Robert Patry

Robert Patry
University of Chicago | UC · Department of Pathology

About

7
Publications
176
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12
Citations

Publications

Publications (7)
Article
It is well known that accumulated changes in microbiota composition over several decades have contributed to an increase in non-communicable chronic diseases. While much effort has been devoted toward achieving a consensus on the microbes, and their products, driving this increase, inconsistent results suggest more research is required to understan...
Preprint
Full-text available
The increasing prevalence of food allergies has been causally associated with the depletion of allergy protective intestinal bacteria. However, few studies have investigated the role of the gut microbiota in promoting allergic responses. In a cohort of infants affected by cow’s milk allergy (CMA), we have identified a patient with a proinflammatory...
Article
Lifestyle-induced changes to the diversity of the commensal microbiota have been causally linked to the increasing prevalence of food allergies and other non-communicable chronic diseases. We have shown that bacteria from the Clostridia class prevent an allergic response to food by eliciting an IL-22 dependent barrier protective response that limit...
Article
Full-text available
A Correction to this paper has been published: https://doi.org/10.1038/s41590-021-00932-2.
Article
We previously reported that a consortium of bacteria from the Clostridia class modulates intestinal barrier function through induction of IL-22 and prevents allergic sensitization to food in mice. We further investigated the properties of these bacteria and the mechanisms that underly their protective impact. Shotgun metagenomic sequencing of the a...
Article
Full-text available
A Correction to this paper has been published: https://doi.org/10.1038/s41590-021-00907-3.
Article
The transition of Sub-Saharan Africans from rural settings and traditional plant-rich diets to urban settings and calorie-dense Westernized diets has led to specific shifts in serum metabolites and activates a heightened proinflammatory state in immune cells.

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