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ATHLETIC THERAPY TODAY SEPTEMBER 2006 69
David O. Draper, EdD, ATC, Column Editor
Top-10 Positional-Release Therapy Techniques
to Break the Chain of Pain, Part 1
OSITIONAL-RELEASE therapy (PRT) is a
treatment technique that is gaining popu-
larity. The purpose of this two-part column
is to briefly explain the theory and appli-
cation of PRT. Then, we will present our top-10 list of
what we think are the most commonly treated tender
points by athletic therapists, five in Part 1 and five in
Part 2, along with general tips, patient self-treatments,
and adjunctive techniques.
PRT, originally termed strain–counterstrain,1 is a
therapeutic technique that uses tender points and a
position of comfort (POC) to resolve the associated
dysfunction. Essentially PRT is the opposite of stretch-
ing. For example, if a patient had a tight, tender area
on the calf, the clinician would dorsiflex the foot to
stretch the calf in an effort to reduce the tightness and
pain. Unfortunately, this might lead to muscle guarding
and increased pain. Using the same example, a clini-
cian who employs PRT would place the tender point
in the position of greatest comfort (plantar flexion),
shortening the muscle, in an effort to relax the tissues
and decrease the tender point.2-4
Dr. Lawrence H. Jones, an osteopathic physician,
was the first to publish a map of tender-point (TP) loca-
tions and their associated treatment positions.1 Jones
proposed that when a muscle is strained by a sudden
unexpected force, its antagonist attempts to stabilize
the joint, resulting in a counterstrain of the muscle in a
resting or shortened position. Before the antagonist is
counterstrained, gamma neural activity is heightened
as a result of its shortened position, making the spindle
more sensitive—propagating development of restric-
tion, sustained contraction, and TP development.1 The
application of PRT relaxes the muscle-spindle mecha-
THERAPEUTIC MODALITIES
Tim Speicher, MS, ATC, CSCS • Sacred Heart University
David O. Draper, EdD, ATC • Brigham Young University
© 2006 Human Kinetics · ATT 11(5), pp. 36-38
nism of the counterstrained tissue, decreasing afferent
gamma and alpha neuronal activity, thereby breaking
the sustained contraction.1 Jones’s original work and PRT
theory have been modified by several practitioners.5-10
The prevailing theory underlying PRT involves
placing tissues in a relaxed shortened state, or POC,
for a period of time (≈90 s) to decrease gamma gain
in order to facilitate restoration of normal tissue length
and tension.1,8-12 Simply put, PRT works to “unkink”
muscle and fascia much like one would a knotted
necklace, by gently twisting and pushing the tissues
together to take tension off the knot. When one link
in the chain is unkinked, others nearby untangle. For
example, when a dominant TP on the posterior tibialis
is treated, the release of an entire chain of TPs along
the length of the muscle can release. Apparently, once
the muscle spindle is unkinked, gamma activity and
neurochemical equilibrium are restored.1,5,11-14
A gentle and passive technique, PRT has been
advocated for the treatment of acute, subacute, and
chronic somatic (whole-body) dysfunction for all
ages.1,7-10 Formal courses are offered in PRT, which
are helpful in gaining competence more quickly, but
the technique is also covered in several texts7-10,15 that
enable self-study.
PRT is an ideal treatment for athletic therapists
to use because injuries with a specific mechanism
respond well to it. There are relatively few contraindi-
cations, including open wounds, sutures, healing frac-
tures, hematoma, hypersensitivity, systemic or local
infection, malignancy, aneurysm, acute rheumatoid
arthritis, and pain during treatment positioning. As a
precaution, monitor the vertebral artery for occlusion
during cervical positioning.
P
70 SEPTEMBER 2006 ATHLETIC THERAPY TODAY
General Treatment Rules for PRT
The following treatment rules should be followed for
PRT:
• Consider the root of the body’s dysfunction.
• Ensure patient and clinician comfort.
• Flex anterior structures; extend posterior struc-
tures.
• Treat dominant TPs first, then proximal, followed by
medial.
• Fasciculation at the TP is the strongest when in an
optimal POC.5
• Treatment should not cause pain.
• After 90 s, slowly release the POC to avoid reengaging
the myotatic reflex.
• 70–100% pain reduction is expected and desired
with the first treatment.
• Rest tissues for 24 hr before resuming vigorous activ-
ity.
• Use established treatment positions as guides—feel
for the POC.
Procedures for PRT
The procedure for applying PRT is as follows:
1. Palpate surrounding and opposing tissues to locate
dominant and other TPs.
2. Document TPs on a standardized scale (extremely
sensitive, very sensitive, moderately sensitive, no
tenderness).
3. Do not try to break up the TP with hard pres-
sure—only dimple the skin (≈1 kg of force).
4. Use one or two finger pads to monitor fasciculation
and TP.
5. Fine-tune position with rotation.
6. Hold the POC until fasciculation decreases signifi-
cantly or ceases.5
7. Average position hold time is 90 s to 3 min.
8. Transient periods of brief tingling, numbness, and
temperature changes might occur.
9. Treat dominant TP and three to five additional TPs
for one session.
