Jing Wang

Zhejiang University, Hang-hsien, Zhejiang Sheng, China

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Publications (810)1529.97 Total impact

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    ABSTRACT: In this paper, the resource allocation for multiuser wiretap Orthogonal Frequency-Division Multiplexing(OFDM) systems is investigated to improve the system power efficiency. In such a multiuser wiretap OFDM system, one legitimate transmitter (Alice), multiple legitimate receivers (Bobs), and one eavesdropper (Eve) are simultaneously considered. The time-domain artificial noise (AN) is firstly involved to guarantee the target secrecy rates of multiple Bobs. Different from the traditional sum secrecy rate maximization problem which may result in the unfair transition for Bobs with bad channel gains and more transmit power consumptions, a non-convex transmit power minimization problem is formulated to jointly optimize the power, subcarrier allocation and AN design, in which a target secrecy rate for each individual Bob is ensured. An iterative algorithm is further proposed to solve this power minimization problem by updating the subcarrier, power allocation and AN design alternatively. Numerical simulation results demonstrate the effectiveness of the proposal, including the improvements of system power efficiency and the fairness of multiple Bobs. 1
    IEEE ICC 2015, London, UK; 06/2015
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    ABSTRACT: In this paper, a new state-of-the-art multi-cell MMSE scheme is proposed for massive MIMO networks, which includes an uplink MMSE detector and a downlink MMSE precoder. The main novelty is that it exploits all available pilots for interference suppression. Specifically, let $K$ and $B$ denote the number of users per cell and the number of orthogonal pilot sequences in the network, respectively, where $\beta = B/K$ is the pilot reuse factor. Then our multi-cell MMSE scheme utilizes all $B$ channel directions, that can be estimated locally at each base station, to actively suppress both intra-cell and inter-cell interference. The proposed scheme is particularly practical and general, since power control for the pilot and payload, imperfect channel estimation and arbitrary pilot allocation are all accounted for. Simulations show that significant spectral efficiency (SE) gains are obtained over the single-cell MMSE scheme and the multi-cell ZF, particularly for large $\beta$ and/or $K$. Furthermore, large-scale approximations of the uplink and downlink SINRs are derived, which are asymptotically tight in the large-system limit. The approximations are easy to compute and very accurate even for small system dimensions. Using these SINR approximations, a low-complexity power control algorithm is also proposed to maximize the sum SE.
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    ABSTRACT: Premature ventricular contractions (PVCs) originate from aortic sinus cusps (ASC) can exhibit preferential conduction to right ventricular outflow tract (RVOT). This study aimed to examine the electrophysiological characteristics for guiding catheter ablation in patients with two morphological types of PVCs that originate from aortic sinus cusps (ASC) or great cardiac vein (GCV). We analyzed ECG from 10 patients with PVCs of two QRS morphologies. The patients who exhibited dominant LBBB QRS morphology and less right bundle branch block (RBBB) morphology were designated as group 1 (n = 7), and those with dominant RBBB QRS morphology were designated as group 2 (n = 3). During PVCs, electro-anatomical mapping was performed in both RVOT and ASC in group 1 and only performed in ASC or GCV in group 2. In group 1, the earliest ventricular activation (EVA) preceding the onset of the QRS complex (V-QRS) was recorded for 27±6ms (range 18 to 36ms) in RVOT and 25±6ms (range 18 to 34ms) in the ASC, while V-QRS was recorded for 28ms, 42ms 42ms in the ASC or GCV in group 2. All patients were successfully ablated at one site finally, including LCC in seven, L-RCC in two, and GCV in one. None of the patients experienced recurrence or complications during the 18.4±5.1 (range 6 to 24 months) months of follow-up. Two QRS morphologies (LBBB and RBBB with inferior axis) in PVCs could be a predictor of PVCs originating from ASC or GCV. This article is protected by copyright. All rights reserved. This article is protected by copyright. All rights reserved.
