody have any idea on how to access such data for research purposes?
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Dear Prof. Farhad Montazeri ,
For example, I saw of Lancet & Nature publications, just register, they will send updated data and research as you agreed to get updated - the ones you chose - to your email registered with them e.g.:
ABOUT THIS ALERT Access to article abstracts is open to all Alert recipients. Access to full-text articles is limited to subscribers who have activated their online access. Activate your online access to your subscriptions at (under elsevier):
@
the Lancet COVID-19 Resource Centre
@
Please add briefing@nature.com to your address book.
Enjoying this newsletter? You can use this form to recommend it* to a friend or colleague — thank you!
@
LitCovid is a curated literature hub for tracking up-to-date scientific information about the 2019 novel Coronavirus
@
Novel Coronavirus Information Center
Elsevier’s free health and medical research on the novel coronavirus (SARS-CoV-2) and COVID-19
@
COVID-19: Epidemiology, virology, and prevention
Hope I understand you correctly, dear Prof.
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Can you tell me "what are the different clinical stages of Mental Health among young boys. " Related to anxiety or depression...If so can you plz tell me those stages.
E. G
Stage 1
(a)
(b)
(C)
Stage(2)
Stage (3)
Stage (4)
Stage (5)
Stage(6)
----++++--
What are the characteristics of these stages?
How as a clinical practitioner do you define these characteristics.?
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As already indicated, the STAGES, according to the Natural History of both psychoptologies, would be AND IT IS unethical and antimoral to see how they appear and evolve WITHOUT INTERVENING IN THEM !; But the LEVELS or DEGREES or STAGES of SEVERITY are universally categorized or hierarchical, from less to more, as: a) ABSENT, b) MILD, c) MODERATE and d) SERIOUS -which is how they are hierarchized, for example, the BDI -Beck Depression Inventory- and BAI -Beck Anxiety Inventory-
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As said in the title, I'm looking for a journal related to my research Twitter Arabic Sentiment Analysis to Detect Depression Using Machine Learning
to publish it it's preferred to be Q1
please help me out.
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Joao Luiz Junho Pereira I don't think the journal you proposed is suitable for what Reem Ml is looking for. In fact the question was about a journal related to Twitter Arabic Sentiment Analysis to Detect Depression Using Machine Learning so it preferred to have a journal that deals with Machine Learning, Artificial Intelligence such as:
- Machine Learning with Applications not impacted but indexed (https://www.journals.elsevier.com/machine-learning-with-applications)
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What do you think are the most important psychological disorders associated with COVID-19?
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Los más comunes son depresión, estrés, miedo, ansiedad, desesperación, tristeza etc
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Friends and colleagues that use psychological questionnaires to measure depressive symptoms: what is your preference between the PHQ-9 and the BDI-II?
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The PHQ-9 has the advantage of being faster to apply because it has fewer items, but the BDI-II is more complete and, therefore, more reliable in addition to having more literature on it: in short, I advise -if you can choose between the two, the BDI-II.
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why the score value in research gate depresses with weekly updated, sometime?
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From my point of view, ResearchGate (RG) is the best attractive academic social networking portal for the scientific community. In practice, RG is one of the biggest social media networks for the scientific community. It is a platform and a gateway for sharing information and experiences between scientists, experts, researchers, and practitioners. Hence, I am grateful to the network opportunities offered by RG for having this access to a broad range of researchers, papers, projects, questions, and discussion threads that otherwise I can't find. In a nutshell, ResearchGate (RG) is a kind of social media with a strong academics/research emphasis.
But, let me point to the following issue. Recommend, Follow, Download an article is just social interaction. Despite it helps to increase the RG scores, it reflects neither the work quality nor the researcher position. This score depends on the institution where you work; this score in the institution where I work hasn't any remote feedback.
I know that the more you participate in various research activities, the more your research interest increases. Nevertheless, as we are researchers, the RG total research of interest shouldn't be our main goal in itself. We should do our job honestly, such as writing valuable articles, participate in the useful discussions that we are really interested in. If we don't bother with this metric, we will see that it will increase.
If we make this metric our goal, it will astray us and then we will jump over some fences to increase our metrics without real internal improvement.
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What are the pharmacological risks?
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Thanks you, Dr. Miky Timothy
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I am a Medical student working on a research concerning mental health on young people. I am looking for a scale with good properties for symptoms of depression and anxiety.
Since the questionnaire includes many other dimension, short version are particularly welcome!
Thanks in advance!
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For Anxiety the "BAI" (Beck Anxiety Inventory) and, for Depression, the "BDI" (Beck Depression Inventory) ... Idem Hamilton
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What are the factors that predispose patients to treatment-resistant depression?
What are the advances that predict antidepressant treatment response for depression?
