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Belle Collaboration,
I. Adachi,
K. Adamczyk,
H. Aihara,
K. Arinstein,
Y. Arita,
D. M. Asner,
T. Aso,
V. Aulchenko,
T. Aushev, [......],
L. M. Zhang,
Z. P. Zhang,
L. Zhao,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc,
N. Zwahlen,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We present a measurement of the time-dependent CP violation parameters in B0
-> pi+ pi- decays. The results are obtained from the final data sample
containing 772 million BBbar pairs collected at the Upsilon(4S) resonance with
the Belle detector at the KEKB asymmetric-energy e+e- collider. We obtain the
CP violation parameters Acp = +0.33 +/- 0.06 (stat) +/- 0.03 (syst) and Scp =
-0.64 +/- 0.08 (stat) +/- 0.03 (syst), where Acp and Scp represent direct and
mixing-induced CP violation, respectively. From an isospin analysis including
results from other Belle measurements, we rule out 23.8 < phi2 < 66.8 degrees
at the 1 sigma level.
02/2013;
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I. Adachi,
K. Adamczyk,
H. Aihara,
K. Arinstein,
Y. Arita,
D. M. Asner,
T. Aso,
V. Aulchenko,
T. Aushev,
T. Aziz, [......],
L. M. Zhang,
Z. P. Zhang,
L. Zhao,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc,
N. Zwahlen,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We present a study of the branching fraction of the decay B^0 -> rho^0 rho^0
and the fraction of longitudinally polarized \rho^0 mesons in this decay. The
results are obtained from the final data sample containing $772 \times 10^{6}$
$B\bar{B}$ pairs collected at the $\Upsilon(4S)$ resonance with the Belle
detector at the KEKB asymmetric-energy e^+e^- collider. We find $166 \pm 59$
B^0 -> rho^0 rho^0 events, corresponding to a branching fraction of ${\cal
B}(B^0 -> rho^0 rho^0) = (1.02\pm 0.30 (\rm stat) \pm 0.22 (\rm syst))\times
10^{-6} $ with a longitudinally polarization fraction $f_L =
0.21^{+0.18}_{-0.22} (\rm stat) \pm 0.11 (\rm syst)$. The branching fraction's
upper limit is ${\cal B}(B^0 -> rho^0 rho^0) < 1.5 \times 10^{-6}$ at 90%
confidence level. We use the longitudinal polarization fraction to determine
the Cabibbo-Kobayashi-Maskawa matrix angle $\phi_2 = (91.0 \pm 7.2)^{\circ}$
through an isospin analysis in the $B\to\rho\rho$ system. We furthermore obtain
${\cal B}(B^0 -> rho^0 f_0) \times {\cal B}(f_0 -> \pi^+\pi^-) = (0.86 \pm 0.27
(\rm stat) \pm 0.15 (\rm syst))\times 10^{-6}$, corresponding to $149 \pm 49$
$B^0 -> rho^0 f_0$ events with a significance of 3.0 standard deviations and
upper limits at 90% confidence level on the (product) branching fractions for
possible decays with the same final state, ${\cal B}(B^0 -> pi^+\pi^-pi^+\pi^-)
< 11.7 \times 10^{-6} $, ${\cal B}(B^0 -> rho^0 pi^+\pi^-) < 12.2 \times
10^{-6}$, ${\cal B}(B^0 -> f_0 pi^+\pi^-) \times {\cal B}(f_0->\pi^+\pi^-) <
3.1 \times 10^{-6}$ and ${\cal B}(B^0 -> f_0 f_0) \times {\cal
B}(f_0->\pi^+\pi^-)^{2} < 0.2 \times 10^{-6}$.
12/2012;
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I. Adachi,
K. Adamczyk,
H. Aihara,
K. Arinstein,
Y. Arita,
D. M. Asner,
T. Aso,
V. Aulchenko,
T. Aushev,
T. Aziz, [......],
L. M. Zhang,
Z. P. Zhang,
L. Zhao,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc,
N. Zwahlen,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We report preliminary results on the analysis of the three-body Y(10860)=> B
barB pi, Y(10860) => (B barB* + c.c.) pi and Y(10860)=> B* barB* pi decays
including an observation of the Y(10860)=> Zb(10610)+- pi-+ => [B barB*+c.c]+-
pi-+ and Y(10860)=> Zb(10650)+- pi-+ => [B* barB*]+- pi-+ decays as
intermediate channels. We measure branching fractions of the three-body decays
to be Br(Y(10860)=> [B barB*+c.c.]+- pi-+)=(28.3+-2.9+-4.6)x10^{-3} and
Br(Y(10860)=> [B* barB*]+- pi-+)=(14.1+-1.9+-2.4)x10^{-3} and set 90% C.L.
