Rheumatic diseases clinics of North America Journal Impact Factor & Information

Publisher: WB Saunders

Journal description

Current impact factor: 2.69

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 2.692
2013 Impact Factor 1.736
2012 Impact Factor 2.096
2011 Impact Factor 3.02
2010 Impact Factor 3.018
2009 Impact Factor 2.594
2008 Impact Factor 1.77
2007 Impact Factor 2.16
2006 Impact Factor 2.568
2005 Impact Factor 2.842
2004 Impact Factor 2.146
2003 Impact Factor 2.776
2002 Impact Factor 3.314
2001 Impact Factor 2.156
2000 Impact Factor 2.257
1999 Impact Factor 2.06
1998 Impact Factor 2.175
1997 Impact Factor 1.99
1996 Impact Factor 2.295
1995 Impact Factor 2.444
1994 Impact Factor 2.225
1993 Impact Factor 1.331
1992 Impact Factor 1.221

Impact factor over time

Impact factor

Additional details

5-year impact 2.47
Cited half-life 8.20
Immediacy index 0.34
Eigenfactor 0.00
Article influence 0.83
Other titles Rheumatic diseases clinics of North America (Online), Rheumatic diseases clinics of North America, Rheumatic disease clinics of North America, Rheumatic disease clinics
ISSN 1558-3163
OCLC 60626445
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

WB Saunders

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Pre-print allowed on any website or open access repository
    • Voluntary deposit by author of authors post-print allowed on institutions open scholarly website including Institutional Repository, without embargo, where there is not a policy or mandate
    • Deposit due to Funding Body, Institutional and Governmental policy or mandate only allowed where separate agreement between repository and the publisher exists.
    • Permitted deposit due to Funding Body, Institutional and Governmental policy or mandate, may be required to comply with embargo periods of 12 months to 48 months
    • Set statement to accompany deposit
    • Published source must be acknowledged
    • Must link to journal home page or articles' DOI
    • Publisher's version/PDF cannot be used
    • Articles in some journals can be made Open Access on payment of additional charge
    • NIH Authors articles will be submitted to PubMed Central after 12 months
    • Authors who are required to deposit in subject-based repositories may also use Sponsorship Option
    • Publisher last reviewed on 03/07/2015
    • 'WB Saunders' is an imprint of 'Elsevier'
  • Classification
    ​ green

