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Energy production within the mitochondria. NAD/H, nicotinamide adenine dinucleotide (reduced); FADH2, flavin adenine dinucleotide (reduced); complex I (NADH dehydrogenase); complex II (succinate dehydrogenase); complex III (cytochrome c reductase); complex IV (cytochrome-c oxidase); CoA, coenzyme A (Forbes & Cooper, 2013).

Energy production within the mitochondria. NAD/H, nicotinamide adenine dinucleotide (reduced); FADH2, flavin adenine dinucleotide (reduced); complex I (NADH dehydrogenase); complex II (succinate dehydrogenase); complex III (cytochrome c reductase); complex IV (cytochrome-c oxidase); CoA, coenzyme A (Forbes & Cooper, 2013).

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... the neuro-protective role played by autophagy in DCAD may be limited to the early stage of the disease (Wu et al., 2019). In addition, the impairment of hippocampal autophagic flux causes cognitive impairment, and the restoration of it may be neuroprotective (Kang et al., 2020). ...
Context 2
... the neuro-protective role played by autophagy in DCAD may be limited to the early stage of the disease (Wu et al., 2019). In addition, the impairment of hippocampal autophagic flux causes cognitive impairment, and the restoration of it may be neuroprotective (Kang et al., 2020). ...