Jing Wang

Zhejiang University, Hang-hsien, Zhejiang Sheng, China

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Publications (793)1468.3 Total impact

  • Wei Tang, Jing Wang
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    ABSTRACT: A facile and efficient hydrothermal route was used to synthesize flower-like ZnO. The morphology and composition of ZnO were characterized by X-ray diffraction (XRD) and scanning electron microscope (SEM). The XRD results that pure ZnO were formed without any other impurity peaks. Uniformly distributed flower-like ZnO were clearly observed from the SEM images. A possible growth model was proposed to simulate the formation of flower-like ZnO. The fabricated ZnO sensors exhibited good response to toluene than to other interfering gases such as acetone, ammonia and benzene. On the perspective of bond dissociation energy discrepancy, the good selectivity of the ZnO sensor to toluene was explained in detail. A reasonable sensing mechanism of ZnO sensor to toluene was proposed in the aspect of charge transfer.
    Sensors and Actuators B Chemical 02/2015; 207. · 3.84 Impact Factor
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    ABSTRACT: The manganese tangstenates (MnWO4) was explored for the first time for the oxide sensing electrode of the yttria-stabilized zirconia electrolyte (YSZ) based mixed-potential type sensor for detecting hydrogen at elevated temperatures. The MnWO4/YSZ/Pt sensor was found to have a good sensitivity to different concentrations of hydrogen from 80 ppm to 960 ppm in the background of 10% O2/N2 at 500 °C. The sensor also indicated excellent selectivity to several possible interferents such as CO, C3H6, C3H8 and NO2. The reproducibility and signal repeatability of the sensor and humidity influence were also studied.
    Sensors and Actuators B Chemical 01/2015; 206:176–180. · 3.84 Impact Factor
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    ABSTRACT: Reversible posterior leukoencephalopathy syndrome (RPLS) is a clinical syndrome based on changes in clinical imaging, and it has been reported to mainly occur in adults. However, it has been recently discovered that RPLS is also prevalent in infant patients, particularly in those using glucocorticoids, immunosuppressant medications and cytotoxic drugs. The current study presents a 5-year-old male with a previous diagnosis of systemic-onset juvenile idiopathic arthritis (SoJIA) and macrophage-activation syndrome who developed posterior reversible encephalopathy syndrome during treatment with glucocorticoids, disease-modifying antirheumatic drugs and biological agent (etanercept) therapy. After ~5 days of treatment, the patient made a complete clinical recovery; the magnetic resonance imaging reviewed 2 weeks later showed that the previous hyper-intensity signal had disappeared and the multiple lesions in the brain had been completely absorbed. The case report shows that, conforming to recent literature, SoJIA in infants should be considered a risk factor for developing RPLS. The clinical manifestations of the disease are multiple, but usually reversible, and the patients mostly have a good prognosis. Rapid diagnosis and treatment is essential as early treatment may prevent progression to irreversible brain damage. By increasing the awareness of RPLS, the patient care may improve and further insight may be gained.
    Biomedical reports. 01/2015; 3(1):55-58.
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    ABSTRACT: Renal insufficiency (RI) is significantly associated with clinical prognosis in patients with heart failure (HF), but direct evidences on the relation between renal function and clinical outcome in patients receiving cardiac resynchronization therapy (CRT) are limited. The aim of the current study was to systematically evaluate the association of baseline and 6-month renal function with cardiac reverse remodeling and long-term outcome after CRT. We retrospectively evaluated 190 consecutive patients who underwent CRT at Fuwai Hospital from January 2008 to April 2013. Renal function tests, echocardiographic measurement, and clinical parameters at baseline and after 6 months of CRT were performed. Primary endpoint events included all-cause mortality, cardiac transplantation, and unplanned hospitalizations for HF. At baseline, compared with normal renal function or mild RI (estimated glomerular filtration rate (eGFR) ≥60 ml×min(-1)×1.73 m(-2)), moderate-to-severe RI (eGFR <60 ml×min(-1)×1.73 m(-2)) exerted a negative influence on cardiac reverse remodeling parameters. At 6-month follow-up, 114 (60.0%) patients were classified as responders and showed significant renal function improvement, whereas renal function deteriorated in non-responders and subsequently 41 (25.6%) patients developed worsening renal function (WRF). During the mean follow-up of (24.3±17.1) months, both patients with baseline eGFR <60 ml×min(-1)×1.73 m(-2) and those with WRF experienced worse event-free survival (P < 0.01, respectively). This analysis identified that baseline eGFR as well as WRF after CRT were found to be independent determinants of the combined endpoints of all-cause mortality and HF-related hospitalizations in CRT recipients.
