Proceedings of the Royal Society B: Biological Sciences Journal Impact Factor & Information

Publisher: Royal Society (Great Britain), Royal Society, The

Journal description

Proceedings B welcomes papers of high quality in any area of biological science. As a fast track journal, Proceedings B specialises in the rapid delivery of the latest research to the scientific community, normally within three months of acceptance. It is published on the 7th and 22nd of each month. Many more good manuscripts are submitted to us, than we have space to print, and we give preference to those that present significant advances of broad interest. Submission of preliminary reports, of papers that merely confirm previous findings, and of papers that are likely to interest only small groups of specialists, is not encouraged. All papers are sent to Editorial Board members for an initial assessment of their suitability, and may be returned to authors without in-depth peer-review if this assessment makes it seem unlikely that they will be accepted.

Current impact factor: 5.05

Impact Factor Rankings

2015 Impact Factor Available summer 2016
2014 Impact Factor 5.051
2013 Impact Factor 5.292
2012 Impact Factor 5.683
2011 Impact Factor 5.415
2010 Impact Factor 5.064
2008 Impact Factor 4.248
2007 Impact Factor 4.112
2006 Impact Factor 3.612
2005 Impact Factor 3.51
2004 Impact Factor 3.653
2003 Impact Factor 3.544
2002 Impact Factor 3.396
2001 Impact Factor 3.192
2000 Impact Factor 3.037
1999 Impact Factor 2.755
1998 Impact Factor 3.033
1997 Impact Factor 2.873

Impact factor over time

Impact factor

Additional details

5-year impact 5.65
Cited half-life 8.50
Immediacy index 0.95
Eigenfactor 0.09
Article influence 2.29
Website Proceedings of the Royal Society B: Biological Sciences website
Other titles Biology letters., Proceedings., Proceedings - Royal Society. Biological sciences, Biological sciences, Proceedings of the Royal Society of London., Proceedings of the Royal Society
ISSN 1471-2954
OCLC 44150803
Material type Document, Periodical, Internet resource
Document type Internet Resource, Computer File, Journal / Magazine / Newspaper

Publisher details

Royal Society, The

  • Pre-print
    • Author can archive a pre-print version
  • Post-print
    • Author can archive a post-print version
  • Conditions
    • Author's pre-print on free public servers
    • Author's post-print on author's personal website or institutional website immediately
    • Author's post-print on institutional repository or not-for-profit open access repository after 12 months embargo
    • Publisher's version/PDF cannot be used
    • Published source must be acknowledged with citation close to title of article
    • Must link to publisher version close to title of article
    • If funding agency rules apply, authors may post articles in PubMed Central 12 months after publication
    • Eligible UK authors may deposit in Open Depot (after 12 months)
    • Publisher last contacted on 21/04/2015
  • Classification
    ​ green