10. Release tissue or joint slowly and reassess.
11. Continue with two or three treatments a week for
6 weeks (on rest days or after physical activity).
If desired pain relief is not attained, reposition and
try again. It is normal to experience muscle soreness up
to 48 hr after treatment as a result of fascial unwinding
of the tissue and release of proinflammatory chemical
mediators.8
Top 10
The sidebar lists the top 10 TPs most commonly treated
by athletic therapists. Figures 1–5 correspond to num-
bers six through ten in the top 10 list.
Clinical Implications
PRT is a valuable clinical tool for the treatment of
somatic dysfunction, but it is not a panacea. It is most
effective when integrated into an overall treatment
plan. Once tissue tension and length are restored and
pain is decreased, the muscle fibers can again func-
tion normally to aid healing. If a muscle is kinked
Top 10 Tender Points Treated by Athletic
Therapists
10. Biceps (Figure 1)
9. Intercostals (Figure 2)
8. Hip flexor (Figure 3)
7. Plantar fascia (Figure 4)
6. Trapezius (Figure 5)
5. Lumbar
4. Posterior tibialis
3. Cervical/Scapular
2. Iliotibial band5
1. Patellar tendon
Figure 1
Biceps. The patient is supine, shoulder abducted with elbow
flexed, dorsum of the hand rests on forehead. Fine-tune with shoulder
abduction or rotation.
ATHLETIC THERAPY TODAY SEPTEMBER 2006 71
for a period of time, weakness is likely to occur, so
strengthening is integral to rehabilitation.
References
1. Jones LH. Spontaneous release by positioning. D.O. 1964;Jan:109-
116.
2. Wang CK, Schauer C. Effect of strain counterstrain on pain and
strength in hip musculature. J Man Manipulative Ther. 2004;12(4):215-
223.
3. Alexander KM. Use of strain–counterstrain as an adjunct for treatment
of chronic lower abdominal pain. Phys Ther Case Rep. 1999;2(5):205-
208.
4. Flynn TW, Lewis C. The use of strain–counterstrain in the treatment
of patients with low back pain. J Man Manipulative Ther. 2001;9(2):92-
98.
5. Speicher TE. Positional release therapy techniques. Paper presented
at: Rocky Mountain Athletic Trainers’ Association Clinical Symposium;
April 22, 2006; Salt Lake City, Utah.
6. Speicher, TE. Positional release therapy techniques. Paper presented
at: National Athletic Trainers’ Association Annual Clinical Symposium;
June 15, 2005; Indianapolis, Ind.
7. Chaitow L. Positional Release Techniques. 2nd ed. London, UK: Churchill
Livingstone; 2002.
8. D’Ambrogio K, Roth, G. Positional Release Therapy: Assessment
and Treatment of Musculoskeletal Dysfunction. St Louis, Mo: Mosby;
1997.
9. Deig D. Positional Release Technique: From a Dynamic Perspective.
Boston, Mass: Butterworth & Heinemann; 2001.
10. Giammatteo T, Weiselfish-Giammatteo S. Integrative Manual Therapy:
For the Autonomic Nervous System and Related Disorders. Berkley, ■:
North Atlantic Books; 1998.
11. Korr IM. Proprioceptor and somatic dysfunction. J Am Osteopath Assoc.
1975;74:638-650.
12. Korr IM. The neural basis of the osteopathic lesion. J Am Osteopath
Assoc. 1947;48:191-198.
13. Bailey M, Dick L. Nociceptive considerations in treating with counter-
strain. J Am Osteopath Assoc. 1992;92(3):334, 337-341.
14. McPartland JM. Travell trigger points—molecular and osteopathic
perspectives. J Am Osteopath Assoc. 2004;104(6):244-249.
15. Jones L, Kusunose R, Goering E. Jones Strain Counterstrain. Boise,
Idaho: Jones Strain Counterstrain, Inc; 1995.
Tim Speicher is a clinical assistant professor at Sacred Heart University
in Fairfield, CT.
David Draper is a professor of athletic training and sports medicine in
the Department of Exercise Sciences at Brigham Young University.
Figure 2
Intercostals. The patient is seated in side position and rests
uninvolved side arm on athletic therapist’s knee. Trunk is flexed toward
tender-point side, rotation and flexion toward tender-point side, head
is toward tender-point side or resting on athletic therapist’s leg, unin-
volved arm hangs at side.
Figure 3
Hip flexor. Patient is supine, hips and knees flexed, ankles
crossed or uncrossed, therapist supported or with physioball. Vary
amounts of hip flexion, lateral flexion, and trunk flexion; move toward
or away from tender point.
Figure 4
Plantar fascia. Patient is prone with knee flexed to ~60°,
dorsum of foot on athletic therapist’s shoulder or knee, marked meta-
tarsal and ankle plantar flexion, calcaneus is compressed toward toes.
Move calcaneus into varus and valgus for fine-tuning.
Figure 5
Trapezius. Patient is supine with head laterally flexed toward
tender point, shoulder abducted to 90°. Shoulder flexion or extension
and rotation are used to fine-tun