    Pacing and Clinical Electrophysiology 05/2015; DOI:10.1111/pace.12652 · 1.25 Impact Factor
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    ABSTRACT: We report measurements of the complex Kerr rotation angle, $\Theta_K$, in thin films of the magnetically doped topological insulator $(\text{Cr}_{0.12}\text{Bi}_{0.26}\text{Sb}_{0.62})_2\text{Te}_3$ as a function of photon energy in the range $0.8$ eV $<\hbar\omega<3.0$ eV. We observe a peak in the real part of $\Theta_K(\omega)$ and corresponding zero crossing in the imaginary part, that we attribute to resonant interaction with a spin-orbit avoided crossing and Dirac band located $\approx$ 1.7 eV above the chemical potential. The resonant enhancement allows measurement of the temperature and magnetic field dependence of $\Theta_K$ in the ultrathin film limit, $d\geq2$ quintuple layers. We find a sharp transition to remnant $\Theta_K=0$ for $d<8$ QL, consistent with theories of the dependence of impurity spin interactions on film thickness and their location relative to topological insulator surfaces.
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    ABSTRACT: The dielectric properties of LaF3 single crystals were investigated in the temperature range from 110 to 773 K and the frequency range from 100 Hz to 10 MHz. Two thermally activated relaxations (R1 and R2) and a dielectric anomaly (A) were observed. The lower temperature relaxation (R1) was ascribed to a polaronic relaxations due to fluorine ions diffusion within the F1 sublattice and fluorine ions hopping in F1 sublattice. The higher temperature relaxation (R2) is Maxwell–Wagner relaxation due to the blocking of electrodes associated with the ionic exchange between F1 and F2,3 sublattices and among the three nonequivalent sublattices. The anomaly appearing in the highest temperature range is related to the inductive effect arising from the coupled electron-ionic inductive response.
    Journal of Materials Science 05/2015; 50(10). DOI:10.1007/s10853-015-8944-x · 2.31 Impact Factor
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    ABSTRACT: In distributed multilevel diversity coding, $K$ correlated sources (each with $K$ components) are encoded in a distributed manner such that, given the outputs from any $\alpha$ encoders, the decoder can reconstruct the first $\alpha$ components of each of the corresponding $\alpha$ sources. For this problem, the optimality of a multilayer Slepian-Wolf coding scheme based on binning and superposition is established when $K\leq 3$. The same conclusion is shown to hold for general $K$ under a certain symmetry condition, which generalizes a celebrated result by Yeung and Zhang.
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    ABSTRACT: Polymorphisms in platelet receptor genes may influence platelet function. This study aimed to assess the impact of five polymorphisms of genes encoding platelet receptors on the risk of ischemic and bleeding events in ST-elevation myocardial infarction (STEMI) patients after percutaneous coronary intervention (PCI). 503 consecutive Chinese patients with STEMI after an uneventful PCI and exposed to standard dual antiplatelet therapy for 12 months were enrolled. Polymorphisms of platelet receptors, GPIa (ITGA2, 807C > T, rs1126643), GPVI (GP6, 13254T > C, rs1613662), PAR-1 (F2R, IVS-14A > T, rs168753) and P2Y12 (P2RY12, 34C > T, rs6785930 and H1/H2 haplotype, 52G > T, rs6809699) were detected by the ligase detection reaction. The follow-up period was 12 months. Overall, 34 (6.8%) ischemic events occurred and 46 (9.1%) major bleedings occurred. Multivariate Cox regression analysis showed the carriage of F2R rs168753 minor allele was an independent predictor of the composite ischemic events (HR 0.387, 95% CI 0.193-0.778, p = 0.008) after adjusted for established risk factors. Multivariate logistic regression model identified that carriage of P2RY12 rs6809699 minor allele (OR 2.71, 95% CI 1.298-5.659, p = 0.008) was an independent predictor of major bleedings. The associations were then validated in a second cohort of 483 STEMI patients. In STEMI patients after PCI, F2R rs168753 minor allele could significantly contribute to the risk of ischemic events, and P2RY12 rs6809699 minor allele could predict bleedings. The genetic testing of platelet receptors can be valuable in predicting adverse events in STEMI patients after PCI.
    Platelets 04/2015; DOI:10.3109/09537104.2015.1034096 · 2.63 Impact Factor
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    ABSTRACT: In recent years, beneficial effects of various ligands of three peroxisome-proliferator-activated receptor (PPAR) isoforms (α,β,γ) have been reported in neurodegenerative diseases through delaying the onset and progression of diseases, reducing lesion size and improving functional recovery. Neural stem cells (NSCs) are assumed as a promising strategy for the treatment of human neurodegenerative diseases. PPARs are supposed to be one group of the key regulators of fate decisions in neural stem cells during development and adulthood, through their impact on the target genes involving cell proliferation, death and differentiation. The neuroprotective role of PPARs is suggested to be closely associated with the inflammation control and regenerative function of NSCs. Nevertheless, the molecular mechanisms remain to be elucidated. Here, we review the current knowledge about the beneficial role of PPARs in NSC development and neurogenesis and attempt to discuss the underlying mechanisms.
