Circulation Cardiovascular Imaging Journal Impact Factor & Information

Publisher: American Heart Association, American Heart Association

Current impact factor: 5.32

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 5.316
2013 Impact Factor 6.752
2012 Impact Factor 5.795
2011 Impact Factor 5.941
2010 Impact Factor 4.757
2009 Impact Factor 1.783

Impact factor over time

Impact factor

Additional details

5-year impact 6.00
Cited half-life 3.30
Immediacy index 1.37
Eigenfactor 0.02
Article influence 2.63
Other titles Circulation / Cardiovascular imaging., Cardiovascular imaging
ISSN 1942-0080
OCLC 316253218
Material type Periodical, Internet resource
Document type Internet Resource, Journal / Magazine / Newspaper

Publisher details

American Heart Association

  • Pre-print
    • Archiving status unclear
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • If required by funding agency or institutional policy
    • 6 months embargo
  • Conditions
    • On Institutional repository or funding agency repository
    • On a non-profit server
    • Publisher's version/PDF cannot be used
    • Publisher will automatically deposit authors post-print in PubMed Central for NIH funded authors after 12 months
    • Publisher will automatically deposit authors post-print in PubMed Central for HHMI and Wellcome Trust funded authors after 6 months
    • Authors may place a 'toll-free' link to teir article on authors' personal website or institutional website without embargo
    • Publisher last contacted on 11/07/2014
  • Classification

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Eletter: Circumferential Strain: The Deja vu in Aortic Stenosis">Aitzaz BS Rai Farrukh Munir, Ehtesham ul Haq, Ahmed Waqas, Sara Bughio, Anum Faisal Khan Green Templeton College, UNIVERSITY of Oxford We read the article with interest1. It is an important contribution to the field. However, we would like to SUGGEST two things. 1) Aortic stenosis causes concentric hypertrophy or sometimes eccentric hypertrophy depending upon flow vortices direction and ventricular anatomy2. In such a situation the concentric muscle fibres would also be involved in addition to longitudinal muscle fibres. In addition to global longitudinal strain (GLS), circumferential strain and possibly radial strain may have predictive value. Though we acknowledge that radial strain has less reproducibility and robustness. 2) Myocardial twist and torsion would also be useful indices to look for. 3) Circumferential strain affects endothelial shear stress and nitric oxide synthase and several other pathways3. 4) GLS does not affect the entire electrical phenomenon and cardiac electrical, mechanical and metabolic remodelling, energetic changes i.e. PCR/ATP ratio, Vascular territories on invasive imaging involve full myocardial thickness. Nevertheless, it's an important contribution to science of aortic stenosis. References: 1. Dahou A, Bartko PE, Capoulade R, Clavel MA, Mundigler G, Grondin SL, Bergler-Klein J, Burwash I, Dumesnil JG, Senechal M, O'Connor K, Baumgartner H, Pibarot P. Usefulness of global left ventricular longitudinal strain for risk stratification in low ejection fraction, low-gradient aortic stenosis: results from the multicenter true or pseudo-severe aortic stenosis study. Circ Cardiovasc Imaging. 2015;8:e002117. 2. Devereux RB, Alonso DR, Lutas EM, Gottlieb GJ, Campo E, Sachs I, Reichek N. Echocardiographic assessment of left ventricular hypertrophy: comparison to necropsy findings. Am J Cardiol. 1986;57:450-8. 3. Qiu Y, Tarbell JM. Interaction between wall shear stress and circumferential strain affects endothelial cell biochemical production. J Vasc Res. 2000;37:147-57. Conflict of Interest:
    Circulation Cardiovascular Imaging 04/2016;
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    Circulation Cardiovascular Imaging 12/2015; 8(1). DOI:10.1161/CIRCIMAGING.114.002970
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    ABSTRACT: Hemodynamically significant coronary artery disease is an important indication for revascularization. Stress myocardial perfusion imaging is a noninvasive alternative to invasive fractional flow reserve for evaluating hemodynamically significant coronary artery disease. The aim was to determine the diagnostic accuracy of myocardial perfusion imaging by single-photon emission computed tomography, echocardiography, MRI, positron emission tomography, and computed tomography compared with invasive coronary angiography with fractional flow reserve for the diagnosis of hemodynamically significant coronary artery disease. The meta-analysis adhered to the Preferred Reporting Items for Systematic Reviews and Meta-analyses statement. PubMed, EMBASE, and Web of Science were searched until May 2014. Thirty-seven studies, reporting on 4721 vessels and 2048 patients, were included. Meta-analysis yielded pooled sensitivity, pooled specificity, pooled likelihood ratios (LR), pooled diagnostic odds ratio, and summary area under the receiver operating characteristic curve. The negative LR (NLR) was chosen as the primary outcome. At the vessel level, MRI (pooled NLR, 0.16; 95% confidence interval [CI], 0.13-0.21) was performed similar to computed tomography (pooled NLR, 0.22; 95% CI, 0.12-0.39) and positron emission tomography (pooled NLR, 0.15; 95% CI, 0.05-0.44), and better than single-photon emission computed tomography (pooled NLR, 0.47; 95% CI, 0.37-0.59). At the patient level, MRI (pooled NLR, 0.14; 95% CI, 0.10-0.18) performed similar to computed tomography (pooled NLR, 0.12; 95% CI, 0.04-0.33) and positron emission tomography (pooled NLR, 0.14; 95% CI, 0.02-0.87), and better than single-photon emission computed tomography (pooled NLR, 0.39; 95% CI, 0.27-0.55) and echocardiography (pooled NLR, 0.42; 95% CI, 0.30-0.59). Stress myocardial perfusion imaging with MRI, computed tomography, or positron emission tomography can accurately rule out hemodynamically significant coronary artery disease and can act as a gatekeeper for invasive revascularization. Single-photon emission computed tomography and echocardiography are less suited for this purpose. © 2015 American Heart Association, Inc.
    Circulation Cardiovascular Imaging 12/2015; 8(1). DOI:10.1161/CIRCIMAGING.114.002666

