Yan Wang

Northwest University, China, Maine, United States

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Publications (442)1087.72 Total impact

  • [Show abstract] [Hide abstract]
    ABSTRACT: To meet the needs of future multilayer ceramic capacitors (MLCCs), thinner dielectric layers are necessary. To achieve this goal, the grain size and uniformity of the MLCC particles must be effectively controlled. In this study, we developed a novel precipitation route to control the dispersion and particle size of Ba0.991Bi0.006TiO3 and Ba0.991Bi0.006TiO3@Nb2O5 particles. We confirmed a core–shell particle structure by means of X-ray diffraction, scanning electron microscopy, and energy-dispersive spectroscopy. The dielectric properties of the ceramics were measured using an LCR meter. We found monodispersed submicron Ba0.991Bi0.006TiO3 particles (∼233 nm) form a core that was coated with a homogeneous Nb2O5 layer (∼7 nm). Dense, fine-grained Ba0.991Bi0.006TiO3-based ceramics (≤250 nm) were obtained by sintering. The ceramics met the X8R requirements, with a Curie temperature of about 140 °C, a maximum dielectric constant of 2336, and a low dielectric loss at room temperature (<2.0%, with a minimum of 0.5%).
    Journal of the European Ceramic Society 09/2015; 35(9). DOI:10.1016/j.jeurceramsoc.2015.03.024 · 2.31 Impact Factor
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    ABSTRACT: We present an efficient approach for the sub-regional multiple-source detection. The essence of this approach is to extract the characteristic components of the signals of interest (SOIs) from the estimated signal-plus-interference subspace by a matrix filter. Compared with some other spatial filtering-based approaches, it has two significant advantages. First, since the power of each source in the signal-plus-interference subspace is normalized, the proposed approach is effective to filter out interferences regardless of their strengths. Second, the matrix filter would not reduce the dimension of SOIs, thus, the proposed approach is able to distinguish multiple SOIs from the output of the matrix filter.
    IEEE Signal Processing Letters 06/2015; 22(7). DOI:10.1109/LSP.2014.2379619 · 1.64 Impact Factor
  • Asian Australasian Journal of Animal Sciences 06/2015; 28(6):782-787. DOI:10.5713/ajas.14.0753 · 0.56 Impact Factor
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    ABSTRACT: A convenient one-pot synthesis of 2-arylbenzofuran-3-carboxylic acids from (E)-2-(2-bromophenyl)-3-phenylacrylic acids via Cu-catalyzed consecutive hydroxylation and aerobic oxidative cycloetherification under microwave conditions has been developed. This protocol employed the reagent combination of Cu(OAc)2, 1,10-phen, and KOH in DMSO/H2O (1:1), all of which are cost-effective, readily available, and easily removable from the reaction mixture. Utilizing this synthetic protocol, various 2-arylbenzofuran-3-carboxylic acids as well as the natural product Moracin M have been synthesized in satisfactory yields under mild conditions.
    The Journal of Organic Chemistry 04/2015; DOI:10.1021/jo502802k · 4.64 Impact Factor
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    ABSTRACT: In order to detect cracks in railroad tracks, various experiments have been examined by Acoustic Emission (AE) method. However, little work has been done on studying rail defect detection at high speed. This paper presents a study on AE detection of rail defect at high speed based on rail–wheel test rig. Meanwhile, Wavelet Transform and Shannon entropy are employed to detect defects. Signals with and without defects are acquired, and characteristic frequencies from them at different speeds are analyzed. Based on appropriate decomposition level and Energy-to-Shannon entropy ratio, the optimal wavelet is selected. In order to suppress noise effects and ensure appropriate time resolution, the length of time window is investigated. Further, the characteristic frequency of time window is employed to detect defect. The results clearly illustrate that the proposed method can detect rail defect at high speed effectively.
    Journal of Sound and Vibration 03/2015; 339. DOI:10.1016/j.jsv.2014.11.021 · 1.86 Impact Factor
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    ABSTRACT: Inhibitor of growth 4 (ING4) is a tumor suppressor. However the role of ING4 in human bladder malignancy is unknown. In this study, ING4 expression in human bladder cancer and its potential effects were studied. ING4 expression in 47 human bladder cancer tissues and paired adjacent normal tissues was detected by Western blotting, quantitative reverse transcription-polymerase chain reaction, and immunohistochemistry. The migration and cell cycle progression of SV-HUC-1 and T24 cells with aberrant ING4 expression were examined. ING4 protein and mRNA were significantly decreased in bladder cancer tissues. ING4 protein level was significantly lower in the group of patients over 50 years of age. ING4 knockdown caused more rapid cell migration and increased the population of SV-HUC-1 and T24 cells in the G2-M phase. Our data suggest a close connection between aberrant ING4 expression and the carcinogenesis of human bladder cells. ING4 may be a potential target for bladder cancer chemotherapy. © 2015 S. Karger AG, Basel.
