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Alcohol Use - Science topic

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I need an insight on whether can "alcohol use" be measured as a mediator between the negative relationship of stress and academic performance among University students.
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Thank you very much for your answer, it was very insightful.
Regards,
Muziwandile
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Hi all,
At the moment I'm writing my bachelorthesis, so my knowledge of statistics is quite limited.
In my first hypothesis I am looking for a relationship between an independent variable ADHD (yes/no) and a continuous dependent variable 'inhibitory control' with repeated measures (2 conditions of a task) by using a mixed ANOVA (so between and within subject).
In my second hypothesis I stated that this relationship is moderated by a dichotomous variable 'alcohol use' (high/low).
I'm having a hard time trying to figure out how to add a moderator to this mixed ANOVA analysis.
Help is greatly appreciated!
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There seems so much wrong in your question in the first place. For an ANOVA, your DEPENDENT variable needs to be metric/continous and your INDEPENDENT variable to be nominal. So, I guess you mixed this up? Or do you mean, that your first IV is "ADHD" (between subjects, 2 levels) and your second IV "Inhibitory Control" (within subjects/repeated measures, 2 levels)? Your DV is then however you operationalized it (e.g. reaction time or an inhibitory control questionnaire etc).
If my assumption is correct, you already have a 2-factorial split plot ANOVA (or sometimes "mixed ANOVA", although this may be confusing since there are also "mixed-effect models", which are something different) with one within subjects (IC) and one between subjects (ADHD) factor. If "alcohol use" shall moderate this, you need to add a third factor to the model, so you'll have a 3-factorial split plot ANOVA with one within and two between subjects factors. But your hypothesis is quite vague "In my second hypothesis I stated that this relationship is moderated by a dichotomous variable 'alcohol use' (high/low).", since the relationship between ADHD and IC is already qualified by an interaction and not a simple correlation. How should alcohol moderate this relationship? Which groups x conditions combinations should be different for participants with low vs. high alcohol use?
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I have one group of alcohol users and have to see the effectiveness of my interventions on their alcohol use (quit or not quit). I apply Pre-post quasi experiment while repeated (at least 2) followups in post interventional phase.
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You can use repeated measures of t-test to check the significant difference between multiple conditions of the same group.
Although, since it is not an experimental design the result will not confirm the efficacy/ effectiveness of the intervention even if there are statistically significant differences. If you can include a control group, and if both the experimental and control group has baseline homogeneity, it is possible to test the efficacy of the intervention with comparison between both groups.
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The data was collected from410 participants and The control variable are age, gender, ethnicity, alcohol use and education. While I have only two dependent variables
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Noted with thankx
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In order to generate a national estimate for prevalence of alcohol use, I divided the country into four strata (male/female an rural/urban) as these are important determinant of alcohol use. I then did a meta-analysis of studies done in the last five years and estimated point estimates for each of the stratum along with 95% CI. To generate the national estimate, I weighted the point estimate to the population proportion and got the national point estimate. My question is on how to generate the 95% CI for this national estimate.
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I think once you have the national point estimate and the population you can calculate the 95% CI by point estimate +/- se of estimate.
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Alcohol consumption is always seen as bad. What are the benefits and risks associated to regular moderate alcohol intake? Can someone be free from religious and social taboos and tell me the facts based on researches and data? I see so many people avoiding even good causes because some alcohol producing company is sponsoring it.
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In many papers, they add a small percentage of water to an alcohol and use it as an organoclay activator like for example [methanol/water]=[95%/5%]. My question is:
Why do they use a mixture instead of a single activator? and why don't we just use water knowing that it has a the highest dielectric constant among all polar solvents?
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I don’t know, and I’m also quite curious. I wonder if it has to do with removing carbon content from the surface?
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It could be be physical, emotional, biological, long or short-term effects etc.
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Dear people,
Can someone kindly share the following paper with me please.
Thank you very much
Alcohol Use and Cardiovascular Disease Risk in Patients With Nonalcoholic Fatty Liver Disease.
Lisa B. VanWagner, Hongyan Ning, Norrina B. Allen, Veeral Ajmera,
Cora E. Lewis, John Jeffrey Carr, Donald M. Lloyd-Jones, Norah A. Terrault, Juned Siddique.
