A case of reversible restless legs syndrome (RLS) and sleep-related eating disorder relapse triggered by acute right leg herpes zoster infection: Literature review of spinal cord and peripheral nervous system contributions to RLS
Minnesota Regional Sleep Disorders Center, Department of Neurology, Hennepin County Medical Center, The University of Minnesota Medical School, Minneapolis, MN 55415, USA.Sleep Medicine (Impact Factor: 3.15). 06/2010; 11(6):583-5. DOI: 10.1016/j.sleep.2009.11.007
Restless legs syndrome (RLS) is thought to be due to abnormalities of iron metabolism in the central nervous system; however, occasional cases are associated with lesions of the spinal cord, spinal rootlets, and peripheral nervous system. This is a case report of RLS exacerbated by shingles with a review of the literature of extra-cerebral lesions or disorders causing or contributing to RLS.
Article: Sleep Eating[Show abstract] [Hide abstract]
ABSTRACT: Pathologic sleep eating, known as sleep-related eating disorder (SRED), is characterized by disordered nocturnal eating that arises from sleep. This disruption in the nocturnal fast can result in adverse health consequences such as inadequate sleep maintenance, weight gain, the consumption of inedible substances, or sleep-related injury. The dysfunctional eating may occur in the setting of sedative-hypnotic medications, in particular the widely prescribed benzodiazepine receptor agonists such as zolpidem, frequently inducing amnesia. SRED is often associated with other sleep disorders, in particular restless legs syndrome, and may be a nonmotor manifestation of this condition.
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