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Memory and the construction of scientific meaning: Michael Faraday's use of notebooks and records

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Research examining the relationship between external artifacts and scientific thinking has highlighted the dynamic role of memory aids. This article explores how the nineteenth-century physicist Michael Faraday (1791-1867) used extensive laboratory notebooks and a highly structured set of retrieval strategies as dynamic aids during his scientific research. The development and dynamic use of memory artifacts are described as part of a distributed, “real-world,” cognitive environment. The processes involved are then related to aspects of expert memory and to the use of model-based reasoning in science. The system demonstrates the importance of epistemic artifacts in scientific cognition and is suggestively related to other cognitive artifacts used in scientific research that rely on similar cognitive processes.

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... However, artifacts do (sometimes quite dramatically) transform our cognitive practices. Lab notebooks, for example, have significantly transformed many of the cognitive practices in laboratories (see, e.g., Tweney and Ayala 2015;Holmes et al. 2003). In sum, lab notebooks rank high on all dimensions, except transformation. ...
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In 1839, when Michael Faraday was 48 and still in the full spate of his genius, he suffered an illness which prevented him doing any work for more than 3 yr. He never fully recovered from this illness and for the rest of his life (he lived to be 75) he complained of his bad memory and of recurrent attacks of giddiness and confusion. A probable cause of the ischaemic attacks could have been atheromatous lesions in the walls of the great vessels, throwing off platelet emboli. Between the ages of 30 and 40, Faraday spent much of his working time in the underground laboratory at the Royal Institution engaged in researches on steel and glass which involved the continual use of a furnace. There is no evidence that he ever suffered from acute carbon monoxide poisoning but exposure to low concentrations of carbon monoxide certainly seems a possibility and this, taken with the recent evidence that such exposure can cause arteriosclerosis affords an explanation for what may have been the initial pathological lesion in Faraday's case.
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