Dietary Sugars Intake and Cardiovascular Health A Scientific Statement From the American Heart Association

Circulation (Impact Factor: 14.95). 09/2009; 120(11):1011-20. DOI: 10.1161/CIRCULATIONAHA.109.192627
Source: PubMed

ABSTRACT High intakes of dietary sugars in the setting of a worldwide pandemic of obesity and cardiovascular disease have heightened concerns about the adverse effects of excessive consumption of sugars. In 2001 to 2004, the usual intake of added sugars for Americans was 22.2 teaspoons per day (355 calories per day). Between 1970 and 2005, average annual availability of sugars/added sugars increased by 19%, which added 76 calories to Americans' average daily energy intake. Soft drinks and other sugar-sweetened beverages are the primary source of added sugars in Americans' diets. Excessive consumption of sugars has been linked with several metabolic abnormalities and adverse health conditions, as well as shortfalls of essential nutrients. Although trial data are limited, evidence from observational studies indicates that a higher intake of soft drinks is associated with greater energy intake, higher body weight, and lower intake of essential nutrients. National survey data also indicate that excessive consumption of added sugars is contributing to overconsumption of discretionary calories by Americans. On the basis of the 2005 US Dietary Guidelines, intake of added sugars greatly exceeds discretionary calorie allowances, regardless of energy needs. In view of these considerations, the American Heart Association recommends reductions in the intake of added sugars. A prudent upper limit of intake is half of the discretionary calorie allowance, which for most American women is no more than 100 calories per day and for most American men is no more than 150 calories per day from added sugars.

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Available from: Rachel K Johnson, Aug 29, 2014
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    • "Sucrose was chosen as the reinforcer to both extend the results of these previous studies with cocaine and gain a better understanding of the neurobiology of food-directed relapse. Given the severity of the obesity epidemic (CDC 2014; WHO 2015), its associated negative health outcomes (Ogden et al. 2007), and also other negative outcomes related to excess food consumption (e.g., sugar; Johnson et al. 2009), further evaluation of factors that maintain feeding behaviors is warranted. In addition, we included both acute (overnight) and chronic (1 month) EE conditions to examine how the extent of EE and/or length of abstinence from self-administration affects neuronal activity. "
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    Brain Structure and Function 06/2015; DOI:10.1007/s00429-015-1074-z · 4.57 Impact Factor
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    • "Similarly, in studies where participants consumed larger portions of sugar-sweetened beverages alongside food, they did not decrease the amount of food consumed to compensate for the increased energy from the beverage (Flood, Roe, & Rolls, 2006; Vartanian, Schwartz, & Brownell, 2007). Evidence suggests that the calories from sugar-sweetened beverages are poorly regulated by the body, so additional portions of sugar-sweetened beverages may uniquely result in a significant increase in total energy intake (Flood et al., 2006; Johnson et al., 2009). Although governments are fundamentally involved in the regulation of many aspects of the food supply, U.S. policies and programs related to serving sizes for children vary or are not clearly communicated . "
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    Appetite 12/2014; 88. DOI:10.1016/j.appet.2014.12.003 · 2.69 Impact Factor
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    • "Among them, behavioral (e.g., smoking , nutrition, and exercise) and pathological (e.g., metabolic syndrome, type 2 diabetes, and dyslipidemia) factors can be pointed out. Epidemiological evidences have shown associations between dietary sugar-intake and increased risk for developing metabolic syndrome [4], type 2 diabetes [5], obesity [5] [6], and body adiposity [7], and the pathophysiology of these complications includes ectopic fat deposition with glucotoxic and lipotoxic actions [8]. To our knowledge there are few studies evaluating direct effects on LPO accessed by plasma MDA concentrations in humans. "
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