The Child Attention-Deficit Hyperactivity Disorder Teacher Telephone Interview (CHATTI): Reliability and validity

The University of Manchester, Manchester, England, United Kingdom
The British Journal of Psychiatry (Impact Factor: 7.99). 02/2004; 184(1):74-8. DOI: 10.1192/bjp.184.1.74
Source: PubMed


The ICD-10 and DSM-IV diagnostic criteria for hyperkinetic disorder and attention-deficit hyperactivity disorder (ADHD) require symptoms or impairment in two or more settings. Thus, information on children's symptoms in school is usually required. This paper presents the Child ADHD Teacher Telephone Interview (CHATTI), an instrument aimed at systematically obtaining this information.
To examine the stability, test-retest reliability and criterion validity of the CHATTI for children referred with a suspected diagnosis of ADHD.
Data were obtained from 79 teachers, of whom 36 were interviewed on two occasions.
Overall, the CHATTI shows good stability, test-retest reliability and criterion validity for symptom scores. Test-retest reliability for some individual items was low. Reliability for the operationalised criteria of 'pervasiveness' (i.e. symptoms at school and home) and 'school impairment' was excellent (kappa=1).
The CHATTI appears to be a promising tool for assessing ADHD symptoms in a school setting and could be useful in clinical as well as research settings.

Full-text preview

Available from:
  • Source
    • "ADHD and other psychiatric diagnoses were assessed by trained psychologists using the Child and Adolescent Psychiatric Assessment (CAPA) parent version,11 a semi-structured interview. Confirmation of pervasiveness of symptoms in school was obtained using the Child Attention-Deficit Hyperactivity Disorder Teacher Telephone Interview (CHATTI)12 or the Conner's Teacher Questionnaire.13 The Wechsler Intelligence Scale for Children–III/IV was used to assess IQ.14,15 "
    [Show abstract] [Hide abstract]
    ABSTRACT: Objective Attention-deficit/hyperactivity disorder (ADHD) and autism spectrum disorder (ASD) often co-occur and share genetic risks. The aim of this analysis was to determine more broadly whether ADHD and ASD share biological underpinnings. Method We compared copy number variant (CNV) data from 727 children with ADHD and 5,081 population controls to data from 996 individuals with ASD and an independent set of 1,287 controls. Using pathway analyses, we investigated whether CNVs observed in individuals with ADHD have an impact on genes in the same biological pathways as on those observed in individuals with ASD. Results The results suggest that the biological pathways impacted by CNVs in ADHD overlap with those impacted by CNVs in ASD more than would be expected by chance. Moreover, this was true even when specific CNV regions common to both disorders were excluded from the analysis. After correction for multiple testing, genes involved in three biological processes (“nicotinic acetylcholine receptor signalling pathway,” “cell division,” and “response to drug”) showed significant enrichment for case CNV hits in the combined ADHD and ASD sample. Conclusion The results of this study indicate the presence of significant overlap of shared biological processes disrupted by large, rare CNVs in children with these two neurodevelopmental conditions.
    Full-text · Article · Jul 2014 · Journal of the American Academy of Child & Adolescent Psychiatry
  • Source
    • "Exclusion criteria included an intelligence quotient (IQ) < 70, epilepsy, fragile X syndrome, fetal alcohol syndrome, maternal drug abuse during pregnancy, primary diagnosis of pervasive developmental disorder, Tourette's syndrome, psychosis, or bipolar disorder, and current treatment with other nonstimulant psychotropic medications. Clinical assessment of DSM IV ADHD diagnoses were confirmed by a child psychiatrist (E.B.) using the parent version of the Child and Adolescent Psychiatric Assessment (CAPA) (Angold et al. 1995) and The Child Attention-Deficit Hyperactivity Disorder Teacher Telephone Interview (CHATTI) (Holmes et al. 2004). Sixty-three (82%) children were diagnosed with combined type ADHD, 7 (9%) were diagnosed with the hyperactive-impulsive subtype, and 7 (9%) were diagnosed with the inattentive subtype. "
    [Show abstract] [Hide abstract]
    ABSTRACT: Objective: A naturalistic, prospective study of the influence of genetic variation on dose prescribed, clinical response, and side effects related to stimulant medication in 77 children with attention-deficit/hyperactivity disorder (ADHD) was undertaken. The influence of genetic variation of the CES1 gene coding for carboxylesterase 1A1 (CES1A1), the major enzyme responsible for the first-pass, stereoselective metabolism of methylphenidate, was investigated. Methods: Parent- and teacher-rated behavioral questionnaires were collected at baseline when the children were medication naïve, and again at 6 weeks while they were on medication. Medication dose, prescribed at the discretion of the treating clinician, and side effects, were recorded at week 6. Blood and saliva samples were collected for genotyping. Single nucleotide polymorphisms (SNPs) were selected in the coding, non-coding and the 3' flanking region of the CES1 gene. Genetic association between CES1 variants and ADHD was investigated in an expanded sample of 265 Irish ADHD families. Analyses were conducted using analysis of covariance (ANCOVA) and logistic regression models. Results: None of the CES1 gene variants were associated with the dose of methylphenidate provided or the clinical response recorded at the 6 week time point. An association between two CES1 SNP markers and the occurrence of sadness as a side effect of short-acting methylphenidate was found. The two associated CES1 markers were in linkage disequilibrium and were significantly associated with ADHD in a larger sample of ADHD trios. The associated CES1 markers were also in linkage disequilibrium with two SNP markers of the noradrenaline transporter gene (SLC6A2). Conclusions: This study found an association between two CES1 SNP markers and the occurrence of sadness as a side effect of short-acting methylphenidate. These markers were in linkage disequilibrium together and with two SNP markers of the noradrenaline transporter gene.
    Full-text · Article · Dec 2013 · Journal of Child and Adolescent Psychopharmacology
  • Source
    • "The interviewers were supervised weekly by a child psychiatrist and inter-rater reliability for ADHD sub-type research diagnosis, assessed using 60 cases, was perfect (j = 1.0). Pervasiveness of symptoms, necessary for a DSM-IV diagnosis, was assessed through questionnaires (DuPaul or Conner's teacher rating scale (Conners et al. 1998; DuPaul 1981)) sent to the school or the Child ADHD Teacher Telephone Interview (ChATTI (Holmes et al. 2004)). "
    [Show abstract] [Hide abstract]
    ABSTRACT: Attention-deficit hyperactivity disorder (ADHD) and autism spectrum disorder (ASD) often co-occur. Factor analyses of ASD traits in children with and without ASD indicate the presence of social and restrictive-repetitive behaviour (RRB) factors. This study used exploratory factor analyses to determine the structure of ASD traits (assessed using the Social Communication Questionnaire) in children with ADHD. Distinct factors were observed for 'social' and 'rigidity' traits, corresponding to previous factor analyses in clinical ASD and population samples. This indicates that the split between social-communicative and RRB dimensions is unaffected by ADHD in children. Moreover, the study also finds that there is some overlap across hyperactive-impulsive symptoms and RRB traits in children with ADHD, which merits further investigation.
    Full-text · Article · Jun 2013 · Journal of Autism and Developmental Disorders
Show more