upper limit Br(Y(10860)=> [B barB]+- pi-+)<4.0x10^{-3}. We also report results
on the amplitude analysis of the three-body Y(10860)=>Y(nS)pi+pi-, n=1,2,3
decays and the analysis of the internal structure of the three-body
Y(10860)=>hb(mP)pi+pi-, m=1,2 decays. The results are based on a 121.4 1/fb
data sample collected with the Belle detector at a center-of-mass energy near
the Y(10860).
09/2012;
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I. Adachi,
K. Adamczyk,
H. Aihara,
K. Arinstein,
Y. Arita,
D. M. Asner,
T. Aso,
V. Aulchenko,
T. Aushev,
T. Aziz, [......],
L. M. Zhang,
Z. P. Zhang,
L. Zhao,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc,
N. Zwahlen,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We report the first observation of Y(5S) -> Y(1,2S) pi0 pi0 decays. Evidence
for the Zb0(10610) with 4.9sigma significance is found in a Dalitz plot
analysis of Y(5S) -> Y(2S) pi0 pi0 decays. The results are obtained with a
121.4 fb^-1 data sample collected with the Belle detector at the Y(5S)
resonance at the KEKB asymmetric-energy e+e- collider.
07/2012;
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[show abstract]
[hide abstract]
ABSTRACT: Mucosal dendritic cells (DC) are intimately associated with the airway epithelium and thus are ideally situated to be first responders to pathogens. We hypothesize that DC drive innate immune responses through early release of tumor necrosis factor (TNF) α, which drives airway epithelial cell responses. In a mouse model, TNFα release was significantly increased following a single exposure to German cockroach (GC) frass, an event independent of neutrophil recruitment into the airways. While lung epithelial cells and alveolar macrophages failed to release TNFα following GC frass exposure, bone marrow-derived DC (BMDC) produced substantial amounts of TNFα suggesting their importance as early responding cells. This was confirmed by flow cytometry of pulmonary myeloid DC. Addition of GC frass-pulsed BMDC or conditioned media from GC frass-pulsed BMDC to primary mouse tracheal epithelial cells (MTEC) or MLE-15 cells induced chemokine (C-C) motif ligand (CCL) 20 and granulocyte macrophage (GM) colony-stimulating factor (CSF), both of which are important for DC recruitment, survival and differentiation. Importantly, DC do not produce CCL20 or GM-CSF following allergen exposure. Blocking TNFα receptor 1 (TNFR1) completely abolished chemokine production, suggesting that BMDC-derived TNFα induced airway epithelial cell activation and enhancement of the innate immune response. Lastly, blocking TNFR1 in vivo resulted in significantly decreased CCL20 and GM-CSF production in the lungs of mice. Together, our data strongly suggest that DC-derived TNFα plays a crucial role in the initiation of innate immune responses through the modification of airway epithelial cell responses.
Journal of Innate Immunity 04/2012; 4(5-6):542-52. · 4.21 Impact Factor
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[show abstract]
[hide abstract]
ABSTRACT: Motor unit number index (MUNIX) measurement has recently achieved increasing attention as a tool to evaluate the progression of motoneuron diseases. In our current study, the sensitivity of the MUNIX technique to changes in motoneuron and muscle properties was explored by a simulation approach utilizing variations on published motoneuron pool and surface electromyogram (EMG) models. Our simulation results indicate that, when keeping motoneuron pool and muscle parameters unchanged and varying the input motor unit numbers to the model, then MUNIX estimates can appropriately characterize changes in motor unit numbers. Such MUNIX estimates are not sensitive to different motor unit recruitment and rate coding strategies used in the model. Furthermore, alterations in motor unit control properties do not have a significant effect on the MUNIX estimates. Neither adjustment of the motor unit recruitment range nor reduction of the motor unit firing rates jeopardizes the MUNIX estimates. The MUNIX estimates closely correlate with the maximum M-wave amplitude. However, if we reduce the amplitude of each motor unit action potential rather than simply reduce motor unit number, then MUNIX estimates substantially underestimate the motor unit numbers in the muscle. These findings suggest that the current MUNIX definition is most suitable for motoneuron diseases that demonstrate secondary evidence of muscle fiber reinnervation. In this regard, when MUNIX is applied, it is of much importance to examine a parallel measurement of motor unit size index (MUSIX), defined as the ratio of the maximum M-wave amplitude to the MUNIX. However, there are potential limitations in the application of the MUNIX methods in atrophied muscle, where it is unclear whether the atrophy is accompanied by loss of motor units or loss of muscle fiber size.