    Current Stem Cell Research & Therapy 04/2015; · 2.86 Impact Factor
  • Industrial & Engineering Chemistry Research 04/2015; 54(14):3664-3677. DOI:10.1021/acs.iecr.5b00567 · 2.24 Impact Factor
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    ABSTRACT: B7-H3 (CD276), known as a member of B7 immunoregulatory family, is a type I transmembrane glycoprotein aberrantly expressed in numerous types of cancer and associated with poor prognosis. However, the role of B7-H3 in oncogenesis and chemosensitivity of mantle cell lymphoma (MCL) remains unknown. We determined the effects of downregulating B7-H3 expression on tumor progression and the sensitivity of chemotherapeutic drug in mantle cell lymphoma. B7-H3 knockdown was performed using lentivirus transduction in the Maver and Z138 mantle cell lymphoma cell lines, respectively. The effects of B7-H3 on cell proliferation, cycle, migration and invasion were investigated by CCK-8 assay, methyl cellulose colony forming assay, PI staining, and Transwell assays in vitro. By establishing Maver and Z138 xenograft models, the effects of B7-H3 on tumorigenicity were observed, and Ki-67 and PCNA was detected by immunohistochemistry. The downregulation of B7-H3 significantly decreased tumor proliferation in MCL in vitro and in vivo. In the B7-H3 knockdown groups of Maver and Z138 xenograft models, the mean inhibition rate of tumor growth was 59.1 and 65.0% (p=0.010 and 0.003), and the expression of both Ki-67 and PCNA were significantly lower, respectively. After B7-H3 silencing, the cell cycles of Maver and Z138 were both arrested at G0/G1 phase, and the cell migration rates and invasion capacity were decreased as well. Moreover, the impacts of B7-H3 RNAi on the antitumor effect of chemotherapy drugs were determined with CCK-8 and Annexin V-FITC/PI assays in vitro and with xenograft models in vivo. The silencing of B7-H3 increased the sensitivity of Maver and Z138 cells to rituximab and bendamustine and enhanced the drug-induced apoptosis, respectively. Our study demonstrates for the first time that B7-H3 promotes mantle cell lymphoma progression and B7-H3 knockdown significantly enhances the chemosensitivity. This may provide a new therapeutic approach to mantle cell lymphoma.
    International Journal of Oncology 04/2015; DOI:10.3892/ijo.2015.2962 · 2.77 Impact Factor
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    ABSTRACT: A multilayer charge generation unit (CGU) has been developed for inverted tandem organic light-emitting diodes (OLEDs). The current efficiency of inverted tandem phosphorescence OLED with (8 nm)/Al (1 nm)/ (0.5 nm) CGU is almost twice as high as that of individual electroluminescent (EL) device. Charges generate at hole-transporting host material/ interface and bulk layer, and electrons are extracted by /electon-transporting layer (ETL) interface. The significantly enhanced performance of the tandem device with CGU compared with a -based one may be attributed to preventing chemical reaction of and by the inserted ultrathin Al layer. Moreover, the current efficiency and EL spectrum of another inverted tandem OLED combining red and green stack further prove the effective charge generation and separation with the multilayer CGU. It is expected that such a CGU is a promising candidate for inverted tandem white OLED for- active matrix organic light-emitting diode display driven by an n-type thin-film transistor backplane.
    Journal of Display Technology 04/2015; 11(4):1-1. DOI:10.1109/JDT.2015.2392118 · 1.69 Impact Factor
  • Jurg Ott, Jing Wang, Suzanne M Leal
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    ABSTRACT: For many years, linkage analysis was the primary tool used for the genetic mapping of Mendelian and complex traits with familial aggregation. Linkage analysis was largely supplanted by the wide adoption of genome-wide association studies (GWASs). However, with the recent increased use of whole-genome sequencing (WGS), linkage analysis is again emerging as an important and powerful analysis method for the identification of genes involved in disease aetiology, often in conjunction with WGS filtering approaches. Here, we review the principles of linkage analysis and provide practical guidelines for carrying out linkage studies using WGS data.
