Scandinavian Journal of Medicine and Science in Sports Impact Factor & Information

Publisher: Wiley

Journal description

Representing the Scandinavian sports medicine and science associations the journal publishes original articles on the traumatologic (orthopaedic) physiologic biomechanic medical (including rehabilitation) sociologic psychologic pedagogic historic and philosophic aspects of sport. Scandinavian Journal of Medicine & Science in Sports is thus multidisciplinary and encompasses all elements of research in sport. Leading authorities are invited to contribute reviews on selected topics. The journal is divided into three sections: I Physiology and Biomechanics; II Medicine Traumatology and Rehabilitation; III Social and Behavioural Aspects of Sports.

Current impact factor: 2.90

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 2.896
2013 Impact Factor 3.174
2012 Impact Factor 3.214
2011 Impact Factor 2.867
2010 Impact Factor 2.794
2009 Impact Factor 2.335
2008 Impact Factor 2.264
2007 Impact Factor 2.295
2006 Impact Factor 1.989
2005 Impact Factor 2.151
2004 Impact Factor 1.717
2003 Impact Factor 0.931
2002 Impact Factor 1.117
2001 Impact Factor 0.899
2000 Impact Factor 0.667
1999 Impact Factor 0.726
1998 Impact Factor 0.83
1997 Impact Factor 0.704
1996 Impact Factor 0.624
1995 Impact Factor 0.39

Impact factor over time

Impact factor

Additional details

5-year impact 3.84
Cited half-life 5.60
Immediacy index 0.76
Eigenfactor 0.01
Article influence 1.06
Website Scandinavian Journal of Medicine & Science in Sports website
Other titles Scandinavian journal of medicine & science in sports (Online), Scandinavian journal of medicine and science in sports
ISSN 1600-0838
OCLC 47858815
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details


  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author cannot archive a post-print version
  • Restrictions
    • 12 months embargo
  • Conditions
    • Some journals have separate policies, please check with each journal directly
    • On author's personal website, institutional repositories, arXiv, AgEcon, PhilPapers, PubMed Central, RePEc or Social Science Research Network
    • Author's pre-print may not be updated with Publisher's Version/PDF
    • Author's pre-print must acknowledge acceptance for publication
    • Non-Commercial
    • Publisher's version/PDF cannot be used
    • Publisher source must be acknowledged with citation
    • Must link to publisher version with set statement (see policy)
    • If OnlineOpen is available, BBSRC, EPSRC, MRC, NERC and STFC authors, may self-archive after 12 months
    • If OnlineOpen is available, AHRC and ESRC authors, may self-archive after 24 months
    • Publisher last contacted on 07/08/2014
    • This policy is an exception to the default policies of 'Wiley'
  • Classification

