Nour H. Abdel-Hamid

Animal Health Research Institute · Department of Brucellosis Research (OIE Reference Lab)

I am working on a project regarding genotype variation of the B. melitensis isolates that have been recovered from small and large ruminants commingling together in the same place. The metadata of the MLVA-16 analysis, precisely bruce 4, 7, 9, and 16 (panel 2b), which have been sent from a certain lab, seems unlogic where different genotypes have been observed among the Brucella isolates recovered from different specimens of the same animal. Does it be logical? Did anyone face something like this?

Since all researches available to me said that creatine monohydrate has no significant effect on triglycerides or T3 and one of these researches showed decreases in T3 and increases in T4, where the authors suggested the causes stands behind that results , yet I didn’t find a potential mechanism responsible for the increase of triglycerides and T3 in sera of broiler chicks at 42nd day and if there is an explain please just attach the references related to that.

Precisely liver function test (AST, ALT, total bilirubin, direct and indirect bilirubin) kidney function test (urea and creatinine), uric acid, lipid profile (LDL, HDL, total cholesterol and triglyceride) and CBC