Sleep and quality of well-being

Department of Psychiatry, University of California San Diego, USA.
Sleep (Impact Factor: 5.06). 01/2001; 23(8):1115-21.
Source: PubMed

ABSTRACT It is commonly believed that sleep duration in the population has been declining gradually. Whereas sleep restriction in the laboratory induces sleepiness and mood disturbances, it is not certain whether a short sleep duration impairs the quality of everyday life.
Using population-based data, we explored whether greater habitual sleep duration is a predictor of better health-related quality of life, measured by the Quality of Well-Being (QWB) scale. The relationships between QWB and several potential correlates were examined in a stepwise linear regression analysis.
Neither subjective nor actigraphic sleep duration were associated with QWB. Greater quality of well-being was associated with greater sleep satisfaction, younger age, less obesity, non-Hispanic White ethnicity, and greater experienced illumination.
These data suggest that increasing sleep duration may not directly improve quality of life, despite evidence that curtailment of nocturnal sleep is associated with fatigue.

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    • "However, our finding that psychomotor slowness was even more pronounced among long sleepers than among short sleepers calls for an explanation. One possible explanation is that long self-reported sleep duration reflects a behaviourally defined long time in bed (Jean-Louis et al., 2000; Kripke, 2004), which, in turn, may reflect a compensatory response to a failure in sleep function(s ). An alternative, but not necessarily exclusionary, explanation is that a long self-reported sleep duration represents a surrogate of pathological processes (Gangwisch et al., 2008), and consequently is a component of the individualÕs subjective symptom formation (Kronholm et al., 2009). "
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    ABSTRACT: Short and long self-reported sleep durations have been found to be associated with several seemingly disparate health risks and impaired functional abilities, including cognitive functioning. The role of long sleep is especially poorly understood in this context. Psychomotor slowness, shown to have analogous associations with cognitive performance and health risks as self-reported long sleep duration, has not been studied together with sleep duration in epidemiological settings. We hypothesized that self-reported habitual sleep duration, especially long sleep, is associated with slow psychomotor reaction time, and that this association is independent of vigilance-related factors. The hypothesis was tested in a sample of 5352 individuals, representing the general adult population. We found a U-shaped association between self-reported sleep duration and psychomotor speed, which prevailed even after controlling for several pertinent confounders. This novel finding can be interpreted to mean that self-reported sleep duration, at least in the case of long sleep, is an indicator of bodily/brain integrity and, taken together with the results of cognitive epidemiology, may provide some new insights into the mechanisms underlying the associations between habitual self-reported sleep duration, health risks and impaired functional abilities.
    Journal of Sleep Research 12/2011; 20(2):288-97. DOI:10.1111/j.1365-2869.2010.00899.x · 2.95 Impact Factor
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    • "Humans spend nearly one third of each day sleeping, and it is a daily process of physiologic restitution and recovery. An epidemiologic study reported a decline in sleep duration over the past few decades by 1.5 to 2 hours [1], with about one third of adults reporting that they sleep less than 6 hours per night, meaning that we live in a sleep-deprived society. U-shaped patterns have been observed for the relationships between sleep duration and all-cause mortality [2] [3] [4], coronary heart disease [5], hypertension [6] [7], obesity [7] [8], and diabetes [9] [10] [11] [12] [13]. "
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    ABSTRACT: U-shaped patterns have been observed for the relationship between sleep duration and diabetes. In addition, prediabetes is associated with the risk of cardiovascular diseases and diabetes. However, there are few studies investigating the relationship between sleep duration and prediabetes/newly diagnosed diabetes. The aim of this study is to examine the relationship between sleep duration and prediabetes/newly diagnosed diabetes in a Taiwanese population. After excluding the subjects with a high risk of obstructive sleep apnea, those with a positive history of diabetes, or those taking hypnotic drugs, a total of 3470 adults were recruited from a health checkup center. Each subject completed a self-administrated structured questionnaire on sleep duration and lifestyle factors. Prediabetes/diabetes was defined following the definition of the American Diabetes Association. Subjects with different sleep durations were classified into short (<6.0 hours), normal (6.0∼8.49 hours), and long sleepers (≥8.5 hours). The proportion of subjects with normal glucose tolerance, prediabetes, and newly diagnosed diabetes was 71.9%, 22.9%, and 5.2%, respectively. There were significant differences in age, sex, weight, education level, body mass index, waist-to-hip ratio, systolic and diastolic blood pressure, alcohol and coffee drinking habits, family history of diabetes, and sleep duration among the 3 glycemic groups. In multinomial regression, both short and long sleepers had a higher risk of newly diagnosed diabetes; and the odds ratio were 1.55 (95% confidence interval, 1.07-2.24) and 2.83 (1.19-6.73), respectively. However, sleep duration was not found to relate to prediabetes. In conclusion, both short and long sleep durations were independently associated with newly diagnosed diabetes, but not with prediabetes.
    Metabolism: clinical and experimental 06/2011; 60(6):799-804. DOI:10.1016/j.metabol.2010.07.031 · 3.61 Impact Factor
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    • "This may modulate the association between sleep characteristics and health risks across different countries. For example, in the United States sleep duration may have decreased during the last decades more strikingly [32] "
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    ABSTRACT: The U-shaped association of self-reported sleep duration with all-cause mortality is generally accepted. Findings on cardiovascular (CVD) mortality and morbidity are inconsistent. We aimed to further clarify the associations of the self-reported sleep duration with CVD mortality and morbidity. In two population based surveys in 1972 and 1977 the levels of coronary risk factors in Finland and habitual sleep duration were measured; 25,025 individuals were followed-up until 2006 by the national register data. The outcome variables were death (for any reason), CVD death, and non-fatal CVD event (non-fatal myocardial infarction or stroke). Participants with former non-fatal CVD event at baseline were excluded from CVD analyses, and socio-demographic and health-related confounders were considered in the final Cox proportional hazard models for both genders. The U-shaped association of total mortality with self-reported sleep duration was confirmed in both genders. The association of CVD mortality with self-reported sleep duration was independent of pertinent cardiovascular risk factors in women. The highest CVD mortality risk was found in both extreme ends of sleep duration distribution (⩽5 and ⩾10h sleepers). Sleep duration is an independent risk factor for CVD mortality and morbidity in women but not in men. The highest CVD mortality risk is associated with the extreme ends of sleep duration distribution. Thus, in epidemiological studies, combining adjacent (6 and 9h) sleep duration groups with the extreme groups may partly mask the mortality risks, especially in the long run.
    Sleep Medicine 02/2011; 12(3):215-21. DOI:10.1016/j.sleep.2010.07.021 · 3.10 Impact Factor
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