    Nature Reviews Genetics 03/2015; DOI:10.1038/nrg3908 · 39.79 Impact Factor
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    ABSTRACT: TGF-β1 secreted abundantly by tumors cells as well as present in the local microenvironment promotes neoplasm invasion and metastasis by triggering the epithelial to mesenchymal transition (EMT). MiR200c has been shown to suppress EMT and to regulate the cellular epithelial and interstitial state conversion, whereas the tumor vaccines are intended to specifically initiate or amplify a host response against evolving tumor cells. Our study aimed at optimizing the antitumor effects of the B16F10/glycosylphosphatidylinositol-interleukin 21 (B16F10/GPI-IL-21) tumor vaccine on melanoma bearing mice by combining the TGF-β1 knockdown and the administration of miR200c agomir. The mice were subcutaneously vaccinated with inactivated B16F10/GPI-IL-21 vaccine and challenged by B16F10 cells transfected with shTGF-β1 (B16F10/shTGF-β1 cells) or B16F10/shTGF-β1 cells with the administration of miR200c agomir. The later combination showed that, when compared with the mice in the control group that received no vaccination, vaccinated mice significantly increased NK and CTL activities, enhanced levels of IFN-γ, and reduced expression of TGF-β1, N-cadherin, Vimentin, Gli1/2, P-Smad2/3 and others involved in promoting expression of EMT-related molecules in tumor areas, and inhibited the melanoma metastasis in lungs and lymph nodes. Altogether, our findings demonstrate that this synergistic anti-cancer regimen effectively induces strong immune response and diminishes the melanoma progression.
    Oncotarget 03/2015; · 6.63 Impact Factor
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    ABSTRACT: Understanding the mechanism by which cell growth, migration, polyploidy, and tumorigenesis are regulated may provide important therapeutic strategies for cancer therapy. Here we identify the Skp2-macroH2A1 (mH2A1)-cyclin-dependent kinase 8 (CDK8) axis as a critical pathway for these processes, and deregulation of this pathway is associated with human breast cancer progression and patient survival outcome. We showed that mH2A1 is a new substrate of Skp2 SCF complex whose degradation by Skp2 promotes CDK8 gene and protein expression. Strikingly, breast tumour suppression on Skp2 deficiency can be rescued by mH2A1 knockdown or CDK8 restoration using mouse tumour models. We further show that CDK8 regulates p27 protein expression by facilitating Skp2-mediated p27 ubiquitination and degradation. Our study establishes a critical role of Skp2-mH2A1-CDK8 axis in breast cancer development and targeting this pathway offers a promising strategy for breast cancer therapy.
    Nature Communications 03/2015; 6:6641. DOI:10.1038/ncomms7641 · 10.74 Impact Factor
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    ABSTRACT: After decades of searching for the dissipationless transport in the absence of any external magnetic field, quantum anomalous Hall effect (QAHE) was recently predicted and experimentally achieved in thin magnetic topological insulator (TI) films, where the quantized chiral edge conduction spontaneously occurred without invoking the formation of discrete Landau levels (LLs). However, the universal phase diagram of QAHE and its relation with quantum Hall effect (QHE) remain to be investigated. In this article, with two-dimensional (2D) quantum confinement, we report the experimental observation of the quantum phase transition between two QAHE states in the 6 quintuple-layer (QL) (Cr0.12Bi0.26Sb0.62)2Te3 film. Consistent with the theoretical prediction, zero Hall conductance plateau and the double-peaked longitudinal conductance at the coercive field are resolved up to 0.3 K, and they manifest the presence of the quantum anomalous Hall insulating state within the magnetic multi-domain network context. Moreover, by studying the angle-dependent quantum transport behaviors, the 2D massive Dirac fermion-featured QAHE phase diagram is mapped out to show that the QAHE state with the first Chern number C1 = 1 is transitioned into the C1 = 0 insulating state, and the conductance tensor follows a universal semicircle relation regardless of the applied magnetic field strength. Our results address that the quantum phase transitions in both QAHE and QHE regimes are in the same universality class, yet the microscopic details are different. In addition, the realization of the quantum anomalous Hall insulator from both the field-driven and angle-dependent experiments unveils new ways to explore quantum phase-related physics and applications.