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Good night. Here you are
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So I'd take need for treatment toward people with Alcohol Use Disorder as one of the indicator of stigma towards people with AUD. What questionnaire would be ideal?
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"uidelines giveNeed for treatment" is a quite wide term, I suppose you mean severity? However, it is not simple to know which type of treatment for which level of severity. The AUDIT
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Please go through these 3 references:
1. An excerpt from wikipedia: (https://en.wikipedia.org/wiki/Lipid) reads:
"A few studies have suggested that total dietary fat intake is linked to an increased risk of obesity (Astrup, 2005; Astrup et al., 2008) and diabetes (Astrup, 2008). However, a number of very large studies, including the Women's Health Initiative Dietary Modification Trial, an eight-year study of 49,000 women, the Nurses' Health Study and the Health Professionals Follow-up Study, revealed no such links (Beresford et al., 2006; Howard et al., 2006). None of these studies suggested any connection between percentage of calories from fat and risk of cancer, heart disease, or weight gain."
2. Watch the video, Sugar the bitter truth by Robert H. Lustig, MD, UCSF professor of pediatrics in the division of endocrinology (https://www.youtube.com/watch?v=dBnniua6-oM).
Analogy: So is it logical to exonerate sugar and indict fat cholesterol as the major causes of heart diseases, increase in blood pressure, obesity, type 2 diabetes etc?
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I spent some time in eastern Africa as an environmental officer, my home was in Nairobi. On an assignment to Uganda, I ran across an old Brit MD and we discussed heart and vascular disease. He commented that the local diet was high in fat but that the prevalence of heart disease was low but had increased following the establishment of sugar plantations and refineries.
The fat may impact the ability of the endothelial cell to generate NO and the sugar generate ROS, thus pro inflammatory transcription factors, hence upregulation of ICAM-1
See, for example: Mitochondrial Reactive Oxygen Species Mediate Lysophosphadylcholine-induced Endothelial Cell Activation, by Xinyuan Li, et al.
Dr Edo McGowan
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Its an extremely common observation that hepatic amoebiasis is extremely common among nothern Sri Lankan population who drink toddy (Palmyrah palm wine). Its also noted the hygiene of taverns that sell toddy are also equally poor. 
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in our observation, amoebic liver abscess is more prevalent in toddy drinkers. In our unit nearly 85 % are toddy drinker. More complicated course noted in toddy drinkers.
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If anyone can help with this I'd greatly appreciate it! I'm trying to consolidate my ideas for a thesis proposal and feeling a bit lost! I would like to investigate how the relationships between self-esteem, alcohol use and mental health outcomes differ between males and females. My hypothesis is that the relationship between self-esteem and alcohol use will be stronger in males than females and the relationship between self-esteem and mental health outcomes will be stronger in females than in males. I will be using the Rosenberg self-esteem scale, the DASS to measure mental health outcomes and the Alcohol Dependence Scale.
Can anyone help with the kind of design I would need to use and the best statistical analysis to use? 
Thanks!
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 Dear Jen,
from a statistical point of view, this sound like a classical moderation hypothesis: you want to check if the magnitude and/or direction of arelationship between variables x and y depends on a variable z. In your idea x=self esteem, y=alcohol use or mental health outcomes, z=gender. For the moderation it is not necessary that z is categorical, it could also be continous.
There are two typical approaches: if you have only one variable/measure for each of your constructs, i.e. you only use Rosenberg's scale for the self esteem measure, than a (moderated) multiple regression is preferrable, because it is very simple to do, analyze and interpret. On the other hand, if you have several measures for one construct (e.g. different self esteem measures to catch different aspects of self esteem; or mental health as a general construct measured with different tools and approaches) then you could/should use structural equation modeling (SEM). Herein you are able to construct and estimate latent variables ("true" selfesteem/mental health) and you can explicitly model which variable interacts with which other ones. But moderation analyses are not implemented in all SEM programs, as far as I know.
I am not so much into SEM, but I have two very good references for multiple regression and moderation analyses:
Darlington, R. B., & Hayes, A. F. (2016). Regression analysis and linear models: Concepts, applications, and implementation. Guilford Publications.
Hayes, A. F. (2013). Introduction to mediation, moderation, and conditional process analysis: A regression-based approach. Guilford Press.