  • Circulation Cardiovascular Imaging 12/2015; 8(1). DOI:10.1161/CIRCIMAGING.114.002636
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    ABSTRACT: Background: In sepsis, whether the assessment of left ventricular global longitudinal systolic strain (GLS) is feasible and prognostically relevant remains controversial. Methods and results: Consecutive patients admitted to a high-dependency observational unit with sepsis or septic shock were evaluated. Left ventricular ejection fraction (EF) by planimetry and peak GLS by 2D speckle tracking were available at admission in 115 of 149 (77%) patients. Compared with patients included in the study, those excluded (n=34, 23%) showed higher proportion of chronic obstructive pulmonary disease (P<0.01), but with comparable clinical characteristics and mortality rates. GLS showed lowest variability for low EF and highest for higher EF. By day-28 follow-up, all-cause mortality was 30% (n=34 and n=19 within 7 days from hospitalization). GLS and EF were both more abnormal in deceased than in those alive by day-28 follow-up (both P<0.05, findings consistent using day-7 follow-up data). GLS showed a borderline relationship with mortality by day-28 follow-up (hazard ratio 1.16/%, P=0.05), whereas EF did not (hazard ratio 0.99/%, P=0.63) accounting for age; the lack of association of all-cause mortality with EF was consistent at day-7 follow-up (hazard ratio 0.94/%, P=0.9), whereas more abnormal GLS correlated significantly with higher mortality rate (hazard ratio 1.30/%, P=0.03) independent to age. Conclusions: In patients with sepsis assisted in a high-dependency observational unit, feasibility of assessments of left ventricular EF and GLS within 24 h from the hospitalization was acceptable and EF showed no prognostic relevance, whereas GLS showed a correlation with mortality rate potentially relevant in shorter more than in longer follow-ups.
    Circulation Cardiovascular Imaging 11/2015; 8(11). DOI:10.1161/CIRCIMAGING.115.003865
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    ABSTRACT: Echocardiography and fluoroscopy are the main techniques for prosthetic heart valve (PHV) evaluation, but because of specific limitations they may not identify the morphological substrate or the extent of PHV pathology. Cardiac computed tomography (CT) and magnetic resonance imaging (MRI) have emerged as new potential imaging modalities for valve prostheses. We present an overview of the possibilities and pitfalls of CT and MRI for PHV assessment based on a systematic literature review of all experimental and patient studies. For this, a comprehensive systematic search was performed in PubMed and Embase on March 24, 2015, containing CT/MRI and PHV synonyms. Our final selection yielded 82 articles on surgical valves. CT allowed adequate assessment of most modern PHVs and complemented echocardiography in detecting the obstruction cause (pannus or thrombus), bioprosthesis calcifications, and endocarditis extent (valve dehiscence and pseudoaneurysms). No clear advantage over echocardiography was found for the detection of vegetations or periprosthetic regurgitation. Whereas MRI metal artifacts may preclude direct prosthesis analysis, MRI provided information on PHV-related flow patterns and velocities. MRI demonstrated abnormal asymmetrical flow patterns in PHV obstruction and allowed prosthetic regurgitation assessment. Hence, CT shows great clinical relevance as a complementary imaging tool for the diagnostic work-up of patients with suspected PHV obstruction and endocarditis. MRI shows potential for functional PHV assessment although more studies are required to provide diagnostic reference values to allow discrimination of normal from pathological conditions.
    Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003703

  • Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003885

  • Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003637
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    ABSTRACT: To prevent left ventricular dysfunction (LVD), surgery is recommended in patients with severe primary mitral regurgitation as soon as ejection fraction (EF) ≤60% or LV end-systolic diameter ≥40 mm. However, LVD may be concealed behind preoperative normal LVEF and LV end-systolic diameter. We sought to identify whether a new composite echocardiographic Doppler marker of the LV ejection according to the LV dilatation may predict postoperative LVD and outcome after mitral valve repair in patients with primary mitral regurgitation. Between 1991 and 2010, patients who underwent mitral valve repair for primary mitral regurgitation were studied. From preoperative echocardiography, we calculated LV ejection index (LVEI) using following formula: LVEI=indexed LV end-systolic diameter/LV outflow tract time-velocity integral. In the 278 patients included, the best correlation with postoperative LVEF was found with LVEI (r=-0.40; P<0.0001), even in patients with preoperative LVEF≥60% (r=-0.46; P<0.0001). In multivariable analysis, LCEI>1.13 was an independent predictor of postoperative LVD (P<0.0001). During a mean follow-up of 10±4.6 years, 67 (29%) deaths occurred. When compared with patients with preserved LVEI, those with LVEI>1.13 had significantly lower both survival and cardiac death-free survival (P=0.017 and P=0.008, respectively). Similar results were found in patients with preoperative LVEF≥60% (P=0.049 and P=0.016, respectively). In Cox proportional hazard model, after meticulous adjustment for cofactors, LVEI>1.13 remains independently associated with death (hazard ratio, 1.64; P=0.039) and cardiac-related death (hazard ratio, 3.27; P=0.026). After mitral valve repair for primary mitral regurgitation, the preoperative LVEI is a new and simple composite parameter of both LV dilatation and LV forward flow able to accurately predict postoperative LVD and outcome. © 2015 American Heart Association, Inc.
    Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.114.003036

  • Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003995
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    ABSTRACT: Left ventricular (LV) mechanical discoordination, often referred to as dyssynchrony, is often observed in patients with heart failure regardless of QRS duration. We hypothesized that different myocardial substrates for LV mechanical discoordination exist from (1) electromechanical activation delay, (2) regional differences in contractility, or (3) regional scar and that we could differentiate electromechanical substrates responsive to cardiac resynchronization therapy (CRT) from unresponsive non-electrical substrates. First, we used computer simulations to characterize mechanical discoordination patterns arising from electromechanical and non-electrical substrates and accordingly devise the novel systolic stretch index (SSI), as the sum of posterolateral systolic prestretch and septal systolic rebound stretch. Second, 191 patients with heart failure (QRS duration ≥120 ms; LV ejection fraction ≤35%) had baseline SSI quantified by automated echocardiographic radial strain analysis. Patients with SSI≥9.7% had significantly less heart failure hospitalizations or deaths 2 years after CRT (hazard ratio, 0.32; 95% confidence interval, 0.19-0.53; P<0.001) and less deaths, transplants, or LV assist devices (hazard ratio, 0.28; 95% confidence interval, 0.15-0.55; P<0.001). Furthermore, in a subgroup of 113 patients with intermediate electrocardiographic criteria (QRS duration of 120-149 ms or non-left bundle branch block), SSI≥9.7% was independently associated with significantly less heart failure hospitalizations or deaths (hazard ratio, 0.41; 95% confidence interval, 0.23-0.79; P=0.004) and less deaths, transplants, or LV assist devices (hazard ratio, 0.27; 95% confidence interval, 0.12-0.60; P=0.001). Computer simulations differentiated patterns of LV mechanical discoordination caused by electromechanical substrates responsive to CRT from those related to regional hypocontractility or scar unresponsive to CRT. The novel SSI identified patients who benefited more favorably from CRT, including those with intermediate electrocardiographic criteria, where CRT response is less certain by ECG alone. © 2015 American Heart Association, Inc.
    Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003744

  • Circulation Cardiovascular Imaging 09/2015; 8(9). DOI:10.1161/CIRCIMAGING.115.003985