Do dietary components have any effects on FTO gene expression in blood cells?

I need to try both the manual method and extraction kit, thank you so much in advance.

Mostly spp. use for plural species and sp. for singular species. In manuscript some time people use spp for plural strains of same species. Can we use spp. for plural strain from single species??? 

I have induced type 2 diabetes mailitus using high fat diet (45% fat) plus multiple low dose of streptozotocin (30mg). So can any please tell me Which type insulin (Short, log, Intermidiate acting or Biphasic insuline) should be used to carry out Insulin Tolerance Test in type 2 diabetic rats?

I am interested in your experience and any advise you can give.

I have a number of items that I need converting from grams per day to serving per day. Indeed, how much is a serving per day potato as grams per day?

Geriatric population increases in the world day by day, most of them have different types of problems such as swallowing, indigestion, CAD, CVD, diabetes etc. due to all these problems there is a need to develop specific foods especially for older population (aged 60 and above). Can anyone recommend research papers relating to this research?

The major risk of overconsumption of a high fibre diet will lead to excessive intake of phytosterols that lead to neurodegeneration. Diets that are high in fibre in the developing world may also contain toxic xenobiotics that may lead to insulin resistance, NAFLD cardiovascular disease and brain damage.

Magnesium is important to lower cholesterol and maintain the peripheral clearance to toxic amyloid beta. Active lifestyles with exercise reduce magnesium levels with increase risk for Type 3 diabetes. Diets with magnesium supplementation may be important to increase fat metabolism in diabetes.

Dear colleagues, during a recent hypothesis-driven study of type 2 diabetes my research group confirmed our prediction that HbA1c and FBG levels are related to two different early pathophysiological subtypes of type 2 diabetes that may be partly overlapping in later stages. In this case HbA1c and FBG should be considered as independent predictors of different pathologies, but not as interchangeable alternatives as suggested by expert committees in diabetes. Outcome may be different protection management and protecting treatment against the disease development.

Editors of Diabetes and Diabetes Care considered that our finding "does not contain sufficient new or novel information" for sending our submission to external peer review. On behalf of my co-authors I myself would like to reach external peers and appeal for your opinion on this forum. I would appreciate very much if anybody on this forum could help us with a reference to similar finding (i.e., with respect to different pathophysiological subtypes associated with HbA1c and FBG), because I could not find such during my literature search.

FYI: Our pathophysiological subtypes are not among rare subtypes like MODY, LADA and hemochromatosis-related diabetes, as well as are not related to pathophysiological (insulinopenic, hyperinsulinemic, or classic) forms developing later in type 2 diabetes.

Unfortunately, I cannot say more about our findings, because do not lose hope to publish them. Your suggestions in a journal that does not use a modern editorial politics for restricting peer reviewing of submissions like in Diabetes and Diabetes Care are very appreciated.

Thank you.