Diseases of Aquatic Organisms Journal Impact Factor & Information

Publisher: Inter Research

Journal description

Diseases affect all facets of life - at the cell, tissue, organ, individual, population and ecosystem level. Since life originated in an aquatic medium, studies of disease phenomena in the wide array of aquatic taxa contribute significantly to the analysis, comprehension, prevention and treatment of diseases in general, including those of organisms now inhabiting terrestrial environments and of humans. DAO aims to fully cover these important research areas

Current impact factor: 1.75

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 1.752
2013 Impact Factor 1.586
2012 Impact Factor 1.734
2011 Impact Factor 2.201
2010 Impact Factor 1.572
2009 Impact Factor 1.687
2008 Impact Factor 1.586
2007 Impact Factor 1.598
2006 Impact Factor 1.509
2005 Impact Factor 1.361
2004 Impact Factor 1.583
2003 Impact Factor 1.263
2002 Impact Factor 1.561
2001 Impact Factor 1.653
2000 Impact Factor 1.556
1999 Impact Factor 1.515
1998 Impact Factor 1.213
1997 Impact Factor 1.183
1996 Impact Factor 1.181
1995 Impact Factor 1.071
1994 Impact Factor 0.906
1993 Impact Factor 0.948
1992 Impact Factor 1.123

Impact factor over time

Impact factor

Additional details

5-year impact 1.99
Cited half-life >10.0
Immediacy index 0.38
Eigenfactor 0.01
Article influence 0.59
Website Diseases of Aquatic Organisms website
Other titles Diseases of aquatic organisms (Online)
ISSN 1616-1580
OCLC 44210633
Material type Periodical, Internet resource
Document type Internet Resource, Journal / Magazine / Newspaper

Publisher details

Inter Research

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Author's pre-print on author's personal website, institutional website or institutional repository
    • Author's post-print on author's personal website or institutional website
    • Author's post-print on institutional repository after 12 months embargo
    • Authors covered by funding agency rules, may post author's post-print in PubMed Central after a 6 months embargo
    • If mandated by a funding agency or institution, the author's post-print may be deposited in institutional repository before 12 months, as long addendum is submitted
    • Publisher's version/PDF may be used after 5 years embargo
    • Publisher copyright and source must be acknowledged
    • Reviewed 31/01/2014
  • Classification
    ​ green