IEEE transactions on neural systems and rehabilitation engineering: a publication of the IEEE Engineering in Medicine and Biology Society 04/2012; 20(3):297-304. · 2.42 Impact Factor
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[show abstract]
[hide abstract]
ABSTRACT: The objective of this study was to assess whether there is evidence of motor unit loss in muscles paralyzed by spinal cord injury (SCI), using a measurement called motor unit number index (MUNIX). The MUNIX technique was applied in SCI (n=12) and neurologically intact (n=12) subjects. The maximum M waves and voluntary surface electromyography (EMG) signals at different muscle contraction levels were recorded from the first dorsal interosseous (FDI) muscle in each subject. The MUNIX values were estimated using a mathematical model describing the relation between the surface EMG signal and the ideal motor unit number count derived from the M wave and surface EMG measurements. We recorded a significant decrease in both maximum M wave amplitude and in estimated MUNIX values in paralyzed FDI muscles, as compared with neurologically intact muscles. Across all subjects, the maximum M wave amplitude was 8.3 ± 4.4 mV for the paralyzed muscles and 14.4 ± 2.0 mV for the neurologically intact muscles (p<0.0001). These measurements, when combined with voluntary EMG recordings, resulted in a mean MUNIX value of 112 ± 71 for the paralyzed muscles, much lower than the mean MUNIX value of 228 ± 49 for the neurologically intact muscles (p<0.00001). A motor unit size index was also calculated, using the maximum M wave recording and the MUNIX values. We found that paralyzed muscles showed a mean motor unit size index value of 80.7 ± 17.7 ìV, significantly higher than the mean value of 64.9 ± 10.1 ìV obtained from neurologically intact muscles (p<0.001). The MUNIX method used in this study offers several practical benefits compared with the traditional motor unit number estimation technique because it is noninvasive, induces minimal discomfort due to electrical nerve stimulation, and can be performed quickly. The findings from this study help understand the complicated determinants of SCI induced muscle weakness and provide further evidence of motoneuron degeneration after a spinal injury.
IEEE transactions on information technology in biomedicine: a publication of the IEEE Engineering in Medicine and Biology Society 04/2012; · 1.69 Impact Factor
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CLEO Collaboration,
J. Insler,
H. Muramatsu,
C. S. Park,
L. J. Pearson,
E. H. Thorndike,
S. Ricciardi,
C. Thomas,
M. Artuso,
S. Blusk, [......],
G. Wilkinson,
D. M. Asner,
G. Tatishvili,
J. Y. Ge,
D. H. Miller,
I. P. J. Shipsey,
B. Xin,
G. S. Adams,
J. Napolitano,
K. M. Ecklund
[show abstract]
[hide abstract]
ABSTRACT: The first measurements of the coherence factor R_{K_S^0K\pi} and the average
strong--phase difference \delta^{K_S^0K\pi} in D^0 \to K_S^0 K^\mp\pi^\pm
decays are reported. These parameters can be used to improve the determination
of the unitary triangle angle \gamma in B^- \rightarrow \widetilde{D}K^-$
decays, where $\widetilde{D}$ is either a D^0 or a D^0-bar meson decaying to
the same final state, and also in studies of charm mixing. The measurements of
the coherence factor and strong-phase difference are made using
quantum-correlated, fully-reconstructed D^0D^0-bar pairs produced in e^+e^-
collisions at the \psi(3770) resonance. The measured values are R_{K_S^0K\pi} =
0.73 \pm 0.08 and \delta^{K_S^0K\pi} = (8.3 \pm 15.2)$^\circ$ for an
unrestricted kinematic region and R_{K*K} = 1.00 \pm 0.16 and \delta^{K*K} =
(26.5 \pm 15.8)$^\circ$ for a region where the combined K_S^0 \pi^\pm invariant
mass is within 100 MeV/c^2 of the K^{*}(892)^\pm mass. These results indicate a
significant level of coherence in the decay. In addition, isobar models are
presented for the two decays, which show the dominance of the K^*(892)^\pm
resonance. The branching ratio {B}(D^0 \rightarrow K_S^0K^+\pi^-)/{B}(D^0
\rightarrow K_S^0K^-\pi^+) is determined to be 0.592 \pm 0.044 (stat.) \pm
0.018 (syst.), which is more precise than previous measurements.
