Jing Wang

University of Jinan (Jinan, China), Chi-nan-shih, Shandong Sheng, China

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Publications (633)960.86 Total impact

  • [Show abstract] [Hide abstract]
    ABSTRACT: Abbreviation of paced QRS duration has been taken as electrical resynchronization imposed by cardiac resynchronization therapy (CRT). However, little is known about alteration in native QRS duration and its correlation with therapeutic response as well as anatomical remodeling post-CRT.
    Journal of interventional cardiac electrophysiology : an international journal of arrhythmias and pacing. 09/2014;
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    ABSTRACT: : Systemic lupus erythematosus (SLE) is an autoimmune disease with uncertain pathogenesis. Endoplasmic reticulum (ER) stress has close correlations with inflammation and/or immune diseases. However, it is unknown whether aberrant ER stress is involved in SLE pathogenesis. We aimed to characterize the ER stress-related genes in patients with SLE and analyzed their correlations with the disease. Peripheral blood leucocytes were isolated from 76 well-characterized patients with SLE and 69 healthy controls. ER stress-related genes were determined at transcription level by absolute quantitative real-time polymerase chain reaction. Stepwise regression and correlation analysis were used to analyze the relationships between SLE disease and ER stress. Abnormal unfolded protein responses were found in patients with SLE with the downregulation of inositol-requiring enzyme 1 (IRE1), pancreatic ER kinase (PERK) and CCAAT/enhancer-binding protein homologous protein (CHOP) and upregulation of XBP1, XBP1s and MANF. In the patients with SLE disease activity index (SLEDAI) <12, PERK and MANF expressions were significantly decreased, compared with the patients with severe SLE (SLEDAI ≥12). However, there was no significant change in ATF6 mRNA expression in the patients with SLE. Negative correlation between IRE1/XBP1 and SLEDAI was observed in lower SLEDAI score group. Negative correlations between CHOP and anti-dsDNA antibody, MANF and antinuclear antibody were observed in high-SLEDAI score group. We also found that antinuclear antibody and anti-dsDNA antibodies correlated with SLEDAI in a weak positive manner. SLEDAI was negatively related with C3 level. SLEDAI and anti-dsDNA antibody showed modestly positive correlation with urine protein. These findings suggest that the abnormal unfolded protein responses, especially IRE1/XBP1 and PERK/CHOP axes, may contribute to SLE pathogenesis, which may be potential diagnosis indicators or treatment targets.
    The American Journal of the Medical Sciences 09/2014; · 1.33 Impact Factor
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    ABSTRACT: After exposure to the atmospheric-pressure air plasma at different discharge powers, the adhesion characteristics of Twaron aramid fibers were investigated. For the 12 s-300 W plasma treatment, the interlaminar shear strength of Twaron fiber reinforced thermoplastic poly(phthalazinone ether sulfone ketone) was increased from 46.0 to 61.7 MPa by 34.1%, and the diffusion of water molecule into the resulting composites was proved to be effectively prevented. These results showed that surface adhesive properties of the plasma-treated aramid fibers were improved. At the power level of 300 W, X-ray photoelectron spectroscopy analysis revealed the increases in concentrations of oxygen and nitrogen polar groups on the fiber surface, and atomic force microscopy observations led to the conclusion that the fiber surface morphology was changed and the surface roughness was greatly increased. These new polar and irregular surface structures accounted for the better adhesion between the fiber and the matrix, while due to the reasonability of this discharge power level applied to the surface modification, the measured fiber tensile strength only decreased by 2.0%. POLYM. COMPOS., 2014. © 2014 Society of Plastics Engineers
    Polymer Composites 09/2014; · 1.48 Impact Factor