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: Carbohydrates (CHO) are a key source of energy for contracting skeletal muscle during strenuous exercise and fatigue during such exercise often coincides with CHO depletion. Our current understanding of the importance of CHO for exercise metabolism has its foundations in classic studies in the early 20th century by Scandinavian physiologists and Bengt Saltin contributed significantly to that tradition. This brief review summarizes our contemporary understanding of key aspects of muscle glycogen and glucose metabolism during exercise, through the lens of seminal studies by Bengt Saltin.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):29-33. DOI:10.1111/sms.12607
  • [Show abstract] [Hide abstract]
    ABSTRACT: Appropriate cardiovascular and hemodynamic adjustments are necessary to meet the metabolic demands of working skeletal muscle during exercise. Alterations in the sympathetic and parasympathetic branches of the autonomic nervous system are fundamental in ensuring these adjustments are adequately made. Several neural mechanisms are responsible for the changes in autonomic activity with exercise and through complex interactions, contribute to the cardiovascular and hemodynamic changes in an intensity-dependent manner. This short review is from a presentation made at the Saltin Symposium June 2–4, 2015 in Copenhagen, Denmark. As such, the focus will be on reflex control of the circulation with an emphasis on the work of the late Dr. Bengt Saltin. Moreover, a concerted effort is made to highlight the novel and insightful concepts put forth by Dr. Saltin in his last published review article on the regulation of skeletal muscle blood flow in humans. Thus, the multiple roles played by adenosine triphosphate (ATP) including its ability to induce vasodilatation, override sympathetic vasoconstriction and stimulate skeletal muscle afferents (exercise pressor reflex) are discussed and a conceptual framework is set suggesting a major role of ATP in blood flow regulation during exercise.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):74-82. DOI:10.1111/sms.12600
  • [Show abstract] [Hide abstract]
    ABSTRACT: The title assigned for my lecture at the Saltin Symposium was “Muscle adaptation to exercise: new paradigms.” The title's topic made me remember that some of Saltin's paradigms for his development of a novel exercise model were either originated by him or modified by him from existing information. Therefore, I deemed it would be instructive for future generations to consider one facet of his 54-year career – human exercise models. I arbitrarily selected to share five examples of new paradigm models initiated by Saltin. They are: bed rest; arms vs legs; one leg vs the other leg; myokine communication from skeletal muscle to other organs/tissues; and 42-day cross-country skiing expedition. I arbitrarily selected the above as examples of novel approaches that he used to the study humans during maximal endurance exercise. Noteworthy though is that Saltin's lifetime demeanor, itself, is a model for other scientists. In final analysis, the world is richer due to his passion to study humans to advance medical science by uncovering mechanisms as to how the human body is constructed to perform endurance types of exercise at maximal intensities and durations.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):49-52. DOI:10.1111/sms.12595
  • [Show abstract] [Hide abstract]
    ABSTRACT: This paper presents an impressionistic summary of the formation, activities, accomplishments, and impact of the Copenhagen Muscle Research Centre (CMRC) from 1994 to 2004. The history of the CMRC is viewed in the context of the goals of the original program, the tradition of excellence in exercise physiology in Copenhagen since the time of August Krogh, and the structure of the center. The key role of Professor Bengt Saltin as a visionary, flexible, and inclusive leader is highlighted.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):22-28. DOI:10.1111/sms.12598
  • [Show abstract] [Hide abstract]
    ABSTRACT: In 1968, Saltin et al. published a landmark paper describing the alterations in VO2max resulting from two sequential interventions – 20 days of bed rest and almost 8 weeks of training. They concluded that bed rest reduced VO2max through reductions in maximal cardiac output, while training enhanced VO2max by an equal combination of increased maximal cardiac output and increased arterio-venous [O2] difference (A-V Δ [O2]). At the time, A-V Δ [O2] was taken as an index of peripheral (skeletal muscle) adaptation. A key interpretive element that was not featured was consideration of how alterations in cardiac output affect the O2 extraction process secondary to changes in red cell transit time through the muscle microcirculation, even in the absence of adaptive changes in the skeletal muscles per se. For the 2015 Saltin Symposium, it was therefore thought appropriate to re-examine the 1968 O2 transport data and re-evaluate the roles central cardiovascular and peripheral muscle changes after bed rest and training allowing for their interaction. The analysis supports the conclusion that bed rest reduced VO2max mainly through reduction in cardiac output, but after training, it is proposed that the 1968 conclusions should be modified: the majority of the increase in VO2max from the control state can be attributed to an improvement in diffusive unloading of O2 from the muscle microcirculation, with a much smaller role for enhanced blood flow.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):83-87. DOI:10.1111/sms.12597
  • [Show abstract] [Hide abstract]
    ABSTRACT: Today the Kenyan dominance in middle- and long-distance running is so profound that it has no equivalence to any other sport in the world. Critical physiological factors for performance in running include maximal oxygen consumption (VO2max), fractional VO2max utilization and running economy (energetic cost of running). Kenyan and non-Kenyan elite runners seem to be able to reach very high, but similar maximal oxygen uptake levels just as there is some indication that untrained Kenyans and non-Kenyans have a similar VO2max. In addition, the fractional utilization of VO2max seems to be very high but similar in Kenyan and European runners. Similarly, no differences in the proportion of slow muscle fibers have been observed when comparing Kenyan elite runners with their Caucasian counterparts. In contrast, the oxygen cost of running at a given running velocity has been found to be lower in Kenyan elite runners relative to other elite runners and there is some indication that this is due to differences in body dimensions. Pulmonary system limitations have been observed in Kenyan runners in the form of exercise-induced arterial hypoxemia, expiratory flow limitation, and high levels of respiratory muscle work. It appears that Kenyan runners do not possess a pulmonary system that confers a physiological advantage. Additional studies on truly elite Kenyan runners are necessary to understand the underlying physiology which permits extraordinary running performances.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):110-118. DOI:10.1111/sms.12573

  • Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(6):735-735. DOI:10.1111/sms.12622
  • [Show abstract] [Hide abstract]
    ABSTRACT: This brief review addresses the regulation of cardiac output (Q) at rest and during submaximal exercise in acute and chronic hypoxia. To preserve systemic O2 delivery in acute hypoxia Q is increased by an acceleration of heart rate, whereas stroke volume (SV) remains unchanged. Tachycardia is governed by activation of carotid and aortic chemoreceptors and a concomitant reduction in arterial baroreflex activation, all balancing sympathovagal activity toward sympathetic dominance. As hypoxia extends over several days a combination of different adaptive processes restores arterial O2 content to or beyond sea level values and hence Q normalizes. The latter however occurs as a consequence of a decrease in SV whereas tachycardia persists. The diminished SV reflects a lower left ventricular end-diastolic volume which is primarily related to hypoxia-generated reduction in plasma volume. Hypoxic pulmonary vasoconstriction may contribute by increasing right ventricular afterload and thus decreasing its ejection fraction. In summary, the Q response to hypoxia is the result of a complex interplay between several physiological mechanisms. Future studies are encouraged to establish the individual contributions of the different components from an integrative perspective.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):53-59. DOI:10.1111/sms.12619
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    ABSTRACT: Performance in most sports is determined by the athlete's technical, tactical, physiological and psychological/social characteristics. In the present article, the physical aspect will be evaluated with a focus on what limits performance, and how training can be conducted to improve performance. Specifically how intensified training, i.e., increasing the amount of aerobic high-intensity and speed endurance training, affects physiological adaptations and performance of trained subjects. Periods of speed endurance training do improve performance in events lasting 30 s–4 min, and when combined with aerobic high-intensity sessions, also performance during longer events. Athletes in team sports involving intense exercise actions and endurance aspects, such as soccer and basketball, can also benefit from intensified training. Speed endurance training does reduce energy expenditure and increase expression of muscle Na+, K+ pump α subunits, which may preserve muscle cell excitability and delay fatigue development during intense exercise. When various types of training are conducted in the same period (concurrent training), as done in a number of sports, one type of training may blunt the effect of other types of training. It is not, however, clear how various training modalities are affecting each other, and this issue should be addressed in future studies.
    Scandinavian Journal of Medicine and Science in Sports 12/2015; 25(S4):88-99. DOI:10.1111/sms.12605