Was Julius Caesar's epilepsy due to neurocysticercosis?

Department of Experimental Pathology, M.B.I.E., Università di Pisa, Medical School, Via Roma, 55, 56126 Pisa Italy.
Trends in Parasitology (Impact Factor: 6.2). 09/2011; 27(9):373-4. DOI: 10.1016/
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Available from: Fabrizio Bruschi
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    ABSTRACT: Neurocysticercosis (neural infection by larvae of Taenia solium) occurs when humans become intermediate hosts of the tapeworm Taenia solium after ingesting its eggs. The disease is now the most common helminthic infection of the nervous system in humans, and its prevalence has risen significantly even in countries where it was formerly considered exotic. The introduction of modern neuroimaging and serologic techniques has improved the diagnosis of neurocysticercosis; furthermore, the development of potent cysticidal drugs has changed the prognosis of most affected patients. Nevertheless, much remains to be learned about this parasitic disease. This book provides a comprehensive and up-to-date review of the various aspects of cysticercosis of the nervous system that will be of interest to all who are involved in the care of patients with this disease. Epidemiology, neuropathology, immunopathogenesis, clinical manifestations, diagnosis, and management are all thoroughly discussed based on current evidence and practice. © Springer-Verlag Berlin Heidelberg 2014. All rights are reserved.
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    ABSTRACT: Julius Caesar (100 - 44 BC) was one of the most charismatic political figures in history. Best remembered for his military achievements, he was also a writer, historian and statesman. Through his constitutional reforms, he played an important role in the events that led to the end of the Roman Republic and the birth of the Roman Empire. Historical sources reveal that Julius Caesar suffered from headaches, seizures and personality changes. In this essay, we highlight the life of Julius Caesar, with emphasis on the potential origin of his sickness. Although a definitive diagnosis obviously cannot be made, as new published studies showed a possible cerebrovascular etiology, a new hypothetical diagnosis is presented. Copyright © 2015 Elsevier Inc. All rights reserved.
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