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Effect of L2 on cGMP release. cGMP release was measured in the coronary effluent 5 min after BNP (10 nM) or L2 (10, 100 or 200 nM) perfusion under normoxic conditions. Data are mean ± SEM. n = 8-14 per group. **, p<0.01, ***, p<0.001 versus control (vehicle). doi:10.1371/journal.pone.0162632.g002
Contexts in source publication
Context 1
... in coronary cGMP release after BNP or L2 perfusion are shown in Fig 2. BNP (10 nM) significantly increased cGMP release in coronary effluent within 5 min compared to control group (17.4 ± 0.1 pmol/μl versus 15.9 ± 0.2 pmol/μl for control group, p<0.01). L2 also increased cGMP to the same extent at 100 and 200 nM (17.9 ± 0.3 pmol/μl and 18.1 ± 0.3 pmol/μl respectively, p<0.001 versus control group) but not at 10 nM. ...
Context 2
... Fig. Effect of L2 on phosphorylated PI3K expression in the ischemic myocardium. Phosphorylation of PI3K was studied after 30 min regional ischemia followed by 20 min reperfusion, in sham operated hearts and in hearts subjected to ischemia-reperfusion and exposed to either vehicle (control), L2 (200 nM) or BNP (10 nM) perfusion, starting 5 min before ...
Context 3
... ERK1/2 was studied after 30 min regional ischemia followed by 20 min reperfusion, in sham operated hearts and in hearts subjected to ischemia-reperfusion and exposed to either vehicle (control), L2 (200 nM) or BNP (10 nM) perfusion, starting 5 min before reperfusion and maintained for 20 min. Tubulin served as molecular weight marker. (TIFF) S6 Fig. Effect of L2 on phosphorylated GSK3β expression in the ischemic myocardium. Phosphorylation of GSK3β was studied after 30 min regional ischemia followed by 20 min reperfusion, in sham operated hearts and in hearts subjected to ischemia-reperfusion and exposed to either vehicle (control), L2 (200 nM) or BNP (10 nM) perfusion, starting 5 min before ...
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