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Was Pasteur Wrong? Human Cells May not may Generate Bacteria

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Was Pasteur Wrong? Human Cells
May Generate Bacteria
Alen J Salerian*
Modern Psychiatry, Athens, Greece
Received: May 22, 2018; Published: May 29, 2018
*Corresponding author: Alen Salerian, Modern Psychiatry, Athens, Greece, Tel: 3 ; Email:
DOI: 10.26717/BJSTR.2018.04.001127
Alen Salerian. Biomed J Sci & Tech Res
Cite this article: Alen Salerian. Was Pasteur Wrong? Human Cells may Generate Bacteria. Biomed J Sci &Tech Res 4(5)- 2018. BJSTR.
MS.ID.001127. DOI: 10.26717/ BJSTR.2018.04.001127.
Editorial Open Access
Editorial
Since 1856 when Louis Pasteur proved that infections origi-
nate from bacteria the germ theory has been the central paradigm

germ theory destined Antoine Bechamp, Pasteur’s arch rivalto sci-
        -
icine. Today not many physicians even heard of Bechamp or his

teria and infections [3,4]. Surprisingly a novel hypotheses” Human
body may produce bacteria “published in Medical Hypotheses [5]

compelling evidence to suggest that although Bechamp failed to of-
fer experimental evidence to support” bacterial growth by transfor-
mation”, he did indeed make a valid discovery that infections may
originate by “pathways independent of contamination “.
ISSN: 2574-1241
Figure 1: Alen Salerian, Pasteur, Bechamp.
History of science has been rich with observations that para-

of the earth drop and being the center of universe lasted over thou-
sand years before the Copernicus discovery. It is easy to prove Pas-
teur wrong for dismissing the existence of” pathways independent
-
teria were born on lifeless earth not by contamination but transfor-
mation of nonliving things to bacteria [6]. Hence, that there are in-
deed pathways independent of contamination for bacterial growth.
Three observations prove that human cells are capable to trans-
form to bacteria consistent with the following evidence:
a) Unicellular organisms transform to different unicellular
organisms [7].
b) Human cells transform to different cells (cancer, stem
cells to bone marrow) [8].
c) Human cells contain all the essentials for life.
Our current paradigm in treating infections -based upon the
premise that contamination is the only pathway for bacterial
-
possibilities.
It is not logical because it has never been proven that that are
no other pathways for bacterial growth. It is physically impossible
because it violates the laws of evolution and history of life on earth.
Burning infections reveal further evidence in support of transfor-
mation of dead or dying human cells to pathogens. Burning wound
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Alen Salerian. Biomed J Sci & Tech Res Volume 4- Issue 5: 2018
Biomedical Journal of
Scientific & Technical Research (BJSTR)
infections seem to be the rule rather than exception in major burns
    
[9]. This high rate of infections stands in contrast to burns routinely
treated with anti-septic solutions and silver sulfadiazine. Also, heat
induced tissue damage is equally lethal for microorganisms. Hence,
the emergence of rare pathogens in burn infections seems consist-
ent with transformation of dead or dying cells to virulent microor-
ganisms.
In retrospect it seems that both Pasteur and Bechamp were
partly wrong and partly correct and both contamination and trans-
formation are valid pathways for bacterial growth and infections.
This discovery may facilitate novel interventions to combat infec-
tions especially those associated with massive tissue losses, burns
and major trauma. It may have other implications in medicine far
beyond infectious disorders for it may lead to better understanding
-
      -