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: The dugong Dugong dugon is classified as Vulnerable to extinction but may be endangered in some regions. Cause of death in stranded dugongs has not been determined in a large proportion of animals examined, with investigations hindered by limited information on dugong health and diseases, and paucity of knowledge of common or endemic pathological findings. Here we describe pathological findings in harvested dugongs from the relatively pristine area of central Torres Strait, and we characterise lesions attributable to drowning. Other recorded lesions were mild and predominated by host reaction to the presence of trematodes within the gastrointestinal tracts, liver and pancreas. Ascarid worm burdens were low in comparison to dugongs from developed coastlines. Hepatocellular lipofuscin and ferritin pigmentation were commonly observed, more pronounced in livers of older animals and concurrent with periportal and bridging fibrosis. Lesions attributable to drowning included incomplete collapse of lungs, dorsal or diffuse pulmonary congestion, mild intra-alveolar haemorrhage and oedema, mild interstitial oedema and rupture of peripheral alveolar septae with acute myofibre fragmentation and degeneration. No accumulation of foam or aspiration of water or particulate matter was observed, suggesting that dugongs 'dry drown'. Morphometric features of normal spleen are also presented. Characterisation of common pathological findings and those attributable to drowning in this species will aid in the interpretation of post mortem findings for the significant number of dugongs found deceased along urbanised coastlines.
    Diseases of Aquatic Organisms 03/2015; 113(2):89-102. DOI:10.3354/dao02825
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    ABSTRACT: As part of a state-wide multispecies survey of amphibian diseases, sampling was conducted at Archbold Biological Station, Venus, Florida, USA, on 15 April 2011. Gross examination of southern toad (Anaxyrus terrestris) larvae was unremarkable, but infections by a mesomycetozoean-like organism were observed in longitudinally sectioned routine haematoxylin and eosin-stained histologic slides. In 100% of the sectioned specimens examined (n = 5), a high density of the organism, representing several developmental stages, was found in the central nervous system, mainly in the spinal cord, brain, retina and optic nerve. No host inflammatory responses were found to be associated with the parasitic infection. Free, mature schizonts were occasionally found in the gill chamber and, more commonly, in the dorsal roof area. No organisms were found in other organs examined histologically, i.e. liver, kidney, heart, alimentary tract, exocrine pancreas and skeletal muscles. Presumptive mesomycetozoean ichthyophonids in anurans are usually reported to be pathogenic, especially affecting skeletal muscle tissue and causing death. To our knowledge, this is the first report of a similar organism infecting primarily the central nervous system in an amphibian.
    Diseases of Aquatic Organisms 03/2015; 113(2):157-162. DOI:10.3354/dao02836
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    ABSTRACT: Fish herpesviruses and their hosts may have coevolved for 400 to 450 million yr. During this coexistence, the hosts have equipped themselves with an elaborate immune system to defend themselves from invading viruses, whereas the viruses have developed strategies to evade host immunity, including the expression of cytokine genes that have been captured from the host. Taking advantage of our experimental model for cyprinid herpesvirus 3 (CyHV-3) persistence in carp, we studied the gene expression of host and virus immune-related genes in each stage of infection: acute, persistent and reactivation phases. IFNγ-1, IFNγ-2, IL-12 and IL-10 host genes, and the CyHV-3 vIL-10 gene (khvIL-10) were highly significantly up-regulated in different phases of CyHV-3 infection. Similarly, host IL-1β was up-regulated in the acute phase of CyHV-3 infection. There was no significant difference in the expression of host TNFα-1 and MHC-II genes during all phases of CyHV-3 infection. Based on the expression profile of carp immune-related genes in each stage of CyHV-3 infection, we propose a possible interaction between carp IL-12, carp IL-10 and khvIL-10 during the course of viral infection. To our knowledge, this is the first report on the expression of cytokine genes during all phases (acute, persistent and reactivation) of CyHV-3 infection.
    Diseases of Aquatic Organisms 03/2015; 113(2):127-135. DOI:10.3354/dao02824
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    ABSTRACT: Vibrio ordalii is the causative agent of atypical vibriosis and has the potential to cause severe losses in salmonid aquaculture, but the factors determining its virulence have not yet been elucidated. In this work, cell-surface-related properties of the isolates responsible for outbreaks in Atlantic salmon were investigated. We also briefly examined whether pathogenicity against fish varied for V. ordalii strains with differing cell-surface properties. Hydrocarbon adhesions indicated the hydrophobic character of V. ordalii, although only 4 of 18 isolates induced haemagglutination in Atlantic salmon erythrocytes. A minority of the studied isolates (6 of 18) and the type strain ATCC 33509T produced low-grade biofilm formation on polyethylene surface after 2 h post-inoculation (hpi), but no strains were slime producers. Interestingly, V. ordalii isolates showed wide differences in hydrophobicity. Therefore, we chose 3 V. ordalii isolates (Vo-LM-03, Vo-LM-18 and Vo-LM-16) as representative of each hydrophobicity group (strongly hydrophobic, relatively hydrophobic and quasi-hydrophilic, respectively) and ATCC 33509T was used in the pathogenicity studies. All tested V. ordalii strains except the type strain resisted the killing activity of Atlantic salmon mucus and serum, and could proliferate in these components. Moreover, all V. ordalii isolates adhered to SHK-1 cells, causing damage to fish cell membrane permeability after 16 hpi. Virulence testing using rainbow trout revealed that isolate Vo-LM-18 was more virulent than isolates Vo-LM-03 and Vo-LM-16, indicating some relationship between haemagglutination and virulence, but not with hydrophobicity.
    Diseases of Aquatic Organisms 02/2015; 113(1):9-23. DOI:10.3354/dao02820