Yuki Ishida

Publications (4)4.4 Total impact

• Article: [A case of Henoch-Schölein purpura with acute pancreatitis].

  Nana Nakayama, Shinji Nagata, Rie Kawase, Yuki Ishida, Takayasu Kuwabara, Takako Tsumura, Shigeru Kimura, Keiji Tsuji, Hiroaki Ogoshi, Mayumi Kaneko, Toru Hidaka
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  ABSTRACT: We report a 11-year-old girl who had abdominal pain, hematemesis and melena. On admission, endoscopy revealed multiple duodenal ulcers and abdominal computed tomography showed dilatation and wall thickness of the jejunum. Because she showed purpura on the lower extremities and a decreased serum level of the XIII factor, she was diagnosed as having Henoch-Schönlein purpura (HSP). On the clinical course, she had acute pancreatitis. Following administration of XIII factor, nafamostal mesilate and predonisolone, the abdominal pain disappeared. Eventually the acute pancreatitis was improved with improvement of purpura. According to this course, this acute pancreatitis was associated with HSP.
  Nippon Shokakibyo Gakkai zasshi The Japanese journal of gastro-enterology 03/2009; 106(2):240-6.
• Article: [A case of H. pylori and API2-MALT1 gene-negative gastric mucosa-associated lymphoid tissue lymphoma with a carcinoma-like signet-ring cell lymphoepithelial lesion].

  Tomonori Sumida, Shinji Nagata, Hiroaki Ohgoshi, Yuki Ishida, Takayasu Kuwabara, Takako Tsumura, Keiji Tsuji, Tohru Hidaka, Mayumi Kaneko, Tadashi Yoshino
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  ABSTRACT: A 60-year-old woman presented in February 2003 with an ulcer on the lesser curvature of the anglus. The endoscopic biopsy specimens showed epithelial signet-ring cell associated with lymphoid infiltration, suggesting a diagnosis of gastric cancer. Histopathological examination confirmed the diagnosis of low-grade B-cell lymphoma of MALT with epithelial signet-ring cell lymphoepithelial lesion, which was negative for H. pylori and t (11;18) (q21;q21) translocation (API2-MALT1 gene). This case was treated with H. pylori eradication and additional radiation therapy, and the tumor was disappeared.
  Nippon Shokakibyo Gakkai zasshi The Japanese journal of gastro-enterology 12/2006; 103(11):1257-63.
• Article: A case of multiple hepatic peribiliary cysts which contributed to the obstructive jaundice and led to liver failure at the young man with von Recklinghausen's disease.

  Haruna Yokomichi, Keiji Tsuji, Yuzo Hayashi, Mayumi Kaneko, Koichi Nakadoi, Yuki Ishida, Takayasu Kuwabara, Takako Tsumuna, Tomonori Sumida, Shinji Nagata, Hiroaki Ohgoshi, Sanshiro Uraki, Tohru Hidaka, Kazuaki Chayama
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  ABSTRACT: We treated a young man with obstructive jaundice, in whom multiple hepatic peribiliary cysts were detected at autopsy. He had been diagnosed with von Recklinghausen's disease in early childhood and had undergone surgery for a ventricular septal defect with massive blood transfusion at the age of 4 years. Examination at the age of 21 revealed prominent splenomegaly and a low platelet count. He underwent splenectomy and liver cirrhosis was confirmed by open biopsy of the liver. He was followed up at a local hospital and was admitted several times. When he was 41 years old, he was transferred to our hospital with severe jaundice and hepatic encephalopathy. Computed tomography showed marked dilation of the intrahepatic bile ducts and liver function tests showed that jaundice was mainly due to an increase of direct bilirubin. Despite endoscopic nasobiliary drainage, plasma exchange, and continuous hemodiafiltration, he died of hepatic failure after 5 days. At autopsy, multiple hepatic peribiliary cysts were found. Although peribiliary cysts have generally been considered to cause no symptoms, this report describes a patient with multiple hepatic peribiliary cysts which appear to have been responsible for the progression of obstructive jaundice.
  Hepatology Research 08/2006; 35(3):222-7. · 2.20 Impact Factor
• Article: A case of congenital absence of the portal vein.

  Keiji Tsuji, Kunio Naoki, Yoshiro Tachiyama, Akira Fukumoto, Yuki Ishida, Takayasu Kuwabara, Tomonori Sumida, Masana Tatsugami, Shinji Nagata, Hiroaki Ohgoshi, Sanshiro Uraki, Tohru Hidaka, Yukio Akagi, Masahiro Ishine, Shuji Saeki, Kazuaki Chayama
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  ABSTRACT: We experienced a girl with congenital absence of the portal vein. She was examined by computed tomography (CT), three-dimensional computed tomographic angiography (3DCTA), digital subtraction angiography and liver biopsy. Nodular regenerative hyperplasia of the liver was detected, presumably due to an abnormal hepatocellular response to absent portal flow. 3DCTA showed that the splenic vein and superior mesenteric vein joined to form a common trunk, which directly entered the right atrium. 3DCTA may be a valuable noninvasive tool for identifying portal malformations.
  Hepatology Research 02/2005; 31(1):43-47. · 2.20 Impact Factor