Two articles about prediction of antidepressant treatment response by using artificial intelligence technology and machine learning algorithms:
  • An electroencephalographic signature predicts antidepressant response in major depression (doi:10.1038/s41587-019-0397-3)
  • Brain regulation of emotional conflict predicts antidepressant treatment response for depression (doi:10.1038/s41562-019-0732-1)
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Because it depends on several causes:
-The type and intensity of such Depression
-The one that, in addition, there is also an Anxiety Chart (Mixed Depressive / Anxious Disorder)
-Whoever is male or female
-The type of Antidepressant used: An MAOI is not the same as a Tricyclic type, an SSRI or a Selective Serotonin and Noradrenaline Reuptake Inhibitor or Epinephrine (SSNRI)
-The one who receives a Combined Treatment with, in addition to Antidepressants, Psychotherapy, eminently Cognitive Behavioral.
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I am working on a paper on unemployment on psychological health of youth. Please in what order can input the following predictors variables using age as the control variable?
Perseverance
Irrational behaviour
Low self esteem
Depression and
Suicidal ideation
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It probably depends on your hypotheses and your design. I'd look at inter correlations between them, to see if multicollinearity might become a problem, but in the end theory and predictions should dictate the order.
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To understand the gut microbiome-brain axis
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It seems yes. I read a study just last week that found that microbiota were associated with depression. Unfortunately, I deleted the source so I can't give you the reference :-( Sorry!
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I have done EIS of SDC (Sm doped ceria) electrolyte from 550 to 750C. I observed the formation of a depressed semicircle. Also the diameter of semicircle decreases with increasing temperature with a decrease in resistance. What could be the possible reasons for formation of depressed semicircle? I am attaching the figure below.
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Thanks, I will study the article you referred. I have tried to construct an equivalent circuit using Zview but it simply leads to Rt(QRep), which is not very helpful to reveal the true information.
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how many of us undergo the Stress of being a researcher ? does it motivates only or it does Depresses at Times ?
please do write your views on it.
Best regards
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It can be nerve-racking at times, but can provide the necessary impetus to create something meaningful.....something that makes a good contribution to the field.
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Hey, all! I'm conducting research including a depression screening as one of our measures - the Centers for Epidemiological Studies Depression scale (CES-D). This tool has cutoff scores: ≥16 indicates clinically relevant symptoms and ≥27 indicates moderate to severe symptomology.
Our team is discussing how to disclose to participants if they had a positive score after our research concludes. We want to inform subjects of anything clinically relevant but not cause harm. How do you all handle this?
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NEVER !: it is an investigation not a clinical practice ... but also, if the anonymity of the participants in it has been rigorously guarded - as required by the APA, the "Lex Artis" and common sense, so that the subjects fill in The Tests - how can the researcher / s know who each Test is or are and their results?
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I do not know if this is a merely coincidence, but over the last number of years doing research, I have seen hundreds of Junior Researchers (either undergrad, master of PhD students) suffering from blood- and energy-suckers coordinators. Perhaps, this is a consequence of multiple factors (such as history of academic world and labor western culture). The fact is, as a Senior Researcher or Project Coordinator, what do you do to avoid or to alleviate mental illness amongst your students?
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The psychological stability has a great impact on the educational level, because it increases his desire for education, which in turn reflects positively on his comprehension and educational attainment, and vice versa if the student's psychological state is disturbed or has a feeling of anxiety, this will lead to poor concentration and a lack of comprehension in general.
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I need a stack of black and white .jpeg/.tiff/.jpg/.png images across time of either action potentials, neurons firing, or brain scans (comparing disease and normal brain, disease progression, etc.) that I can colorize and overlay for a project in a data visualization course. It wouldn't be published and only for submission to the instructor.
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Something like this for action potentials ?
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I'm doing a research with my team on Depression in India. We need a dataset of social media posts of Indian people. We want to do Sentiment Analysis on these posts and find out some insights which are India specific.
  • We have tried Facebook(but its very hard to scrape data from it).
  • We have tried Reddit(but got very less posts, also it doesn't tell the country)
  • We looked twitter, but not fruitful.
It is possible that we can get data from the above three more efficiently, but do not know how?
If there is any forum related to depression, or mental health, it will also work.
We've also posted related question here:
  1. https://www.reddit.com/r/redditdev/comments/k2stws/getting_post_from_a_subreddit_only_from_users_of/
  2. https://www.reddit.com/r/datasets/comments/k2tx96/looking_for_social_media_posts_of_indian_people/
TL;DR: Looking for Indian dataset of social media posts of depresses/anxious people.
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Mohamed Elhadad Thanks for your suggestion, will sure look into that.
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Dear colleagues
I'm interesting in vermiculture and vermicompost technology. However, I noticed that there were some negative impact effects of adding vermicompost to soil, and depressing growth rate of plants.
