Dong-Seung Kang

Publications (3)9.61 Total impact

• Article: Combination of 1064-nm Q-switched neodymium: yttrium-aluminum-garnet laser with low fluence and 578-/511-nm copper bromide laser for nipple-areolar hyperpigmentation.

  Eui Hyung Lee, Jin-Soo Kang, Dong-Seung Kang, Choong Seop Han, Sung Bin Cho
  The Journal of Dermatology 01/2012; 39(1):110-2. · 1.49 Impact Factor
• Article: Facilitated scalp measuring using phototrichogram with a headband and tapeline.

  Eui Hyung Lee, Jin-Soo Kang, Dong-Seung Kang, Choong Seop Han, Sang Ho Oh, Sung Bin Cho
  [show abstract] [hide abstract]
  ABSTRACT: The authors have indicated no significant interest with commercial supporters.
  Dermatologic Surgery 05/2011; 37(8):1150-2. · 1.80 Impact Factor
• Article: Increased expression of endothelial cell adhesion molecules due to mediator release from human foreskin mast cells stimulated by autoantibodies in chronic urticaria sera.

  Kwang Hoon Lee, Ji Young Kim, Dong-Seung Kang, Yoo Jean Choi, Won-Jae Lee, Jai Youl Ro
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  ABSTRACT: Histamine-releasing antibodies that act against the epitope of the alpha chain of Fc(epsilon)RI (anti-Fc(epsilon)RI(alpha) antibody) that may affect pathogenesis in serum of patients with chronic urticaria. We assessed the capability of anti-Fc(epsilon)RI(alpha) antibody in sera from patients with chronic urticaria to release histamine and cytokines, and to induce the expression of endothelial cell adhesion molecules. We also assessed the release of inflammatory mediators from cultured foreskin mast cells, and expression of endothelial cell adhesion molecules on human dermal microvascular endothelial cells. Cells were pretreated with mast cell-conditioned media: culture media of mast cells treated with sera from chronic urticaria patients containing anti-Fc(epsilon)RI(alpha) antibody. Histamine release from human foreskin mast cells challenged with sera, increased after both 20 min and 16 h intervals. Leukotriene D4 release also increased at both 20 min and 16 h. Tumor necrosis factor-alpha increased significantly in foreskin mast cell culture challenged with sera of chronic urticaria patients. After the stimulation of human dermal microvascular endothelial cells with the conditioned media, the expression of intercellular cell adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin increased significantly. Treatment of the conditioned media with anti-tumor necrosis factor-alpha monoclonal antibody partially inhibited the expression of intercellular cell adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin. The data suggest that sera from patients with chronic urticaria containing anti-Fc(epsilon)RI(alpha) antibody release mediators and tumor necrosis factor-alpha by activating human foreskin mast cells. This release can play a pathogenic role in chronic urticaria by activating endothelial cells, in part due to the actions of tumor necrosis factor-alpha from mast cells.
  Journal of Investigative Dermatology 05/2002; 118(4):658-63. · 6.31 Impact Factor