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    ABSTRACT: To compare cardiovascular magnetic resonance (CMR) characteristics between arrhythmogenic right ventricular cardiomyopathy (ARVC) patients with syncope and without syncope and explore CMR parameters related with syncope. A consecutive series of 80 patients with ARVC were divided in two groups according to history of syncope prior to CMR examinations. The biventricular function and volumes were calculated and indexed by body surface area. Fatty infiltration and late-gadolinium enhancement (LGE) were self-quantitatively analyzed according to segmental model. Patients with syncope had statistically significant greater left ventricular end-diastolic volume index (LVEDVI) (79.6 ± 23.0 vs. 69.0 ± 17.9 mL/m(2), P = 0.030), right ventricular end-diastolic volume index (RVEDVI) (122.0 ± 30.0 vs. 107.4 ± 21.8 mL/m(2), P = 0.017), and LGE incidence (52.2 vs. 21.1 %, P = 0.006) than that of patients without syncope. Patients with syncope had a trend towards greater number of segments with LGE (8.6 ± 4.2 vs. 6.6 ± 3.1, P = 0.199) than that of patients without syncope in subgroup analyses of patients with LGE, but no statistical significance was reached. Multivariate regression analysis showed the presence of LGE was independently associated with syncope in patients with ARVC (odds ratios 8.827, 95 % confidence interval 1.945-40.068, P = 0.005). CMR is helpful in detection and management of the patients with ARVC. Patients with syncope had significantly higher LVEDVI, RVEDVI and LGE incidence, and larger studies with follow-up data are needed to elucidate the relationship between LGE and syncope in patients with ARVC.
    The international journal of cardiovascular imaging. 07/2014;
  • Chen Shen, Yupeng Sun, Jing Wang, Yun Lu
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    ABSTRACT: Carbon nanodots (CDs) of uniform size were prepared simply by the hydrothermal decomposition of folic acid (FA) precursor at various reaction temperatures. The CDs formed via dehydration of FA at 260 °C contributed the strongest photoluminescence (PL) signal and the highest quantum yield at about 68%, without assistance of any passivation agent. The effects of preparation conditions on PL behavior of CDs have been investigated in detail, and the quantum yield of the CDs was found to be associated strongly with sample crystallinity. Moreover, because the obtained CDs also exhibited high luminescence stability, abundant surface functional groups and good biocompatibility, there are many promising applications in printing ink, ion detection, pH sensors and cell imaging.
    Nanoscale 06/2014; · 6.73 Impact Factor
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    ABSTRACT: The variation of the substrates of verapamil-sensitive idiopathic left ventricular tachycardia (ILVT) was not understood. The purpose of this study was to investigate the variation of electroanatomic substrate [slow conduction zone (SCZ) and left ventricular conduction system (LVCS)] in ILVT and control individuals and markers of successful ablation.
    Journal of cardiovascular medicine (Hagerstown, Md.). 06/2014;
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    ABSTRACT: Background Fragmented QRS (fQRS) marks inhomogeneous activation and asynchronous cardiac contraction. It has been proved that cardiac resynchronization therapy (CRT) could reverse geometrical remodeling as well as correct electrical dyssynchrony. We aimed to investigate whether fQRS changed corresponding to the therapeutic response to CRT.Methods Patients who underwent de novo CRT implantation previously and had ≥1 follow-up between August 2012 and September 2013 in our hospital were investigated. Intrinsic electrocardiogram was recorded and fQRS in any lead was calculated. Response to CRT was defined as absolute improvement in left ventricular ejection fraction by ≥10% or by improvement >1 New York Heart Association class and without heart failure hospitalization.ResultsA total of 75 patients (48 male, mean ages, 61 ± 9 years) were included in this study. At a median follow-up of 13 months, 57 patients had response to CRT. Responders had narrowed QRS (from 167 ± 23 ms to 158 ± 19 ms, P = 0.003) and reduced fQRS post-CRT. Nonresponders had QRS prolonging (from 151 ± 26 ms to 168 ± 16 ms, P = 0.033) and increase in fQRS. Eleven of 12 patients with reduced fQRS were responders and 8 of 12 with increased fQRS were nonresponders. Both changes in QRS and fQRS correlated strongly with CRT response (r = 0.389, P = 0.001 and r = 0.403, P = 0.000, respectively). Reduction of fQRS in ≥1 leads had high specificity (95%) in association to responders, though in low sensitivity (19%).Conclusions The changes in fQRS associated with therapeutic response to CRT. Regression of fQRS could be a maker of electrical reverse remodeling following CRT.