Publications in this journal

  • Rheumatic diseases clinics of North America 08/2015; 41(3):xiii. DOI:10.1016/j.rdc.2015.06.002
  • Rheumatic diseases clinics of North America 08/2015; 41(3):xv-xvi. DOI:10.1016/j.rdc.2015.06.001
  • [Show abstract] [Hide abstract]
    ABSTRACT: Lung disease commonly occurs in connective tissue diseases (CTD) and is an important cause of morbidity and mortality. Imaging is central to the evaluation of CTD-associated pulmonary complications. In this article, a general discussion of radiologic considerations is followed by a description of the pulmonary appearances in individual CTDs, and the imaging appearances of acute and nonacute pulmonary complications. The contribution of imaging to monitoring disease, evaluating treatment response, and prognostication is reviewed. Finally, we address the role of imaging in the challenging multidisciplinary evaluation of interstitial lung disease where there is an underlying suspicion of an undiagnosed CTD. Copyright © 2015 Elsevier Inc. All rights reserved.
    Rheumatic diseases clinics of North America 05/2015; 41(2):167-196. DOI:10.1016/j.rdc.2014.12.001
  • [Show abstract] [Hide abstract]
    ABSTRACT: The major manifestations of antiphospholipid syndrome (APS) are caused by thrombosis within the venous or arterial vasculature, whereas the vascular lesions in systemic vasculitis result from an inflammatory infiltrate in the vessel wall. There is an association between vascular thrombosis and inflammation, however, as vasculitis can occur in APS and thromboembolic complications are seen in systemic vasculitis. Although differentiating between vasculitis and antiphospholipid-associated thrombosis can be difficult, it may be crucial to do so given the different therapeutic implications for immunosuppression or anticoagulation. This article explores the relationship between thrombosis and inflammation as it relates to APS and systemic vasculitis.
    Rheumatic diseases clinics of North America 02/2015; 41(1):109-123. DOI:10.1016/j.rdc.2014.09.009
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    ABSTRACT: This article provides an update on the diagnosis and management of the antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides, granulomatosis with polyangiitis (formerly Wegener), microscopic polyangiitis, and eosinophilic granulomatosis with polyangiitis (formerly Churg-Strauss). Focus is on new schemes of classification and the importance of ANCAs in the diagnosis and prognosis of these systemic vasculitides. Current therapeutic strategies consisting of glucocorticoids in conjunction with conventional or biologic agents for both induction of remission and remission maintenance are outlined. Future research directions include investigation of the optimal duration and frequency of maintenance therapy and development of targeted therapeutic agents.
    Rheumatic diseases clinics of North America 02/2015; 41(1):1-19. DOI:10.1016/j.rdc.2014.09.003
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    ABSTRACT: Primary angiitis of the central nervous system (PACNS) is a rare disease, although it is increasingly recognized both in adults and children. Little is known about pathogenesis, but efforts at classification into subtypes are being made, and the distinction of PACNS from reversible cerebral vasoconstriction syndrome has been a major advance. The prognosis for improvement, or at least stabilization, of neurologic function is good with prompt and aggressive treatment, but the diagnosis continues to be challenging. Refinement of treatment strategies is needed. Multicenter collaboration may be crucial to make additional progress via randomized trials.
    Rheumatic diseases clinics of North America 02/2015; 41(1):47-62. DOI:10.1016/j.rdc.2014.09.004
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    ABSTRACT: The need to distinguish true primary systemic vasculitis from its multiple potential mimickers is one of the most challenging diagnostic conundrums in clinical medicine. This article reviews 9 challenging vasculitis mimickers: fibromuscular dysplasia, calciphylaxis, segmental arterial mediolysis, antiphospholipid syndrome, hypereosinophilic syndrome, lymphomatoid granulomatosis, malignant atrophic papulosis, livedoid vasculopathy, and immunoglobulin G4-related disease.
    Rheumatic diseases clinics of North America 02/2015; 41(1):141-160. DOI:10.1016/j.rdc.2014.09.011
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    ABSTRACT: Small vessel vasculitis in the skin manifests with palpable purpura on the lower extremities. This clinical presentation prompts a complete physical examination, history, and review of systems, as well as biopsies for routine processing and direct immunofluorescence to confirm the diagnosis. The presence of vasculitis in other organs, associated underlying conditions, and the severity of cutaneous manifestations dictate management. The majority of cases are self-limited, and overall the prognosis is favorable. Still, a subset of patients can have serious complications and chronic or recurrent disease.
    Rheumatic diseases clinics of North America 02/2015; 41(1):21-32. DOI:10.1016/j.rdc.2014.09.006