    Chinese medical journal. 12/2014; 127(23):4036-42.
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    ABSTRACT: Aberrant activation of the Wnt/beta-catenin signaling pathway is an important factor in the development of nasopharyngeal carcinoma (NPC). Previous studies have demonstrated that the developmental gene sex-determining region Y (SRY)-box 1 (SOX1) inhibits cervical and liver tumorigenesis by interfering with the Wnt/-catenin signaling pathway. However, the role of SOX1 in NPC remains unclear. This study investigates the function of SOX1 in NPC pathogenesis. A quantitative methylation-specific polymerase chain reaction revealed that SOX1 was down-regulated and its promoter was hypermethylated in NPC cell lines and tissues. Ectopic expression of SOX1 in NPC cells suppressed colony formation, proliferation and migration in vitro and impaired tumor growth in nude mice. Restoration of SOX1 expression significantly reduced epithelial-mesenchymal transition, enhanced cell differentiation and induced cellular senescence. Conversely, transient knockdown of SOX1 by siRNA in these cells partially restored cell proliferation and colony formation. Notably, SOX1 was found to physically interact with beta-catenin and reduce its expression independent of proteasomal activity, leading to inhibition of Wnt/beta-catenin signaling and decreased expression of downstream target genes. SOX1 decreases the expression of beta-catenin in a proteasome-independent manner and reverses the malignant phenotype in NPC cells.
    Molecular cancer. 11/2014; 13(1):257.
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    ABSTRACT: Dental pulp/dentin regeneration using dental stem cells combined with odontogenic factors may offer great promise to treat and/or prevent premature tooth loss. Here, we investigate if BMP9 and Wnt/β-catenin act synergistically on odontogenic differentiation. Using the immortalized SCAPs (iSCAPs) isolated from mouse apical papilla tissue, we demonstrate that Wnt3A effectively induces early osteogenic marker alkaline phosphatase (ALP) in iSCAPs, which is reduced by β-catenin knockdown. While Wnt3A and BMP9 enhance each other's ability to induce ALP activity in iSCAPs, silencing β-catenin significantly diminishes BMP9-induced osteo/odontogenic differentiation. Furthermore, silencing β-catenin reduces BMP9-induced expression of osteocalcin and osteopontin and in vitro matrix mineralization of iSCAPs. In vivo stem cell implantation assay reveals that while BMP9-transduced iSCAPs induce robust ectopic bone formation, iSCAPs stimulated with both BMP9 and Wnt3A exhibit more mature and highly mineralized trabecular bone formation. However, knockdown of β-catenin in iSCAPs significantly diminishes BMP9 or BMP9/Wnt3A-induced ectopic bone formation in vivo. Thus, our results strongly suggest that β-catenin may play an important role in BMP9-induced osteo/ondontogenic signaling and that BMP9 and Wnt3A may act synergistically to induce osteo/odontoblastic differentiation of iSCAPs. It's conceivable that BMP9 and/or Wnt3A may be explored as efficacious biofactors for odontogenic regeneration and tooth engineering.