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How to analyse this data totaling 210 and grouped differently by different variables. 210 incidences are classified on 14 parameters . For example the first parameter has 7 sub categories and second parameter has 4 categories...so on,,,,total 89 sub-parameters to 14 main parameters. Number of sub-parameters min 3 to maximum. For example 210 incidents from last 11 years is classified on basis of time ( 7 time zones in 24 hours) again same data of 210 is classified on the basis of age group( 5 sub categories).. What kind of statistical analysis or data modelling will indicate most important combinations of parameters which are responsible for these incidents
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How to derive weights for each variable?
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I've been unable to find any published research on this question. It would seem that increasing the convenience of alcohol consumption at sporting events would increase consumption, but it's not clear that the increase would be substantial.
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There is no such thing as a study that showed increased access to alcohol resulted in decreased usage.
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I have recently attempted to gain permission for the substance abuse attitude survey (SAAS) by JN Chapell, 1985 as well as the alcohol and alcohol problems perception questionnaire (AAPPQ) and have had no response from numerous requests for permission to use these tools. 
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I'm looking into doing some behavioural testing (Y-maze and open-field) to assess any persistent cognitive effects in a mouse model of alcohol abuse then look at any corresponding brain changes.
My question is when is best to do the behavioural testing to try and isolate the chronic effects of alcohol rather than acute intoxication or withdrawal. Ie should alcohol be removed prior to behavioural testing? If so how long should be allowed before testing should to avoid behavioural changes due to withdrawal?
Any thoughts greatly appreciated!
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Dear Claude, Good question.  Step one is determining what you want to study. Do you want to study the long-term effects of a single administration of alcohol or do you want to study addiction? These are two very different things. For the first, you indeed wait for the alcohol effects to wean off. But you seem to be wanting to study addiction. You can choose to study  addiction in the maintenance phase (so the animal is still getting alcohol at set time intervals) or when they are not using it (during or after withdrawal). 
You can schedule your test at a time that the animal is just expecting it (so there would not be alcohol in the body) or e.g after a few days of withdrawal. An animal that has gone through withdrawal is very different to an animal that uses alcohol repeatedly.
When you are interested in alcohol misuse, you can use different models to induce it. You do not need to give it each day but can also choose a binge pattern. You get damage and alcohol searching, but have more time to look at long-term effects without alcohol in their system. 
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My thesis focuses on PSAs and their level of influence on college students. Specifically, if they influence the student's drinking/driving habits and if this influence is more or less effective for students who belong to a Greek organization.
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I ran a program for the worst DUI offenders and used CBT for one year-no miracles
came out. It was for 3+Duis. Then research showed the population actually averaged 4.5 DUIs-worst of the worst.  CBT good for future research.
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I am working in a community that 80-100 off-premise licensees.  Is there research to support a minimum number of compliance checks in a community?  The Sheriff's Office will be conducting the checks.  Thanks!
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I only wanted to link with you as our areas of   interest are similar-alcohol related issues
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I am working in a community that 80-100 off-premise licensees.  Is there research to support a minimum number of compliance checks in a community?  The Sheriff's Office will be conducting the checks.  Thanks in advance!
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With some types of beer my blood pressure rises rather badly after drinking a single bottle. This is due to my having a problem of sodium retention. Does anyone know of any research comparing the salt contents of different brands of beer ? It would be a fairly simple exercise. I am particularly interested in Brazilian beers, though I believe many people elsewhere must suffer with the same problem that I do – and especially those who drink more than one bottle.
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It may not be readily practicable to compare the salt content of different beer brands due to patent or trademark rights. However we can measure the diuretic effects of different beer types and possible the blood pressure responses.
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I am looking for strong medical evidence that supports the following statement: "Moderate alcohol consumption (1-2 times a week) is good for health"
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the Chief Medical OfficerS
indeed Lucy it is a greta piece
the J shape curve is a bias! 
Stockwell, T., Zhao, J., Panwar, S., Roemer, A., Naimi, T., Chikritzhs, T. ( 2016) Do "moderate" drinkers have Reduced mortality risk? A systematic review and meta-analysis of alcohol consumption and all-cause mortality. Journal of Studies on Alcohol and Drugs, 77, 185-98.