Is there any one noticed that or face problems with using vermicompost as organic fertilizers
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M.K. Tripathi
I do agreed with Dr. M.K. Tripathi sir
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Hi,
for a facial emotion recognition detection project, a friend asked me if I know where he could find a dataset with faces of depressed people.
I found this deep learning facial emotion recognition project:
based on theDexter Miranda’s photo project, "The Face We Make":
But, the photos are not specifically about "depressive faces" (more about emotions).
On Kaggle, I found a better dataset:
And the JAFFE dataset:
So, I would like to know if there are "scientific or psychological dataset" with photos of faces expressing different emotions with depressive symptoms?
Thanks,
Laurent Berry
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Thanks a lot...
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I am interested in the literary origins of noir crime writing.
Can anyone recommend the very best, or 'must read', works on either the novelist Ernest Hemingway or the subject of the 'Forgotten Man' (that is American veterans of WWI and the mass unemployed of the Great Depression)?
Many Thanks!
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Its a pity this has not heretofore been answered. I do not know of essays etc but there were a number of writers who filled in the gap but not necessarily by alluding to the war as such or in depth. Scott Fitzgerald's stories of social and personal dissociation demonstrate the effects of the conflict.
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Dear Colleauges,
What is the rationale for studying dopamine’s role in Major Depressive Disorder?
Major Depressive Disorder (depression) is a multifactorial, biologically and symptomatically heterogeneous condition. Amongst the numerous—and potentially equally valid—proposals concerning its aetiology (e.g., inflammation, genetics, stress-related mechanisms, etc.), one of the longest-standing ideas is the “monoamine hypothesis” stating that one or more dysfunctional monoamine systems are key contributors in the development of this psychiatric disorder. Historically, most of the research focus has been on the role of serotonin and noradrenalin. Although this might not be “The” biological explanation for depression, a dysfunctional monoamine system, dopamine system in particular, is likely to contribute to the emergence of the two “sine qua non” symptoms associated with depression: on one hand, depressed mood and reduced motivation/perseverance and on the other, anhedonia and loss of interest in what were previously pleasurable activities. There is growing interest in dopamine’s role in clinical depression, especially, in the way it works as a modulator of the brain’s reward systems. Furthermore, there is pre-clinical evidence that A10 dopaminergic neurons projecting from the midbrain ventral tegmental area to the nucleus accumbens and dorsolateral prefrontal cortical areas over several routes are associated with motivation, exploration, appetitive learning, reward-driven behaviours, and a depressive-like phenotype in experimental models of depression. The reward systems might also be implicated in aversive stimuli, a recent notion which needs to be further explored.
This Special Issue of Brain Sciences aims to bring together some of the current ideas on the function or dysfunction of dopamine and dopaminergic transmission in depression, by examining the most recent evidence from advanced clinical and experimental research. It will gather insights from well-acclaimed experts in the field towards answering the ultimate question: what is the true role of dopamine in Major Depression?
For more information on contributing to this Special Issue, please go to:
Thank You!
Dr. Màté D. Döbrössy Guest Editor
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Нехватка дофамина и повышение глутаминергические рецепторы увеличивают риск болезни Паркинсона, на механизм развитие депрессии основной роль играет серотонинергические медиаторы
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Are feelings of emptiness commonly experienced even within non-clinical populations? If so, are these experiences less salient due to a lack of personal awareness about the problem? Or, are people reluctant to disclose this feeling due to societal stigma?
In other words, is emptiness a major problem in modern society, and, if so, why is it so inconspicuous?
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Different believes have different meaning for "Emptiness". In Buddhism, Emptiness is the kernel of everything, living or non-living. All human feelings are illusion, and only emptiness is essential and eternal.
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Why the number of secondary depressions on existing chromosomes varies?
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Thank you dear hadeel.
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As millions are getting depressed losing health and job and loniliness ,anew hope is begining to build up.really we need to this encouragement.we need all hygene ,distant physically to immune ourself from epidemic .this is new oxygen and new life really.so How can we fight depression and fear?lets hope that vwe recovered from deadly disease ,to get new life ,freedom and happiness.
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Once people can master (balance) their ego (materialistic desires) and turn to their soul (creative purpose), happiness and bliss will set in.
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Evidence is accumulating that glutamate abnormalities may also play a role in depression. But how does the glutamate level change? In chronic mild unexpected stress models, the glutamate level is decreased. But in the early stress, the glutamate level is increased. In my opinion, the early stress results in the increased glutamate, which leads to glutamate-mediated toxicity and damage the neurons. Later, the loss of neurons lead to decreased glutamate.