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    ABSTRACT: A highly efficient and stable electron injection layer (EIL) for inverted organic light-emitting diodes (OLEDs) is developed. A 1-nm-thick Al is deposited between indium tin oxide cathode and commonly used Cs2CO3 EIL, which can significantly improve the stability. The Al may react with evaporated Cs2CO3 and form a much stabler Al-O-Cs complex, avoiding Cs oxidization by air according to X-ray photoemission spectroscopy measurement. When the Al is evaporated after Cs2CO3 layer, although such Al-O-Cs complex also forms, the inferior electron injection at Al/4,7-diphenyl-1,10-phenanthroline interface leads to a joule heat-induced resistance that adversely affects the air-stability of device. It is expected that the developed Al/Cs2CO3 EIL promotes high efficiency and stable active-matrix OLEDs based on n-type thin film transistor.
    ACS Applied Materials & Interfaces 03/2015; 7(12). DOI:10.1021/am506300c · 5.90 Impact Factor
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    ABSTRACT: To compare the efficacy and toxicity of 10 mg/m² or 8 mg/m² idarubicin (Ida) combined with cytarabine (IA"3+7"regimen) as induction chemotherapy for adult patients with newly diagnosed acute myeloid leukemia (AML). From June 2004 to October 2013, 335 adult AML (non acute promyelocytic leukemia) patients receiving the IA regimen as induction chemotherapy were enrolled, including 198 cases with 10 mg/m² Ida and 137 cases with 8 mg/m² Ida for 3 days. We compared the hematologic response, hematologic side effects and prognosis between the two regimens. Except for 4 early deaths, the complete remission (CR) rate after the first cycle of induction chemotherapy was 72.5%, 10.0% partial remission (PR) and 82.5% overall remission (OR) rate. The CR and OR rates were higher in the 10 mg/m² Ida group than the 8 mg/m² Ida group (CR: 78.9% vs 63.5%, P=0.003; OR: 88.2% vs 75.4%, P=0.007). Multivariate analysis showed that female, HGB≥100 g/L, FLT3-ITD mutation negative and 10 mg/m² Ida were favorable factors for CR. All patients presented cytopenias of grade Ⅳ. There was no differences on the recovery time of ANC≥0.5×10⁹/L and PLT≥20×10⁹/L after induction chemotherapy. Within a median follow-up of 14 (1-118) months, 98 (29.3%) patients relapsed, 92 (27.5%) died. The disease-free survival (DFS) and overall survival (OS) at 3 years were 53.2% and 58.9%, respectively. DFS and OS at 3-year were 34.2% and 37.4% in the chemotherapy cohort, 74.5% and 81.2% in the transplant cohort. 10 mg/m² Ida was an independent favorite factor for DFS (P=0.040) and OS (P=0.007). As compared to 8 mg/m², 10 mg/m² Ida significantly improved the CR, with the same extent of hematological side effects, and was an independent favorite factor for DFS and OS.
    Zhonghua xue ye xue za zhi = Zhonghua xueyexue zazhi 03/2015; 36(3):225-9. DOI:10.3760/cma.j.issn.0253-2727.2015.03.011
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    ABSTRACT: No-reflow is associated with an adverse outcome and higher mortality in patients with ST-segment elevation acute myocardial infarction (STEMI) who undergo percutaneous coronary intervention (PCI) and is considered a dynamic process characterized by multiple pathogenetic components. The aim of this study was to investigate the effectiveness of a combination therapy for the prevention of no-reflow in patient with acute myocardial infarction (AMI) undergoing primary PCI. A total of 621 patients with STEMI who underwent emergency primary PCI were enrolled in this study. Patients with high risk of no-reflow (no-flow score ≥ 10, by using a no-flow risk prediction model, n = 216) were randomly divided into a controlled group (n = 108) and a combination therapy group (n = 108). Patients in the controlled group received conventional treatment, while patients in combination therapy group received high-dose (80 mg) atorvastatin pre-treatment, intracoronary administration of adenosine (140 µg/min per kilogram) during PCI procedure, platelet membrane glycoprotein IIb/IIIa receptor antagonist (tirofiban, 10µg/kg bolus followed by 0.15 µg/kg per minute) and thrombus aspiration. Myocardial contrast echocardiography was performed to assess the myocardial perfusion 72 h after PCI. Major adverse cardiac events (MACE) were followed up for six months. Incidence of no-reflow in combination therapy group was 2.8%, which was similar to that in low risk group 2.7% and was significantly lower than that in control group (35.2%, P < 0.01). The myocardial perfusion (A × β) values were higher in combination therapy group than that in control group 72 h after PCI. After 6 months, there were six (6.3%) MACE events (one death, two non-fatal MIs and three revascularizations) in combination therapy group and 12 (13.2%) (four deaths, three non-fatal MIs and five revascularizations, P < 0.05) in control group. Combination of thrombus aspiration, high-dose statin pre-treatment, intracoronary administration of adenosine during PCI procedure and platelet membrane glycoprotein IIb/IIIa receptor antagonist reduce the incidence of no-reflow after primary PCI in patients with acute myocardial infarction who are at high risk of no-reflow.