Stockwell, T., Greer, A., Fillmore, K., Chikritzhs., T, Zeisser, C. (2012) How good is the science? BMJ, 344, e2276.
the alcohol lobby fooled too many (and paid to many also)
Jackson, R., Broad, J., Connor, J., Wells, S. (2005) Alcohol and ischaemic heart disease: probably no free lunch. Lancet, 366, 1911–2.
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I am looking for an article with the following info: college graduates are more likely to drink alcohol to cope, while college students are more likely to drink alcohol for social and/pr enhancement purposes.
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Sorry. I have attached files. I hope they will be useful for you. 
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If you have, I would be delighted to receive a reference.
Particularly interested in research done in the last 2 years.
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Hi! I've found some articles about this topic, but they are in spanish. If you don't care about it, let me know and I will send you the links.
However, I'm interested in english articles too, so I will follow this question.
Luck!
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I am conducting a secondary data analysis on a dataset that used 10 measures of cognitive function and I am interested in knowing which measure(s) are more susceptible than others to short or long-term alcohol consumption. Here are the 10 measures used in the study: 
-Cognitive 3MS Part 1
-Cognitive 3MS Part 2
-Cognitive CVLT
-Cognitive FingerTap
-Cognitive DSCT
-Cognitive FAS A
-Cognitive FAS F
-Cognitive FAS S
-a cognitive FAS Total
-Cognitive Checklist
Thank you. 
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Binge drinking has been shown to affect impulsivity measure on a variety of cognitive tasks, e.g. Reflection Impulsivity  Information as measured by the Sampling Task (IST; CANTAB Cambridge Cognition Ltd.).
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[Edited question:]
Sorry, I wasn't being specific enough in my original question (but thanks for your responses so far!). Things like personality and genetics don't really change from day to day, so they can't directly correlate with / predict / cause someone to drink on one day but not another. I'm looking for antecedent causal variables that can fluctuate from day to day, and thereby cause fluctuating behavior from day to day. There are some daily-drinking-diary studies out there on this kind of thing (e.g., daytime experiences of negative social interactions leading to more drinking that evening), and to achieve greater specificity I want to parse "more drinking" into the binary variable "if drank" and the interval variable "how much drank, provided drinking occurred." Different daytime events may be differentially stronger predictors of these two criterion variables, and I'm looking to learn what people know about this possibility. In any research, have these variables been parsed before with respect to the causes of a single drinking episode? Aside from established research, what are your best guesses? Thanks!
[Original question:]
I'm aware of the frequency/quantity literature on characterizing global drinking traits, but would like to hone in on what causes whether or not a person will drink on a given day versus how much they will drink on a given day, provided they have at least one drink. Studies on daily determinants of other kinds of potentially problematic behavior or experiential avoidance would also be useful (e.g., drug use, binge eating); I'm primarily interested in functional and methodological approaches to this kind of distinction. Thanks!   
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I would suggest looking at secondary analyses from Project MATCH or the COMBINE study. Then look at predictors of percentage of days abstinent (a marker of frequency) and drinks per drinking day.
We recently looked at this in a depressed alcohol dependent sample (Foulds et al, Alcohol and Alcoholism,doi: 10.1093/alcalc/agv122) and found the personality trait novelty seeking predicted more heavy drinking on drinking days. 
Personality measures are probably worth considering for your analyses.
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I am looking for widely used/validated self-report measures of substance use (both alcohol and drug use) to use for an upcoming project examining the structure of externalizing psychopathology. I already am planning to use the AUDIT and SMAST to assess alcohol use and already have a measure of drug use called the Drug Use Survey that I am planning to use. 
However, I would like to include another 2-3 drug- and alcohol-related measures as well. I would prefer to use dimensional (rather than categorical) assessment measures if possible, and would like at least 1-2 of these other measures to assess substance use other than alcohol (so other illicit substances).
Thank you for your help!
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Thank you both for your suggestions and comments!
Kasey
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Can anyone please help me to know what volume of liquid alcoholic diet should be given per animal per day to induce osteopenia or to check the adverse effects of alcohol?
