Archives of Oral Biology (ARCH ORAL BIOL)

Publisher: European Organization for Research on Fluorine and Dental Caries Prevention, Elsevier

Journal description

The Archives of Oral Biology publishes papers concerned with advances in knowledge of every aspect of the oral and dental tissues and bone over the whole range of vertebrates, whether from the standpoint of anatomy, bacteriology, biophysics, chemistry, DNA biotechnology, epidemiology, genetics, immunology, molecular biology, palaentology, pathology, physiology or otherwise.

Current impact factor: 1.74

Impact Factor Rankings

2016 Impact Factor Available summer 2017
2014 / 2015 Impact Factor 1.735
2013 Impact Factor 1.88
2012 Impact Factor 1.549
2011 Impact Factor 1.603
2010 Impact Factor 1.463
2009 Impact Factor 1.649
2008 Impact Factor 1.379
2007 Impact Factor 1.554
2006 Impact Factor 1.655
2005 Impact Factor 1.288
2004 Impact Factor 1.158
2003 Impact Factor 1.098
2002 Impact Factor 1.047
2001 Impact Factor 0.973
2000 Impact Factor 0.845
1999 Impact Factor 1.04
1998 Impact Factor 0.938
1997 Impact Factor 0.735
1996 Impact Factor 0.84
1995 Impact Factor 0.967
1994 Impact Factor 0.959
1993 Impact Factor 0.972
1992 Impact Factor 1.034

Impact factor over time

Impact factor
Year

Additional details

5-year impact 1.89
Cited half-life >10.0
Immediacy index 0.25
Eigenfactor 0.01
Article influence 0.49
Website Archives of Oral Biology website
Other titles Archives of oral biology
ISSN 0003-9969
OCLC 2484813
Material type Periodical, Internet resource
Document type Journal / Magazine / Newspaper, Internet Resource

Publisher details

Elsevier

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Authors pre-print on any website, including arXiv and RePEC
    • Author's post-print on author's personal website immediately
    • Author's post-print on open access repository after an embargo period of between 12 months and 48 months
    • Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months
    • Author's post-print may be used to update arXiv and RepEC
    • Publisher's version/PDF cannot be used
    • Must link to publisher version with DOI
    • Author's post-print must be released with a Creative Commons Attribution Non-Commercial No Derivatives License
    • Publisher last reviewed on 03/06/2015
  • Classification
    green

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Objectives: Considering the evident relationship between periodontitis and cardiovascular diseases in humans, we aimed to study the in vitro vascular reactivity of aorta rings prepared from rats with ligature-induced periodontitis. Methods: Seven days after the induction of unilateral periodontitis, the animals were euthanised; rings were prepared from the descending abdominal aortas and mounted in tissue baths for the in vitro measurement of the isometric force responses to norepinephrine (NE) and acetylcholine (ACh), as well as in the presence of inhibitors of nitric oxide synthase (NOS) and cycloxygenase (COX) isoenzymes. Aortic COX and NOS gene expressions were analysed by RT-PCR, as well as protein COX-2 expression by Western blot. Results: Periodontitis resulted in significant alveolar bone loss and did not affect arterial pressure. However, both NE-induced contraction and ACh-induced relaxation were significantly decreased and related to the presence of endothelium. Diminished eNOS and augmented COX-2 and iNOS expressions were found in the aortas from rats with periodontitis, and the pharmacological inhibition of COX-2 or iNOS improved the observed vasomotor deficiencies. Conclusions: We can thus conclude that periodontitis induces significant endothelial dysfunction in rat aorta which is characterized by decreased eNOS expression and mediated by upregulated iNOS and COX-2 products.
    No preview · Article · Dec 2015 · Archives of Oral Biology
  • [Show abstract] [Hide abstract]
    ABSTRACT: BMP-2 induces osteoblast differentiation and activates osteoclast formation. Here, we investigated the role of Smad1, a molecule that signals downstream of BMP-2, in mediating the effects of BMP-2 on osteoclast differentiation induced by 1,25-dihydroxyvitamin D3 (1,25(OH)2D3) in osteoblasts. The effects of 1,25(OH)2D3 and BMP-2 in osteoclasts were examined using polymerase chain reaction and Western blotting to measure changes in target gene and protein expression. Immunostaining was carried out to investigate the localization of the vitamin D receptor (VDR) in the nucleus in response to BMP-2. Stimulation with both 1,25(OH)2D3 and BMP-2 resulted in significantly greater osteoclast formation and receptor activator of nuclear factor κB ligand (RANKL) mRNA expression compared to stimulation with 1,25(OH)2D3 alone. In addition, expression of the VDR protein was increased, enhancing the activity of 1,25(OH)2D3. Interestingly, knockdown of Smad1 resulted in reduced osteoclast formation, RANKL mRNA expression, and VDR protein expression compared with control cells. Costimulation with 1,25(OH)2D3 and BMP-2 enhanced VDR localization in the nucleus. We found that BMP-2 induced Smad1 activation, thereby influencing the localization of VDR in the nucleus in the presence of 1,25(OH)2D3 and resulting in increased RANKL mRNA expression. These effects ultimately resulted in enhanced osteoclast differentiation. Copyright © 2015 Elsevier Ltd. All rights reserved.
    No preview · Article · Jun 2015 · Archives of Oral Biology
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    ABSTRACT: To evaluate the in vitro effectiveness of APDI with a 660nm laser combined with methylene blue (MB), toluidine blue ortho (TBO) and malachite green (MG) dyes to inactivate Staphylococcus aureus (ATCC 25923) biofilms in compact and cancellous bone specimens. Eighty specimens of compact and 80 of cancellous bone were contaminated with a standard suspension of the microorganism and incubated for 14 days at 37°C to form biofilms. After this period, the specimens were divided into groups (n=10) according to established treatment: PS-L- (control - no treatment); PSmb+L-, PStbo+L-, PSmg+L- (only MB, TBO or MG for 5min in the dark); PS-L+ (only laser irradiation for 180s); and APDImb, APDItbo and APDImg (APDI with MB, TBO or MG for 180s). The findings were statistically analyzed by ANOVA at 5% significance levels. All experimental treatments showed significant reduction of logCFU/mL S. aureus biofilms when compared with the control group for compact and cancellous bones specimens; the APDI group's treatment was more effective. The APDI carried out for the compact specimens showed better results when compared with cancellous specimens at all times of application. For the group of compact bone, APDImg showed greater reductions in CFU/mL (4.46log10). In the group of cancellous bone, the greatest reductions were found in the APDImb group (3.06log10). APDI with methylene blue, toluidine blue ortho and malachite green dyes and a 660nm laser proved to be effective in the inactivation of S. aureus biofilms formed in compact and cancellous bone. Copyright © 2015. Published by Elsevier Ltd.
    No preview · Article · May 2015 · Archives of Oral Biology