03/2012;
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[show abstract]
[hide abstract]
ABSTRACT: Diabetes mellitus inducing a leading cause of morbidity are widespread in the entire globe. The present study was to investigate the antidiabetic potency and mechanism of a proteoglycan extract, named FYGL (Fudan-Yueyang-G. lucidum), from the fruiting bodies of Ganoderma Lucidum as published recently, using streptozotocin-induced type 2 diabetic mellitus (T2DM) rats.
The T2DM model rats were treated with FYGL as well as metformin and rosiglitazone. The levels of plasma glucose and insulin were measured, and the expression and activity of the protein tyrosine phosphatase 1B (PTP1B) and the tyrosine phosphorylation level of the insulin receptor (IR) 3-subunit in the livers and skeletal muscles of the T2DM rats were analyzed by immunoprecipitation and Western blotting methods. In addition, the levels of free fatty acid and serum lipid profile were measured using commercial kits for those trailed rats.
The decrease in fasting plasma glucose and the increase in insulin concentration dose- and time-dependently in the T2DM rats treated by FYGL, comparable with that by the clinical drugs, metformin and rosiglitazone. The levels of the PTP1B expression and activity were decreased, and the tyrosine phosphorylation level of the IR 1-subunit was increased in the skeletal muscles of the T2DM rats. Furthermore, FYGL significantly decreased the levels of free fatty acid, triglyceride, total cholesterol and low density lipoprotein-cholesterol as well as increased the level of high density lipoprotein-cholesterol.
It is suggested that the hypoglycemic mechanisms of FYGL are caused by inhibition of the PTP1B expression and activity, consequently, regulation of the tyrosine phosphorylation level of the IR 13-subunit. As those results, FYGL also controlled the plasma biochemistry indexes relative to the type 2 diabetes-accompanied metabolic disorders. This is possibly the first report on the underlying mechanisms responsible for the antidiabetic effect of Ganoderma lucidum.
European review for medical and pharmacological sciences 02/2012; 16(2):166-75. · 1.04 Impact Factor
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[show abstract]
[hide abstract]
ABSTRACT: The protein-protein interactions between viral and host proteins play an essential role in plant virus infection and host defense. The potyviral nuclear inclusion protein a protease (NIa-Pro) is involved in various steps of viral infection. In this study, the host proteins interacting with papaya ringspot virus (PRSV) NIa-Pro were screened in a Carica papaya L. plant cDNA library using a Sos recruitment two-hybrid system (SRS). We confirmed that the full-length EIF3G, FBPA1, FK506BP, GTPBP, MSRB1, and MTL from papaya can interact specifically with PRSV NIa-Pro in yeast, respectively. These proteins fufill important functions in plant protein translation, biotic and abiotic stress, energy metabolism and signal transduction. In this paper, we discuss possible functions of interactions between these host proteins and NIa-Pro in PRSV infection and their role in host defense. Keywords: Sos recruitment two-hybrid system; papaya ringspot virus; NIa-Pro; protein-protein interaction.
Acta virologica 01/2012; 56(1):25-30. · 0.68 Impact Factor
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[show abstract]
[hide abstract]
ABSTRACT: Specific noncovalent interactions that are indicative of attractive, directional intermolecular forces have always been of key interest to medicinal chemists in their search for the "glue" that holds drugs and their targets together. With the rapid increase in the number of solved biomolecular structures as well as the performance enhancement of computer hardware and software in recent years, it is now possible to give more comprehensive insight into the geometrical characteristics and energetic landscape of certain sophisticated noncovalent interactions present at the binding interface of protein receptors and small ligands based on accumulated knowledge gaining from the combination of two quite disparate but complementary approaches: crystallographic data analysis and quantum-mechanical ab initio calculation. In this perspective, we survey massive body of published works relating to structural characterization and theoretical investigation of three kinds of strong, specific, direct, enthalpy-driven intermolecular forces, including hydrogen bond, halogen bond and salt bridge, involved in the formation of protein-ligand complex architecture in order to characterize their biological functions in conferring affinity and specificity for ligand recognition by host protein. In particular, the biomedical implications of raised knowledge are discussed with respect to potential applications in rational drug design.