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Looking for some comments on your experience of using these two drugs in the above setting. Benzodiazepines have been recommended, but can sometimes be inadvertently deliriogenic! Despite adequate patient selection, there appears to be some inter-patient variability with Dexmedetomidine and clearly there is a cost implication that comes with it. Although Clonidine is cheaper, it has issues with rebound hypertension and being not as effective in my experience so far. Would be interested to hear your thoughts.  
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Dear Anand Padmakumar,
Check this out:
Role of α2-agonists in the Treatment of Acute Alcohol Withdrawal
Andrew J Muzyk PharmD, Jill A Fowler PharmD, Daryn K Norwood PharmD, Allison Chilipko PharmD
DisclosuresThe Annals of Pharmacotherapy. 2011;45(5):649-657.
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I'm looking for research regarding alcohol consumption in post-industrial or ex-mining communities.  I am looking to study alcohol consumption in post-industrial communities that are currently experiencing decline/have experienced decline in South Yorkshire, England.
Mark Jayne and Gil Valentine (2010) have done a similar study in Stoke-on-Trent and in a Rural Community in Cumbria, but i struggle to find similar studies that have analysed communities such as ex-coalfield villages in Barnsley, Rotherham and Doncaster. 
I am really interested in studying how the industry and how it's decline in areas in South Yorkshire have effected alcohol consumption and 'alcohol culture' in the area.   Areas that have high unemployment, low educational prospects and poor health. 
Thanks in advance for your replies.
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Hi Everyone,
Thanks for the replies. They've been really helpful! I'll check out Barnsley Drug and Alcohol service and equivalent services in other boroughs (if there are any).  I'll also get in touch with David Beckingham at Liverpool.
Justin 
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I am interested in finding relevant research on the spread of alcohol use attitudes and behaviours through adolescent peer/friendship/social networks. I'm particularly interested in longitudinal studies that have separated out the effects of selection versus influence. Can anyone point me in the direction of relevant published research? 
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We have a major problem in North America with intoxicated patients involved in major trauma, other than their injuries, and there are rarely any legal implications. The literature reports that 60-90% of drivers who are evaluated by a trauma service are not charged with impaired driving. They follow through cracks in the system. Has anyone addressed this in your center?
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as for Turkey, any driver involved in accidents (and who are intoxicated by alcohol or any other drugs) are taken in custody after discharge from ED or hospital until freed by court order.  if there is/are patients with serious injuries/death involved in the accident, usually prosecution office demand for jail time until the next trial (wihch usually takes 2-3 months).  i once asked an attorney general about the rationale for this practice and he said whether the driver is guilty or not, any alcohol involvement increases the chance for getting a sentence, and this practice is considered to "ease the pain of the" of the deceased ones.
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Dear all, I’m looking for a questionnaire measuring degree of alcohol cravin « here and now ». My difficulty is that I work with French-speaking population, so I need a qestionnaire validated in French. Can you suggest something ? Thanks a lot.
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I know it is a bit late but the most used scale is the Obsessive and Compulsive Drinking Scale.
Annsseau, M., Besson, J., Lejoyeux, M., Pinto E., Landry, U., Cornes, M., Deckers, F.,
Potgieter, A. & Ades, J. (2000). A French translation of the obsessive-compulsive
drinking scale for craving in alcohol-dependent patients: A validation study in Belgium,
France, and Switzerland. European Addiction Research, 6, 51-56.
If you want, I can send it to you.
However, as craving is more conceptualized as an intense desire, the compulsive part is not relevant. May and colleagues have created an interresting transdiagnostic scale (Craving Experience Questionnaire) but it is in English. I'm currently doing research to create a more holistic scale.
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I want to compare with SPSS the two groups (alcoholic admissions vs non alcoholic ones) regarding which has a longer stay in hospitals and which acquires more charges. Appreciate your help!
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Length of stay and hospitals costs are invariably highly skewed. You can try doing a log transformation, followed by a t-test. Alternatively, you could try a Mann-Whitney U-test on the original data. In terms of a regression approach, you could use the SPSS generalized linear model procedure  with a gamma distribution and log link function. This often fits length of stay data quite well.
Adrian
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I have the graduated frequency questionnaire which consists of the 6 questions however I have no clue on how to analyze it- meaning how are we going to conclude that a person is a heavy, moderate, light drinker? Or if there is dependence? Please help. Quite urgent.
