European Child & Adolescent Psychiatry (EUR CHILD ADOLES PSY)

Publisher: European Society of Child and Adolescent Psychiatry, Springer Verlag

Journal description

European Child and Adolescent Psychiatry is Europe's only peer-reviewed journal entirely devoted to child and adolescent psychiatry. It aims to further a broad understanding of psychopathology in children and adolescents. Empirical research is its foundation and clinical relevance is its hallmark. Contributions are encouraged from around the world. All papers will be reviewed by at least three expert referees. Indexing and Abstract Services - Applied Social Sciences Index & Abstracts (ASSIA) - Cumulative Index to Nursing & Allied Health Literature (CINAHL) - Excerpta Medica Embase - Index Medicus - Institute for Scientific Information (ISI) - Current Contents/Social & Behavioral Sciences - Research Alert - Social Sciences Citation Index (SSCI) - Social SciSearch - Medline Database - Mental Health Abstracts - PsycINFO and PsycLIT (APA - American Psychological Association) - Sociological Abstracts

Current impact factor: 3.34

Impact Factor Rankings

2016 Impact Factor Available summer 2017
2014 / 2015 Impact Factor 3.336
2013 Impact Factor 3.554
2012 Impact Factor 3.699
2011 Impact Factor 2.821
2010 Impact Factor 1.622
2009 Impact Factor 1.651

Impact factor over time

Impact factor
Year

Additional details

5-year impact 3.74
Cited half-life 6.50
Immediacy index 0.63
Eigenfactor 0.01
Article influence 1.13
Website European Child & Adolescent Psychiatry website
Other titles European child & adolescent psychiatry (Online), European child and adolescent psychiatry
ISSN 1018-8827
OCLC 42809473
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

Springer Verlag

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Author's pre-print on pre-print servers such as arXiv.org
    • Author's post-print on author's personal website immediately
    • Author's post-print on any open access repository after 12 months after publication
    • Publisher's version/PDF cannot be used
    • Published source must be acknowledged
    • Must link to publisher version
    • Set phrase to accompany link to published version (see policy)
    • Articles in some journals can be made Open Access on payment of additional charge
  • Classification
    green