    Urologia Internationalis 03/2015; DOI:10.1159/000364832 · 1.15 Impact Factor
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    ABSTRACT: Plasminogen activator inhibitor-1 (PAI-1) plays a key role in tissue remodeling and tumor development by suppression of plasminogen activator function. Glucocorticoids (GCs) and transforming growth factor beta (TGF-β) signal pathways cross-talk to regulate gene expression, but the mechanism is poorly understood. Here we investigated the mechanism and significance of co-regulation of PAI-1 by TGF-β and dexamethasone (DEX), a synthetic glucocorticoid in ovarian cancer cells. We found TGF-β and DEX showed rapidly synergistic induction of PAI-1 expression, which contributed to the early pro-adhesion effects. The synergistic induction effect was accomplished by several signal pathways, including GC receptor (GR) pathway and TGF-β-activated p38MAPK, ERK and Smad3 pathways. TGF-β-activated p38MAPK and ERK pathways cross-talked with GR pathway to augment the expression of PAI-1 through enhancing DEX-induced GR phosphorylation at Ser211 in ovarian cancer cells. These findings reveal possible novel mechanisms by which TGF-β pathways cooperatively cross-talk with GR pathway to regulate gene expression. Copyright © 2015. Published by Elsevier Ireland Ltd.
    Molecular and Cellular Endocrinology 03/2015; DOI:10.1016/j.mce.2015.03.005 · 4.24 Impact Factor
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    ABSTRACT: High-grade serous ovarian cancer (HGSC) is among the most lethal forms of cancer in women. Excessive genomic rearrangements, which are expected to create fusion oncogenes, are the hallmark of this cancer. Here we report a cancer-specific gene fusion between BCAM, a membrane adhesion molecule, and AKT2, a key kinase in the PI3K signaling pathway. This fusion is present in 7% of the 60 patient cancers tested, a significant frequency considering the highly heterogeneous nature of this malignancy. Further, we provide direct evidence that BCAM-AKT2 is translated into an in-frame fusion protein in the patient's tumor. The resulting AKT2 fusion kinase is membrane-associated, constitutively phosphorylated, and activated as a functional kinase in cells. Unlike endogenous AKT2, whose activity is tightly regulated by external stimuli, BCAM-AKT2 escapes the regulation from external stimuli. Moreover, a BCAM-AKT2 fusion gene generated via chromosomal translocation using the CRISPR/Cas9 system leads to focus formation in both OVCAR8 and HEK-293T cell lines, suggesting that BCAM-AKT2 is oncogenic. Together, the results indicate that BCAM-AKT2 expression is a new mechanism of AKT2 kinase activation in HGSC. BCAM-AKT2 is the only fusion gene in HGSC that is proven to translate an aberrant yet functional kinase fusion protein with oncogenic properties. This recurrent genomic alteration is a potential therapeutic target and marker of a clinically relevant subtype for tailored therapy of HGSC.
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    ABSTRACT: Candida albicans is both a commensal microorganism in healthy individuals and a major fungal pathogen causing high mortality in immunocompromised patients. Yeast-hypha morphological transition is a well known virulence trait of C. albicans. Host innate immunity to C. albicans critically requires pattern recognition receptors (PRRs). In this review, we summarize the PRRs involved in the recognition of C. albicans in epithelial cells, endothelial cells, and phagocytic cells separately. We figure out the differential recognition of yeasts and hyphae, the findings on PRR-deficient mice, and the discoveries on human PRR-related single nucleotide polymorphisms (SNPs).