prompted to investigate whether some infections result from trans-
formation triggered by adverse environmental conditions-such as
sudden temperature changes and especially exposure to cold-may
lead to epidemics by host to host transmission.
References
1. Pasteur L (1880) On the extension of the germ theory to the etiology of
certain common diseases.
2. Comptes Rendus de l’Academic des Sciences Xc Ernst (Ttrans). pp. 1033-
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3. Manchester KL, Louis Pastor (2007) fermentation and a rival. South
African journal of science 103(9): 377-380.
4. Hume ED (1924) Bechamp or Pasteur? A lost chapter in the history of
biology. Covici Mv Gee, Chicago.
5. BechampA, LesMicrozymas: Heterogeny, histogeny, physiology and
pathology. Librarie JB Baillierre, Paris.
6. Salerian AJ (2017) Human body may produce bacteria. Medical
Hypotheses 103: 131-132.
7. VanHolde KE (1980) the origins of life and evolution. Allan R Liss, New
York, USA.
8. Krause DS, Thiese ND, Collector ML (2001) Multi-Organ, Multi-Lineage
Engraftment by a Single Bone Marrow-Derived Stem Cell. Cell 105(3):
369-377.
9. Rosenow EC (1914) Test mutations within the streptococcus enamel
proper school. Journal of Infectious Disorders 14(1): 1-32.
10. Macedo JLS Santos JB (2005) Bacterial and fungal colonization of burn
wounds. Mem Inst Oswaldo Cruz 100(5): 535-539.
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... Introduction Unicellular organisms (prokaryota) including bacteria transformed from lifeless matter 3.5 billion years ago [1][2][3][4][5]. Organic compounds were produced by artificial methods [6]. ...
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... Scientific observations suggest pathways independent of contamination may produce infections [1,2]. For instance, unicellular organisms (prokaryota) including bacteria transformed from lifeless matter 3.5 billion years ago [3][4][5]. ...
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“Human body may produce bacteria” proposes that human body may produce bacteria and represent an independent source of infections contrary to the current paradigm of infectious disorders proposed by Louis Pasteur in 1880. The following observations are consistent with this hypothesis: A. Bidirectional transformations of both living and nonliving things have been commonly observed in nature. B. Complex multicellular organisms harbor the necessary properties to produce bacteria (water, nitrogen and oxygen). C. Physical laws suggest any previously observed phenomenon or action will occur again (life began on earth; a non living thing). D. Animal muscle cells may generate energy (fermentation). E. Sterilized food products (i.e. boiled eggs), may produce bacteria and fungus under special conditions and without any exposure to foreign living cells. “Human body may produce bacteria” may challenge the current medical paradigm that views human infectious disorders as the exclusive causative byproducts of invading foreign cells. It may also introduce new avenues to treat infectious disorders.
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Purification of rare hematopoietic stem cell(s) (HSC) to homogeneity is required to study their self-renewal, differentiation, phenotype, and homing. Long-term repopulation (LTR) of irradiated hosts and serial transplantation to secondary hosts represent the gold standard for demonstrating self-renewal and differentiation, the defining properties of HSC. We show that rare cells that home to bone marrow can LTR primary and secondary recipients. During the homing, CD34 and SCA-1 expression increases uniquely on cells that home to marrow. These adult bone marrow cells have tremendous differentiative capacity as they can also differentiate into epithelial cells of the liver, lung, GI tract, and skin. This finding may contribute to clinical treatment of genetic disease or tissue repair.
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A prospective study of fungal and bacterial flora of burn wounds was carried out from February 2004 to February 2005 at the Burns Unit of Hospital Regional da Asa Norte, Brasília, Brazil. During the period of the study, 203 patients were treated at the Burns Unit. Wound swab cultures were assessed at weekly intervals for four weeks. Three hundred and fifty four sampling procedures (surface swabs) were performed from the burn wounds. The study revealed that bacterial colonization reached 86.6% within the first week. Although the gram-negative organisms, as a group, were more predominant, Staphylococcus aureus (28.4%) was the most prevalent organism in the first week. It was however surpassed by Pseudomonas aeruginosa form third week onwards. For S. aureus and P. aeruginosa vancomycin and polymyxin were found to be the most effective drugs. Most of the isolates showed high level resistance to antimicrobial agents. Fungi were found to colonize the burn wound late during the second week postburn, with a peak incidence during the third and fourth weeks. Species identification of fungi revealed that Candida tropicalis was the most predominant, followed by Candida parapsilosis. It is crucial for every burn institution to determine the specific pattern of burn wound microbial colonization, the time-related changes in the dominant flora, and the antimicrobial sensitivity profiles. This would enable early treatment of imminent septic episodes with proper empirical systemic antibiotics, without waiting for culture results, thus improving the overall infection-related morbidity and mortality.
Article
On the extension of the germ theory to the etiology of certain common diseases / Louis Pasteur ; translated by H. C. Ernst Note: The University of Adelaide Library eBooks @ Adelaide.
  • K L Manchester
  • Louis Pastor
Manchester KL, Louis Pastor (2007) fermentation and a rival. South African journal of science 103(9): 377-380.
Bechamp or Pasteur? A lost chapter in the history of biology
  • E D Hume
Hume ED (1924) Bechamp or Pasteur? A lost chapter in the history of biology. Covici Mv Gee, Chicago.
Test mutations within the streptococcus enamel proper school
  • E C Rosenow
Rosenow EC (1914) Test mutations within the streptococcus enamel proper school. Journal of Infectious Disorders 14(1): 1-32.