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Molecular Carcinogenesis 12/2008; 48(5):465-78. · 3.16 Impact Factor
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Molecular Carcinogenesis 08/2008; 47(7):554-71. · 3.16 Impact Factor
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Molecular Carcinogenesis 03/2008; 47(9):707-32. · 3.16 Impact Factor
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Molecular Carcinogenesis 07/2007; 46(6):415-35. · 3.16 Impact Factor
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Molecular Carcinogenesis 01/2006; 44(4):300-16. · 3.16 Impact Factor
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Molecular Carcinogenesis 04/2003; 36(3):101-14. · 3.16 Impact Factor
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Molecular Carcinogenesis 03/2002; 33(2):67-80. · 3.16 Impact Factor
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ABSTRACT: A method is described for the isolation of epithelial cells from normal human esophagus and the establishment of the epithelial cells in monolayer cell culture using irradiated 3T3 fibroblast feeder layers as a substrate. The monolayer cultures were subsequently used to study the effects of a tumor promoter and carcinogen on human esophageal epithelial cells in vitro.
Methods in Cell Science 01/1986; 10(4):227-232.
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ABSTRACT: Explants of adult human bronchus were cultured in CMRL-1066 medium supplemented with heat-inactivated fetal bovine serum,
hormones, antibiotics, and either 0.0, 0.1 1, or 10 mM putrescine. The outgrowth of bronchial epithelial cells was stimulated in medium containing 1 mM putrescine, a concentration that partially inhibited the outgrowth of fibroblasts. In medium containing 10 mM putrescine, the outgrowth of epithelial cells was similar to that observed in the control medium, but the outgrowth of fibroblasts
was completely inhibited for periods of at least 4 weeks. When 10 mM putrescine was added to cultures of bronchial fibroblasts, the fibroblasts were not killed. These results suggest that human
bronchial epithelial cells have a higher requirement for putrescine for growth than fibroblasts, and the molecular basis for
this observation is under investigation.
In Vitro Cellular & Developmental Biology - Plant 01/1980; 16(5):399-406. · 1.50 Impact Factor
