Plant life and reproduction are constantly threatened by biotic and abiotic pressures generated by pathogenic bacteria, fungi, viruses, and oomycetes, as well as environmental variables (Panstruga et al., 2009). The majority of plant pathogenic
microorganisms, such as bacteria and fungi, enter the apoplast and extract nutrients through a variety of enzymatic and physical methods. The plant's principal goal is to be healthy and productive by defending itself against pathogens via physical and chemical barriers such as the cell wall, waxes, hairs, antimicrobial enzymes, and secondary metabolites. Pathogens have the ability to break through these barriers and get access to their hosts. For successful defence, the plant must be able to identify microbial assault. When a pathogen is identified, the plant responds by inducing apoplastic defence to block microbial enzymes, reinforce cell walls, or poison the pathogen. The microbial phytopathogens must have some sort of interaction with the cell wall. Necrotrophic pathogens, which destroy cells and feed on dead tissues, macerate plant tissues by secreting a large number of hydrolytic enzymes that break down cell wall polymers (Laluk and Mengiste, 2010). Biotrophic and hemibiotrophic infections, on the other hand, must interact with their hosts by generating specialised structures, such as the haustorium produced by oomycete mildews, powdery mildew, and rust-causing fungus (Szabo and Bushnell, 2001). Due to invasion difficulties, the characteristics of the plant cell wall have altered, activating a defensive barrier for those bacteria that have evolved a way to overcome the produced barriers. Plants have evolved an innate immunity system known as pattern triggered immunity (PTI), which is based on a set of plasma membrane-anchored pattern-recognition receptors (PRRs) that detect a set of microbial molecules known as microbial/pathogen associated molecular patterns (MAMPs/PAMPs) and prevents pathogen development (Boller and He, 2009).