    Journal of Geriatric Cardiology 03/2015; 12(2):135-42. DOI:10.11909/j.issn.1671-5411.2015.02.003 · 1.06 Impact Factor
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    ABSTRACT: Transmembrane TNF-α (tmTNF-α) acts both as a ligand, delivering 'forward signaling' via TNFR, and as a receptor, transducing 'reverse signaling'. The contradiction of available data regarding the effect of tmTNF-α on insulin resistance may be due to imbalance in both signals. Here, we demonstrated that high glucose-induced impairment of insulin-stimulated glucose uptake by 3T3-L1 adipocytes was concomitant with decreased tmTNF-α expression and increased soluble TNF-α (sTNF-α) secretion. However, when TACE was inhibited, preventing the conversion of tmTNF-α to sTNF-α, this insulin resistance was partially reversed, indicating a salutary role of tmTNF-α. Treatment of 3T3-L1 adipocytes with exogenous tmTNF-α promoted insulin-induced phosphorylation of IRS-1 and Akt, facilitated GLUT4 expression and membrane translocation, and increased glucose uptake while addition of sTNF-α resulted in the opposite effect. Furthermore, tmTNF-α downregulated the production of IL-6 and MCP-1 via NF-κB inactivation, as silencing of A20, an inhibitor for NF-κB, by siRNA, abolished this effect of tmTNF-α. However, tmTNF-α upregulated adiponectin expression through the PPAR-γ pathway, as inhibition of PPAR-γ by GW9662 abrogated both tmTNF-α-induced adiponectin transcription and glucose uptake. Our data suggest that tmTNF-α functions as an insulin sensitizer via forward signaling. Copyright © 2015. Published by Elsevier Ireland Ltd.
    Molecular and Cellular Endocrinology 02/2015; 406. DOI:10.1016/j.mce.2015.02.023 · 4.24 Impact Factor
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    ABSTRACT: Polo-like kinases (PLKs) family has long been known to be critical for cell cycle and recent studies have pointed to new dimensions of PLKs function in the nervous system. Our previous study has verified that the levels of PLK3 in the brain are severely down-regulated in prion-related diseases. However, the associations of PLKs with prion protein remain unclear. In the present study, we confirmed that PrP protein constitutively interacts with PLK3 as determined by both in vitro and in vivo assays. Both the kinase domain and polo-box domain of PLK3 were proved to bind PrP proteins expressed in mammalian cell lines. Overexpression of PLK3 did not affect the level of wild-type PrP, but significantly decreased the levels of the mutated PrPs in cultured cells. The kinase domain appeared to be responsible for the clearance of abnormally aggregated PrPs, but this function seemed to be independent of its kinase activity. RNA-mediated knockdown of PLK3 obviously aggravated the accumulation of cytosolic PrP. Moreover, PLK3-overexpression in a scrapie infected cell line caused notable reduce of PrP(Sc) level in a dose-dependent manner, but had minimal effect on the expression of PrP(C) in its normal partner cell line. Our findings here confirmed the molecular interaction between PLK3 and PrP and outlined the regulatory activity of PLK3 on the degradation of abnormal PrPs, even its pathogenic isoform PrP(Sc). We therefore assume that the recovery of PLK3 in the early-stage of prion infection may be helpful to prevent the toxic accumulation of PrP(Sc) in the brain tissues. Copyright © 2015. Published by Elsevier Ltd.