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Parents may develop symptoms of distress and dysfunctional cognitions in response to their child's exposure to traumatic events. Additionally, they may also be affected by their own traumatic experiences. This study investigated the frequency of traumatic experiences and of symptoms of posttraumatic stress and depression in a sample of parents of children and adolescents with posttraumatic stress disorder (PTSD). Furthermore, we explored the association of parental symptoms with their dysfunctional cognitions related to their child's trauma. Parents (N = 113) of children and adolescents with PTSD completed the Posttraumatic Diagnostic Scale (PDS), the Beck depression inventory (BDI-II), the State-Trait Anxiety Inventory, and the Posttraumatic Cognitions Inventory. Correlations between symptom measures and dysfunctional cognitions were calculated. The majority (78.8 %) of the parents reported their own potentially traumatic experiences. Furthermore, 33.6 % evaluated their child's trauma as the worst event, 34.5 % rated their own experiences as their worst event, and 26.5 % indicated that their own worst traumatic event was the same type as their child's trauma. The frequency of clinically elevated parental symptoms on the PDS was 48.6 %, and on the BDI-II 32.7 %. Parental symptoms were independent of the reference person of the parental traumatic index event. However, they did correlate significantly with their dysfunctional cognitions (between r = 0.44 and 0.69, p < 0.01). Many parents report their own traumatic experiences and a significant proportion has its own clinically relevant symptoms of distress. Parental psychological symptoms are moderately associated with their dysfunctional cognitions. The results emphasize the need to consider parental distress when treating pediatric PTSD.
    No preview · Article · Feb 2016 · European Child & Adolescent Psychiatry
  • [Show abstract] [Hide abstract]
    ABSTRACT: Children of parents diagnosed with bipolar disorder (BD), termed high-risk offspring (HRO), are at greater risk of developing psychiatric disorders compared to healthy children of healthy parents (HCO). Gray matter volume (GMV) abnormalities have been observed in HRO, however, these reports are inconsistent. We posit that this variability may be attributed to differences in methodology among offspring studies; in particular, the presence of psychiatric symptoms in HRO. Here, we directly compared GMVs between symptomatic and asymptomatic HRO, and HCO. High-resolution T1-weighted MR images were collected from 31 HRO (18 symptomatic and 13 asymptomatic) and 20 age- and sex-matched HCO. HRO had at least one parent diagnosed with BD. Symptomatic HRO were defined as having a psychiatric diagnosis other than BD, while asymptomatic HRO were required to be free of any psychiatric diagnosis. Scans were processed using voxel-based morphometry methods and between group analyses were performed in SPM. Compared to HCO, the HRO group showed decreased GMV in the right inferior orbitofrontal, right middle frontal, and bilateral superior and middle temporal regions. Both symptomatic and asymptomatic HRO groups showed decreased GMV in these regions separately when compared to HCO. When comparing symptomatic and asymptomatic HRO, GMVs were comparable in all regions except the lateral occipital cortex. Our study compared symptomatic and asymptomatic HRO directly. In doing so, we provided further support for the presence of discrete GMV deficits in HRO, and confirmed that these deficits are present irrespective of the presence of symptoms in HRO.
    No preview · Article · Jan 2016 · European Child & Adolescent Psychiatry
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    ABSTRACT: We aimed to assess the prevalence, incidence, age-of-onset and diagnostic stability of threshold and subthreshold anorexia nervosa (AN) and bulimia nervosa (BN) in the community. Data come from a prospective-longitudinal community study of 3021 subjects aged 14-24 at baseline, who were followed up at three assessment waves over 10 years. Eating disorder (ED) symptomatology was assessed with the DSM-IV/M-CIDI at each wave. Diagnostic stability was defined as the proportion of individuals still affected with at least symptomatic eating disorders (EDs) at follow-ups. Baseline lifetime prevalence for any threshold ED were 2.9 % among females and 0.1 % among males. For any subthreshold ED lifetime prevalence were 2.2 % for females and 0.7 % for males. Symptomatic expressions of EDs (including core symptoms of the respective disorder) were most common with a lifetime prevalence of 11.5 % among females and 1.8 % among males. Symptomatic AN showed the earliest onset with a considerable proportion of cases emerging in childhood. 47 % of initial threshold AN cases and 42 % of initial threshold BN cases showed at least symptomatic expressions of any ED at any follow-up assessment. Stability for subthreshold EDs and symptomatic expressions was 14-36 %. While threshold EDs are rare, ED symptomatology is common particularly in female adolescents and young women. Especially threshold EDs are associated with a substantial risk for stability. A considerable degree of symptom fluctuation is characteristic especially for subthreshold EDs.
    No preview · Article · Jan 2016 · European Child & Adolescent Psychiatry

  • No preview · Article · Dec 2015 · European Child & Adolescent Psychiatry
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    ABSTRACT: The author of this correspondence has recently published findings of an ecological investigation wherein herbicide use was proposed as an instrumental variable that predicts healthcare utilization among subjects with severe ADHD impairment via individual exposure to nitrous oxide (N2O) emissions. Vitamin D deficiency, a possible risk factor for autism spectrum disorders (ASD) as discussed by Wang et al., may be a homeostatic response to increasing chronic environmental N2O exposures. The author explains how deficient and insufficient states of vitamin D may promote vagal withdrawal and tolerance to increasing opioid exposures in the environment and how these characteristics are particularly relevant in neurodevelopmental disorders, like ASD and ADHD.
    No preview · Article · Dec 2015 · European Child & Adolescent Psychiatry

  • No preview · Article · Nov 2015 · European Child & Adolescent Psychiatry