Questions related to Audiology
I am doing a cross-sectional study on audiology patients in South Africa. As I cannot find information regarding the population size, does anyone have any suggestions on how to calculate a possible sample size?
The study will only consist of one population i.e. audiology patients, however will later need to compare scores obtained state vs private patients. Is there a way I can calculate a good sample size which will provide appropriate generalization?
Any suggestions would be helpful, thank you.
Die Frage ist sehr allgemein, da ich aus der Medientechnik und nicht aus der Audiologie komme: Ich suche hinsichtlich Verstärkung, Kompression, Störschallunterdrückung/Binauralität und Frequenzverschiebung Studien und Ergebnisse, die anhand von Hörtests Vergleiche unterschiedlicher Herangehensweisen anstellen. Ich kann leider nur teilweise Informationen finden.
Für jede Hilfe bin ich dankbar! Frohe Weihnachten!
There are conflicts in the research papers I have reviewed regarding the best clinical testing protocol for the monitoring of noise or music induced hearing loss and I would be grateful if information could be provided regarding the the most effective battery of audiological tests required for the monitoring of noise/music induced hearing losses.
The biomedical industry provide support for medical diagnostics & monitoring, cardiology, audiology, neurology, plus..
Phonetically Balance word list ,malayalam is available in ISHA battery;however the full article in which the list was published isnt available.Hence we couldnt find out whether or not psychometric function was done for the list.It would be helpful if someone could suggest any other PB word list in malayalam for which the psychometric function was done or direct me to the orginal article in which the list was published.
A 17 year male presented with right progressive deafness 2 years ago. There is frequent wax impaction with many attempts to remove it. No history of previous ear surgery or trauma. On examination there is severe narrowing of the right external ear canal from bulging of posterior canal wall with wax impaction, the tympanic membrane was not appear even when the wax is removed. Tuning fork tests show Rinne test negative in right ear and positive in left ear, Weber test was lateralized to the right ear. No disfigurement of the right pinna or postauricular region.
CT scan of the temporal bones revealed widening and ground glass of the most temporal bone, right inner ear is not involved by the lesion. Pure tone audiogram identified 50 dB conductive deafness in right ear and normal hearing in left ear.
What are the options of treatment for such patient?
I'm trying to find a credible source for identifying the approximate number/percentage of human beings who can hear beyond the "normal" range (20 Hz to 20 kHz.). I found many articles offering numbers, but they have no source materials/references to back them up.
From what I've read to date, it seems that people with autism can hear sounds that fall into the ultrasonic range, and younger people can also occasionally hear those normally inaudible sounds. Some people who simply have sensitive hearing (no apparent special conditions and in any age range). But I need a credible source - a research study, data source or scientific organization - that can represent a close approximation of this population size (and make up, if that's possible) is what I need.
e.g. "Approximately 4% of human beings can hear audio signals in the range of 20 -30 kHz.."
I am currently working on my 3rd year dissertation in audiology and my questions is relating to cochlear implants and bimodal stimulation. Can anybody recommend any books or research articles/journals on cochlear implants or bimodal stimulation (the use of on HA and a CI).
*Cognition and Hearing: Educator Survey*
This international survey is for those who teach students about the relation between hearing and cognition, broadly construed. Of interest is current practice in higher education as well as how a new textbook on cognition and hearing might supplement or improve that practice.
Educators may teach students in higher education under acoustics technology, audiology, engineering acoustics, psychology, or neuroscience, as well as in speech, hearing, and phonetic sciences. Analyses will steer textbook planning to serve this community and their students.
Taking it that, as an educator, you have now decided that the survey concerns you, please:
* Reflect for a moment on the relative value of all approaches to cognition and hearing.
* Think of up to three textbooks that you use in your teaching on cognition and hearing.
This survey has seven sections starting with an introductory section. Thinking of up to three textbooks, there follow three separate sections - each of which is optional, depending on how many textbooks you use. A fifth section concerns the future textbook. A sixth section surveys your views on current and future practice. A final section relates to your valuable experience of this survey at this crucial piloting phase, as well as any further feedback.
