Can anyone help me find sequence information for the HIN1 and HSR203j genes in Nicotiana Benthamiana?

Is there a way to analyse any possible inhibition effects of one pathogen on the other or possibly any other interactions between them ? e.g. one pathogen might just weaken the plant defenses without causing any symptoms, therefore facilitating easiest infection of the symptom-causing pathogen...

Can the hypersensitive response occur as part of PAMP-Triggered Immunity (PTI) as well as Effector-Triggered Immunity (ETI)?

What are the potential effects of on interactions between nonvectors and vectors on the spread of plant pathogens in managed and natural ecosystems?

Previous studies have shown that different herbivore and plant pathogen species elicit different hormone responses in plants.

Mediated by plant defenses, could herbivores and plant pathogens interact to affect the spread of the disease across landscapes?

Any thoughts?

Polyamine and ethylene biosynthesis.

Broadly, SA induces defense against biotrophic pathogens, whereas JA activates defense against necrotrophic pathogens. Are there any reports regarding the synergistic relation of salicylic and jasmonic acids in pathogen defense, especially against necrotrophs?

Many thanks in advance.

Are resistance (R) genes expressed in response to pathogen attack - or only defense genes - as part of the plant immune response? R proteins act as receptors to recognize pathogen effectors and an interaction between R protein and effector stimulates Effector Triggered Immunity (ETI) which itself involves the expression of defense related genes. However, are R genes also expressed as part of ETI?

Are plant pathogenesis-related protein genes, mlo genes, and SNARE protein defense genes classified as resistance (R) genes? Or do they fall into a different category?

Is BRASSINOSTEROID INSENSITIVE 1-Associated receptor Kinase 1 (BAK1) a plant resistance (R) gene?