Thank you
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Dear Dr Singh
to define the different categories of drinkers the WHO classification according utlize the NCR (consumption level risk), which is set according to the daily consumption and / or consumption throughout the week if this is intermittent. Thus:
Low Risk Drinking Women 1-20 gr / day and 1-40 g / day in men
Consumption of Medium Risk in Women: 20-40 gr / day and 40-60 g / day in men
High-Risk Drinking in Women: 40-60 gr / day and 60-100 g / day in men
Very High Risk Drinking:> & 0 g / day in women and> 100 g day in men.
  The concept of dependency involves other clinical and psychosocial parameters defining the DSM V or ICD10, allowing diagnosis. In any case, an alcohol consumption Very High Risk perfectly as could include alcoholism.
In any case I leave the reference for more information on measures of consumption that define risk groups according to WHO
I hope that the information has been useful
Greetings.
EMA treatment guidelines (WHO International guide for monitoring alcohol consumption and related harm. © WHO, 2000);Rehm et al. Eur Addict Res 2001;7:138–147.
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In my study I used the questionnaire "Physical Self Perception Profile 25" (Fox and Corbin) on alcoholic patients. I would like to know wether the socres are similar to healthy adults and/or people with alcohol problems; Any references? What values are considered high/low/normal?
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Dear Alexandra, these are considerations you should have done before you started your study. One possibility would be to look for a control group and to examine them with the questionnaire. Or you will look for papers in which the PSPP was used and the results for certain patient groups are reported.
Find attached a pdf with a short cv of Professor Ken Fox the author of the PSPP. You find his email in the cv and you can directly ask him whether he would provide you with some infomation/ data you are looking for. Good look fur your further work.
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While we can find a lot of information on drinking patterns we have not found much on actual attitudes to drinking. Does anyone have some sources of information please?
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If you haven't already come across it you should look at the research sponsored by Alcohol Research UK. I am not sure that there is anything specific on Ireland but there is definitely work on adolescent attitudes in the UK
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I am looking for literature which specifically evaluates the efficacy of health promotion messages relating to safe drinking limits. By conventional, I mean messages which are designed to increase awareness of what constitutes ‘safe’ drinking levels and to increase understanding of existing government recommendations regarding weekly/daily unit intake maxima. Many thanks.
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Regarding: conventional ‘safe consumption level’ alcohol-related health promotion messages...My research is a text based intervention to reduce alcohol related harms. I comment here because the content & timing of the personal text messages in my research is controlled by each participant. This approach challenges the efficacy of "standard" health promotion messages - as I found out when I saw the messages participants had created for themselves.
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We have laws in some Australian states but I'm interested in what is happening in other countries/states/provinces.
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Постановление Верховного Суда РФ от 23.09.2013 N 80-АД13-4
Требование: Об отмене постановления о привлечении к ответственности, предусмотренной частью 2.1 статьи 14.16 КоАП РФ за розничную продажу несовершеннолетнему алкогольной продукции.
Решение: Производство по делу прекращено на основании пункта 6 части 1 статьи 24.5 КоАП РФ в связи с истечением срока давности привлечения к административной ответственности.
ВЕРХОВНЫЙ СУД РОССИЙСКОЙ ФЕДЕРАЦИИ
ПОСТАНОВЛЕНИЕ
от 23 сентября 2013 г. N 80-АД13-4
Судья Верховного Суда Российской Федерации Меркулов В.П., рассмотрев надзорную жалобу Наумовой Л.И. на постановление мирового судьи судебного участка N 2 Барышского административного района Ульяновской области от 25 февраля 2013 г., решение судьи Барышского городского суда Ульяновской области от 2 апреля 2013 г. и постановление заместителя председателя Ульяновского областного суда от 15 мая 2013 г., вынесенные в отношении Наумовой Л.И. по делу об административном правонарушении, предусмотренном частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях,
установил:
постановлением мирового судьи судебного участка N 2 Барышского административного района Ульяновской области от 25 февраля 2013 г., оставленным без изменения решением судьи Барышского городского суда Ульяновской области от 2 апреля 2013 г. и постановлением заместителя председателя Ульяновского областного суда от 15 мая 2013 г., Наумова Л.И. признана виновной в совершении административного правонарушения, предусмотренного частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях, и подвергнута административному наказанию в виде административного штрафа в размере 3 000 рублей.