With enthusiasm for your educational aims, please, I ask you to take a few minutes to complete the survey at the link below. Thank you very much in advance!
Survey link: https://goo.gl/forms/avLf9wZlZoS7cMTP2
To investigate the status of patients with increased ESR in blood and how it results in hearing loss.
The arcuate fasciculus develops a leftward asymmetry on the human brain of a healthy subject. Are there any studies about this on congenital deaf subjects?
We know that;
a. outer ear and middle ear has 3000 Hz resonance frequency
b. High frequencies is more sensitive to the barriers,
but the conductive hearing loss make low frequency hearing loss.
Hi, I have a patient. she is 4 years old. Her hearing loss has been diagnosed 1 and half year a go. She has a severe to profound hearing loss. Parents claim that she was OK before and she gradually has lost her hearing. As there was not any previous hearing evaluation (even no hearing screening at birth!), we can not confirm that. She received hearing aid and auditory rehabilitation right away. Since then she has had 3 sudden reduction of hearing to profound hearing loss (parents recognize that because she do not react to sound at all with her hearing aid).
Otologist prescribed corton therapy and ketotifen... for two weeks in first two episodes. She had cold in one of them. She showed recovery after that. Today she came to me with same problem (again sudden reduction of hearing to profound and no reaction to sound).
What do you think is the underlying cause? (some thing is wrong for sure)
Can it be an autoimmune disease? (she seems totally normal and her blood test is normal)
Parents ask me is there a neural problem or cochlear? (How can I be sure?!)
Parents ask me if cochlear implant will resolve the problem?
Please help us. Thank you
hello, in my experiments some participants' auditory P1 responses are below the base line. in terms of wave form I am confident that it is a P1, but below the base line, the same holds also for N1, which is sometimes above the baseline (but it is a N1, depending on latency and wave form characteristics)
what can I do for that? this leads to loss of data.
Also, how reliable are BIC responses? Stollman et al (1996) article mentions a detection rate of 95-97%. Any personal or clinical experiences?
In literature, Well known that RIAR is not elicited from Stapedius Muscle. Because It can be only seen in İpsi way and with high intensity. Even though profound sensorineural hearing loss present, RIAR could be detectable.
In attachment article, Researchers think; "RIAR comes into existence when the stapedius muscle could not affect the ossicular system." and "The pressure effect of acoustic stimuli stimulates a mechanical opposite reaction of the tympanic membrane and mobile malleus-incus system."
In my clinical practice, I observed this reflex pattern from many patients. Not only from patient who has cochlear pathology, but also from patient who has conductive pathology . Practically, I do not use this observation for any comment.
When I repeat a. reflex measurement at the different instrument. I can't see RIAR! For example, I can see RIAR in Interacoustics AT235 device but I can't see RIAR in GSI Tympstar. I thought, This is maybe stimulus artifact by the manufacturer immitance system.
Do you have any experience about RIAR? What is your opinion?
Many individuals are suffering from tinnitus accompanying sensorineural hearing loss. That tinnitus is continuous, day and night, and with the same pitch and intensity.
Are there any research literature on the Validity of Selective Audiroty attention test (SAAT)? I have a problem in Validity of the test. I want to use Dichotic digit test for validity , but ask the listener to attend just to Right ear, and then compare the score of SAAT and Dichotic digit test. Any suggestion, any test?
Management of ANSD patients is still a challenge for audiologists. In countries where people can't afford CI, can hearing aids with proper fitting strategies employed benefit individuals with ANSD?
I just heard about some normal hearing test persons still hearing sounds or even detected words at about 65 dB in free field though their air conduction was completely blocked (headphones, earbuds; not sure, but I guess you know what I mean). I found out that in free-field bone conduction perception has to be 40-50 dB higher than air conduction to be perceived as equally loud. Is this possible? And if it is, are there any papers about such a scenario?