Publications in this journal

  • [Show abstract] [Hide abstract]
    ABSTRACT: The waterflea Daphnia is a model to investigate the genetic basis of phenoty-pic plasticity resulting from one differentially expressed genome. Daphnia develops adaptive phenotypes (e.g. morphological defences) thwarting predators, based on chemical predator cue perception. To understand the genomic basis of phenotypic plasticity, the description of the precedent cellular and neuronal mechanisms is fundamental. However, key regulators remain unknown. All neuronal and endocrine stimulants were able to modulate but not induce defences, indicating a pathway of interlinked steps. A candidate able to link neuronal with endocrine responses is the multi-functional amine dopamine. We here tested its involvement in trait formation in Daphnia pulex and Daphnia longicephala using an induction assay composed of predator cues combined with dopaminergic and cholin-ergic stimulants. The mere application of both stimulants was sufficient to induce morphological defences. We determined dopamine localization in cells found in close association with the defensive trait. These cells serve as centres controlling divergent morphologies. As a mitogen and sclerotiza-tion agent, we anticipate that dopamine is involved in proliferation and structural formation of morphological defences. Furthermore, dopamine pathways appear to be interconnected with endocrine pathways, and control juvenile hormone and ecdysone levels. In conclusion, dopamine is suggested as a key regulator of phenotypic plasticity.
    Proceedings of the Royal Society B: Biological Sciences 09/2015; 282(20151440). DOI:10.1098/rspb.2015.1440
  • [Show abstract] [Hide abstract]
    ABSTRACT: Researchers have long been interested in the evolution of culture and the ways in which change in cultural systems can be reconstructed and tracked. Within the realm of language, these questions are increasingly investigated with Bayesian phylogenetic methods. However, such work in cultural phylogenetics could be improved by more explicit quantification of reconstruction and transition probabilities. We apply such methods to numerals in the languages of Australia. As a large phylogeny with almost universal 'low-limit' systems, Australian languages are ideal for investigating numeral change over time. We reconstruct the most likely extent of the system at the root and use that information to explore the ways numerals evolve. We show that these systems do not increment serially, but most commonly vary their upper limits between 3 and 5. While there is evidence for rapid system elaboration beyond the lower limits, languages lose numerals as well as gain them. We investigate the ways larger numerals build on smaller bases, and show that there is a general tendency to both gain and replace 4 by combining 2 + 2 (rather than inventing a new unanalysable word 'four'). We develop a series of methods for quantifying and visualizing the results.
    Proceedings of the Royal Society B: Biological Sciences 09/2015; 282(1815). DOI:10.1098/rspb.2015.1278
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    ABSTRACT: There is a connection between nutrient inputs, energy-sensing pathways, lifespan variation and aging. Despite the role of metabolic enzymes in energy homeostasis and their metabolites as nutrient signals, little is known about how their gene expression impacts lifespan. In this report, we use P-element mutagenesis in Drosophila to study the effect on lifespan of reductions in expression of seven central metabolic enzymes, and contrast the effects on normal diet and dietary restriction. The major observation is that for five of seven genes, the reduction of gene expression extends lifespan on one or both diets. Two genes are involved in redox balance, and we observe that lower activity genotypes significantly extend lifespan. The hexokinases also show extension of lifespan with reduced gene activity. Since both affect the ATP/ADP ratio, this connects with the role of AMP-activated protein kinase as an energy sensor in regulating lifespan and mediating caloric restriction. These genes possess significant expression variation in natural populations, and our experimental genotypes span this level of natural activity variation. Our studies link the readout of energy state with the perturbation of the genes of central metabolism and demonstrate their effect on lifespan.
    Proceedings of the Royal Society B: Biological Sciences 09/2015; 282(1815). DOI:10.1098/rspb.2015.1646
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    ABSTRACT: There is extensive evidence from model systems that disrupting associations between co-adapted mitochondrial and nuclear genotypes can lead to deleterious and even lethal consequences. While it is tempting to extrapolate from these observations and make inferences about the human-health effects of altering mitonuclear associations, the importance of such associations may vary greatly among species, depending on population genetics, demographic history and other factors. Remarkably, despite the extensive study of human population genetics, the statistical associations between nuclear and mitochondrial alleles remain largely uninvestigated. We analysed published population genomic data to test for signatures of historical selection to maintain mitonuclear associations, particularly those involving nuclear genes that encode mitochondrial-localized proteins (N-mt genes). We found that significant mitonuclear linkage disequilibrium (LD) exists throughout the human genome, but these associations were generally weak, which is consistent with the paucity of population genetic structure in humans. Although mitonuclear LD varied among genomic regions (with especially high levels on the X chromosome), N-mt genes were statistically indistinguishable from background levels, suggesting that selection on mitonuclear epistasis has not preferentially maintained associations involving this set of loci at a species-wide level. We discuss these findings in the context of the ongoing debate over mitochondrial replacement therapy.
    Proceedings of the Royal Society B: Biological Sciences 09/2015; 282(1815). DOI:10.1098/rspb.2015.1704
  • [Show abstract] [Hide abstract]
    ABSTRACT: Self-fertilization and admixture of genotypes from different populations can have major fitness consequences in native species. However, few studies have addressed their potential roles in invasive species. Here, we used plants of Mimulus guttatus from seven native North American, three invasive Scottish and four invasive New Zealand populations to address this. We created seeds from self-fertilization, within-population outcrossing, between-population outcrossing within the same range, and outcrossing between the native and invasive ranges. A greenhouse experiment showed that native and invasive plants of M. guttatus suffered to similar degrees from inbreeding depression, in terms of asexual reproduction and biomass production. After outcrossing with plants from other populations, M. guttatus benefited from heterosis, in terms of asexual and sexual reproduction, and biomass production, particularly when plants from native and invasive populations were crossed. This suggests that, when novel genotypes of M. guttatus from the native North American range will be introduced to the invasive ranges, subsequent outcrossing with M. guttatus plants that are already there might further boost invasiveness of this species.
    Proceedings of the Royal Society B: Biological Sciences 09/2015; 282(1815). DOI:10.1098/rspb.2015.1487