    Biomaterials 11/2014; · 8.31 Impact Factor
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    ABSTRACT: One of the greatest obstacles to current cancer treatment efforts is the development of drug resistance by tumors. Despite recent advances in diagnostic practices and surgical interventions, many neoplasms demonstrate poor response to adjuvant or neoadjuvant radiation and chemotherapy. As a result, the prognosis for many patients afflicted with these aggressive cancers remains bleak. The insulin-like growth factor (IGF) signaling axis has been show to play critical role in the development and progression of various tumors. Many basic science and translational studies have shown that IGF pathway modulators can have promising effects when used to treat various malignancies. There also exists a substantial body of recent evidence implicating IGF signaling dysregulation in the dwindling response of tumors to current standard-of-care therapy. By better understanding both the IGF-dependent and –independent mechanisms by which pathway members can influence drug sensitivity, we can eventually aim to use modulators of IGF signaling to augment the effects of current therapy. This review summarizes and synthesizes numerous recent investigations looking at the role of the IGF pathway in drug resistance. We offer a brief overview of IGF signaling and its general role in neoplasia, and then delve into detail about the many types of human cancer that have been shown to have IGF pathway involvement in resistance and/or sensitization to therapy. Ultimately, our hope is that such a compilation of evidence will compel investigators to carry out much-needed studies looking at combination treatment with IGF signaling modulators to overcome current therapy resistance.
    Genes & Diseases. 11/2014;
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    ABSTRACT: T-wave alternans (TWA) represents myocardial instability. The present study was to determine the impact of cardiac resynchronization therapy (CRT) on TWA and left ventricular ejection fraction (LVEF) in heart failure patients.
    11/2014;
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    ABSTRACT: Although N-terminal pro-brain natriuretic peptide (NT-proBNP) is a useful screening test of impaired right ventricular (RV) function in conditions affecting the right-sided cardiac muscle, the role of NT-proBNP remains unclear in patients with arrhythmogenic right ventricular cardiomyopathy (ARVC). This study was designed to clarify the relation between the plasma NT-proBNP level and the RV function evaluated by cardiovascular magnetic resonance (CMR) imaging. We selected 56 patients with confirmed ARVC only when their blood specimens for NT-proBNP measurements were collected within 48 hours of a CMR scan. The NT-proBNP level was significantly higher in patients with RV dysfunction than in patients without RV dysfunction (median of 655.3 [interquartile range 556.4 to 870.0] vs 347.0 [interquartile range 308.0 to 456.2] pmol/L, p <0.001). The NT-proBNP levels were positively correlated with RV end-diastolic and end-systolic volume indices (r = 0.49 and 0.70, respectively) and negatively correlated with RV ejection fraction (r = -0.76, all p <0.001), which remained significant after adjustment for age, gender, and body mass index. The area under the receiver-operating characteristic curve for NT-proBNP was 0.91 (95% confidence interval 0.80 to 0.97, p <0.001). The cut-off value of NT-proBNP (458 pmol/L) was associated with sensitivity, specificity, and positive and negative predictive values of 91%, 89%, 67%, and 98%, respectively. In conclusion, NT-proBNP is a useful marker for the detection of RV dysfunction and associated with extent of RV dilatation and dysfunction determined by CMR in patients with ARVC. Copyright © 2014 Elsevier Inc. All rights reserved.
    The American journal of cardiology. 11/2014;
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    ABSTRACT: Abstract The treatment outcome and development of new mutations in imatinib- and/or nilotinib-failure Ph+ leukemia patients with highly nilotinib-resistant mutations (Y253H, E255K/V and F359V/C) were assessed on dasatinib. A total of 111 Ph+ leukemia patients were grouped into 3 cohorts by baseline BCR-ABL kinase domain mutation status: no mutation (n=44), non-nilotinib-resistant mutations (n=26), or nilotinib-resistant mutations (n=41). The frequencies of hematological, cytogenetic and molecular responses and clinical resistance to dasatinib were similar among the 3 cohorts during dasatinib therapy. In dasatinib-resistant patients, new mutations were most frequently observed in the cohort with nilotinib-resistant mutations (P=0.001). Multivariate analyses revealed that the advanced disease and harboring the Y253H or F359V mutation before dasatinib were independent predictors of developing new mutations. We concluded that Ph+ leukemia patients with the Y253H or F359V mutation have a high likelihood of developing new mutations in the setting of dasatinib resistance.