Six months seems to be the consensus age for doing VRA. But how well does VRA obtained at 6 months correlate with later threshold measures? Is a VRA measure at 8 months significantly more accurate than one at 6 months?
As monaural auditory thresholds may be different, how can we measure binaural loudness discomfortable levels by presenting pure tones through earphones?
An adult patient with few months of hyperacusis and sound hypersensitivity, audiological assessments are within normal ranges;
It started to affect his Psychic stability.
An adult person complaints of difficulty hearing in background noise. Pure tone audiometry and speech audiometry reveals normal findings with good speech discrimination scores. ABR and OAE results normal. What can be the further investigations required? and possible interventions
We know about the several causes for Tullio's phenomenon. I would like to the treatment strategy for the same with any Clinical and research experiences?
Is there a way, in which music or sound can influence a living system - especially the human body - directly and not only as a result of interactions between the hearing organs, ear, skin and bones, and brain activities?
What is the advantage of using dBnHL over dBpeSPL. The description of dBnHL, I understand that it is calculated by taking the difference between dB peSPL and behavioral threshold @ one repetition rate. If we are calculating at one rate how has this value been generalized for other reputation rate (30.1/sec, 90.1/sec). From the psycho-acoustics it is understood that behavioral threshold is better at higher rate (90.1/sec) than lower rate. Are there any standards which specify which rate should be used and why.
What does the term "contralateral reflex" with respect to the right ear in the case of acoustic stapedial measurements in routine clinical situations mean?
Since there are two different views regarding which ear is to be the stimulus ear and probe ear, please specify.
And while testing reflex decay in the right ear, which contralateral reflex threshold is to be taken?
I'm trying to find any articles/information that could provide me with information on how proficient audiologists are in using South African sign language once then leave university.
Why is the maturation of high frequency faster after birth? Why does the base respond to low frequency before birth?
Could anyone provide me a solution on what test to be used to differentiate between hyperacusis (with hearing loss) and recruitment, from an audiological point of view.
Is there any condition where the B type tympanogram is present , with the normal ear canal volume (0.48) and thye acoustic relexes are present across the frequencies at 95- 100 dB? ( while the other ear yielded a B type tympanogram -ear canal volume 0.42 with reflexes absent due to middle ear fluid.)
This is to give evidence to rehab advice given in clinic.
I'm looking for data on consonant confusions for in-congruent (McGurk-like) stimuli. I have found several studies that use a small subset of consonant pairs. Is there anything close to exhaustive? Something that pairs each English consonant with each other English consonant?
When a patient reports a complaint of not being able to hear properly, and when evaluated, reveals idiopathic bilateral moderate-to-severe SNHL with acoustic reflexes at elevated levels, what can be the possible recommendations and the appropriate retest duration to suggest for?
The 'speech banana' plotted on audiograms show the highest concentration of energy (frequencies and intensities) of speech sounds. How was this being determined? Can anyone suggest to me the original article on this study?
The wavelengths of the same frequency are substantially different in air and water. Do humans utilize wavelength in auditory pitch perception? This would also apply to rooms/places with substantially different air temperatures, and has implications for understanding auditory localization.
Public health, medicare, medicaid, insurance and tax burden created by premature hearing loss induced by the use of ear buds, earphones, or loud noises that are avoidable. Is prevention the only solution?
Auditory neuropathy pathology is not clearly evident. Site of lesion could be at cochlear level, synaptic level, central connections level and cortical level. Absent acoustic reflexes and abnormal ABR wave patterns or absent ABR waves may point to the higher level of desynchronization (brain stem).
Are individuals with type 1 or type 2 diabetes just as likely to develop hearing loss or is it purely dependent on glycaemic control?
What about children with diabetes, is the prevalence of hearing loss and clinical outlook different for this population?
Reduction in hearing sensitivity can result from reduced number of excitatory cochlear hair cells or increased inhibitory effect of OCB (olivocochlear bundle).
Mixed hearing loss is a challenge in some cases when tympanogram is type A and acoustic reflexes are elicited ipsi and contralaterally.