Current Medicinal Chemistry 01/2012; 19(2):226-38. · 4.86 Impact Factor
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M Bischofberger,
H Hayashii,
K Adamczyk,
H Aihara,
V Aulchenko,
A M Bakich,
V Balagura,
E Barberio,
K Belous,
A Bozek, [......],
P Wang,
M Watanabe,
Y Watanabe,
E Won,
H Yamamoto,
Y Yamashita,
C Z Yuan, P Zhou,
V Zhulanov,
T Zivko
[show abstract]
[hide abstract]
ABSTRACT: We report on a search for CP violation in τ(±)→K(S)(0)π(±)ν(τ) decays using a data sample of 699 fb(-1) collected by the Belle experiment at the KEKB electron-positron asymmetric-energy collider. The CP asymmetry is measured in four bins of the invariant mass of the K(S)(0)π(±) system and found to be compatible with zero with a precision of O(10(-3)) in each mass bin. Limits for the CP violation parameter Im(η(S)) are given at the 90% confidence level. These limits are |Im(η(S))| < 0.026 or better, depending on the parametrization used to describe the hadronic form factors, and improve upon previous limits by 1 order of magnitude.
Physical Review Letters 09/2011; 107(13):131801. · 7.37 Impact Factor
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V Bhardwaj,
K Trabelsi,
J B Singh,
S-K Choi,
S L Olsen,
I Adachi,
K Adamczyk,
D M Asner,
V Aulchenko,
T Aushev, [......],
K M Williams,
B D Yabsley,
Y Yamashita,
C Z Yuan,
C C Zhang,
Z P Zhang,
V Zhilich, P Zhou,
V Zhulanov,
A Zupanc
[show abstract]
[hide abstract]
ABSTRACT: We report a study of B→(J/ψγ)K and B→(ψ'γ)K decay modes using 772×10⁶ B ̅B events collected at the Υ(4S) resonance with the Belle detector at the KEKB energy-asymmetric e(+)e(-) collider. We observe X(3872)→J/ψγ and report the first evidence for χ(c2)→J/ψγ in B→(X_{c ̅cγ)K decays, while in a search for X(3872)→ψ'γ no significant signal is found. We measure the branching fractions, B(B(±)→X(3872)K(±))B(X(3872)→J/ψγ)=(1.78(-0.44)(+0.48)±0.12)×10(-6), B(B(±)→χ(c2)K(±))=(1.11(-0.34)(+0.36)±0.09)×10(-5), B(B(±)→X(3872)K(±))B(X(3872)→ψ'γ)<3.45×10⁶ (upper limit at 90% C.L.), and also provide upper limits for other searches.
Physical Review Letters 08/2011; 107(9):091803. · 7.37 Impact Factor
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A Vossen,
R Seidl,
I Adachi,
H Aihara,
T Aushev,
V Balagura,
W Bartel,
M Bischofberger,
A Bondar,
M Bračko, [......],
C H Wang,
M-Z Wang,
P Wang,
Y Watanabe,
E Won,
B D Yabsley,
Y Yamashita,
V Zhilich, P Zhou,
V Zhulanov
[show abstract]
[hide abstract]
ABSTRACT: The interference fragmentation function translates the fragmentation of a quark with a transverse projection of the spin into an azimuthal asymmetry of two final-state hadrons. In e(+)e(-) annihilation the product of two interference fragmentation functions is measured. We report nonzero asymmetries for pairs of charge-ordered π(+)π(-) pairs, which indicate a significant interference fragmentation function in this channel. The results are obtained from a 672 fb(-1) data sample that contains 711 × 10(6) π(+)π(-) pairs and was collected at and near the Υ(4S) resonance, with the Belle detector at the KEKB asymmetric-energy e(+)e(-) collider.