    Virulence 02/2015; DOI:10.1080/21505594.2015.1014270 · 3.32 Impact Factor
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    ABSTRACT: One of the challenging problems in drug discovery is to identify the novel targets for drugs. Most of the traditional methods for drug targets optimization focused on identifying the particular families of "druggable targets", but ignored their topological properties based on the biological pathways. In this study, we characterized the topological properties of human anticancer drug targets (ADTs) in the context of biological pathways. We found that the ADTs tended to present the following seven topological properties: influence the number of the pathways related to cancer, be localized at the start or end of the pathways, interact with cancer related genes, exhibit higher connectivity, vulnerability, betweenness, and closeness than other genes. We first ranked ADTs based on their topological property values respectively, then fused them into one global-rank using the joint cumulative distribution of an N-dimensional order statistic to optimize human ADTs. We applied the optimization method to 13 anticancer drugs, respectively. Results demonstrated that over 70% of known ADTs were ranked in the top 20%. Furthermore, the performance for mercaptopurine was significant: 6 known targets (ADSL, GMPR2, GMPR, HPRT1, AMPD3, AMPD2) were ranked in the top 15 and other four out of the top 15 (MAT2A, CDKN1A, AREG, JUN) have the potentialities to become new targets for cancer therapy. Copyright © 2015. Published by Elsevier Inc.
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    ABSTRACT: The MyoD1, MyoG, Myf5, and Mrf4 proteins belong to the family of muscle regulatory factors (MRFs) and play important roles in skeletal muscle hyperplasia and hypertrophy. We hypothesized that exercise would affect MRFs mRNA and protein abundance in postnatal chicken skeletal muscle driving molecular changes that could ultimately lead to increased muscle fiber diameter. At day (d) 43, twelve hundred chickens with similar body weight were randomly assigned to cage, pen, and free-range groups. The MRFs mRNA abundance was measured in pectoralis major and thigh muscle at d56, d70, and d84, and the protein levels of MRFs were determined from the thigh muscle at d84. The results showed no significant difference in mRNA of the MRFs among the three groups at d56 (P>0.05). At d84, chicken in the pen and free-range group showed higher MyoD1, MyoG, Myf5, and Mrf4 mRNA abundance compared to the caged chickens (P<0.05). Free-range chickens had higher Mrf4 and MyoG expression than those in penned ones (P<0.05). Protein abundances of all four factors were lowest in the caged group, and Mrf4 and MyoG protein quantities were greatest in free-range chickens (P<0.05), but Myf5 and MyoD1 protein abundance did not differ between penned and caged groups. The results suggested that exercise up-regulated MRFs expression in the postnatal skeletal muscles, which led to an increase in muscle fiber diameter, and eventually affected the meat quality of the skeletal muscles in adult chickens. Copyright © 2015. Published by Elsevier B.V.
    Gene 02/2015; 561(2). DOI:10.1016/j.gene.2015.02.044 · 2.08 Impact Factor
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    ABSTRACT: Background/Aims: Although it has been widely accepted that Enterovirus 71 (EV71) enters permissive cells via receptor-mediated endocytosis, the details of entry mechanism for EV71 still need more exploration. This study aimed to investigate the role of lipid rafts in the early stage of EV71 Infection. Methods: The effect of cholesterol depletion or addition of exogenous cholesterol was detected by immunofluorescence assays and quantitative real-time PCR. Effects of cholesterol depletion on the association of EV71 with lipid rafts were determined by flow cytometry and co-immunoprecipitation assays. Localization and internalization of EV71 and its receptor were assayed by confocal microscpoy and sucrose gradient analysis. The impact of cholesterol on the activation of phosphoinositide 3'-kinase/Akt signaling pathway during initial virus infection was analyzed by Western-blotting. Results: Disruption of membrane cholesterol by a pharmacological agent resulted in a significant reduction in the infectivity of EV71. The inhibitory effect could be reversed by the addition of exogenous cholesterol. Cholesterol depletion post-infection did not affect EV71 infection. While virus bound equally to cholesterol-depleted cells, EV71 particles failed to be internalized by cholesterol-depleted cells. EV71 capsid protein co-localized with cholera toxin B, a lipid-raft-dependent internalization marker. Conclusion: Lipid rafts play a critical role in virus endocytosis and in the activation of PI3K/Akt signaling pathway in the early stage of EV71 infection. © 2015 S. Karger AG, Basel.