    Annals of Noninvasive Electrocardiology 06/2014; · 1.08 Impact Factor
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    ABSTRACT: Manipulation of the organic coatings of nanoparticles such as quantum dots (QDs) to enhance specific applications may also affect their interaction and uptake by different organisms. In this study, poplar trees (Populus deltoides × nigra) were exposed hydroponically to 50-nM CdSe/CdZnS QDs coated with cationic polyethylenimine (PEI) (35.3 ± 6.6 nm) or polyethylene glycol of anionic poly(acrylic acid) (PAA-EG) (19.5 ± 7.2 nm) to discern how coating charge affects nanoparticle uptake, translocation and transformation within woody plants. Uptake of cationic PEI-QDs was 10 times faster despite their larger hydrodynamic size and higher extent of aggregation (17 times larger than PAA-EG-QDs after 11-day incubation in the hydroponic medium), possibly due to electrostatic attraction to the negatively charged root cell wall. QDs cores aggregated upon root uptake, and their translocation to poplar shoots (negligible for PAA-EG-QDs and 0.7 ng Cd/mg stem for PEI-QDs) was likely limited by the endodermis. After 2-day exposure, PEI and PAA-EG coatings were likely degraded from the internalized QDs inside the plant, leading to the aggregation of the metallic cores and a "red-shift" of fluorescence. The fluorescence of PEI-QD aggregates was stable inside the roots through the 11-day exposure period. In contrast, the PAA-EG-QD aggregates lost fluorescence inside the plant after 11 days probably due to destabilization of the coating, even though these QDs were stable in the hydroponic solution. Overall, these results highlight the importance of coating properties in the rate and extent to which nanoparticles are assimilated by plants and potentially introduced into food webs.
    Environmental science & technology. 05/2014;
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    ABSTRACT: Ecological evidence indicates a worldwide trend of dramatically decreased soil Ca(2+) levels caused by increased acid deposition and massive timber harvesting. Little is known about the genetic and cellular mechanism of plants' responses to Ca(2+) depletion. In this study, transcriptional profiling analysis helped identify multiple extracellular Ca(2+) ([Ca(2+) ]ext ) depletion-responsive genes in Arabidopsis thaliana L., many of which are involved in responses to other environmental stresses. Interestingly, a group of genes encoding putative cytosolic Ca(2+) ([Ca(2+) ]cyt ) sensors were significantly upregulated, implying that [Ca(2+) ]cyt has a role in sensing [Ca(2+) ]ext depletion. Consistent with this observation, [Ca(2+) ]ext depletion stimulated a transient rise in [Ca(2+) ]cyt that was negatively influenced by [K(+) ]ext , suggesting the involvement of a membrane potential-sensitive component. The [Ca(2+) ]cyt response to [Ca(2+) ]ext depletion was significantly desensitized after the initial treatment, which is typical of a receptor-mediated signaling event. The response was insensitive to an animal Ca(2+) sensor antagonist, but was suppressed by neomycin, an inhibitor of phospholipase C. Gd(3+) , an inhibitor of Ca(2+) channels, suppressed the [Ca(2+) ]ext -triggered rise in [Ca(2+) ]cyt and downstream changes in gene expression. Taken together, this study demonstrates that [Ca(2+) ]cyt plays an important role in the putative receptor-mediated cellular and transcriptional response to [Ca(2+) ]ext depletion of plant cells.
    Journal of Integrative Plant Biology 05/2014; · 3.75 Impact Factor
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    ABSTRACT: To analyze the endocardial, myocardial, and epicardial longitudinal systolic strain (LSsys) in the left ventricle (LV) segments and walls in patients with cardiac involvement due to primary amyloidosis (AL-CA) and hypertrophic cardiomyopathy (HCM).
