Awards and achievements (6) View all

  • Feb 2014
    NIH F32 NRSA Fellowship
  • Jun 2013
    F.A.S.E.B. Ion Channel Conference Travel Award
  • Dec 2012
    William N. Pearson Fellowship


Perturbation to the function of the human cardiac sodium channel (NaV1.5) can result in arrhythmias and in some cases stroke or death. Inactivation of the channel utilizes specific elements of the NaV1.5 C-terminus and calmodulin to translate changes in intracellular calcium. Combining structural biology and electrophysiology this research will elucidate important molecular details of this process and provide insight into the molecular basis of specific cardiac life threatening arrhythmias.

5 Following View all

6 Followers View all