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    ABSTRACT: The sex-determining region Y-box 4 (SOX4), a transcription factor, is involved in various developmental processes. It has been reported in multiple human cancers. However, the prognostic value and its exact role in chondrosarcoma remain poorly understood. In the current study, SOX4 was overexpressed in 28 of 92 (30.4 %) interpretable chondrosarcoma patients compared with 3 of 43 (6.9 %) interpretable chondroma cases (P = 0.003). Its overexpression in chondrosarcoma was significantly associated with histological grade (P < 0.001) and the presence of tumor recurrence (P = 0.041). In addition, SOX4 overexpression was notably correlated with c-MYC (P = 0.011) and P53 (P = 0.029) expression as well as high Ki67 labeling index (LI) (P < 0.001) in our cohort. More importantly, we found that SOX4 was an unfavorable independent prognostic factor for chondrosarcoma patients with low histological grade. Functionally, SOX4 silencing significantly suppressed the proliferation, migratory, and invasive capacity of SW1353 cells, suggesting an oncogenic role of SOX4 in chondrosarcoma in vitro. In an attempt of characterizing SOX4 overexpression mechanism, we identified miR-30a as a tumor suppressor that directly targets SOX4 in chondrosarcoma cells. Clinically, miR-30a expression was negatively correlated with SOX4 expression in chondrosarcoma cases. In all, we identified that SOX4 was oncogenic in chondrosarcoma and negatively regulated by miR-30a in vitro. Importantly, SOX4 overexpression may serve as a prognostic marker for patients with low-histological-grade chondrosarcoma.
    Tumor Biology 01/2015; DOI:10.1007/s13277-014-3026-2 · 2.84 Impact Factor
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    ABSTRACT: Abstract Candida albicans is the most common cause of hematogenously disseminated candidiasis, and this disease is particularly prevalent in immunocompromised patients. The mortality of invasive candidiasis remains 40% to 50% even with the proper treatment with current antifungal drugs. Recently, with the better understanding of host-fungus interactions, notable progress has been made in antifungal vaccine research. Most antifungal vaccines exert protection by inducing either (or both) B-cell and T-cell responses. Here we summarize the current available information on C. albicans vaccines, highlight the obstacles that researchers identified, and offer several suggestions.
    Virulence 01/2015; DOI:10.4161/21505594.2014.983015 · 3.32 Impact Factor
  • Asian Australasian Journal of Animal Sciences 01/2015; DOI:10.5713/ajas.14.0554 · 0.56 Impact Factor
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    ABSTRACT: Due to increasing incidence and mortality of invasive fungal infections, discovery and development of new generations of antifungal agents represents a challenging task. On the basis of our previously reported triazole-benzyloxypiperidinyl lead compound, a series of novel triazole antifungal agents containing piperdine-oxadiazoleside chains were rationally designed and synthesized. Most of the target compounds showed excellent inhibitory activity against clinically important fungal pathogens. In particular, compounds 6g (MIC = 0.031 ug/mL) and 11b (MIC = 0.016 ug/mL) were highly active against Candida albicans including fluconazole-resistant strains. Moreover, they showed inhibitory activity against hyphal formation with low toxicity, which were promising leads for further development.
    Medicinal Chemistry Communication 12/2014; DOI:10.1039/C4MD00505H · 2.63 Impact Factor
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    ABSTRACT: Human Rad 9 (hRad9), part of the Rad9-Hus1-Rad1 complex plays an important role in DNA damage repair as an up-stream regulator of checkpoint signaling, however little is known about its role in response to chemotherapy of breast cancer and whether hRad9 inhibition can potentiate the cytotoxic effects of chemotherapy on breast cancer cells remains to be elucidated. Fifty cases of breast cancer receiving neoadjuvant therapy were collected. All these cases were revised and classified into chemotherapy sensitive (CS) or chemotherapy resistant (CR) group according to the Miller and Payne (MP) grading system. Immunohistochemically, hRad9 positive tumours showed nuclear and/or cytoplasmic staining. hRad9 over-expression was associated with an impaired neoadjuvant chemotherapy response. A significant correlation was found between expression of hRad9 and Cyclin D1. In vitro, hRad9 was knocked down using siRNA in breast cancer cell line MCF-7 and MDA-MB-231. Deregulated expression of Rad9 accompanied by down expression of chk1 enhanced the sensitivity of human breast cancer cells to doxorubicin. Our work suggests that hRad9 might be a potential predictor for the response to chemotherapy in patients with breast cancer and its clinical value as a target for improving chemosensitivity needs further exploration.