В надзорной жалобе, поданной в Верховный Суд Российской Федерации, Наумова Л.И. просит отменить постановление мирового судьи судебного участка N 2 Барышского административного района Ульяновской области от 25 февраля 2013 г., решение судьи Барышского городского суда Ульяновской области от 2 апреля 2013 г. и постановление заместителя председателя Ульяновского областного суда от 15 мая 2013 г., вынесенные в отношении нее по делу об административно правонарушении, предусмотренном частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях, считая их незаконными.
Изучение материалов дела об административном правонарушении и доводов жалобы Наумовой Л.И. свидетельствует о наличии оснований для удовлетворения данной жалобы.
В соответствии с частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях (в редакции Федерального закона от 21 июля 2011 г. N 253-ФЗ) розничная продажа несовершеннолетнему алкогольной продукции, если это действие не содержит уголовно наказуемого деяния, влечет наложение административного штрафа на граждан в размере от 3000 до 5000 рублей; на должностных лиц - от 10 000 до 20 000 рублей; на юридических лиц - от 80 000 до 100 000 рублей.
Как усматривается из материалов дела, 13 ноября 2012 г. в 16 часов 25 минут Наумова Л.И., работающая продавцом у <...> П. в магазине, расположенном по адресу: Ульяновская область, Барышский район, <...>, реализовала алкогольную продукцию - одну бутылку пива "Большая кружка" объемом 1,5 литра с содержанием этилового спирта 4% несовершеннолетнему М.
В соответствии с частью 1 статьи 4.5 Кодекса Российской Федерации об административных правонарушениях срок давности привлечения к административной ответственности за совершение административного правонарушения, предусмотренного частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях, составляет два месяца, а в случае рассмотрения дела судьей - три месяца.
Обстоятельства, явившиеся основанием для привлечения Наумовой Л.И. к административной ответственности, имели место 13 ноября 2012 г. Следовательно, по данному делу об административном правонарушении срок давности привлечения ее к административной ответственности истек 13 февраля 2013 г.
Таким образом, постановление мирового судьи судебного участка N 2 Барышского административного район Ульяновской области от 25 февраля 2013 г. было вынесено за пределами срока давности привлечения Наумовой Л.И. к административной ответственности.
Из системного толкования части 1 статьи 4.5, пункта 6 части 1 статьи 24.5 Кодекса Российской Федерации об административных правонарушениях следует, что истечение срока давности привлечения к административной ответственности является обстоятельством, исключающим производство по делу об административном правонарушении.
При таких обстоятельствах постановление мирового судьи судебного участка N 2 Барышского административного района Ульяновской области от 25 февраля 2013 г., решение судьи Барышского городского суда Ульяновской области от 2 апреля 2013 г. и постановление заместителя председателя Ульяновского областного суда от 15 мая 2013 г., вынесенные в отношении Наумовой Л.И. по делу об административно правонарушении, предусмотренном частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях, подлежат отмене, а производство по делу об административном правонарушении - прекращению на основании пункта 6 части 1 статьи 24.5 Кодекса Российской Федерации об административных правонарушениях.
На основании изложенного, руководствуясь статьями 30.13 и 30.17 Кодекса Российской Федерации об административных правонарушениях, судья Верховного Суда Российской Федерации
постановил:
надзорную жалобу Наумовой Л.И. удовлетворить.
Постановление мирового судьи судебного участка N 2 Барышского административного района Ульяновской области от 25 февраля 2013 г., решение судьи Барышского городского суда Ульяновской области от 2 апреля 2013 г. и постановление заместителя председателя Ульяновского областного суда от 15 мая 2013 г., вынесенные в отношении Наумовой Л.И. по делу об административно правонарушении, предусмотренном частью 2.1 статьи 14.16 Кодекса Российской Федерации об административных правонарушениях, отменить.
Производство по делу об административном правонарушении прекратить на основании пункта 6 части 1 статьи 24.5 Кодекса Российской Федерации об административных правонарушениях.
Судья Верховного Суда
Российской Федерации
В.П.МЕРКУЛОВ