    The International Journal of Biochemistry & Cell Biology 02/2015; 62. DOI:10.1016/j.biocel.2015.02.011 · 4.24 Impact Factor

Publication Stats

5k Citations
1,529.97 Total Impact Points


  • 2014–2015
    • Zhejiang University
      Hang-hsien, Zhejiang Sheng, China
    • Chinese PLA General Hospital (301 Hospital)
      Peping, Beijing, China
    • Beijing Centers for Disease Control and Prevention
      Peping, Beijing, China
    • Zhejiang Gongshang University
      Hang-hsien, Zhejiang Sheng, China
    • The Third People's Hospital
      Shen-ch’üan-shih, Zhejiang Sheng, China
    • Chinese Center For Disease Control And Prevention
      Peping, Beijing, China
    • University of Science and Technology, Beijing
      Peping, Beijing, China
    • Anhui University
      Luchow, Anhui Sheng, China
    • 307 Hospital of the Chinese People's Liberation Army
      Peping, Beijing, China
    • Harbin Institute of Technology
      • School of Food Science and Engineering
      Charbin, Heilongjiang Sheng, China
  • 2013–2015
    • Stanford University
      • Department of Physics
      Palo Alto, California, United States
    • Hui Zhou University
      Kao-lan-hsien, Gansu Sheng, China
    • Wannan Medical College
      Wu-hu-shih, Anhui Sheng, China
    • Shanghai University of Traditional Chinese Medicine
      Shanghai, Shanghai Shi, China
    • Peking University School of Stomatology
      Peping, Beijing, China
    • Chinese Academy of Agricultural Sciences
      Peping, Beijing, China
    • College of Veterinary Sciences and Animal Husbandry
      Aizwal, Mizoram, India
    • University of Electronic Science and Technology of China
      Hua-yang, Sichuan, China
    • Zhejiang University of Technology
      Ch’u-chou, Zhejiang Sheng, China
    • University of Jinan (Jinan, China)
      Chi-nan-shih, Shandong Sheng, China
  • 2012–2015
    • National Center for Nanoscience and Technology
      Peping, Beijing, China
    • Lanzhou University
      • School of Stomatology
      Kao-lan-hsien, Gansu Sheng, China
    • Beijing Institute of Microbiology and Epidemiology
      Peping, Beijing, China
    • GuangDong University of Technology
      • Faculty of Environmental Science and Engineering
      Shengcheng, Guangdong, China
  • 2011–2015
    • Beijing Fuwai Hospital
      Peping, Beijing, China
    • Chinese Academy of Medical Sciences
      Peping, Beijing, China
    • The Chinese University of Hong Kong
      • Department of Information Engineering
      Hong Kong, Hong Kong
  • 2010–2015
    • University of Texas MD Anderson Cancer Center
      • Department of Molecular and Cellular Oncology
      Houston, Texas, United States
    • Northeast Normal University
      Hsin-ching, Jilin Sheng, China
    • Beijing University of Technology
      Peping, Beijing, China
    • Beijing Medical University
      • Department of Infectious Diseases
      Peping, Beijing, China
  • 2008–2015
    • Peking University Third Hospital
      Peping, Beijing, China
    • Dalian University of Technology
      • • State Key Laboratory of Structural Analysis for Industrial Equipment
      • • School of Chemical Engineering
      Lü-ta-shih, Liaoning, China
    • Chinese Academy of Sciences
      • • State Key Laboratory of Environmental Geochemistry
      • • Institute of Psychology
      • • Key Laboratory of Nutrition and Metabolism
      Peping, Beijing, China
    • Henan University
      • Institute of Immunology
      K’ai-feng-shih, Henan Sheng, China
    • Nanchang University
      Nan-ch’ang-shih, Jiangxi Sheng, China
  • 2007–2015
    • Southeast University (China)
      • Department of Cardiology
      Nan-ching-hsü, Jiangxi Sheng, China
    • University of the Sciences in Philadelphia
      Philadelphia, Pennsylvania, United States
    • Brigham and Women's Hospital
      • Department of Medicine
      Boston, Massachusetts, United States
  • 2006–2015
    • Tongji Medical University
      • Department of Immunology
      Wu-han-shih, Hubei, China
  • 2005–2015
    • Peking University People's Hospital
      Peping, Beijing, China
  • 1996–2015
    • Tsinghua University
      • Department of Electronic Engineering
      Peping, Beijing, China