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I have worked a lot with mice and must say that they are surprisingly good models for the human being. One evolutionistic theory claims that the rat is our closest cousin, not the ape.
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Anyone has a pdf form of Children''s Depression Inventory 2 (CDI2)
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Children's Depression Inventory (CDI and CDI 2)
  • January 2015
  • DOI:
  • 10.1002/9781118625392.wbecp419
  • In book: The Encyclopedia of Clinical Psychology
  • 📷Maria Kovacs
  • request fulltext from here on RG
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Hello everyone! I am researching the molecular mechanisms which drive depression in the context of gut microbiota. From what I know, depression's pathogenesis primarily relies on Tryptophan metabolism. On one hand, the pathway is perturbed to the kynurenic pathway which leads to the formation of kynurenic metabolites (KYNA, QA, XA, etc). These kynurenic metabolites exert neurotoxic effects which ultimately drive depression phenotypes. They are also able to affect the enteric nervous system, the gut millieu, and immune system which contribute to depression.
On the other hand, Tryptophan metabolism can also be driven towards serotonin synthesis. Inflammatory bowel disease and its likes (Crohn's disease, colitis) have been shown to have increased serotonin (evidenced by increased TPH expression) and decreased SERT expression. Serotonin has been shown to be pro-inflammatory and this supports the inflammatory theory of depression.
While these explanations do not completely contradict each other and that they may simultaneously contribute to depression, an "irony" still exist that one says a perturbation towards kynurenine while the other is towards serotonin. Either way, Tryptophan is consumed and surely leads towards an increased bias to one pathway (ie. increased serotogenic or increased kynurenic). Hence, do you know of any articles which settle this apparent contradiction? Or perhaps there is something I incorrectly understand?
Thank you!
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Simon N Young thank you for your response.
1. Yes, they cannot cross the BBB but kynurenine and tryptophan can. Trp can be converted by microglia and astrocytes using IDO to kynurenine. From thereon, they can be metabolized into these acids. Is this not a mechanism which kynurenine metabolites exert negative effects on the brain?
2. Thank you for this. I , too, am more familiar with the low serotonin associated with depression. I just asked about high serotonin and IBS because it conflicts this explanation. Perhaps inflammation and other factors caused by IBS influence increased depression incidence found in patients.
And yes, MDD is indeed multifactorial. I am interested in how gut microbiota possibly exert influence in the etiology of depression via Trp metabolism.
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Hi all,
I am doing a longitudinal study using depressive score as my DV. It is an observational study. No variables are manipulated. I have currently collected baseline (Time 1) and first follow-up (Time 2) data. I have a number of variables as predictors of the depressive score and I would like to identify what hypothesized variables collected at baseline would predict the Time 2 depressive score, and possibly be significant predictors for depressive scores at other time points in the future.
Do I
1) put predictors at baseline as IV, change score of depressive score as DV in the multiple regression model?
or
2) put predictors at baseline and baseline (Time 1) depressive score as IV, and Time 2 depressive score as DV in the multiple regression model?
I have actually tried both methods, it ended up that while using method 1, the predictors of change score are the same as those of Time 1 depressive score.
Whlie using method 2, no predictors (not even baseline depressive score) at baseline are significant predictors of Time 2 depressive score.
I wonder how I should have analyzed and interpreted the data. Thanks a lot!
Anna
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Remember, especially with no manipulation, correlation does not equal causation. So, "predicttion" may be a bit of a stretch.
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In your personal experience,How do you manage your life and make a balance between work, family and other related sections!?:)
I would be thrilled to have your points!
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Equally, dear Dr. Hossein
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After being affected with that viruse , most of the patients feel depressed and sad . How can we , psychologically, encourage them ?
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Interested discussion. Please follow up....👍
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please guide me about the sample size calculation for this study
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Hi! It depends on whether the researcher is using probability or non probability sampling and the population size. Usually the sources used to decide the sample size is following Hair et al 2010, G power calculator etc.
Reading the following material could be helpful as well. Thanks
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I am trying to discern whether, in the brain (structure & function) of people with bipolar disorder/MDD/schizophrenia who experienced childhood trauma, there are:
1. already differences in children's brain structure & function that trauma further modifies and the person develops a mental illness OR (genetics first then trauma)
2. trauma changes the structure and function of the brain that the child's genetics further modifies and the person develops a mental illness (trauma first then genetics)
Has anyone researched this & if so, can you please share your findings or references you know of?
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Thank you for your insightful comment. I have read Cassiers et al (2018) as well as 3 other insightful articles: 1) Nemeroff CB.(2016). Paradise Lost: The Neurobiological and Clinical Consequences of Child Abuse and Neglect. In Neuron; 2) Aas M et al. (2019) Childhood maltreatment and polygenic risk in bipolar disorders. In Bipolar Disorders; and 3) Stevelink et al. (2019) Childhood abuse and white matter integrity in bipolar disorder patients and healthy controls. In European Neuropsychopharmacology.