    05/2014; 34(5):609-16.
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    ABSTRACT: Attention-deficit/hyperactivity disorder (ADHD) is a common childhood-onset psychiatric disorder with high heritability. In recent years, numerous molecular genetic studies have been published to investigate susceptibility loci for ADHD. These results brought valuable candidates for further research, but they also presented great challenge for profound understanding of genetic data and general patterns of current molecular genetic studies of ADHD since they are scattered and heterogeneous. In this review, we presented a retrospective review of more than 300 molecular genetic studies for ADHD from two aspects: (1) the main achievements of various studies were summarized, including linkage studies, candidate-gene association studies, genome-wide association studies and genome-wide copy number variation studies, with a special focus on general patterns of study design and common sample features; (2) candidate genes for ADHD have been systematically evaluated in three ways for better utilization. The thorough summary of the achievements from various studies will provide an overview of the research status of molecular genetics studies for ADHD. Meanwhile, the analysis of general patterns and sample characteristics on the basis of these studies, as well as the integrative review of candidate ADHD genes, will propose new clues and directions for future experiment design.
    Psychiatry research. 05/2014;
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    ABSTRACT: Bone metastases from hepatocellular carcinoma (HCC) seem to be increasing. Previous studies showed that soluble factors secreted by host cells and direct cell-to-cell interactions contributed to the preferential metastasis and growth of cancer cells in bone, while the underlying mechanism(s) of the metastasis of HCC in the bone are poorly understood. Here, we determined the effect of HS-5 cells on Huh7 cell proliferation, and investigated the role of CCL5 from HS-5 cells on the development of Huh7 cells. In addition, the underlying mechanisms on the influence in Huh7 cells were investigated. Our results showed that HS-5 cells could promote the proliferation, migration and invasion of Huh7 cells, and inhibited apoptosis. CCL5 downregulation was able to inhibit the effects of HS-5 cells on Huh7 cell migration and invasion via the PI3K-Akt signaling pathway and reduce MMP-2 expression. Therefore, these findings suggest that CCL5 secreted from MSCs can promote the migration and invasion of Huh7 cells and could be an important factor in HCC related to occurrence of bone metastases.
    International Journal of Oncology 05/2014; · 2.66 Impact Factor
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    ABSTRACT: To investigate differences in genetic risk factors for rheumatoid arthritis (RA) in Han Chinese as compared with Europeans. A genome-wide association study was conducted in China with 952 patients and 943 controls, and 32 variants were followed up in 2,132 patients and 2,553 controls. A transpopulation meta-analysis with results from a large European RA study was also performed to compare the genetic architecture across the 2 ethnic remote populations. Three non-major histocompatibility complex (non-MHC) loci were identified at the genome-wide significance level, the effect sizes of which were larger in anti-citrullinated protein antibody (ACPA)-positive patients than in ACPA-negative patients. These included 2 novel variants, rs12617656, located in an intron of DPP4 (odds ratio [OR] 1.56, P = 1.6 × 10(-21) ), and rs12379034, located in the coding region of CDK5RAP2 (OR 1.49, P = 1.1 × 10(-16) ), as well as a variant at the known CCR6 locus, rs1854853 (OR 0.71, P = 6.5 × 10(-15) ). The analysis of ACPA-positive patients versus ACPA-negative patients revealed that rs12617656 at the DPP4 locus showed a strong interaction effect with ACPAs (P = 5.3 × 10(-18) ), and such an interaction was also observed for rs7748270 at the MHC locus (P = 5.9 × 10(-8) ). The transpopulation meta-analysis showed genome-wide overlap and enrichment in association signals across the 2 populations, as confirmed by prediction analysis. This study has expanded the list of alleles that confer risk of RA, provided new insight into the pathogenesis of RA, and added empirical evidence to the emerging polygenic nature of complex trait variation driven by common genetic variants.
