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    • "59, see also Oliver, 2006). Healthy diet and active lifestyle are better predictors of health outcomes, rather than excess weight (Campos et al., 2006). The overweight and obese population is heterogeneous with some obese persons having better metabolic profiles than normal weight individuals (Primeau et al., 2011). "
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    ABSTRACT: Because body weight is largely seen as controllable, weight stigma—the social devaluation of those who are overweight—is not subject to the social norms that condemn open expression of racism and sexism. Indeed, rejection of peers based on perceptions of excess weight is normative. Since weight stigma is internalized, popular views (and often the views of physicians) have suggested that increasing the salience of weight stigma might produce a reduction in overeating and/or an increase in physical activity. However, that perspective is not rooted in scientific evidence. Recent randomized controlled designs demonstrate that stigma may promote overeating. Correlational evidence suggests that self-reported stigma experience is associated with risk for binge eating and decreased interest in physical exercise and dieting, for children and adults. In addition to reviewing these research studies, this paper examines the potential for intersectionality of stigma across multiple social identities and considers alternatives to stigmatizing weight loss interventions.
    Full-text · Article · Jan 2016 · Appetite
    • "These ideals are further bolstered through media coverage which suggests that weight loss is easy (Blaine and McElroy, 2002) and through the association of weight control and thinness with healthiness (Davies, 1998) and weight loss with personal responsibility (Ricciardelli et al., 2010). Some argue that contemporary concerns around obesity are actually attempts to regulate and manage 'deviant' populations (such as working class people and women) and are not really interested in health improvement (Evans et al., 2008), and may be in response to anxieties around social changes, such as changes in gender roles (Campos et al., 2006). "
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    ABSTRACT: The media play a key role in promoting the thin ideal. A qualitative study, in which we used in depth interviews and thematic analysis, was undertaken to explore the attitudes of 142 obese individuals toward media portrayals of the thin ideal. Participants discussed the thin ideal as a social norm that is also supported through the exclusion of positive media portrayals of obese people. They perceived the thin ideal as an ‘unhealthy’ mode of social control, reflecting on their personal experiences and their concerns for others. Participants’ perceptions highlighted the intersections between the thin ideal and gender, grooming and consumerism. Participants’ personal responses to the thin ideal were nuanced – some were in support of the thin ideal and some were able to critically reflect and reject the thin ideal. We consider how the thin ideal may act as a form of synoptical social control, working in tandem with wider public health panoptical surveillance of body weight.
    No preview · Article · Nov 2015 · Social Science [?] Medicine
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    • "HVR rats co-selected for their obtaining a total body fat percentage similar to LVR when each had an ad lib access to WD. Our findings and interpretations may be translatable to the human observation of fit, but fat, in which metabolic health improves in obese with exercise that does not lower total body fat [54] [55] [56]. While different species and outcomes, they reinforce each's findings that a little physical activity in a sedentary population has important health benefits. "
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    ABSTRACT: Metabolic disease risk is influenced by genetics and modifiable factors, such as physical activity and diet. Beginning at 6 wks of age, rats selectively bred for high (HVR) versus low voluntary running distance (LVR) behaviors were housed in a complex design with or without voluntary running wheels being fed either a standard or Western (WD, 42% kcal from fat and added sucrose) diet for 8wks. Upon intervention completion, percent body fat, leptin, insulin, and mediobasal hypothalamic mRNAs related to appetite control were assessed. Wheel access led to differences in body weight, food intake, and serum leptin and insulin. Intriguing, percent body fat, leptin, and insulin did not differ between HVR and LVR lines in response to the two levels of voluntary running, regardless of diet, after the 8 wk. experiment despite HVR eating more calories than LVR regardless of diet and voluntarily running 5-7 times further in wheels than LVR. In response to WD, we observed increases in Cart and Lepr mediobasal hypothalamic mRNA in HVR, but no differences in LVR. Npy mRNA was intrinsically greater in LVR than HVR, while wheel access led to greater Pomc and Cart mRNA in LVR versus HVR. These data suggest that despite greater consumption of WD, HVR animals respond similarly to WD as LVR as a result, in part, of their increased wheel running behavior. Furthermore, high physical activity in HVR may offset the deleterious effects of a WD on adiposity despite greater energy intake in this group.
    Full-text · Article · Sep 2015 · Physiology & Behavior
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