Wilson Tárraga

Wilson Tárraga
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Wilson verified their affiliation via an institutional email.
Verified
Wilson verified their affiliation via an institutional email.
  • Doctor of Philosophy
  • PostDoc Position at National Scientific and Technical Research Council

About

8
Publications
244
Reads
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12
Citations
Current institution
National Scientific and Technical Research Council
Current position
  • PostDoc Position
Additional affiliations
National Scientific and Technical Research Council
Position
  • PhD Student

Publications

Publications (8)
Article
Full-text available
Silica-binding peptides (SBPs) are increasingly recognized as versatile tools for various applications spanning biosensing, biocatalysis, and environmental remediation. This review explores the interaction between these peptides and silica surfaces, offering insights into how variables such as surface silanol density, peptide sequence and compositi...
Article
Full-text available
Safe implementation of nanotechnology-based products in biomedical applications necessitates an extensive understanding of the (bio)transformations that nanoparticles undergo in living organisms. The long-term fate in the body is a crucial...
Article
Full-text available
This article contains data for the self-association of pyrene-labelled single Cys-mutants of apolipoprotein A-I (apoA-I). Mathematical models were developed to characterize the self-association events at different cysteine positions on apoA-I obtained as a function of protein concentration based on the multi-parametric spectrum of pyrene, particula...
Article
ApoA-I is the main protein of HDL which has anti-atherogenic properties attributed to reverse cholesterol transport. It shares with other exchangeable apolipoproteins a high level of structural plasticity. In the lipid-free state, the apolipoprotein amphipathic α-helices interact intra- and inter-molecularly, providing structural stabilization by a...
Article
The cholesterol-ester transfer protein (CETP) exchanges lipids between high-density lipoproteins (HDLs) and low-density lipoproteins (LDLs). The excessive transport of lipids from HDLs to LDLs mediated by this protein can cause an alteration in the deposition of lipoproteins onto the arterial walls, thus promoting the development of arteriosclerosi...

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