    Arthritis & rheumatology (Hoboken, N.J.). 05/2014; 66(5):1121-1132.
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    ABSTRACT: To observe Modified Zhisou Powder (MZP) on the lung function of chronic obstructive pulmonary disease (COPD) model rats of northwest China cold dryness syndrome (NCCDS).
    05/2014; 34(5):556-61.
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    ABSTRACT: Perfusion MRI has the potential to provide pathophysiological biomarkers for the evaluating, staging and therapy monitoring of prostate cancer. The objective of this study was to explore the feasibility of noninvasive arterial spin labeling (ASL) to detect prostate cancer in the peripheral zone and to investigate the correlation between the blood flow (BF) measured by ASL and the pharmacokinetic parameters Ktrans (forward volume transfer constant), kep (reverse reflux rate constant between extracellular space and plasma) and ve (the fractional volume of extracellular space per unit volume of tissue) measured by dynamic contrast-enhanced (DCE) MRI in patients with prostate cancer. Forty-three consecutive patients (ages ranging from 49 to 86 years, with a median age of 74 years) with pathologically confirmed prostate cancer were recruited. An ASL scan with four different inversion times (TI = 1000, 1200, 1400 and 1600 ms) and a DCE-MRI scan were performed on a clinical 3.0 T GE scanner. BF, Ktrans, kep and ve maps were calculated. In order to determine whether the BF values in the cancerous area were statistically different from those in the noncancerous area, an independent t-test was performed. Spearman's bivariate correlation was used to assess the relationship between BF and the pharmacokinetic parameters Ktrans, kep and ve. The mean BF values in the cancerous areas (97.1 ± 30.7, 114.7 ± 28.7, 102.3 ± 22.5, 91.2 ± 24.2 ml/100 g/min, respectively, for TI = 1000, 1200, 1400, 1600 ms) were significantly higher (p < 0.01 for all cases) than those in the noncancerous regions (35.8 ± 12.5, 42.2 ± 13.7, 53.5 ± 19.1, 48.5 ± 13.5 ml/100 g/min, respectively). Significant positive correlations (p < 0.01 for all cases) between BF and the pharmacokinetic parameters Ktrans, kep and ve were also observed for all four TI values (r = 0.671, 0.407, 0.666 for TI = 1000 ms; 0.713, 0.424, 0.698 for TI = 1200 ms; 0.604, 0.402, 0.595 for TI = 1400 ms; 0.605, 0.422, 0.548 for TI = 1600 ms). It can be seen that the quantitative ASL measurements show significant differences between cancerous and benign tissues, and exhibit strong to moderate correlations with the parameters obtained using DCE-MRI. These results show the promise of ASL as a noninvasive alternative to DCE-MRI. Copyright © 2014 John Wiley & Sons, Ltd.
    NMR in Biomedicine 05/2014; · 3.45 Impact Factor
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    ABSTRACT: B cell activation is regulated through the interplay of the BCR with the inhibitory coreceptor FcγRIIB and the activating coreceptor CD19. Recent studies suggest that Ag-driven BCR microclusters are efficiently converted to a signaling active state on colocalization with CD19 microclusters. Using total internal reflection fluorescence microscopy-based, high-resolution, high-speed live-cell and molecule imaging approaches, we show that when co-ligated to the BCR, the FcγRIIB can inhibit B cell activation by blocking the colocalization of BCR and CD19 microclusters within the B cell immunological synapse. Remarkably, this inhibitory function of FcγRIIB is dependent not on its well-characterized ITIM-containing cytoplasmic domain, but its transmembrane domain. Indeed, human primary B cells from systemic lupus erythematosus patients homozygous for gene encoding the loss-of-function transmembrane domain mutant FcγRIIB-I232T fail to block the synaptic colocalization of the BCR with CD19, leading to dysregulated recruitment of downstream signaling molecule p-PI3K to membrane proximal signalosome. This inhibitory function of FcγRIIB in impairing the spatial-temporal colocalization of BCR and CD19 microclusters in the B cell immunological synapse may help explain the hyper-reactive features of systemic lupus erythematosus patient B cells in reported studies. These observations may also provide new targets for therapies for systemic autoimmune disease.