  1. In Cassiers et al (2018), their review of the literature supports the hypothesis that the brain changes are a protective adaptation in response to abuse, they did not discuss the genetic components that may be predisposing to or responsive to abuse so it doesn't directly answer the question.
  2. In Nemeroff (2016), he suggests (based on my assessment) some brain changes are due to the moderating effects of genetics (so genetics first) (eg, carriers of 1 or 2 copies of the "short" allele of the serotonin transporter promoter polymorphism have greater rates of depression vs those with the "long" allele homozygotes with equal childhood trauma) and other changes are in response to the trauma and predispose the person to the mental illness (eg, reduced hippocampal volume in depressed women with a history of childhood maltreatment but not in equally depressed women without maltreatment).
  3. Aas M et al (2019) reported that polygenic risk score (PRS) and CTQ were inversely correlated so those with lower PRS reported more severe abuse and vice versa, so the based on this (my interpretation) is that in a brain/body with more "imbalances" due to genetics (brain structure/function and physiology that is predisposed to BD), abuse does not need to be severe to make the changes sufficient to cause BD wherein people with low PRS have brain structures and physiology is more "balanced", greater severity of abuse is needed to sufficiently change the brain structures and physiology to cause BD. They did not also look at brain or physiology so we can only infer based on genetics.
  4. In Stevelink et al (2019), their study findings suggest "that childhood abuse results in poor white matter integrity in a subset of people who are then possibly more vulnerable to development of psychiatric disorders, including bipolar disorder" and "that childhood abuse, in particular, is associated with FA, possibly due to its effects on HPA-axis activity." But as pointed out in the limitations, their cross-sectional study design does not allow for establishing causation "Based on our results, we cannot differentiate whether decreased integrity of white matter in patients is caused by childhood abuse, or whether this decreased integrity was already present prior to experiencing childhood abuse."
I am interested in causation and the interplay of genetics and childhood trauma as I am trying to write a book on childhood trauma and how the different types of trauma at different times explains the behaviors of survivors, the mental health challenges caused by childhood trauma and toxic stress, and how people can "overcome" (I hate this word but can't think of another) trauma. My story of childhood trauma, developing several mental illnesses including bipolar disorder, and how I was able to get a PhD and have a successful career while growing and healing is being used as the backdrop to tell the scientific story to a lay audience.
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Hi Everyone,
I want to analyse how well two scales agree for detecting changes in wellbeing. I look at the Bland-Altman Method. One scale is a simple score from 0 - 10 for wellbeing. The other scale is the well established Beck’s Depression Inventory-II (goes from 0 - 63) . I wonder how to analyse the agreement?
Thanks!
Fred
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Hello,
This link talk about: Agreement Between Two Ratings with Different Ordinal Scale
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I am a physician and visiting research scientist at yale and together with my coauthors have published several studies in the field of psychology through Network Modeling and related analyses using r packages like bootnet, qgraph, NetworkComparisonTest, etc. I thought it might be a good idea to discuss different subjects related to this field, in a group. I am also interested in collaboration with other teams working in the field. Let's share our ideas, questions and suggestions in this group.
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Makes sense. Thanks for letting me know.
I hope followers of this discussion forum check your study and package,
which address an interesting topic in network analysis. Let's keep in touch!
Best Wishes,
Farhad
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I have a student conducting research on coping mechanisms used by males with partners suffering from Post Part Depression. It has been very difficult getting participants to complete the survey. Any suggestions on where this can best be posted to improve participation?
Already tried social media sites and groups focused on male issues with limited response.
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Hi. I think you will have constant challenges getting validated data with a health topic relating to women, but studying it from a partner/male perspective.
All the best, though.
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With everyone having their own struggle and a different definition of being 'stressed'. Does problems of 23 years old smaller than that of 46 years?
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The uniqueness of depression is that the base of depression, on the basis of which depression occurs, is present in 99% of the population. 99% of the population are susceptible to depression, since the cause of depression is a violation of certain physiological processes in the human body, which of course are common to all people (all people in the body have the same physiological processes). Therefore, at 10, 20 or 40 years, depression is one and the same basic disorder, which can manifest itself in different ways due to different stresses at different ages (stresses are triggers of depression at any age).
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I am currently working on a database and I have tried various methods of machine learning and deep learning, all leads to poor classification/prediction. However, this might show that the two groups have no significant difference. For example, the database I have is a collection of audio speech and their associated depression level. I am assuming that the culture of where I am from and the strong religious believe, prevented the society from going too deep into depression. However, I need to find some literature to back up this statement. But, it seems that there are no significant difference between the acoustics of depressed and non depressed. Can I write these poor results in a paper? Any tips?