    Leukemia & lymphoma. 11/2014;
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    ABSTRACT: Based on ab initio calculations, we predict that a monolayer of Cr-doped (Bi,Sb)2Te3 and GdI2 heterostructure is a quantum anomalous Hall insulator with a non-trivial band gap up to 38 meV. The principle behind our prediction is that the band inversion between two topologically trivial ferromagnetic insulators can result in a non-zero Chern number, which offers a better way to realize the quantum anomalous Hall state without random magnetic doping. In addition, a simple effective model is presented to describe the basic mechanism of spin polarized band inversion in this system. Moreover, we predict that 3D quantum anomalous Hall insulator could be realized in (Bi2/3Cr1/3)2Te3/GdI2 superlattice.
    11/2014;
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    ABSTRACT: PIK3R3, an isoform of class IA phosphoinositide 3-kinase (PI3K), specifically interacts with cell proliferation regulators, such as retinoblastoma and proliferation cell nuclear antigen, to promote cell proliferation. However, the mechanisms behind the upstream signaling pathway of PIK3R3 remain unclear to date. This study showed that PIK3R3 expression was regulated by transforming growth factor-β (TGF-β) signaling and that PIK3R3 mediated the TGF-β-induced inhibition of lung adenocarcinoma cell proliferation. TGF-β down-regulated PIK3R3 expression in lung adenocarcinoma cells. However, this TGF-β-induced inhibition of cell proliferation can be attenuated by PIK3R3 overexpression. In addition, TGF-β can attenuate the transcriptional activity of NKX2.1, a transcription factor that binds to the promoter of PIK3R3. This result indicated that TGF-β regulated PIK3R3 expression by targeting NKX2.1. We confirmed the correlation between NKX2.1 and PIK3R3 in clinical samples. Therefore, the TGF-β/NKX2.1/PIK3R3 axis is crucial in the TGF-β-induced inhibition of cell proliferation, and the NKX2.1/PIK3R3 axis might become a target in TGF-β receptor-repressed lung adenocarcinoma. © 2014 Wiley Periodicals, Inc.
    Molecular Carcinogenesis 11/2014; · 4.27 Impact Factor
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    ABSTRACT: Background:The effect of adiposity on response to cardiac resynchronization therapy (CRT) and long-term outcome in patients undergoing CRT has not been previously reported. This study assessed the impact of baseline body mass index (BMI) on cardiac reverse remodeling and prognosis following CRT.Methods and Results:A total of 247 CRT patients were included and divided into 4 groups according to baseline BMI. During 6-month follow-up, overweight and obese patients (BMI, 24-28 kg/m(2), ≥28 kg/m(2), respectively) were inclined to have better clinical and echocardiographic improvements (P<0.05) as well as higher response rate (P<0.001) than underweight and normal weight patients (BMI, <18.5 kg/m(2), 18.5-24 kg/m(2), respectively). During long-term follow-up, overweight and obese patients had lower all-cause mortality (P=0.015) and combined endpoint of death or HF hospitalizations (P=0.001) than underweight and normal weight patients. Compared with normal weight patients, underweight patients had a 2.29-fold increase in risk of combined endpoint events whereas overweight and obese patients had a reduction in the risk of death (66% and 58%, respectively) and combined endpoint events (52% and 38%, respectively).Conclusions:Patients with obesity and overweight derived more benefit from CRT. Higher BMI was independently associated with better clinical outcome in CRT patients.