    The Journal of Immunology 04/2014; · 5.52 Impact Factor
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    ABSTRACT: The incidence of stent restenosis (SR) has risen with as more patients are being treated with drug-eluting stents (DESs). Trimetazidine has multiple favorable effects on the cardiovascular system. Here, we aimed to evaluate whether chronic treatment with trimetazidine reduced the incidence of SR. From January 2009 to December 2011 at Chinese PLA General Hospital, 768 patients were enrolled and randomized into the trimetazidine treatment group (TG, n=384) and control group (CG, n=384). After DES implantation, all patients were treated with regular medication. In the TG, trimetazidine was administrated at 20mg tid for at least 30days. All patients received follow-up angiography 9-13months after discharge. Major adverse cardiac and cerebrovascular events (MACCEs) were recorded. Six hundred thirty-five patients were included in the final analysis (TG, n=312; CG, n=323). SR occurred in 49 (7.7%) patients. The TG had a lower incidence of SR compared to the CG (4.2% vs. 11.1%, p=0.001). At the 30-day follow-up, the TG exhibited a higher left ventricular ejection fraction than the CG (65.4±10.7 vs. 63.1±10.4, p=0.006). The incidence of MACCEs was also lower in the TG at the 1-year follow-up (6.1% vs. 10.8%, p=0.032). Further multivariate analysis revealed that trimetazidine treatment was a predictor for SR (OR: 0.376; 95% CI: 0.196-0.721; p=0.003). Trimetazidine treatment effectively reduced the incidence of SR and MACCEs after DES implantation at the 1-year follow-up.
    International journal of cardiology 04/2014; · 6.18 Impact Factor
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    ABSTRACT: To assess the value of E/(e'×s) in estimating left ventricular diastolic dysfunction in patients with coronary heart disease by dual Doppler echocardiograph.
    Zhonghua xin xue guan bing za zhi. 04/2014; 42(4):305-8.
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    ABSTRACT: The aim of this study is to determine the value of matrix metalloproteinase 9 (MMP-9) in diagnosis of osteosarcoma (OS). A systematic review and meta-analysis was conducted using Medline, Embase, ISI Web of Knowledge, the Cochrane Library, Scopus, BioMed Central, Science Direct, China Biomedical literature Database (CBM) and China National Knowledge Internet (CNKI) from inception through Aug 29, 2013. Articles written in English or Chinese that investigated the accuracy of MMP-9 for the diagnosis of OS were included. Pooled sensitivity, specificity and the area under the receiver operating characteristic curve (AUC) were determined. I(2) was used to test heterogeneity and source of heterogeneity was investigated by meta-regression (tested with Meta-DiSc and STATA 12.0 statistical software). A total of 3729 articles were retrieved, of which 18 were included, accounting for 892 patients. Overall, the pooled sensitivity, specificity and AUC were 0.78 (95% CI 0.730 - 0.83), 0.90 (95% CI 0.79 - 0.95), and 0.87 (95% CI 0.83 - 0.89), respectively. The studies had substantial heterogeneity (I(2)=84%, 95% CI 65 - 100)96%, 95% CI 94 - 99). Assay kit subgroup was the main source of the heterogeneity. Although MMP-9 was identified as a potential biomarker for OS, more studies were clearly needed to establish its diagnostic value.