Thank you
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Hello Dr Nik, Yes sure you can write a paper and publish it in Scopus journals...
I can present some tips for you:
1- You can change the Chunk size of the voice sample and then check the results again (Usually when the chunk size (sec) is big, the results will increase (accuracy). While when the Chunk size is small, it sounds like a challenge to get high results.
2- Also, when the databases are equal (e.g., depressed and non depressed databases), the results often become higher
2- Give some significant points in the conclusion section in terms of this area.
3- Write the paper in very good English.
4- Moreover, check out these articles may help you in this area:
Thesis ''Automatic assessment of depression from speech: paralinguistic analysis, modelling and machine learning''
And all the best (:
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Hello, I am working with the Beck Depression Inventory and Beck Anxiety Inventory. I am trying to establish cut points for depressed/not depressed and anxious/not anxious. I know that I need to use an ROC curve to find the cut points, but I am not sure of the process to get there. Can anyone provide directions on going from survey questions/answers (I have 22 participants who have completed both surveys; no missing data) to establishing the ROC curve?
Thank you very much
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ROC (Receiver Operating Characteristic) analysis (Hajian-Tilaki, 2013) is a graphical representation of the effectiveness of the prediction model by drawing the characteristics of qualitative binary classifiers created from the model using many different cut-off points. Calculate the Youden Index for best sensitivity and specificity values.
One (mine) example of use of ROC Analysis in research:
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It is believed that there are several depressed people of COVID-19 lockdown. How a psychologist could support them amid the lockdown?
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E-questionnaire to the family & the cases. I think that is may be help. psychiatrist and psychotherapist can help more.
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I would like to enrol into a master's program at one of these universities and write my master's thesis on a topic involving the above three components.
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Following.
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Is there any physiological value or index that show us a stress or anxiety level? For example in an experimental reseach, when we want to find out computer games effect on stress, depression or anxiety, How can we detect these disorders level by using with physiological indicators?
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Yes you can detect stress, anxiety and depression by using those parameters. Here is a paper on detecting stress by machine learning techniques using ECG.
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Dear all
Hope you are all safe and healthy. We are doing a study on Phubbing and its association with depression, anxiety, and couple satisfaction. Phubbing is an under-researched area despite the increasing prevalence of problematic internet use and the associated mental health consequences. Since we need a large number of respondents, we are posting here and requesting you all to kindly spare a couple of minutes to go through it.
We assure that it will only take a few minutes to complete the questions.
We also encourage you to share the study.
Feel free to contact us for any doubts
Here is the link:
PS: It can be filled by singles too, as couple satisfaction is only a part of the study.
Best regards
Pooja
Karthick
Bigya
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Neil Dagnall Thank you for your interest in our study. Please feel free to participate and share . This is the link
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Most of the commentary about the current COVID-19 induced economic challenges use either the 2008 global financial crisis (GFC) or the 1929 Great Depression (https://www.bbc.com/news/business-52236936) as reference points. The reference points most relate to; 1) the severity of the economic challenges and; b) the time it will take to recover from the current challenge, in relation to either 1929 or 2008.
In the outlook for the recovery period, it assumed that the recovery will either be in the form of V shape or an L Shape compared to previous recovery shapes.
The questions are as follows:
  1. Are there parallels between either the 2008 GFC or the 1929 Great Depression and the COVID-19 induced economic challenges? Can we possibly draw any parallels?
  2. If there is, in what way are these the same and in what way are they different, except for the obvious and "well documented" drivers and causes.
  3. What shape do you think the recovery will take/assume? V or L?
  4. How long will the recovery take, either as measured in number of years, quarters or months? and Why?
For ease of reference, see some of the links below
NB: The significance of the BBC reference is the quote (s) attributed to the MD of the International Monetary Fund (IMF).
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Well Dear Prof. Kheepe Lawrence Moremi
In 2008 vzla didn´t feel any finantial crisis (oil barrel ~ 100 US $) so for me 2008 GFC is not a reference point. Now happens the same, we don´t feel any crisis, since our internal hyperinflation started 3 years ago & the venezuelan oil industry was already brought to its knees at that time. So I take as a temporal reference the 1929 GD.
Interesting to see an hyperinflation chart poweder by Prof. Hanke:
Our hyperinflationary misfortune is a little more than a year ahead of the rest of the world according to Prof. Hanke chart (02/2019 we had a peak). But I can tell you something for sure: humankind can live only with 1/3 pound of white rice & a cooked green banana daily per person. No toothpaste & teeth falling out, all alive until a lack of medications, either the bug, kill the mayority.
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I am writing a research proposal on psychological aspects of IBD (anxiety/depression...) what should I use as my themes for my lit review?