    Circulation journal : official journal of the Japanese Circulation Society. 10/2014;
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    ABSTRACT: To address issues of image authenticity verification and integrity protection, a fragile watermarking method for stereo image authentication is proposed with stereo matching technique. According to stereo matching of left and right images, non-overlapping blocks of left image are classified into non-matchable and matchable ones. Right image blocks and non-matchable blocks of left image are categorized into smooth, texture and complex blocks considering their roughness. Then, the corresponding alterable-length watermark is computed based on the results of block classification, and the destroyed blocks are recovered by using the corresponding alterable-length watermark. Besides, matchable tampered blocks of left image are recovered with the matched contents in right image. A detection technique with the part of alterable-length watermark is used to increase accuracy of tamper localization at the receiver. The disparity can be used to recover the tampered matchable block. Therefore, the quality of watermarked images is improved because of low embedding capacity. Additionally, the alterable-capacity watermark provides suitable information for smoother images with fewer bits and we obtain high quality restoration. Experimental results show that the proposed method can not only localize the tampered regions precisely but also recover the tampered regions with higher quality compared with the state-of-the-art methods.
    AEU - International Journal of Electronics and Communications 10/2014; · 0.55 Impact Factor
  • Organic Electronics 10/2014; 15(10):2408–2413. · 3.84 Impact Factor
  • Organic Electronics 10/2014; 15(10):2492–2498. · 3.84 Impact Factor
  • Source
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    ABSTRACT: The search for topologically non-trivial states of matter has become an important goal for condensed matter physics. Here, we give a theoretical introduction to the quantum anomalous Hall (QAH) effect based on magnetic topological insulators in two-dimension (2D) and three-dimension (3D). In 2D topological insulators, magnetic order breaks the symmetry between the counter-propagating helical edge states, and as a result, the quantum spin Hall effect can evolve into the QAH effect. In 3D, magnetic order opens up a gap for the topological surface states, and chiral edge state has been predicted to exist on the magnetic domain walls. We present the phase diagram in thin films of a magnetic topological insulator and review the basic mechanism of ferromagnetic order in magnetically doped topological insulators. We also review the recent experimental observation of the QAH effect. We discuss more recent theoretical work on the coexistence of the helical and chiral edge states, multi-channel chiral edge states, the theory of the plateau transition, and the thickness dependence in the QAH effect.
    09/2014;
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    ABSTRACT: Abbreviation of paced QRS duration has been taken as electrical resynchronization imposed by cardiac resynchronization therapy (CRT). However, little is known about alteration in native QRS duration and its correlation with therapeutic response as well as anatomical remodeling post-CRT.
    Journal of Interventional Cardiac Electrophysiology 09/2014; · 1.39 Impact Factor
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    ABSTRACT: : Systemic lupus erythematosus (SLE) is an autoimmune disease with uncertain pathogenesis. Endoplasmic reticulum (ER) stress has close correlations with inflammation and/or immune diseases. However, it is unknown whether aberrant ER stress is involved in SLE pathogenesis. We aimed to characterize the ER stress-related genes in patients with SLE and analyzed their correlations with the disease. Peripheral blood leucocytes were isolated from 76 well-characterized patients with SLE and 69 healthy controls. ER stress-related genes were determined at transcription level by absolute quantitative real-time polymerase chain reaction. Stepwise regression and correlation analysis were used to analyze the relationships between SLE disease and ER stress. Abnormal unfolded protein responses were found in patients with SLE with the downregulation of inositol-requiring enzyme 1 (IRE1), pancreatic ER kinase (PERK) and CCAAT/enhancer-binding protein homologous protein (CHOP) and upregulation of XBP1, XBP1s and MANF. In the patients with SLE disease activity index (SLEDAI) <12, PERK and MANF expressions were significantly decreased, compared with the patients with severe SLE (SLEDAI ≥12). However, there was no significant change in ATF6 mRNA expression in the patients with SLE. Negative correlation between IRE1/XBP1 and SLEDAI was observed in lower SLEDAI score group. Negative correlations between CHOP and anti-dsDNA antibody, MANF and antinuclear antibody were observed in high-SLEDAI score group. We also found that antinuclear antibody and anti-dsDNA antibodies correlated with SLEDAI in a weak positive manner. SLEDAI was negatively related with C3 level. SLEDAI and anti-dsDNA antibody showed modestly positive correlation with urine protein. These findings suggest that the abnormal unfolded protein responses, especially IRE1/XBP1 and PERK/CHOP axes, may contribute to SLE pathogenesis, which may be potential diagnosis indicators or treatment targets.
