Gillian England-MasonThe University of Calgary | HBI · Department of Paediatrics
Gillian England-Mason
PhD
About
17
Publications
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162
Citations
Citations since 2017
Introduction
Additional affiliations
September 2018 - May 2020
Publications
Publications (17)
Background
Previous research reports associations between prenatal exposure to phthalates and childhood behavior problems; however, the neural mechanisms that may underlie these associations are relatively unexplored.
Objective
This study examined microstructural white matter as a possible mediator of the associations between prenatal phthalate ex...
Background
Environmental health research has reported mixed findings on the associations between prenatal exposure to phthalates and parent-ratings of child behavioral problems.
Objective
We examined the consistency of the associations between prenatal urinary phthalate concentrations and child behavior scores across two standardized instruments –...
Background:
Prenatal exposure to phthalates has been associated with adverse health and neurodevelopmental outcomes. DNA methylation (DNAm) alterations may be a mechanism underlying these effects, but prior investigations of prenatal exposure to phthalates and neonatal DNAm profiles are limited to placental tissue and umbilical cord blood.
Object...
Background:
Although emotion socialization parenting interventions are supported by a growing body of literature, their effects have yet to be systematically examined. The present systematic review and meta-analysis assesses the evidence for emotion socialization parenting interventions for parents of young children.
Methods:
Six electronic data...
BACKGROUND: Early bisphenol exposure may have consequences for executive function development, but less is known about potential sex effects. We hypothesized that early bisphenol A (BPA) and bisphenol S (BPS) exposures would be associated with sex-dependent changes in preschool executive function.
METHODS: A subsample of the Alberta Pregnancy Outc...
Folate and choline are methyl donor nutrients that may play a role in fetal brain development. Animal studies have reported that prenatal folate and choline supplementation are associated with better cognitive outcomes in offspring and that these nutrients may interact and affect brain development. Human studies that have investigated associations...
Background
Experiences of childhood maltreatment are associated with a variety of negative outcomes throughout individuals' lives as well as disadvantaged cognitive and socioemotional development among their offspring. The mechanisms through which some children show resilience against the intergenerational transmission of risk, however, are less we...
Women’s nutritional status during pregnancy can have long-term effects on children’s brains and cognitive development. Folate and choline are methyl-donor nutrients and are important for closure of the neural tube during fetal development. They have also been associated with brain and cognitive development in children. Animal studies have observed...
Exposure to adverse childhood experiences (ACEs) is associated with oxytocin dysregulation in women, such as decreased peripheral oxytocin concentrations, but little is known about vulnerability markers for oxytocin dysregulation in mothers exposed to ACEs. Identifying vulnerability markers may help inform future targets for prevention and interven...
Background
Maternal adverse childhood experiences (ACEs) are associated with difficulties in parent-child relationships; however, current research has primarily used cross-sectional designs. Parent and child behavior may be differentially affected by ACEs as children develop and caregiving demands change. Furthermore, diminished executive function...
Purpose of Review
Emotion regulation is recognized as a core underlying dimension common across psychiatric disorders and could be conceptualized as a transdiagnostic feature (i.e., mechanism underlying comorbidity). This review highlights recent research examining emotion regulation in children with attention-deficit/hyperactivity disorder (ADHD)...
Emotion regulation patterns are shaped, in part, by the quality of interpersonal interactions during early development. Given the important contribution of caregiver emotion socialization, parenting programs targeting these socialization practices represent an effective method of preventative intervention for childhood difficulties with emotion reg...
Objectives:
This study investigated associations between three types of child maltreatment (exposure to intimate partner violence, sexual, and physical abuse) and multimorbidity (chronic physical conditions, pain conditions, and mental disorders) in adults.
Methods:
Multinomial logistic regression was used to analyze weighted data from the 2012...
Child abuse and neglect can lead to difficulties regulating responses to threatening and emotional situations. Exposure to childhood maltreatment has been linked to conflicting findings of both attention biases toward and away from threat-related information. The aim of the current study was to investigate whether emotion regulation moderated the a...
Projects
Projects (4)
The first aim of this line of inquiry is to better understand vulnerability factors (e.g., emotion regulation) for hormonal dysregulation, mental health problems, and parenting difficulties. Through ongoing research, the second aim is to examine the associations between parent mental health, epigenetic modifications (e.g., DNA methylation alterations), and children's development.
The focus of this work is to identify different environmental factors, such as parenting practices (e.g., emotion coaching), that shape emotion regulation development and evaluate evidence-based interventions for promoting adaptive emotion regulation in young children.
The goal of these projects is to examine the associations between early environmental exposures, such as endocrine-disrupting chemicals (e.g., bisphenols, phthalates), and neurodevelopmental outcomes in young children. This line of inquiry also examines potential biological pathways (e.g., epigenetic modifications) that may mediate these associations.