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look at the idea emphasized in the literature review of the disease
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HI,
this is Farhana, currently studying Bachelor of Science in Computer Science and Engineering in BRAC University in Bangladesh.
My thesis project is on "Depression and normal condition" and I wanna find a clean EEG data set about Depression and normal condition. is it possible for you to share your data set with me?
Thank you
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Depression, also called major depressive disorder or clinical depression, is a mood disorder that causes a persistent feeling of sadness and loss of interest.
Have you ever struggle with depression? Please advise how depression can be overcome?
Thank you in advance for sharing your knowledge and experience.
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"Yoga" and "meditation" are the best ways to overcome depression.
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We are in the middle of a crisis, in the quarantines. Irresponsible behavior has brought us to our current state. We did not learn any lessons learned from viruses that preceded and resembled the COVID-19 virus. Today's topic is not the reason that led us to this situation but commenting on the measures we have taken. The study of (Bishwajit et al., 2017) examined the effects of physical activity on depression. Their study had a representative number of middle- and older-aged subjects (7204). They concluded that a lower frequency of vigorous physical activity was significantly associated with higher rates of depression diagnosed. Depression symptoms and physical inactivity are factors that are closely correlated with obesity (Garimella et al., 2016). The elderly population has a prevalence of anxiety and depression around 10 and 12 %, these findings are caused as a consequence of different factors. Health-related quality of life and physical function play an important role in depression and anxiety (Sousa et al., 2017). The logical conclusion is that physical activity can reduce the levels of depression. Many studies have addressed this topic. Throughout history, our race has evolved. From the beginning of the cognitive, through the agricultural and industrial revolution to the present, we can observe a trend of decline in physical activity. This trend was accompanied by the appearance of metabolic and chronic diseases. Chronic diseases are major killers in the modern era. Physical inactivity is the primary cause of most chronic diseases. (Booth et al., 2011). Physical activity primarily prevents, or delays, chronic diseases, implying that chronic disease need not be an inevitable outcome during life.
This brief introduction is just a small overview of the literature that has examined the topics of physical inactivity, depression, and chronic illnesses.
Because we are in quarantine, and our movement is restricted and in some environments disabled we face many difficulties. Speaking personally and listening to people from my surroundings, from a psychological point of view, quarantine has a rather negative impact on people. With this, the media and the internet, which is full of misinformation, make people panic.
The following questions are:
  • Is quarantine an ethical solution?
  • How will this inactivity affect people?
  • How will inactivity affect obesity, chronic diseases, and ultimately, mortality?
Reference:
Bishwajit, G., O’Leary, D. P., Ghosh, S., Yaya, S., Shangfeng, T., & Feng, Z. (2017). Physical inactivity and self-reported depression among middle-and older-aged population in South Asia: World health survey. BMC geriatrics, 17(1), 100.
Booth, Frank W., Christian K. Roberts, and Matthew J. Laye. "Lack of exercise is a major cause of chronic diseases." Comprehensive Physiology 2, no. 2 (2011): 1143-1211.
Garimella, R. S., Sears, S. F., & Gehi, A. K. (2016). Depression and physical inactivity as confounding the effect of obesity on atrial fibrillation. The American journal of cardiology, 117(11), 1760-1764.
Sousa, R. D. D., Rodrigues, A. M., Gregório, M. J., Branco, J. D. C., Gouveia, M. J., Canhão, H., & Dias, S. S. (2017). Anxiety and depression in the Portuguese older adults: prevalence and associated factors. Frontiers in medicine, 4, 196.
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You have touched on an interesting problem of long-term and epidemic-related effects. No one is dealing with them right now because of the current situation but in a few months, we will see a terrifying summary of what happens.
Patients have worse control of chronic diseases due to difficult access to health care and almost not existing private health care sector. Quarantine is associated with a lack of physical activity, social life and developing bad eating habits. It will result in a higher suicide rate, higher chronic diseases mortality.
In my opinion, two weeks of restricted quarantine won't lead to increased obesity. Although the quarantine should last as long as the pandemic will end, and we don't know how long it'll take...
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Many people suffering from Typ one diabetes mellitus, could be identify as almost depressive as well.
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Dear Bettina,
I suppose this has to do with money and time. As psychological treatment are long and time-consuming, a general practitioner would not like to vaste time on
depression besides diabetes. And the nurses lack time. After surgery you really have to beg for being in the hospital for some extra hours and you are not always granted these. Who cares then about your depression?
Mit freundlichen Grüßen und vielen Dank für die schönen Worte,
Béatrice
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Treatment of Bipolar Depression
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Cognitive behavioral therapy as well as somebody who walks with them is also helpful if you fear that an antidepressant will provoke mania. The more physical training and talking the better. These patients are usually rather alone while relatives and friends are tired and burdened from their ups and downs. Already group therapy is helpful.