    The American Journal of the Medical Sciences 09/2014; · 1.33 Impact Factor
  • 09/2014; 35(9):842-844.

Publication Stats

4k Citations
1,468.30 Total Impact Points

Institutions

  • 2014
    • Zhejiang University
      • College of Pharmaceutical Sciences
      Hang-hsien, Zhejiang Sheng, China
    • Chongqing Medical University
      Ch’ung-ch’ing-shih, Chongqing Shi, China
    • Soochow University (PRC)
      • Department of Materials Science and Engineering
      Wu-hsien, Jiangsu Sheng, China
    • State Key Laboratory of Medical Genetics of China
      Ch’ang-sha-shih, Hunan, China
    • The Third People's Hospital
      Shen-ch’üan-shih, Zhejiang Sheng, China
  • 2012–2014
    • Beijing University of Chemical Technology
      Peping, Beijing, China
    • Stanford University
      • Department of Physics
      Palo Alto, California, United States
    • Xin Hua Hospital Affiliated to Shanghai Jiao Tong University School of Medicine
      Shanghai, Shanghai Shi, China
    • China Agricultural University
      • State Key Laboratory for Agrobiotechnology
      Beijing, Beijing Shi, China
    • Beijing Medical University
      • Department of Radiology
      Peping, Beijing, China
    • Nanchang University
      Nan-ch’ang-shih, Jiangxi Sheng, China
  • 2011–2014
    • Beijing Fuwai Hospital
      Peping, Beijing, China
    • Tongji University
      • College of Material Science and Engineering
      Shanghai, Shanghai Shi, China
  • 2009–2014
    • Peking Union Medical College Hospital
      Peping, Beijing, China
    • Shanghai Jiao Tong University
      • Department of Electronic Engineering
      Shanghai, Shanghai Shi, China
    • École Polytechnique Fédérale de Lausanne
      • Polymers Laboratory
      Lausanne, Vaud, Switzerland
    • Renmin University of China
      Peping, Beijing, China
    • Wuhan Union Hospital
      Wu-han-shih, Hubei, China
  • 2008–2014
    • Dalian University of Technology
      • School of Chemical Engineering
      Lü-ta-shih, Liaoning, China
    • Samsung
      Sŏul, Seoul, South Korea
    • Chongqing University
      • School of Mechanical Engineering
      Chongqing, Chongqing Shi, China
    • Huawei Technologies Co. Ltd.