Physical Review Letters 08/2011; 107(7):072004. · 7.37 Impact Factor
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Belle Collaboration,
A. Vinokurova, A. Kuzmin,
S. Eidelman,
K. Arinstein,
V. Aulchenko,
T. Aushev,
A. M. Bakich,
V. Balagura,
E Barberio, [......],
E. Won,
B. D. Yabsley,
Y Yamashita,
C. Z. Yuan,
Z P Zhang,
V. Zhilich, P Zhou,
V. Zhulanov,
A. Zupanc,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We report the results of a study of $B^{\pm}\to K^{\pm}\eta_c$ and
$B^{\pm}\to K^{\pm}\eta_c(2S)$ decays followed by $\eta_c$ and $\eta_c(2S)$
decays to $(K_SK\pi)^0$. The results are obtained from a data sample containing
535 million $B\bar{B}$-meson pairs collected by the Belle experiment at the
KEKB $e^+e^-$ collider. We measure the products of the branching fractions
${\mathcal B}(B^{\pm}\to K^{\pm}\eta_c){\mathcal B}(\eta_c\to K_S
K^{\pm}\pi^{\mp})=(26.7\pm 1.4(stat)^{+2.9}_{-2.6}(syst)\pm 4.9(model))\times
10^{-6}$ and ${\mathcal B}(B^{\pm}\to K^{\pm}\eta_c(2S)){\mathcal
B}(\eta_c(2S)\to K_S
K^{\pm}\pi^{\mp})=(3.4^{+2.2}_{-1.5}(stat+model)^{+0.5}_{-0.4} syst))\times
10^{-6}$. Interference with the non-resonant component leads to significant
model uncertainty in the measurement of these product branching fractions. Our
analysis accounts for this interference and allows the model uncertainty to be
reduced. We also obtain the following charmonia masses and widths:
$M(\eta_c)=(2985.4\pm 1.5(stat)^{+0.5}_{-2.0}(syst))$ MeV/$c^2$,
$\Gamma(\eta_c)=(35.1\pm 3.1(stat)^{+1.0}_{-1.6}(syst))$ MeV/$c^2$,
$M(\eta_c(2S))=(3636.1^{+3.9}_{-4.2}(stat+model)^{+0.7}_{-2.0}(syst))$
MeV/$c^2$,
$\Gamma(\eta_c(2S))=(6.6^{+8.4}_{-5.1}(stat+model)^{+2.6}_{-0.9}(syst))$
MeV/$c^2$.
05/2011;
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J Li,
H Aihara,
K Arinstein,
V Aulchenko,
T Aushev,
T Aziz,
A M Bakich,
V Balagura,
E Barberio,
K Belous, [......],
E Won,
B D Yabsley,
Y Yamashita,
D Zander,
C C Zhang,
Z P Zhang,
V Zhilich, P Zhou,
V Zhulanov,
A Zupanc
[show abstract]
[hide abstract]
ABSTRACT: We report the observation of B(s)⁰→J/ψf₀(980) and evidence for B(s)⁰→J/ψf₀(1370), which are CP eigenstate decay modes. These results are obtained from 121.4 fb⁻¹ of data collected at the Υ(5S) resonance with the Belle detector at the KEKB e(+)e⁻ collider. We measure the branching fractions B(B(s)⁰→J/ψf₀(980); f₀(980)→π(+)π⁻)=(1.16(-0.19)(+0.31)(stat)(-0.17)(+0.15)(syst)(-0.18)(+0.26)(N(B(s)((*))B(s)((*)))))×10⁻⁴ with a significance of 8.4σ, and B(B(s)⁰→J/ψf₀(1370); f₀(1370)→π(+)π⁻)=(0.34(-0.14)(+0.11)(stat)(-0.02)(+0.03)(syst)(-0.05)(+0.08)(N(B(s)((*))B(s)((*)))))×10⁻⁴ with a significance of 4.2σ. The last error listed is due to uncertainty in the number of produced B(s)((*))B(s)((*)) pairs.
Physical Review Letters 03/2011; 106(12):121802. · 7.37 Impact Factor
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H. Guler,
H. Aihara,
K. Arinstein,
V. Aulchenko,
T. Aushev,
A. M. Bakich,
V. Balagura,
E. Barberio,
K. Belous,
V. Bhardwaj, [......],
Y. Watanabe,
K. M. Williams,
E. Won,
Y. Yamashita,
Z. P. Zhang,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc
[show abstract]
[hide abstract]
ABSTRACT: Using 535×106 B-meson pairs collected by the Belle
detector at the KEKB e+e- collider, we measure
branching fractions of
(7.16±0.10(stat)±0.60(syst)×10-4 for
B+→J/ψK+π+π-
and (4.31±0.20(stat)±0.50(syst))×10-4 for
B+→ψ'K+π+π-.