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During test of cycle_gate, we ask people to walk, then they pay attention to it and their way of walking is changed, also darkness can change it too.
but that is question, whether mental illness such as depression change the cycle_gate?that could help us to find therapies for these diseases.
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Yes,affects,Of course it affect our cycle_gate. Depression (major depressive disorder) is a common and serious medical illness that negatively affects our feels. Depression causes feelings of sadness and/or a loss of interest in activities once enjoyed. It can lead to a variety of emotional and physical problems and can decrease a person’s ability to function at work and at home.
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Planning an intervention for infertile depressed and anxious people, for guidence purpose I want to go through those interventions if you know any. thank You
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Logotherapy is a successful intervention. Finding the present meaning in life, can be short-term or long-term. Resolving the fear of death by finding a lasting faith. Social support, love, which a man suggested should be available in the pharmacy,
and insight and acceptance that there are things that exceeds our understanding.
A very quick and effective intervention is listening to classic music.
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Is there a scale available free of cost and without copyright issues to evaluate self-efficacy for primary health workers ?
Similarly , are they scales available for evaluating change in social , health and economic outcome in depressed individuals located in low middle income countries ?
If the scales are not available , on constructing these scales can we develop a structured questionnaire assessing social , health and economic outcome in depressed individuals ? If so , what constructs should we undertake ?
Thank you
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http://userpage.fu-berlin.de/~health/selfscal.htm here is the page to self-efficacy scales in many different languages
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I am currently starting to write a literature review for my Doctoral Thesis. As I am writing, I am wondering about the acceptability of using only three books as references for a chapter (not the whole discussion) in my literature review.
The reason why I am doing because I found these three books are recent (2012, 2014, & 2015) and already consist of abundant information for the chapter.
I am writing a chapter about Depression and these three books has already given me information about 1) Types of Depression 2) Causes of Depression (from Biopsychosocial aspect) 3) Effect of Depression 4) Best Available Treatments 5) Prognosis of Depression. Furthermore, for me, these sub-chapters are already enough for one chapter only. Considering that there are still many to come.
Any critique or suggestions are welcome.
Thank you
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Dear Wisnu
Whilst it is good that you have a solid reading base from the books you have mentioned, it is often the case that books have taken some time to produce and therefore have older primary sources. I think it would be good for you to conduct a scoping search as well, you may identify some of the latest literature from a quick search. It is always good to look for any current systematic reviews or commentary reviews that fit your topic in general (i.e. search of topic names + systematic review, review), in addition look for the latest papers as they may be useful and can often signpost you to other relevant literature. In addition books tend to have their own overall narrative that reflects the thinking of an author (or authors), whereas it would be expected that you would take a more critical and objective stance and this would be helped by a wider search of the literature.
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I need to apply LiDAR for fault growth analysis project to study the presence of Strike-slip faults in strain linkage extensional regime.
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Try to see the following publication: Magmatic cycles pace tectonic and morphological expression of rifting (Afar depression, Ethiopia)S. Medynski, Raphaël Pik, Pete Burnard, S Dumont, Raphael Grandin, Alice Williams, Pierre-Henri Blard, Irene Schimmelpfennig, C Vye-Brown, Lyderic France, et al.
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I've seen at symposia that some people have success recruiting for research studies on subreddits like r/depression or r/anxiety. Does anyone have experience with this? I have an upcoming study where I think this recruitment method would be useful. Has anyone run into IRB issues with this recruitment method? If you've had success with this recruitment method, how did you go about making your posts?
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I've used the "subreddit" r/samplesize for recruitment, its dedicated directly for this purpose (instructions on how to post, etc., are contained in the sidebar). I got about 200 participants in of a month using this approach, and my IRB did not ask any questions.
I think posting recruitment ads on other subreddits (related to disease states, etcc) is at the discretion of the moderators. I asked about one and was told no.
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I would like to write an academic essay on Depression among youth using Participatory Action Research and its applicability in our country but I dont have the idea how to make my ideas flow. Im starting to build an outline but it seems lacking and disastrous. Do u have any idea what are the essential information and data to include so that my paper will be clear and convincing? You can also include suggests readings that I could review. I highly appreciate any form of help.
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Two other articles of mine might help:
`Action research in graduate management research programs', Higher Education, vol. 23, pp. 195-208, March 1992. With O. Zuber-Skerritt.
Zuber-Skerritt, O. and Perry, C. 2002, ‘Action research within organisations and university thesis writing’ Organisational Learning, vol. 9, no. 4, pp. 171-179.
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Why Have Researchers so far failed to identify common GENE MUTATIONS that contribute to risk for MAJOR DEPRESSION?