Audiological Medicine

Publisher: Società italiana di audiologia, Informa Healthcare

Journal description

The study of audiological medicine is evolving, creating a collaboration with other physicians involved in the diagnosis and treatment of communication disorders. Audiological Medicine has been conceived from the inspiration of the International Association of Physicians in Audiology. The journal aims to provide not only a forum for clinical audiologists in continuing medical education, but also to a wider scientific fields including professionals within communication disorders. The journal will publish 4 issues in its first year. The volume will be divided as 2 "regular" issues and 2 "special " issues. A regular issue will contain not only research papers, but also a basic science review paper, an editorial, one radiological case, one clinical case, a historical note, a technical note, a pathological note, book reviews, letters to the Editor and the news of IAPA and other audiological societies. The special issues will be monographic issues. Topics will include: Genetics of hearing; Inner ear pharmacology, inner ear plasticity; Tinnitus; toneurological evaluation of children; Otoneurological evaluation of head trauma; Standardization of vestibular tests. Audiological Medicine has been selected as the official journal of the Società ltaliana di Audiologia (SIA).

Current impact factor: 0.00

Impact Factor Rankings

Additional details

5-year impact 0.00
Cited half-life 0.00
Immediacy index 0.00
Eigenfactor 0.00
Article influence 0.00
Website Audiological Medicine website
Other titles Audiological medicine (Online)
ISSN 1651-3835
OCLC 56974446
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

Informa Healthcare

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • On author's personal website or institution website
    • Publisher copyright and source must be acknowledged
    • On a non-profit server
    • Must link to publisher version
    • Publisher's version/PDF cannot be used
    • NIH funded authors may post articles to PubMed Central for release 12 months after publication
    • Wellcome Trust authors may deposit in Europe PMC after 6 months
  • Classification
    ​ yellow

Publications in this journal

  • Source
    [Show abstract] [Hide abstract]
    ABSTRACT: OBJECTIVE: In chinchillas, moderate doses of carboplatin administered systemically selectively destroy inner hair cells and type I vestibular hair cells; however, it is unclear whether this unique damage pattern persists if carboplatin is applied directly to the cochlea, how quickly the damage develops and what cell death pathways are involved. STUDY DESIGN: To address these questions, carboplatin (5 mg/ml, 50 µl) was applied to the round window. RESULTS: Carboplatin caused a rapid decline in distortion product otoacoustic emissions, significantly increased compound action potential thresholds and caused massive inner hair cell loss and less severe outer hair cell loss. Hair cell loss was initially more severe in the base than the apex of the cochlea, but by 28 days post-treatment most cochlear hair cells were missing and hair cell density in the utricle, saccule and lateral crista was greatly reduced. At one day post-treatment, many hair cell nuclei were condensed or fragmented indicative of apoptosis, and expressed initiator caspase-8 and executioner caspase-3, but not initiator caspase-9. Carboplatin-treated animals circled towards the treated ear and during the swim test rolled towards the treated ear. CONCLUSION: These results indicate that local application of carboplatin causes loss of hair cells that begins near the base of the cochlea and spreads towards the apex with increasing survival time. Hair cell loss is initiated by caspase-8 followed by executioner caspase-3.
    Audiological Medicine 12/2009; 7(4):189-199. DOI:10.3109/16513860903335795
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    ABSTRACT: Abstract The scans of two patients with lipomas of the internal auditory canal are presented. The first patient was referred to our department from a local hospital where a vestibular schwannoma had been diagnosed following a magnetic resonance (MR) scan. A subsequent MR scan suggested the presence of an intracanalicular lipoma rather than a vestibular schwannoma as previously diagnosed. This case illustrates the importance of performing unenhanced T1 weighted (T1W) axial sequences prior to giving gadolinium. Fat suppression (FAT SAT) scan techniques are also helpful in differentiating lipomas from vestibular schwannomas which have indistinguishable clinical presentations (Dalley et al, 1986; Olson et al, 1978). The second patient was a 60-year-old lady who presented with vertigo, reduced hearing, tinnitus and a sensation of pressure in her right ear. An MR scan showed the presence of an intracanalicular lipoma. Conservative management was adopted for both patients. In case 1 there has been no evidence of any growth of the lesion on MR scans over a seven year period and only a slight deterioration in his hearing thresholds. There was no previous imaging of the second patient, but there has been no significant alteration in her hearing since her initial assessment seven years previously. A repeat MR scan performed one year after the initial diagnosis showed no change in the size of the lesion. There have been only three previously reported cases of lipomas of the internal auditory meatus (IAM)/cerebello-pontine angle (CPA) with documented radiologic follow-up of five years or more, one of which showed a 15% increase in size at 8 years (Bigelow et al, 1998). This brings the total number of cases reports with long- term radiologic follow-up to four.
    Audiological Medicine 08/2002; 11(2):110.
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    ABSTRACT: Quinine induces a reversible cochlear hearing loss. We are reporting a study where stable plasma concentrations (total 12-24 mol/l, free 1.4-4 mol/l) were maintained in a healthy volunteer using a computer controlled infusion pump. Concentration levels were maintained for long periods of time, allowing detailed audiologic testing throughout the dynamic range during pronounced hearing threshold shifts. 1. The pure tone threshold shift was almost flat over 1000 4000 Hz, with a maximum of 46 dB at 4000 Hz at the highest concentration achieved. 2. The evoked otoacoustic emissions shifted in accord with pure tone shift. 3. The overall slope of the input output function of the emissions changed from 0.35 to 1.1 at the most. 4. The stapedius reflex thresholds were unaffected. 5. The brain stem audiometry findings changed in agreement with a recruiting cochlear hearing loss. 6. Upward spread of masking changed substantially. 7. The hearing loss could be modelled to the total and free plasma quinine concentration, according to an exponential model previously suggested by us. This study further supports the view that quinine exerts its reversible hearing impairment by interfering with the function of the outer hair cell.
    Audiological Medicine 01/1995; 4:12-24.