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Intuitively, this kind of infection would be detrimental to the host because there are more things exploiting it. But assuming that there is interference competition between the two species, isn’t it possible for them to lower each other’s pathogenicity because of the mutual reduction in fitness?

As we know that charcoal rot appear during stress period. Afterall pest and disease creates stress to plant.

Is there any relation between them?

What could be the probable techniques applied from transcriptomics to study metabolomics of plant pathogen interaction ?

Need suggestion.

More specifically, will a plant virus achieve greater inoculation success if the vector feeds on a growing plant part vs. a senescent part?

I've heard that fast growing parts of a plant may be more susceptible to infection from viruses, but haven't been able to find anything empirical backing this up.

I was analyzing several examples of plant pathogen interaction (pathosystems) and I was wondering if cell expansion comes first or second to cell division. Of course, I must considered that everything happens after a PAMP sequence of events and also to possible recognition of pathogen by plant cells.

Does anybody have some clues?

Thanks and Regards,

Dr. Joao Paulo R. Marques

As we know that the Macrophomina is the pycnidial stage, so where exactly the pycnidial stage form naturally.

In decoy model, in susceptible plant which is without R-prt or guard prt decoy prt plays the role of a sink and reduces the number of successful effector-operative gaudees interactions. While this causes reduction in pathoen fitness why the decoy prt should undergo evasive evolution which translates into higher pathogen fitness?

I want to determine the resting spore population present in Barley roots. if anybody can suggest how a single resting spore of Polymyxa graminis looks like, while resting spore suspension prepared by crushing the roots.

I've been working with an insect pest of tea plantations and I want to differentiate biochemically between the host (principally being tea) and a non host plant (with no record of insecticidal property). I wish to start with extraction of compounds through chromatographic techniques and then subject the isolated compounds to GC-MS, NMR and IR spectrometry to find out the nature of the compounds that differentiate them from being attacked by the pest.

Hello.

Nowadays, I am studying on seed storage protein in wheat and their relatives.

HMW glutenin is well-known and it has been studied a lot, but LMW glutenin has studied less than HMW glutenin although LMW has a relatively shorter sequence than HMW because it is harder to distinguish LMW bands than HMW bands.

Glu-1 encodes HMW glutenins, and a lot of Glu-1 such as Glu-A1, Glu-B1, Glu-D1 in wheat, Glu-H1 in barley, and Glu-R1 in rye have been characterized.

However, I couldn't find Glu-R3 in Secale cereale. I could find only one study about Glu-R3 in Secale sylvestre. But as far as I know, Secale sylvestre is not a cultivated rye but a kind of wild rye. Also, there is no Glu-R3 protein or nucleotide information of S. cereale in NCBI database. 

I want to know LMW glutenin in Secale cereale because it can affect to the quality of 1RS wheat-rye translocation varieties. I can't find any related reference. Please help me.