    Scientific Reports 12/2014; 4:7548. DOI:10.1038/srep07548 · 5.08 Impact Factor
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    ABSTRACT: In this paper, to monitor the border in real-time with high efficiency and accuracy, we applied the compressed sensing (CS) technology on the border monitoring wireless sensor network (WSN) system and proposed a reconstruction method based on approximately l0 norm and fast gradient descent (AL0FGD) for CS. In the frontend of the system, the measurement matrix was used to sense the border information in a compressed manner, and then the proposed reconstruction method was applied to recover the border information at the monitoring terminal. To evaluate the performance of the proposed method, the helicopter sound signal was used as an example in the experimental simulation, and three other typical reconstruction algorithms 1)split Bregman algorithm, 2)iterative shrinkage algorithm, and 3)smoothed approximate l0 norm (SL0), were employed for comparison. The experimental results showed that the proposed method has a better performance in recovering the helicopter sound signal in most cases, which could be used as a basis for further study of the border monitoring WSN system.
    PLoS ONE 12/2014; 9(12):e112932. DOI:10.1371/journal.pone.0112932 · 3.53 Impact Factor
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    ABSTRACT: In an attempt to discover a new generation of triazole antifungal agents, a series of triazole–thiazolidinedione hybrids were designed and synthesized by molecular hybridization of the antifungal agent fluconazole and rosiglitazone (an antidiabetic). Most of the target compounds showed good to excellent inhibitory activity against a variety of clinically important fungal pathogens. In particular, compounds (Z)-5-(2,4-dichlorobenzylidene)-3-(2-(2,4-difluorophenyl)-2-hydroxy-3-(1H-1,2,4-triazol-1-yl)propyl)thiazolidine-2,4-dione) (15 c), (Z)-3-(2-(2,4-difluorophenyl)-2-hydroxy-3-(1H-1,2,4-triazol-1-yl)propyl)-5-(furan-3-ylmethylene)thiazolidine-2,4-dione (15 j), and (Z)-3-(2-(2,4-difluorophenyl)-2-hydroxy-3-(1H-1,2,4-triazol-1-yl)propyl)-5-(furan-3-ylmethylene)thiazolidine-2,4-dione (15 r) were highly active against Candida albicans, with MIC80 values in the range of 0.03–0.15 μM. Moreover, compounds 15 j and 15 r were found to be effective against four fluconazole-resistant clinical isolates; these two compounds are particularly promising antifungal leads for further optimization. Molecular docking studies revealed that the hydrogen bonding interactions between thiazolidinedione and CYP51 from C. albicans are important for antifungal activity. This study also demonstrates the effectiveness of molecular hybridization in antifungal drug discovery.
    ChemMedChem 12/2014; 9(12). DOI:10.1002/cmdc.201402320 · 3.05 Impact Factor
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    ABSTRACT: Acoustic Emission (AE) technique is an effective nondestructive detecting method, and has a promising application for rail defect detection. So far, little attention has been paid to propagation distances, types, and depths of AE sources, which are important for rail defect detection accurately. This paper presents an experimental study on the simulated AE sources with different propagation distances, types and depths for rail defect detection. Three simulated AE sources with different frequencies are seeded on the cross section of rail, and the depths of AE sources are changed in the vertical direction. After receiving AE signals, wavelet transform and Rayleigh–Lamb equations are utilized to extract time–frequency features and modes. Based on the wavelet transform with corresponding group-velocity curves, the influences of different propagation distances, the features of different source types and the rules of different source depths are examined. It is concluded that the features of AE sources with different propagation distances, types and depths can be obtained by AE technique for rail defect detection. It is very useful to analyze and detect defects in rail defect detection.