    Clinica chimica acta; international journal of clinical chemistry 04/2014; · 2.54 Impact Factor
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    ABSTRACT: To evaluate the accuracy of the time interval between the onset of early transmitral flow velocity (E) and that of early diastolic mitral annular velocity (e') (TE-e') measured by dual Doppler echocardiography in the diagnosis of diastolic dysfunction. Seventy-seven coronary heart disease patients with preserved systolic function underwent a echocardiographic study. Early transmitral flow velocity E and early diastolic mitral annular velocity e' were simultaneously recorded by dual Doppler echocardiography. The E/e' and TE-e' were calculated and compared with the left ventricular end diastolic pressure (LVEDP), which was measured invasively. The validity of TE-e', E/e' and their combination was analyzed in estimating left ventricular dysfunction (LVEDP ≥12 mmHg). The single-beat E/e' and TE-e' were correlated with the LVEDP (r=0.79 and 0.42, respectively, P<0.001). Receiver-operating characteristic analysis showed that the optimal cut-off for TE-e' was 38 ms (sensitivity 54%, specificity 79%, AUC 0.71) and that for E/e' was 9.2 (sensitivity 74%, specificity 81%, AUC 0.87) for diagnosis of left ventricular diastolic dysfunction. The combined cut-offs of TE-e'≥38 ms and E/e'≥9.2 had a sensitivity of 100% and a specificity of 62% in diagnosing left ventricular diastolic dysfunction with an AUC of 0.96. In patients with coronary heart disease, the simultaneous recording of TE-e' by dual Doppler echocardiography can accurately estimate diastolic dysfunction, and its combination with E/e' can further improve the diagnostic accuracy.
    Nan fang yi ke da xue xue bao = Journal of Southern Medical University 03/2014; 34(3):349-53.

Publication Stats

2k Citations
960.86 Total Impact Points

Institutions

  • 2014
    • University of Jinan (Jinan, China)
      Chi-nan-shih, Shandong Sheng, China
    • Inner Mongolia University
      Suiyüan, Inner Mongolia, China
    • 307 Hospital of the Chinese People's Liberation Army
      Peping, Beijing, China
    • Chongqing Medical University
      Ch’ung-ch’ing-shih, Chongqing Shi, China
  • 2012–2014
    • Beijing University of Chemical Technology
      Peping, Beijing, China
    • Stanford University
      • Department of Physics
      Stanford, CA, United States
    • Beijing Medical University
      • Department of Radiology
      Peping, Beijing, China
    • Shenyang Pharmaceutical University
      Feng-t’ien, Liaoning, China
    • Anhui Medical University
      • Institute of Dermatology
      Luchow, Anhui Sheng, China
    • Beijing Institute of Microbiology and Epidemiology
      Peping, Beijing, China
  • 2011–2014
    • Tongji University
      • College of Material Science and Engineering
      Shanghai, Shanghai Shi, China
    • Beijing Fuwai Hospital
      Peping, Beijing, China
    • Dalian Ocean University
      Lü-ta-shih, Liaoning, China
  • 2010–2014
    • Chinese Academy of Sciences
      • Institute of Psychology
      Peping, Beijing, China
  • 2009–2014
    • Peking Union Medical College Hospital
      Peping, Beijing, China
    • Chinese Academy of Agricultural Sciences
      Peping, Beijing, China
    • École Polytechnique Fédérale de Lausanne
      • Polymers Laboratory
      Lausanne, Vaud, Switzerland
    • Renmin University of China
      Peping, Beijing, China
    • Shanghai Jiao Tong University
      • Department of Electronic Engineering
      Shanghai, Shanghai Shi, China
  • 2008–2014
    • Peking University
      • • Academy for Advanced Interdisciplinary Studies
      • • Department of Biomedical Engineering (BME)
      Peping, Beijing, China
    • Dalian University of Technology
      • School of Chemical Engineering
      Lü-ta-shih, Liaoning, China
    • Samsung
      Sŏul, Seoul, South Korea
    • Chongqing University
      • School of Mechanical Engineering
