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Hair growth and the fluid factor

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Abstract

It is accepted that the detailed mechanisms of changes in human hair growth patterns are poorly understood. It is this uncertainty that has encouraged many charlatans to operate in this area. From my perspective as an engineer, however, there is a simple mechanism that makes sense of these changes. I suggest this mechanism is closely connected with the evolution and function of hair. The fact that the presence of this mechanism can be demonstrated in the male pattern baldness scenario, raises a number of serious questions relating to fundamental physiology and the mechanisms of some serious diseases. As an amateur, it is difficult for me to gain access to up-to-date research data, so my references are derived from textbooks. I would therefore welcome comments from professionals who are involved in the areas indicated in this paper.

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Article
Androgen-related changes in hair growth represent something of a mystery. Through the action of dihydrotestosterone (DHT), hair growth is increased in specific areas of the body. Elevated levels of DHT produce a general increase over the larger part of the body, often accompanied by hair loss in specific areas of the scalp. Because of this 'opposite' effect, a genetic difference in the hair follicles is proposed. This view is supported through the success of the 'plug graft' transplantation technique. However, this is unsatisfactory, because transplantation procedures that should work well according to this theory, ultimately fail. There is an alternative 'mechanism', that demonstrates its origins in the prime function of hair as an insulator. This simple mechanism makes sense of all the recognized effects of DHT in the dermal system, and throughout the body. In DHT-related hair growth it can be directly observed. The implication is that DHT achieves its effects through a primary physiological action that can be easily tested given the necessary expertise. Given existing knowledge, such a proven action of DHT would have serious implications for further understanding of female susceptibility to autoimmune disease.
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