      Bao'an, Guangdong, China
  • 2007–2014
    • Sun Yat-Sen University Cancer Center
      Shengcheng, Guangdong, China
  • 2006–2014
    • Tongji Medical University
      • Department of Immunology
      Shanghai, Shanghai Shi, China
    • Southeast University (China)
      • Department of Cardiology
      Nan-ching-hsü, Jiangxi Sheng, China
    • Peking University
      • • Academy for Advanced Interdisciplinary Studies
      • • Institute of Hematology
      Peping, Beijing, China
  • 2005–2014
    • Peking University People's Hospital
      Peping, Beijing, China
  • 2004–2014
    • Sun Yat-Sen University
      • • Department of Chemical Engineering
      • • State Key Laboratory of Oncology
      Shengcheng, Guangdong, China
    • Chinese Academy of Sciences
      • • Institute of Psychology
      • • Key Laboratory of Rare Earth Chemistry and Physics
      Peping, Beijing, China
  • 2013
    • Ningbo University
      Ning-po, Zhejiang Sheng, China
    • Wannan Medical College
      Wu-hu-shih, Anhui Sheng, China
    • Jiangxi University of Traditional Chinese Medicine
      Nan-ch’ang-shih, Jiangxi Sheng, China
    • Hui Zhou University
      Kao-lan-hsien, Gansu Sheng, China
    • University of Electronic Science and Technology of China
      Hua-yang, Sichuan, China
  • 2012–2013
    • Beijing University of Posts and Telecommunications
      • State Key Laboratory of Switching and Networking
      Peping, Beijing, China
  • 2011–2013
    • Sichuan University
      • • State Key Laboratory of Oral Diseases
      • • West China School of Stomatology
      Chengdu, Sichuan Sheng, China
    • Tongji Hospital
      Wu-han-shih, Hubei, China
    • Lanzhou University Second Hospital
      Kao-lan-hsien, Gansu Sheng, China
  • 2010–2013
    • Northeast Institute of Geography and Agroecology
      • Institute of Psychology
      Peping, Beijing, China
    • Huazhong Agricultural University
      • College of Science
      Wuhan, Hubei, China
    • Beijing University of Technology
      Peping, Beijing, China
    • Tianjin Medical University Cancer Institute and Hospital
      T’ien-ching-shih, Tianjin Shi, China
    • University of Missouri - Kansas City
      • Division of Cell Biology and Biophysics
      Kansas City, Missouri, United States
  • 2009–2013
    • KTH Royal Institute of Technology
      • • School of Information and Communication Technology (ICT)
      • • Department of Microelectronics and Applied Physics (MAP)
      Tukholma, Stockholm, Sweden
  • 2008–2013
    • Huazhong University of Science and Technology
      • State Key Laboratory of Coal Combustion (SKLCC)
      Wu-han-shih, Hubei, China
  • 2007–2013
    • Shenyang Pharmaceutical University
      • • College of Pharmacy
      • • School of Pharmaceutics
      • • Department of Pharmacy
      Feng-t’ien, Liaoning, China
    • Peking University Third Hospital
      Peping, Beijing, China
  • 1–2013
    • Tsinghua University
      • Department of Electronic Engineering
      Peping, Beijing, China
  • 2011–2012
    • Anhui Medical University
      • • Institute of Dermatology
      • • Department of Pediatrics
      Luchow, Anhui Sheng, China
    • Capital Medical University
      • Department of Ophthalmology
      Peping, Beijing, China
    • Jilin University
      • College of Physics
      Yung-chi, Jilin Sheng, China
    • Lanzhou University
      • School of Stomatology
      Lanzhou, Gansu Sheng, China
  • 2008–2012
    • Liaoning Research Institute of Family Planning
      Feng-t’ien, Liaoning, China
    • Henan University
      • • Laboratory of Cellular and Molecular Immunology
      • • Institute of Immunology
      Kaifeng, Henan Sheng, China
  • 2006–2012
    • Nanjing University
      • • School of Medicine
      • • Department of Chemical Engineering
      Nanjing, Jiangsu Sheng, China
  • 2010–2011
    • Dalian Ocean University
      Lü-ta-shih, Liaoning, China
    • Beijing FivePlus Molecular Medicine Institute
      Peping, Beijing, China
    • Harbin Medical University
      Charbin, Heilongjiang Sheng, China
  • 2009–2011
    • University of Texas MD Anderson Cancer Center
      • Department of Molecular and Cellular Oncology
      Houston, TX, United States
  • 2008–2011
    • Beijing Genomics Institute
      Bao'an, Guangdong, China
  • 2008–2009
    • Nokia Siemens Networks
      Esbo, Southern Finland Province, Finland