BioMed Research International
superoxide anion in the reaction of reduced phenazine metho-
sulfate and molecular oxygen,” Biochemical and Biophysical
Research Communications, vol. , no. , pp. –, .
 D. C. Garratt, e Quantitative Analysis of Drugs,vol.,
Chapman and Hall, .
 C. Henriquez, C. Aliaga, and E. Lissi, “Formation and decay
of the ABTS derived radical cation: a comparison of dier-
ent preparation procedures,” International Journal of Chemical
 I. F. F. Benzie and J. J. Strain, “e ferric reducing ability of
plasma (FRAP) as a measure of ’antioxidant power’: e FRAP
assay,” Analytical Biochemistry,vol.,no.,pp.–,.
 O. P. Sharma and T. K. Bhat, “DPPH antioxidant assay revisited,”
 L. L. Stookey, “Ferrozine—a new spectrophotometric reagent
for iron,” Analytical Chemistry,vol.,no.,pp.–,.
 L. Wang, M. Bassiri, R. Naja et al., “Hypochlorous acid as
acid: a component of the inorganic armamentarium of innate
immunity,” Journal of Burns and Wounds,vol.,pp.–,.
 O. I. Aruoma and B. Halliwell, “Action of hypochlorous acid
on the antioxidant protective enzymes superoxide dismutase,
catalase and glutathione peroxidase,” Biochemical Journal,vol.
, no. , pp. –, .
 M. Prost, “Process for the determination by means of free
radicals of the antioxidant properties of a living organism or
a potentially aggressive age,” U.S. patent application ,
 United States Department of Agriculture and Agricultural
Research Service, “USDA National Nutrient Database for Stan-
dard Reference, Release ,” Nutrient Data Laboratory Home
 A. S. Levey, J. P. Bosch, J. B. Lewis, T. Greene, N. Rogers, and D.
Roth, “A more accurate method to estimate glomerular ltration
rate from serum creatinine: a new prediction equation,” Annals
of Internal Medicine,vol.,no.,pp.–,.
 C. S. Alisi and G. O. C. Onyeze, “Nitric oxide scavenging
ability of ethyl acetate fraction of methanolic leaf extracts of
Chromolaena odorata (Linn),” African Journal of Biochemistry
 R. J. Mahler and M. L. Adler, “Clinical review : type diabetes
mellitus: update on diagnosis, pathophysiology, and treatment,”
Journal of Clinical Endocrinology and Metabolism,vol.,no.,
pp. –, .
 F. ielecke and M. Boschmann, “e potential role of green
tea catechins in the prevention of the metabolic syndrome—a
 A. Luximon-Ramma, V. S. Neergheen, T. Bahorun et al.,
“Assessment of the polyphenolic composition of the organic
extracts of Mauritian black teas: a potential contributor to their
antioxidant functions,” BioFactors,vol.,no.–,pp.–,
 H. Raza and A. John, “In vitro protection of reactive oxy-
gen species-induced degradation of lipids, proteins and -
deoxyribose by tea catechins,” Food and Chemical Toxicology,
vol. , no. , pp. –, .
 L. N. Grinberg, H. Newmark, N. Kitrossky, E. Rahamim,
M. Chevion, and E. A. Rachmilewitz, “Protective eects of
tea polyphenols against oxidative damage to red blood cells,”
Biochemical Pharmacology, vol. , no. , pp. –, .
of green tea extract on obese women: a randomized, double-
blind, placebo-controlled clinical trial,” Clinical Nutrition,vol.
 S. I. Koo and S. K. Noh, “Green tea as inhibitor of the intestinal
absorption of lipids: potential mechanism for its lipid-lowering
eect,” Journal of Nutritional Biochemistry,vol.,no.,pp.–
extract rich in catechin polyphenols and caeine in increasing
-h energy expenditure and fat oxidation in humans,” Ameri-
can Journal of Clinical Nutrition,vol.,no.,pp.–,
 M. A. Potenza, F. L. Marasciulo, M. Tarquinio et al., “EGCG,
a green tea polyphenol, improves endothelial function and
insulin sensitivity, reduces blood pressure, and protects against
myocardial I/R injury in SHR,” American Journal of Physiology,
vol. , no. , pp. E–E, .
 Y. Huang, A. Zhang, C. Lau, and Z. Chen, “Vasorelaxant eects
of puried green tea epicatechin derivatives in rat mesenteric
artery,” Life Sciences, vol. , no. , pp. –, .
and Z. Y. Chen, “Involvement of endothelium/nitric oxide in
vasorelaxation induced by puried green tea (-)epicatechin,”
Biochimica et Biophysica Acta,vol.,no.,pp.–,.
eriault, “Green tea leaf extract improves lipid and glucose
homeostasis in a fructose-fed insulin-resistant hamster model,”
Journal of Ethnopharmacology,vol.,no.-,pp.–,.
 Y. Kobayashi, M. Suzuki, H. Satsu et al., “Green tea polyphenols
inhibit the sodium-dependent glucose transporter of intestinal
epithelial cells by a competitive mechanism,” Journal of Agricul-
tural and Food Chemistry, vol. , no. , pp. –, .
 H. Tsuneki, M. Ishizuka, M. Terasawa, J. Wu, T. Sasaoka, and I.
proteomic patterns in diabetic (db/db) mice and on glucose
metabolism in healthy humans,” BMC Pharmacology,vol.,
article , .
domized controlled trial for an eect of green tea consumption
on insulin resistance and inammation markers,” Journal of
 K. H. Van het Hof, H. S. M. De Boer, S. A. Wiseman, N. Lien, J.
A. Weststrate, and L. B. M. Tijburg, “Consumption of green or
black tea does not increase resistance of low-density lipoprotein
to oxidation in humans,” American Journal of Clinical Nutrition,
vol. , no. , pp. –, .
 S. Samman, B. Sandstr
or rosemary extract added to foods reduces nonheme-iron
absorption,” American Journal of Clinical Nutrition,vol.,no.
 E. Kim, S. Ham, M. K. Shigenaga, and O. Han, “Bioac-
tive dietary polyphenolic compounds reduce nonheme iron
transport across human intestinal cell monolayers,” Journal of
 D. N. Sarma, M. L. Barrett, M. L. Chavez et al., “Safety of green
tea extracts: a systematic review by the US Pharmacopeia,” Drug
 J. Frank, T. W. George, J. K. Lodge et al., “Daily consumption
of an aqueous green tea extract supplement does not impair