We perform amplitude analyses to determine the resonant structure of the
K+π+π- final state in
B+→J/ψK+π+π-
and
B+→ψ'K+π+π-
and find that the K1(1270) is a prominent component of both
decay modes. There is significant interference among the different
intermediate states, which leads, in particular, to a striking
distortion of the ρ line shape due to the ω. Based on the
results of the fit to the
B+→J/ψK+π+π-
data, the relative decay fractions of the K1(1270) to Kρ,
Kω, and K*(892)π are consistent with previous
measurements, but the decay fraction to K0*(1430)
is significantly smaller. Finally, by floating the mass and width of the
K1(1270) in an additional fit of the
B+→J/ψK+π+π-
data, we measure a mass of
(1248.1±3.3(stat)±1.4(syst))MeV/c2 and a width
of (119.5±5.2(stat)±6.7(syst))MeV/c2 for the
K1(1270).
Physical review D: Particles and fields 01/2011; 83(3):32005.
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M. Bischofberger,
H. Hayashii,
K. Adamczyk,
H. Aihara,
V. Aulchenko,
A. M. Bakich,
V. Balagura,
E Barberio,
K. Belous,
A. Bozek, [......],
P Wang,
M Watanabe,
Y Watanabe,
E. Won,
H Yamamoto,
Y Yamashita,
C. Z. Yuan, P Zhou,
V. Zhulanov,
T. Zivko
[show abstract]
[hide abstract]
ABSTRACT: We report on a search for CP violation in tau -> K^0_S pi nu_tau decays using
a data sample of 699 fb^{-1} collected in the Belle experiment at the KEKB
electron-positron asymmetric-energy collider. The CP asymmetry is measured in
four bins of the invariant mass of the K^0_S pi system and found to be
compatible with zero with a precision of O(10^{-3}) in each mass bin. Limits
for the CP violation parameter Im(eta_S) are given at a 90 % confidence level.
These limits are |Im(eta_S)|<0.026 or better, depending on the parameterization
used to describe the hadronic form factors and improve upon previous limits by
one order of magnitude.
01/2011;
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G. Pakhlova,
I. Adachi,
H. Aihara,
K. Arinstein,
T. Aushev,
T. Aziz,
A. M. Bakich,
V. Balagura,
E. Barberio,
A. Bay, [......],
E. Won,
Y. Yamashita,
C. Z. Yuan,
Z. P. Zhang,
V. Zhilich, P. Zhou,
V. Zhulanov,
T. Zivko,
A. Zupanc,
O. Zyukova
[show abstract]
[hide abstract]
ABSTRACT: We report a measurement of exclusive
e+e-→Ds(*)+Ds(*)-
cross sections as a function of center-of-mass energy near the
Ds(*)+Ds(*)- threshold with
initial-state radiation. The analysis is based on a data sample
collected with the Belle detector with an integrated luminosity of
967fb-1.
Physical review D: Particles and fields 12/2010; 83(1):11101.
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W. Dungel,
C. Schwanda,
I. Adachi,
H. Aihara,
T. Aushev,
T. Aziz,
A. M. Bakich,
V. Balagura,
E. Barberio,
M. Bischofberger, [......],
Y. Watanabe,
R. Wedd,
K. M. Williams,
E. Won,
Y. Yamashita,
D. Zander,
Z. P. Zhang, P. Zhou,
V. Zhulanov,
T. Zivko
[show abstract]
[hide abstract]
ABSTRACT: This article describes a determination of the Cabibbo-Kobayashi-Maskawa matrix element |Vcb| from the decay B0→D*-ℓ+νℓ using 711 fb-1 of Belle data collected near the Υ(4S) resonance. We simultaneously measure the product of the form factor normalization F(1) and the matrix element |Vcb| as well as the three parameters ρ2, R1(1) and R2(1), which determine the form factors of this decay in the framework of the heavy quark effective theory. The results, based on about 120 000 reconstructed B0→D*-ℓ+νℓ decays, are ρ2=1.214±0.034±0.009, R1(1)=1.401±0.034±0.018, R2(1)=0.864±0.024±0.008 and F(1)|Vcb|=(34.6±0.2±1.0)×10-3. The branching fraction of B0→D*-ℓ+νℓ is measured at the same time; we obtain a value of B(B0→D*-ℓ+νℓ)=(4.58±0.03±0.26)%. The errors correspond to the statistical and systematic uncertainties. These results give the most precise determination of the form factor parameters and F(1)|Vcb| to date. In addition, a direct, model-independent determination of the form factor shapes has been carried out.
Phys. Rev. D. 12/2010; 82(11).