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    ABSTRACT: Cogan and Behcet syndromes are considered large vessel vasculitides. Both are rare diseases, with varied clinical manifestations affecting multiple organ systems. Although both have hallmark symptoms (ocular and vestibuloauditory inflammation in Cogan syndrome and aphthous ulcers in Behcet syndrome), neither has confirmatory diagnostic testing. Delayed diagnosis can result in poor outcomes. In both syndromes, large vessel arterial inflammation may result in severe morbidity and mortality. Treatment strategies in both syndromes vary based on organ system involvement and severity of manifestations. In this article, the epidemiology, proposed pathogenesis, manifestations, and the most current treatment paradigms for these syndromes are reviewed.
    Rheumatic diseases clinics of North America 02/2015; 41(1):75-91. DOI:10.1016/j.rdc.2014.09.007
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    ABSTRACT: Cryoglobulins are immunoglobulins that precipitate at temperatures less than 37°C. They occur secondary to infectious, autoimmune, and malignant processes. In the Brouet classification, type I cryoglobulinemia is caused by hyperviscosity, whereas type II and III manifestations are caused by vasculitis in target organs (primarily skin, peripheral nerves, and kidney). New classification criteria were recently proposed that may help with study and treatment of cryoglobulinemic vasculitis (CryoVas). Hepatitis C virus is the most common cause of CryoVas and treatment with antivirals can be curative in mild cases, whereas rituximab is highly effective in treating active vasculitis in more severe cases.
    Rheumatic diseases clinics of North America 02/2015; 41(1):93-108. DOI:10.1016/j.rdc.2014.09.008
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    ABSTRACT: Polyarteritis nodosa (PAN) is a systemic disease, but variants are cutaneous PAN and single-organ disease. Histologic confirmation of vasculitis in medium-sized arteries is desirable, and biopsies should be obtained from the symptomatic and least invasive sites. Angiography can show multiple microaneurysms in the viscera. Treatment includes high-dose corticosteroids, which are combined with immunosuppressive agents when internal organs are involved and with life-threatening disease. Once remission is achieved, maintenance agents are initiated. PAN is becoming a rare disease. International collaborative efforts are under way to establish better diagnostic and classification for all vasculitides, including PAN.
    Rheumatic diseases clinics of North America 02/2015; 41(1):33-46. DOI:10.1016/j.rdc.2014.09.005
  • Article: Preface.
    Rheumatic diseases clinics of North America 01/2015; 41(1):xiii-xiv. DOI:10.1016/j.rdc.2014.10.003
  • Rheumatic diseases clinics of North America 12/2014; 40(4):xiii. DOI:10.1016/j.rdc.2014.09.002
  • Rheumatic diseases clinics of North America 11/2014; 41(1):xi. DOI:10.1016/j.rdc.2014.10.002
  • Rheumatic diseases clinics of North America 08/2014; 40(3):xiii-xiv. DOI:10.1016/j.rdc.2014.05.004
  • Rheumatic diseases clinics of North America 07/2014; 40(3):xi. DOI:10.1016/j.rdc.2014.06.001
  • [Show abstract] [Hide abstract]
    ABSTRACT: Gout is a common disorder with clinical signs and symptoms resulting from inflammatory responses to monosodium urate crystals deposited in tissues from extracellular fluids saturated for urate. Long-term management of gout focuses on nonpharmacologic and pharmacologic means to achieve and maintain serum urate levels in a subsaturating range. Despite a firm understanding of gout pathophysiology, means to achieve certain diagnosis, and a variety of effective therapies, treatment outcomes remain suboptimal. In this review, available nonpharmacologic and pharmacologic therapies for chronic gout are discussed and a framework is provided for successful achievement and maintenance of goal-range serum urate levels.
    Rheumatic diseases clinics of North America 05/2014; 40(2):357-374. DOI:10.1016/j.rdc.2014.01.012
  • [Show abstract] [Hide abstract]
    ABSTRACT: Calcium crystal arthritis is often unrecognized, poorly managed, and few effective therapies are available. The most common types of calcium crystals causing musculoskeletal syndromes are calcium pyrophosphate (CPP) and basic calcium phosphate (BCP). Associated syndromes have different clinical presentations and divergent management strategies. Acute CPP arthritis is treated similarly to acute gouty arthritis, whereas chronic CPP and BCP arthropathy may respond to strategies used for osteoarthritis. Calcific tendonitis is treated with a variety of interventions designed to dissolve BCP crystals. A better understanding of the causes and larger well-planned trials of current therapies will lead to improved care.
    Rheumatic diseases clinics of North America 05/2014; 40(2):343-356. DOI:10.1016/j.rdc.2014.01.010
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    ABSTRACT: This article presents an overview of the treatment of acute gout. Nonpharmacologic and pharmacologic treatments, monotherapy versus combination therapy, suggested recommendations, guidelines for treatment, and drugs under development are discussed.
    Rheumatic diseases clinics of North America 05/2014; 40(2):329-341. DOI:10.1016/j.rdc.2014.01.008