Subacute sclerosing panencephalitis

Division of Neurology, Banaras Hindu University, Varanasi.
Indian pediatrics (Impact Factor: 1.04). 05/1998; 35(4):337-44.
Source: PubMed
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Available from: Ravindra Kumar Garg, Mar 29, 2015
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    • "The case is of particular relevance as annual prevalence of SSPE has been reported to be high at 21 cases per million people in some settings.[14] While the prevalence has been reported to be lower in developed countries, SSPE is of relevance to these countries as well in context of the migrated population. "
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    ABSTRACT: Catatonia is not a usual clinical presentation of subacute sclerosing panencephalitis (SSPE), especially in the initial stages of illness. However, there is only one reported case of SSPE presenting as catatonia among children. In this report, however, there were SSPE-specific changes on EEG and the catatonia failed to respond to lorazepam. We describe a case of SSPE in a child presenting as catatonia that presented with clinical features of catatonia and did not have typical EEG findings when assessed at first contact. He responded to lorazepam and EEG changes emerged during the course of follow-up.
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    ABSTRACT: An unusual case of panencephalitis in a 4-year-old Japanese boy, with onset at three months after measles infection and rapid progression to a comatose state in approximately one month, is described. A rapid rise in serum measles antibody titre after the onset of the symptoms, and the appearance of various abnormal antibodies in the serum, were noted. Pathologically, the brain showed sclerosing polio- and leucoencephalitis with diffuse gliosis and sporadic intranuclear inclusions. The process is suggested to be intermediate or transitional between acute measles encephalitis and SSPE.
    Preview · Article · Jun 1977 · Acta Neuropathologica
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    ABSTRACT: We present the unique case of a 2-year-old girl with congenital athyreosis who acquired primary measles virus infection at the age of 18 months, coincidentally with an Epstein-Barr virus infection. First neurologic symptoms of subacute sclerosing panencephalitis appeared 5 months later, and the girl died within 6 months after a rapid progressive illness. Factors possibly predisposing to this extraordinary disease course—primary measles virus infection at an early age and lack of evidence for immuno-deficiency—are discussed.
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