Kenshiro Shida

Publications (3)5.85 Total impact

• Article: [A case of carbon monoxide poisoning by explosion of coal mine presenting as visual agnosia: re-evaluation after 40 years].

  Akiko Takaiwa, Kenichiro Yamashita, Takuo Nomura, Kenshiro Shida, Takayuki Taniwaki
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  ABSTRACT: We re-evaluated a case of carbon monoxide poisoning presenting as visual agnosia who had been injured by explosion of Miike-Mikawa coal mine 40 years ago. In an early stage, his main neuropsychological symptoms were visual agnosia, severe anterograde amnesia, alexia, agraphia, constructional apraxia, left hemispatial neglect and psychic paralysis of gaze, in addition to pyramidal and extra pyramidal signs. At the time of re-evaluation after 40 years, he still showed visual agnosia associated with agraphia and constructional apraxia. Concerning visual agnosia, recognition of the real object was preserved, while recognition of object photographs and picture was impaired. Thus, this case was considered to have picture agnosia as he could not recognize the object by pictorial cues on the second dimensional space. MRI examination revealed low signal intensity lesions and cortical atrophy in the bilateral parieto-occipital lobes on T1-weighted images. Therefore, the bilateral parieto-occipital lesions are likely to be responsible for his picture agnosia.
  Nō to shinkei = Brain and nerve 12/2005; 57(11):997-1002.
• Source

  Available from: Shozo Tobimatsu

  Article: Electrophysiological correlates of associative visual agnosia lesioned in the ventral pathway.

  Takao Yamasaki, Takayuki Taniwaki, Shozo Tobimatsu, Kenji Arakawa, Hiroshi Kuba, Yoshihisa Maeda, Yasuo Kuwabara, Kenshiro Shida, Yasumasa Ohyagi, Takeshi Yamada, Jun-ichi Kira
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  ABSTRACT: Visual agnosia has been well studied by anatomical, neuropsychological and neuroimaging studies. However, functional changes in the brain have been rarely assessed by electrophysiological methods. We carried out electrophysiological examinations on a 23-year-old man with associative visual agnosia, prosopagnosia and cerebral achromatopsia to evaluate the higher brain dysfunctions of visual recognition. Electrophysiological methods consisted of achromatic, chromatic and category-specific visual evoked potentials (CS-VEPs), and event-related potentials (ERPs) with color and motion discrimination tasks. Brain magnetic resonance imaging revealed large white matter lesions in the bilateral temporo-occipital lobes involving the lingual and fusiform gyri (V4) and inferior longitudinal fasciculi due to multiple sclerosis. Examinations including CS-VEPs demonstrated dysfunctions of face and object perception while sparing semantic word perception after primary visual cortex (V1) in the ventral pathway. ERPs showed abnormal color perception in the ventral pathway with normal motion perception in the dorsal pathway. These electrophysiological findings were consistent with lesions in the ventral pathway that were detected by clinical and neuroimaging findings. Therefore, CS-VEPs and ERPs with color and motion discrimination tasks are useful methods for assessing the functional changes of visual recognition such as visual agnosia.
  Journal of the Neurological Sciences 07/2004; 221(1-2):53-60. · 2.35 Impact Factor
• Article: Computerized tomographies of 34 patients at the chronic stage of acute carbon monoxide poisoning

  Eisuke Kono, Ryosuke Kono, Kenshiro Shida
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  ABSTRACT: The brains of 34 patients at the chronic stage of acute carbon monoxide poisoning (CO poisoning) were examined using computerized tomography (CT). Ventricular and sulcal dilatations were measured quantitatively, with picture analysis of CT for the measurement of ventricular dilatation. Significant ventricular and sulcal dilatations were found in all cases of the CO group compared with age-matched controls, and bilateral low density areas in the globus pallidus were seen in 9 of the patients. There were significant correlations between duration of initial unconsciousness and the ventricular dilatation or cortical atrophy. Such dilatations were considered to be due to the cerebral damage in the acute stage.
  European Archives of Psychiatry and Clinical Neuroscience 08/1983; 233(4):271-278. · 3.49 Impact Factor