Psychopathy is a personality construct that has witnessed a surge in research interest since its initial conceptualization more than eight decades ago. Throughout this period, a prominent area of focus has been on socio-emotional deficits. Yet, the current body of research exhibits considerable heterogeneity, leaving conflicting results and multiple unanswered questions. In an effort to reconcile contradictory findings, explore socio-emotional deficits more holistically, and advance the conceptualization of psychopathy, this dissertation was conducted along with three studies.
The first study (Chapter 2: Study I) within this doctoral thesis is a comprehensive meta-analysis that synthesized the bulk of evidence concerning the relationship between psychopathy and both empathy and alexithymia. A total of 72 articles comprising more than 15,000 participants from 19 different countries were included in the analyses. The results suggest deficits not only in affective and cognitive empathy, but also in the understanding of one’s own emotions, i.e., alexithymia. Notably, the pooled correlations varied across psychopathy factors in terms of magnitude and direction, underscoring the importance of recognizing psychopathy as a multidimensional construct.
The second study (Chapter 3: Study II) explored whether alexithymia serves as a mediator in the relationship between psychopathy and empathy deficits, as well as between psychopathy and emotion dysregulation. This was tested across two diverse samples, one drawn from the general population and the other from a German forensic hospital. The results of our path analyses suggest that alexithymia contributes to the lack of empathy and emotion dysregulation in psychopathic individuals. However, these findings were again specific to certain psychopathy factors (i.e., meanness and disinhibition), with boldness indicating adaptive features by being linked to lower levels of alexithymia and fewer socio-emotional deficits overall.
In a final study (Chapter 4: Study III), we investigated the potential for overcoming psychopathy-related empathy deficits through explicit instructions to engage in affective perspective taking. To this end, we tested 87 participants from the community, using an experimental paradigm while simultaneously recording their physiological arousal. Although we observed a disconnect between behavioral and physiological measures of empathic concern, our results imply that empathy is not an automatic response in individuals with pronounced levels of meanness, leading to diminished empathic concern. Yet, this can be overcome when individuals are instructed to engage in affective perspective taking. Consequently, psychopathy does not appear to indicate a fundamental inability to empathize, but rather reflects a reduced propensity to do so automatically.
Taken together, the findings presented in this dissertation advance our comprehension of the various socio-emotional impairments in psychopathy. Specifically, four key conclusions can be drawn. Firstly, affective deficits in psychopathy are substantial, including not only impairments in sharing the emotions of others, but also in understanding one’s own feelings. Secondly, these deficits extend to cognitive empathy. Thirdly, empathic concern does not appear to be absent in individuals with psychopathy per se, but instead does not occur automatically, which may be due to a lack of motivation. Lastly, psychopathy is a multidimensional personality construct that must be viewed as a constellation of distinct traits in order to truly capture all its intricacies. As such, this dissertation offers significant implications for future research and clinical practice.