  • 2013–2014
    • Wuhan Institute Of Virology
      Wu-han-shih, Hubei, China
    • Vanderbilt University
      • Department of Biomedical Informatics
      Нашвилл, Michigan, United States
    • Ningbo University
      Ning-po, Zhejiang Sheng, China
  • 2012–2014
    • Rice University
      • Department of Civil and Environmental Engineering
      Houston, Texas, United States
  • 2011–2014
    • Sun Yat-Sen University Cancer Center
      • Department of Radiation Oncology
      Shengcheng, Guangdong, China
    • Inner Mongolia University
      Suiyüan, Inner Mongolia, China
    • Tongji University
      • College of Material Science and Engineering
      Shanghai, Shanghai Shi, China
  • 2009–2014
    • Peking Union Medical College Hospital
      Peping, Beijing, China
    • Shanghai Jiao Tong University
      • Department of Electronic Engineering
      Shanghai, Shanghai Shi, China
    • Renmin University of China
      Peping, Beijing, China
    • West Georgia Obstetrics and Gynecology
      Georgetown, Georgia, United States
  • 2008–2014
    • Liaoning Research Institute of Family Planning
      Feng-t’ien, Liaoning, China
    • Beijing University of Chemical Technology
      Peping, Beijing, China
    • Huazhong Agricultural University
      • • National Key Laboratory of Crop Genetic Improvement
      • • College of Science
      • • College of Plant Sciences and Technology
      Wu-han-shih, Hubei, China
  • 2007–2014
    • Shenyang Pharmaceutical University
      • • School of Traditional Chinese Materia Medica
      • • School of Pharmaceutics
      • • College of Traditional Chinese Medicine
      • • Department of Pharmacy
      Feng-t’ien, Liaoning, China
    • Beijing University of Posts and Telecommunications
      • State Key Laboratory of Switching and Networking
      Peping, Beijing, China
    • Sun Yat-Sen University
      • State Key Laboratory of Oncology
      Shengcheng, Guangdong, China
    • Jilin University
      • • College of Chemistry
      • • Department of Chemistry
      Yung-chi, Jilin Sheng, China
    • Huazhong University of Science and Technology
      • • Department of Dermatology
      • • Department of Chemical Engineering
      • • State Key Laboratory of Coal Combustion (SKLCC)
      Wu-han-shih, Hubei, China
  • 2006–2014
    • Peking University
      • Institute of Hematology
      Peping, Beijing, China
  • 2012–2013
    • China-Japan Friendship Hospital
      Peping, Beijing, China
    • Chang'an University
      Xi’an, Guangdong, China
    • Anhui Medical University
      • Institute of Dermatology
      Luchow, Anhui Sheng, China
    • Chang Gung University
      Hsin-chu-hsien, Taiwan, Taiwan
  • 2010–2013
    • Beijing FivePlus Molecular Medicine Institute
      Peping, Beijing, China
    • Chongqing Center for Disease Control and Prevention
      Ch’ung-ch’ing-shih, Chongqing Shi, China
    • Sichuan University
      • • State Key Laboratory of Oral Diseases
      • • Department of Orthodontics
      Chengdu, Sichuan Sheng, China
  • 2009–2013
    • KTH Royal Institute of Technology
      • • School of Information and Communication Technology (ICT)
      • • Department of Microelectronics and Applied Physics (MAP)
      Tukholma, Stockholm, Sweden
  • 2006–2013
    • Chinese Academy of Inspection and Quarantine
      Peping, Beijing, China
  • 2010–2012
    • Capital Medical University
      • Department of Ophthalmology
      Peping, Beijing, China
  • 2008–2011
    • Beijing Genomics Institute
      Bao'an, Guangdong, China
  • 2008–2010
    • Shanghai Institutes for Biological Sciences
      Shanghai, Shanghai Shi, China
  • 2007–2010
    • Beijing Information Science and Technology University
      Peping, Beijing, China
  • 2008–2009
    • Chongqing University
      • Department of Industrial Engineering
      Ch’ung-ch’ing-shih, Chongqing Shi, China
  • 2005–2007
    • University of Pennsylvania
      • • School of Engineering and Applied Science
      • • Department of Mechanical Engineering and Applied Mechanics
      Philadelphia, Pennsylvania, United States
  • 2002–2007
    • National Tsing Hua University
      • Department of Electronic Engineering
      Hsin-chu-hsien, Taiwan, Taiwan