      Chongqing, Chongqing Shi, China
  • 2007–2014
    • Southeast University (China)
      • Department of Cardiology
      Nan-ching-hsü, Jiangxi Sheng, China
    • University of the Sciences in Philadelphia
      Philadelphia, Pennsylvania, United States
  • 2006–2014
    • Tongji Medical University
      • Department of Immunology
      Shanghai, Shanghai Shi, China
    • Harbin Institute of Technology
      • School of Food Science and Engineering
      Charbin, Heilongjiang Sheng, China
    • University of Nevada, Las Vegas
      • Department of Mechanical Engineering
      Las Vegas, NV, United States
  • 2013
    • Wannan Medical College
      Wu-hu-shih, Anhui Sheng, China
    • Shanghai University of Traditional Chinese Medicine
      Shanghai, Shanghai Shi, China
    • Chang Gung University
      Hsin-chu-hsien, Taiwan, Taiwan
    • China Agricultural University
      • College of Food Science and Nutritional Engineering
      Beijing, Beijing Shi, China
    • Hui Zhou University
      Kao-lan-hsien, Gansu Sheng, China
    • University of Electronic Science and Technology of China
      Hua-yang, Sichuan, China
    • Rice University
      • Department of Civil and Environmental Engineering
      Houston, TX, United States
    • Changchun Institute of Optics, Fine Mechanics and Physics
      Hsin-ching, Jilin Sheng, China
  • 2012–2013
    • Beijing University of Posts and Telecommunications
      • State Key Laboratory of Switching and Networking
      Peping, Beijing, China
  • 2011–2013
    • Tongji Hospital
      Wu-han-shih, Hubei, China
    • Sichuan University
      • • State Key Laboratory of Oral Diseases
      • • West China School of Stomatology
      Chengdu, Sichuan Sheng, China
  • 2010–2013
    • Huazhong Agricultural University
      • College of Science
      Wuhan, Hubei, China
  • 2009–2013
    • KTH Royal Institute of Technology
      • • School of Information and Communication Technology (ICT)
      • • Department of Microelectronics and Applied Physics (MAP)
      Tukholma, Stockholm, Sweden
  • 2008–2013
    • Huazhong University of Science and Technology
      Wu-han-shih, Hubei, China
  • 2006–2013
    • Chinese Academy of Inspection and Quarantine
      Peping, Beijing, China
  • 1–2013
    • Tsinghua University
      • Department of Electronic Engineering
      Peping, Beijing, China
  • 2011–2012
    • Lanzhou University
      • School of Stomatology
      Lanzhou, Gansu Sheng, China
    • Capital Medical University
      • • Department of Cardiology
      • • Department of Ophthalmology
      Beijing, Beijing Shi, China
  • 2008–2012
    • Henan University
      • • Laboratory of Cellular and Molecular Immunology
      • • Institute of Immunology
      Kaifeng, Henan Sheng, China
    • Nanchang University
      Nan-ch’ang-shih, Jiangxi Sheng, China
    • Liaoning Research Institute of Family Planning
      Feng-t’ien, Liaoning, China
  • 2007–2012
    • Jilin University
      • • College of Chemistry
      • • Department of Chemistry
      Yung-chi, Jilin Sheng, China
  • 2006–2012
    • Nanjing University
      • • School of Medicine
      • • Department of Chemical Engineering
      Nanjing, Jiangsu Sheng, China
  • 2005–2012
    • Northeast Institute of Geography and Agroecology
      • • Institute of Psychology
      • • Graduate School
      Beijing, Beijing Shi, China
  • 2010–2011
    • Harbin Medical University
      Charbin, Heilongjiang Sheng, China
    • Beijing FivePlus Molecular Medicine Institute
      Peping, Beijing, China
  • 2008–2011
    • Beijing Genomics Institute
      Bao'an, Guangdong, China
  • 2008–2009
    • Nokia Siemens Networks
      Esbo, Southern Finland Province, Finland
  • 2005–2007
    • University of Pennsylvania
      • • School of Engineering and Applied Science
      • • Department of Mechanical Engineering and Applied Mechanics
      Philadelphia, Pennsylvania, United States