    Applied Acoustics 12/2014; 86:80–88. DOI:10.1016/j.apacoust.2014.06.004 · 1.07 Impact Factor

Publication Stats

4k Citations
1,087.72 Total Impact Points


  • 2013–2015
    • Northwest University
      China, Maine, United States
    • Baylor College of Medicine
      • Department of Molecular & Cellular Biology
      Houston, Texas, United States
    • Tsinghua University
      • Department of Chemistry
      Peping, Beijing, China
    • Hunan University
      • College of Environmental Science and Engineering
      Ch’ang-sha-shih, Hunan, China
  • 2007–2015
    • Sichuan Agricultural University
      Hua-yang, Sichuan, China
  • 2005–2015
    • Second Military Medical University, Shanghai
      • Faculty of Naval Medicine
      Shanghai, Shanghai Shi, China
    • Peking University Health Science Center
      Peping, Beijing, China
  • 1998–2015
    • Harbin Institute of Technology
      • • Department of Control Science and Engineering
      • • School of Astronautics
      Charbin, Heilongjiang Sheng, China
  • 2014
    • University of Jinan (Jinan, China)
      Chi-nan-shih, Shandong Sheng, China
    • Yangzhou University
      • Medical College
      Chiang-tu, Jiangsu Sheng, China
    • Alpert Medical School - Brown University
      • Department of Medicine
      Providence, Rhode Island, United States
  • 2013–2014
    • Peking University Cancer Hospital
      Peping, Beijing, China
  • 2011–2014
    • Liaoning Normal University
      • School of Physics and Electronic Technology
      Lü-ta-shih, Liaoning, China
    • China Pharmaceutical University
      • School of Pharmacy
      Nanjing, Jiangxi Sheng, China
    • Chinese Academy of Sciences
      • National Laboratory of Biomacromolecules
      Peping, Beijing, China
    • Civil Aviation Institute of China
      Peping, Beijing, China
    • Columbia University
      • Department of Medicine
      New York, New York, United States
  • 2010–2014
    • Hebei Medical University
      Chentow, Hebei, China
    • Shanghai Jiao Tong University
      • State Key Laboratory of Medical Genomics
      Shanghai, Shanghai Shi, China
    • State Key Laboratory of Medical Genetics of China
      Ch’ang-sha-shih, Hunan, China
  • 2009–2014
    • Sichuan University
      • • College of Electronics and Information Engineering
      • • State Key Laboratory of Polymer Material Engineering
      Hua-yang, Sichuan, China
  • 2008–2014
    • Fourth Military Medical University
      • • School of Stomatology
      • • Department of Dermatology
      Xi’an, Liaoning, China
    • Harbin Medical University
      • • Department of Microbiology
      • • College of Bioinformatics Science and Technology
      • • Department of Gynecology
      Charbin, Heilongjiang Sheng, China
  • 2007–2014
    • Beijing Normal University
      • College of Chemistry
      Peping, Beijing, China
  • 2006–2014
    • Henan University
      • • Institute of Environmental and Analytical Sciences
      • • Key Laboratory of Special Functional Materials, Ministry of Education
      K’ai-feng-shih, Henan Sheng, China
    • Shanghai Putuo District People's Hospital
      Shanghai, Shanghai Shi, China
  • 2011–2013
    • Ruijin Hospital North
      Shanghai, Shanghai Shi, China
  • 2010–2013
    • Harbin Engineering University
      Charbin, Heilongjiang Sheng, China
  • 2009–2013
    • Capital Medical University
      Peping, Beijing, China
    • Nankai University
      • Institute of Polymer Chemistry
      Tianjin, Tianjin Shi, China
  • 2008–2013
    • Anqing Normal University
      Nganking, Anhui Sheng, China
  • 2003–2013
    • Nanjing University
      • • State Key Laboratory of Coordination Chemistry
      • • Coordination Chemistry Institute
      • • Department of Chemical Engineering
      • • Department of Chemistry
      Nan-ching, Jiangsu Sheng, China
  • 2012
    • Nanjing Xiaozhuang University
      • School of Biochemical and Environmental Engineering
      Nan-ching, Jiangsu Sheng, China
    • Guilin Medical University
      Ling-ch’uan, Guangxi Zhuangzu Zizhiqu, China
    • University of Science and Technology of China
      • School of Life Sciences
      Luchow, Anhui Sheng, China
  • 2011–2012
    • Tongji Hospital
      Wu-han-shih, Hubei, China
    • Jiangnan University
      • School of Environmental and Civil Engineering
      Wu-hsi, Jiangsu Sheng, China
  • 2009–2012
    • Shandong University
      • School of Medicine
      Chi-nan-shih, Shandong Sheng, China
  • 2007–2008
    • Nanjing University of Aeronautics & Astronautics
      • Department of Applied Chemistry
      Nan-ching, Jiangsu Sheng, China
  • 2005–2007
    • Peking University People's Hospital
      Peping, Beijing, China
  • 2003–2006
    • University of Utah
      • Department of Chemistry
      Salt Lake City, Utah, United States
  • 2004–2005
    • Peking University
      • Institute of Hematology
      Peping, Beijing, China
    • Xinjiang Medical University
      Ouroumtchi, Xinjiang Uygur Zizhiqu, China