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Intentional Destruction of Life on Earth

DOI:10.14738/assrj.87.10597
Authors:
J. Marvin Herndon at Transdyne Corporation, California
J. Marvin Herndon
  • Transdyne Corporation, California
Mark Whiteside at Florida Department of Health in Monroe County
Mark Whiteside
  • Florida Department of Health in Monroe County
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Abstract and Figures

The title might suggest that this is a fictional work about an invasion from outer space. Wrong. It is a true recitation of forensic scientific and medical investigations of a near-global, near-daily conspicuous activity whose actual nature and intent is cloaked in secrecy and disinformation.
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Advances)in)Social)Sciences)Research)Journal)–)Vol.)8,)No.)7)
Publication)Date:!"#$%!&'(!&)&*!
DOI+*),*-./0123345,0.,*)'6.,!
Herndon, J. M., & Whiteside, M. (2021). Intentional Destruction of Life on Earth. Advances in Social Sciences Research Journal, 8(7).
295-309.
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Intentional!Destruction!of!Life!on!Earth!
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J.)Marvin)Herndon,)Ph.D.)
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Mark)Whiteside,)M.D.,)M.P.H.)
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of$a$near-global,$near-daily$conspicuous$activity$whose$actual$nature$and$intent$is$
cloaked$in$secrecy$and$disinformation.$
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Full-text available
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Full-text available
  • Mar 2020
Coral reefs provide many benefits to the society, including ecological nest for aquatic species, medicine ingredient, and protection of coastlines from flooding and storms. Organic and inorganic UV filters (UVfs) are key ingredients in a variety of sunscreen and personal care products, which are massively released to natural waters by marine tourism. Reports are alarming UVfs cause fast bleaching of coral reefs in the Persian Gulf at a tiny concentration (less than 10 µl L-1). Here, for the first time, the evidence pertaining to the impacts of UVfs on corals in the Persian Gulf is highlighted. Based on the existing field reports, laboratory experiments and increasing trend f tourism, corals of Kish Island in Iran are seriously threatened. Here, Kish is a study model that highlights the alarming impacts of UVfs on the endangered population of corals in all Gulf countries. This study found a positive relation between the number of visitors to the island and the bleaching rate of stony coral.
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Technology Bill of Rights Needed to Protect Human and Environmental Health and the U. S. Constitutional Republic
Article
  • Jul 2020
For the protection of humanity and the planetary environment in general, and American citizens in particular, what is needed, we posit, is a set of new Constitutional Amendments that collectively form a second Bill of Rights, a Technology Bill of Rights, to protect our freedoms, health, air, water, agriculture, and the planetary environment from deliberate perversion and alteration. We describe the rationale for said Technology Bill of Rights that would: (1) Prohibit the application of any technique or method for changing – through the deliberate manipulation of natural processes – the dynamics, composition or structure of the Earth, including its biota, lithosphere, hydrosphere and atmosphere, or of outer space; (2) Prohibit the application of any technique, including software-based process or platform or method for violating individuals free speech, censoring, altering, editing, deleting, excluding, blacklisting, or engaging in any activities that potentially bias votes or deceive the public on matters of health and/or environmental harm; and, (3) Prohibit activities of such scale and nature that would intentionally or unintentionally alter the complex but delicate balance in nature by and between myriad biota and their environments that makes our planet habitable for life. Whereas the meaning of (1) and (2) above is clear, (3) necessitates further clarification that may be inferred from the following non-exclusive examples of prohibited activities: ● Use of metallic and/or nano-particulate additions to aircraft fuel; ● Excessive launching of satellites, numbering in the tens of thousands, whose rocket exhaust might damage the ozone layer; ● Excessive exposure of humans and other biota to electromagnetic radiation; ● Use of electromagnetic radiation to heat the ionosphere; ● Pollution of air, land, water, agriculture, and aquaculture by particulates, toxic chemicals, heavy metals, radioactive nuclides, and bio-toxins; and, ● Strict oversight of biotechnology/bioengineering, including prohibition of gain-of-function experiments with potential pandemic pathogens.
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Iron-rich air pollution nanoparticles: An unrecognised environmental risk factor for myocardial mitochondrial dysfunction and cardiac oxidative stress
Article
  • Jun 2020
Exposure to particulate air pollution is a major environmental risk factor for cardiovascular mortality and morbidity, on a global scale. Both acute and chronic cardiovascular impacts have so far been attributed to particulate-mediated oxidative stress in the lung and/or via ‘secondary’ pathways, including endothelial dysfunction, and inflammation. However, increasing evidence indicates the translocation of inhaled nanoparticles to major organs via the circulation. It is essential to identify the composition and intracellular targets of such particles, since these are likely to determine their toxicity and consequent health impacts. Of potential major concern is the abundant presence of iron-rich air pollution nanoparticles, emitted from a range of industry and traffic-related sources. Bioreactive iron can catalyse formation of damaging reactive oxygen species, leading to oxidative stress and cell damage or death. Here, we identify for the first time, in situ, that exogenous nanoparticles (∼15 – 40 nm diameter) within myocardial mitochondria of young, highly-exposed subjects are dominantly iron-rich, and co-associated with other reactive metals including aluminium and titanium. These rounded, electrodense nanoparticles (up to ∼ 10 x more abundant than in lower-pollution controls) are located within abnormal myocardial mitochondria (e.g. deformed cristae; ruptured membranes). Measurements of an oxidative stress marker, PrPC and an endoplasmic reticulum stress marker, GRP78, identify significant ventricular up-regulation in the highly-exposed vs lower-pollution controls. In shape/size/composition, the within-mitochondrial particles are indistinguishable from the iron-rich, combustion- and friction-derived nanoparticles prolific in roadside/urban environments, emitted from traffic/industrial sources. Incursion of myocardial mitochondria by inhaled iron-rich air pollution nanoparticles thus appears associated with mitochondrial dysfunction, and excess formation of reactive oxygen species through the iron-catalyzed Fenton reaction. Ventricular oxidative stress, as indicated by PrPC and GRP78 up-regulation, is evident even in children/young adults with minimal risk factors and no co-morbidities. These new findings indicate that myocardial iron overload resulting from inhalation of airborne, metal-rich nanoparticles is a plausible and modifiable environmental risk factor for cardiac oxidative stress and cardiovascular disease, on an international scale.
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